ライフサイエンスコーパス: unexposed

1 0/82; 97.6% exposed vs. 12,940/13,971; 92.6% unexposed).

2 l effect is knowing who is truly exposed and unexposed.

3 ly to be infected with a pathogen than those unexposed.

4 onotherapy, or combination therapy, or being unexposed.

5 distributions, usually 100% exposed and 100% unexposed.

6 the first trimester) and 29 293 infants were unexposed.

7 WGS was performed on 31 isolates: 14 unexposed, 14 exposed to azithromycin </=30 days before

8 WGS was performed on 31 isolates: 14 unexposed, 14 exposed to azithromycin </=30 days before

9 n uptake (exposed: 25.74 +/- 8.36 mL/kg/min; unexposed: 26.82 +/- 8.36 mL/kg/min) than did control su

10 unexposed infants (6.4% exposed versus 4.4% unexposed; adjusted odds ratio [OR] = 1.38, 95% confiden

11 ear resistant to infection, while previously unexposed adults remain susceptible.

12 were associated with SCC compared with those unexposed after controlling for important confounders in

13 -exposed but uninfected children, and 56 HIV-unexposed and -uninfected children aged 2-10 years old i

14 R = 1.4; 95% CI: 0.9, 2.3) and brain (n = 67 unexposed and 10 ever exposed cases; RR = 1.8; 95% CI: 0

15 exposure for cancers of the kidney (n = 157 unexposed and 17 ever exposed cases; RR = 1.4; 95% CI: 0

16 is was detected in 2.1% (n = 26/1257) of HIV-unexposed and 2.7% (n = 16/599) of HIV-exposed infants.

17 d with meningioma risk in women only (n = 38 unexposed and 9 exposed cases; RR = 2.4; 95% CI: 1.1, 5.

18 els did not differ significantly between AED-unexposed and AED-exposed children.

19 cumulative incidence difference between the unexposed and exposed children was 0.04% (CI, -0.09% to

20 Mean follow-up was 6.7 years in both unexposed and exposed children.

21 hich consist of estimating genetic effect in unexposed and exposed individuals separately can be of i

22 We reared F1 offspring of unexposed and predator-exposed F0 males under 'control'

23 nalyzed the antibody repertoire from healthy unexposed and previously MARV-infected individuals to as

24 posed but uninfected (HEU) children; and 182 unexposed and uninfected (HUU) children.

25 HIV-exposed and uninfected children with HIV-unexposed and uninfected children at ages 12, 24, 48, an

26 HIV-unexposed and uninfected children matched for age, sex,

27 NK cells were contrasted among 29 HIV-unexposed and uninfected controls (5-19 years), 23 HIV-e

28 dity and mortality than children who are HIV-unexposed and uninfected despite safer breastfeeding and

29 ere similar to those for children in the HIV-unexposed and uninfected reference group at all timepoin

30 months, compared with children who were HIV-unexposed and uninfected.

31 n of LCSs can be correctly characterized as "unexposed" and to investigate whether instructions to av

32 maternal tobacco smoke; [Formula: see text] unexposed) and in a study of adult [Formula: see text] c

33 children will significantly differ from HIV-unexposed-and-uninfected (HUU) children.

34 exposed animals receiving vehicle only, and unexposed animals receiving virus.

35 Furthermore, compared with HIV-unexposed, antibody titers >=330 mIU/mL (ie, presumed se

36 hildren (168 HIV-exposed uninfected, 564 HIV-unexposed) at 24 months.

37 children (61 HIV-exposed uninfected, 199 HIV-unexposed) at 6 months and in 732 (73%) children (168 HI

38 less with increasing experience than that of unexposed bees, with exposed bees collecting between 47%

39 rs and take longer to complete the task than unexposed bees.

40 nfant mortality, comparing exposed births to unexposed births.

41 ildren to 860 echocardiograms from 140 HAART-unexposed but HIV-infected children from the Pulmonary a

42 Our results showed that unexposed CAST/EiJ and C57BL/6J mice have very different

43 Importantly, in unexposed CAST/EiJ mice, which acquired relatively less

44 exposed to mechanical stretch, compared with unexposed cells.

45 central compartment (cytoplasm) of control (unexposed) cells.

46 ren (22484 male and 11030 female) and 159619 unexposed children (105812 male and 53807 female) in the

47 Compared with HIV-unexposed children (2860 mIU/mL), measles antibody geome

48 A total of 1,655 HIV-unexposed children (64% of those eligible) were recruite

49 ressants vs 2.03 per 1000 person-years among unexposed children (between-group difference, 2.48 [95%

50 y to be hospitalized in early childhood than unexposed children (hospitalization rates per 1000 perso

51 y to be hospitalized in early childhood than unexposed children (hospitalization rates per 1000 perso

52 r received an ADHD diagnosis more often than unexposed children (Odds Ratio (OR) = 1.31, 95% confiden

53 in the brain between ferumoxytol-exposed and unexposed children and young adults with arteriovenous m

54 iffer between HIV-exposed uninfected and HIV-unexposed children at 6 months.

