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1 le elements that have expanded presumably by unequal crossing over.
2 receding meiotic recombination, resulting in unequal crossing over.
3 enotypes through infrequent recombination or unequal crossing over.
4 tion slippage or, more likely, intra-allelic unequal crossing over.
5 rgoing concerted evolution by the process of unequal crossing over.
6 ve elements that undergo increased levels of unequal crossing-over.
7 d strand slippage during DNA replication and unequal crossing-over.
8 of the gene cluster by a process of meiotic unequal crossing-over.
9 heir origins through replication slippage or unequal crossings over.
10 at substitutions can alter the mechanisms of unequal crossing-over, altering the way concerted evolut
11 ene RPP8 and two RPP8 homologs indicate that unequal crossing over and gene conversion may have contr
13 e occurred as a result of repeated events of unequal crossing over and pericentric inversions during
15 Reticulate evolutionary processes, such as unequal crossing-over and gene conversion, are known to
20 s directly measured the frequency of meiotic unequal crossing-over (approximately 3 x 10(-6)), leadin
22 array's formation, and propose a model using unequal crossing-over as the primary mechanism of array
24 of the deletions are shown to have arisen by unequal crossing over between CLCNKB and the nearby rela
25 bsequent repair, and cannot be attributed to unequal crossing over between resistance gene homologs.
26 chimeric gene, which was likely derived from unequal crossing over between RPP8-Ler and RPH8A ancesto
28 -Tooth disease type 1A (CMT1A), results from unequal crossing over between two >98% identical 24 kb r
29 for the derivation of this haplotype invokes unequal crossing over between two known ancestral KIR ha
30 independent events that likely resulted from unequal crossing-over between segmental duplications.
32 ique P450 enzyme, CYP337B3, which arose from unequal crossing-over between two parental P450 genes, r
33 versity, where extensive gene conversion and unequal crossing over contribute to highly similar gene
34 eferentially paternal in origin, arises from unequal crossing over due to homologous recombination be
35 HNPP) deletion are reciprocal products of an unequal crossing-over event between misaligned flanking
36 ng DNA was used to estimate the frequency of unequal crossing-over events (6.3 x 10(-5) events per ge
37 cial syndrome/DiGeorge syndrome results from unequal crossing-over events between two 240-kb low-copy
41 ization of molecular genetic events, such as unequal crossing over, gene conversion and crossover asy
42 ions, duplication of the KIR3DL1/S1 locus by unequal crossing over has enabled individuals to carry a
43 xchange of flanking markers, consistent with unequal crossing over; however, although the number of D
45 o the high probability of misreplication and unequal crossing-over in the repeated segment of the gen
46 riants suggested that most of them arose via unequal crossing over, indicating that rp3 is a complex
48 concerted evolution, although the degree of unequal crossing over may differ among complex satellite
50 Thus, if these reactions occur in humans, unequal crossing-over or gene conversion may also contri
51 CNVs and disease-associated CNCs is meiotic unequal crossing over, or nonallelic homologous recombin
52 massive exon-shuffling mechanism, involving unequal crossing over possibly in concert with retrotran
53 loss of Rp1-D resistance can be explained by unequal crossing over that occurred mostly within coding
55 ave been diversified by gene duplication and unequal crossing over, thereby generating haplotypes wit
56 re for the evolution of a T-box subfamily by unequal crossing over to form a two-gene cluster that wa
57 ons caused by illegitimate recombination and unequal crossing over were major driving forces in the e
58 lication) or interallelic recombination with unequal crossing over, whereas both mechanisms appear to
59 t proximal and distal CMT1A-REPs and promote unequal crossing over, which occurs 10 times more freque
60 ents duplicating two to five LRRs because of unequal crossing-over within or between RGC2 genes at on