55 l outcomes of HIV-exposed uninfected and HIV-unexposed children during their first 2 years of life.

56 opmental delay were higher than those of HIV-unexposed children for receptive language (adjusted odds

57 p, 394 (1.0%) vaccine-exposed and 330 (1.1%) unexposed children had a diagnosis of ASD.

58 Differences between HIV-exposed and unexposed children on any of the outcomes were minimal a

59 portions with seroprotective titers than HIV-unexposed children, indicating a potential downside of A

60 d childhood obesity for both HIV-exposed and unexposed children, suggesting that promoting optimal nu

61 mpared to HIV-exposed but uninfected and HIV-unexposed children, which may play a role in HIV-associa

62 pared between HIV-exposed uninfected and HIV-unexposed children.

63 In separate virus-injected animals, unexposed cochleas were extracted for qRT-PCR.

64 This was a French multicenter exposed-unexposed cohort study enrolling patients admitted in 55

65 an difference between the exposed cohort and unexposed cohort was estimated in a model that included

66 96 940 subjects who were not exposed to CT (unexposed cohort) was gathered.

67 e of 8.3 events per 1000 person-years in the unexposed cohort, giving an adjusted IRR of 2.14 (95% CI

68 o omalizumab compared with a disease-matched unexposed cohort.

69 significantly different between the FHT and unexposed cohorts (HR, 0.99; 95% confidence interval [CI

70 9% vs. 2.6%, P = 0.49) in MPA exposed versus unexposed cohorts of children.

71 Birth weight was similar in exposed and unexposed cohorts of children; 3381 +/- 681 g vs. 3429 +

72 Results Comparison of exposed and unexposed cohorts showed that there was no statistically

73 ount for differences between the exposed and unexposed cohorts, a propensity score for being prescrib

74 for long periods of time were distinct from unexposed communities.

75 ubsequent hospitalization and 159619 matched unexposed control children.

76 These patients were matched to 423 410 unexposed control patients with no recorded exposure to

77 to tobacco only (without cannabis) with 113 unexposed control subjects on the basis of age and gende

78 ls were analyzed in healthy adolescents, Mtb-unexposed control subjects, and patients with pulmonary

79 s found among exposed subjects compared with unexposed control subjects.

80 significant difference between OD cases and unexposed controls (AFR z = 5.55, p = 2.9 x 10(-8)) and

81 silience/Recovery group, n = 26), and trauma-unexposed controls (Control group, n = 54).

82 er operating characteristic analysis in M.tb-unexposed controls and microbiologically confirmed pulmo

83 In total, 485 HEU children and 2495 HIV-unexposed controls were included.

84 rential gene expression (>8-fold increase vs unexposed controls).

85 /tobacco exposed, 32 tobacco exposed, and 71 unexposed controls).

86 3 opioid-exposed controls, and 32,500 opioid-unexposed controls, including participants of European a

87 Compared with unexposed controls, nicotine increased NGF, FN1, ET-1, C

88 8) was positively associated with OD (OD vs. unexposed controls, p = 3.2 x 10(-5); OD vs. exposed con

89 sed controls, p = 0.054) and OE (exposed vs. unexposed controls, p = 3.6 x 10(-5)).

90 1) was positively associated with OD (OD vs. unexposed controls, p = 8.1 x 10(-5); OD cases vs. expos

91 lly guided cardiovascular procedures than in unexposed controls, raising the need to spread the cultu

92 donors (respectively), as well as in 2 of 15 unexposed controls.

93 s status, and general practice to up to four unexposed controls.

94 tal samples were collected and compared with unexposed controls.

95 s9291211 was associated with OE (exposed vs. unexposed controls; EUR z = -5.39, p = 7.2 x 10(-8)).

96 bidity and mortality compared with their HIV-unexposed counterparts.

97 At birth, exposed offspring were fostered to unexposed dams.

98 known to increase disease susceptibility in unexposed descendants in the absence of detectable genet

99 to non-mutagenic agents can exert effects in unexposed descendants.

100 Unexposed donors had no MDRO-positive cultures.

101 d disease and 14 with severe disease) and 16 unexposed donors, using interferon-gamma-based assays wi

102 In unexposed ears, NT3 overexpression did not affect thresh

103 rs may spawn more aggressive phenotypes than unexposed ER+ tumors, in particular, basal subtypes that

104 ures in a parental (F0) generation to affect unexposed F1 or F2 generations (multigenerational and tr

105 roughout pregnancy and lactation predisposes unexposed F4 male descendants to obesity when dietary fa

106 ed uninfected children scored lower than HIV-unexposed for receptive language (adjusted mean differen

107 recipients were compared with nonrecipients (unexposed) for whom transfusion was delayed or not given

108 acco smoke in pregnancy; [Formula: see text] unexposed) from the Maternal and Child Health Study (MAC

109 les (n = 3), their counterparts in the next, unexposed, generation (n = 3 + 3) and also in adult sper

110 in the F0 exposed generation and subsequent unexposed generations (F1-F3).

111 ttack relative to grandoffspring of control, unexposed grandparents.

112 -years) versus 36 390 (8.9%) patients in the unexposed group (20.5 per 1000 person-years) resulting i

113 similar time adequately sedated as a matched unexposed group although no reduction in morphine or ben

114 72-75%) of the study time compared with the unexposed group at 70% (95% CI, 67-72%) giving a ratio o

115 The unexposed group was a random sample of ESBL-PE-negative

116 iology Score II with septic shock survivors (unexposed group).

117 SRIs during pregnancy; and 31207 were in the unexposed group, ie, had mothers without a psychiatric d

118 ng farmers without HPEEs as the reference or unexposed group.

119 R 0.6-4.3) versus 3.2 years (1.3-6.1) in the unexposed group.

120 ening were higher when compared to the HAART-unexposed group.

121 ion in exposed individuals compared with the unexposed group.

122 icated groups compared with offspring in the unexposed group.

123 st as part of their sedation regimen, and an unexposed group.

124 ect separating the pseudo-exposed and pseudo-unexposed groups, a DRS model is evaluated by its abilit

125 Comparing the HAART-exposed and HAART-unexposed groups, any HAART exposure was positively asso

126 o the likelihood of membership in exposed or unexposed groups.

127 dds ratios were based on exposed compared to unexposed groups.

128 population into "pseudo-exposed" and "pseudo-unexposed" groups so that differences on observed covari

129 ncer (n = 808 deaths; for >12.1 ppm-year vs. unexposed, hazard ratio (HR) = 1.43, 95% CI: 1.13, 1.81;

130 ood of patients with COVID-19 and SARS-CoV-2-unexposed healthy donors.

131 to detect SARS-CoV-2-specific T cells in 18 unexposed healthy individuals.

132 asles vaccination at 4.5 years of age in HIV-unexposed, HEU, and HIV-infected children with CD4+ >=25

133 4) and OS (65.0% v 61.9%; P = .613) to those unexposed; however, there was a suggestion of lower TRM

134 .02) and pneumonitis (for >3.12 ppm-year vs. unexposed, HR = 4.73, 95% CI: 1.42, 15.76; P for trend =

135 HIV-unexposed (HU) (n = 212) and HIV-exposed, uninfected (HE

136 pare their growth with that of breastfed HIV-unexposed (HU) children drawn from the same community.

137 d developmental delays compared to their HIV-unexposed (HU) counterparts.

138 uninfected (HEU) infants as compared to HIV-unexposed (HU) infants born in a high-income country (HI

139 d vaccine responses in HEU compared with HIV-unexposed (HUU) infants.

140 l was exposed to 2% oil, and the behavior of unexposed in mixed groups, in terms of movement speed an

141 ant infections) among pregnancies exposed vs unexposed in utero to biologics, thiopurines, or a combi

142 s to the fore the dark implications of TTOM, unexposed in Veissiere et al.

143 xposed to S. Typhi shed less than previously unexposed individuals (OR, 0.30; 95% CI, 0.1-0.8; P = .0

144 individuals was 67.4, compared with 92.5 in unexposed individuals (P = 0.027).

145 ated individual IEs binding IgM from malaria-unexposed individuals by fluorescence-activated single-c

146 n was observed in a sample of 3,245 nicotine-unexposed individuals from the same discovery cohort, co

147 duals with lower-complexity ACHD and 497 983 unexposed individuals in the UK Biobank (median age at e

148 ity (which compare risks between exposed and unexposed individuals) do not factor in the population-s

149 e of SARS-CoV-2-specific T cell responses in unexposed individuals, exposed family members, and indiv

150 CoV-2-reactive CD4(+) T cells in ~40%-60% of unexposed individuals, suggesting cross-reactive T cell

151 eactive CD4(+) T cells have been reported in unexposed individuals, suggesting preexisting cross-reac

152 profoundly impact the activation readout in unexposed individuals.

153 numbers of cytokine-producing cells even in unexposed individuals.

154 re common among HIV-exposed infants than HIV-unexposed infants (39.6% vs 28.9%; adjusted relative ris

155 were at increased risk for PTB compared with unexposed infants (6.4% exposed versus 4.4% unexposed; a

156 g lithium-exposed infants versus 0.18% among unexposed infants (adjusted risk ratio, 2.66; 95% CI, 1.

157 -6 but lower I-FABP than HIV-exposed and HIV-unexposed infants (P < .001).

158 CRP was higher in HIV-exposed than HIV-unexposed infants (P = .02).

159 CRP remained higher in HIV-exposed vs HIV-unexposed infants (P = .04).

160 during the first trimester as compared with unexposed infants and, in secondary analyses, with infan

161 The HEU and HIV-unexposed infants had a similarly high prevalence of CMV

162 Additional analyses showed that exposed and unexposed infants had generally similar Apgar scores at

163 pregnancy relative to a comparison group of unexposed infants of mothers with histories of psychiatr

164 EU infants have higher inflammation than HIV-unexposed infants until at least 6 months, which may con

165 malformations comparing exposed infants with unexposed infants was 1.25 (95% CI=0.13-12.19).

166 infants exposed to lithium as compared with unexposed infants was 1.65 (95% confidence interval [CI]

167 us (HIV)-exposed, uninfected infants (vs HIV-unexposed infants) were at increased risk of hospitalize

168 , 95% CI 1.17-1.67, p < 0.001) compared with unexposed infants.

169 194 HIV-exposed uninfected (HEU) and 197 HIV-unexposed infants.

170 ospitalization in perinatal HIV-exposed and -unexposed infants.

171 ns was 1.4% for exposed infants and 1.1% for unexposed infants.

172 idity, and impaired growth compared with HIV-unexposed infants.

173 al virus-associated hospitalization than HIV-unexposed infants.

174 sured covariates while comparing exposed and unexposed infants.

175 icant differences in BWT between exposed and unexposed infants: very preterm -61.54 g (+/-SE 28.62, P

176 lting from bladder cancer (for >2.56 ppm vs. unexposed, lagged 10-year HR = 2.96, 95% CI: 1.38, 6.34;

177 pe than a male whose Y chromosome is from an unexposed male.

178 al-time-PCR analysis indicated that CRHOEdev unexposed males exhibit significant changes in Crhr2 exp

179 ous abortion, compared with 563 among 13,246 unexposed matched women.

180 able isotopes (delta(15)N and delta(13)C) in unexposed mayflies.

181 proved nutrition in early life compared with unexposed men.

182 re foci of invasive cancer than pancreata of unexposed mice (controls); radiation reduced survival ti

183 ouse strains, C57BL/6J and CAST/EiJ, both in unexposed mice and in mice exposed to a model DNA-damagi

184 Fkbp51 in forebrain of CRHOEdev exposed and unexposed mice were examined 7 days after predator stres

185 38% reduction in CcO activity compared with unexposed mice.

186 rmore, we report that Toxoplasma-exposed and unexposed monocytes are transcriptionally distinguished

187 d all colorectal adenomas (39.0% vs 19.0% in unexposed; mOR = 3.29; 95% CI: 2.16-5.03; P < .001).

188 valence of villous adenomas (5.5% vs 1.3% in unexposed; mOR = 6.28; 95% CI: 2.02-19.53; P = .001) and

189 ), biologics (n = 642), or both (n = 227) vs unexposed (n = 379).

190 In this study, HIV-unexposed (n = 95), HEU (n = 84), HIV/Immed-ART-12 (n =

191 sed neonates (5.2%, 95% CI 3.8-6.9) than HIV-unexposed neonates (1.4%, 95% CI 0.9-2.0).

192 ely to have neonatal GBS disease compared to unexposed neonates.

193 greater severe mental illness rates than did unexposed offspring (moderate smoking during pregnancy:

194 s associated with all outcomes compared with unexposed offspring (preterm birth odds ratio [OR], 1.47

195 s 4.98% in exposed offspring versus 4.96% in unexposed offspring (risk difference, 0.02% [95% CI, -0.

196 rth, 30 years), 6.98% of exposed vs 4.78% of unexposed offspring were preterm, 2.54% of exposed vs 2.

197 Exposed offspring were compared with 197 588 unexposed offspring.

198 7 (4.97%) exposed offspring and 9443 (4.78%) unexposed offspring.

199 perm morphology compared to the lineage from unexposed parents.

200 parent was exposed; the control lineage had unexposed parents.

201 -group comparison of ferumoxytol-exposed and unexposed participants and a within-group (subgroup) com

202 idence intervals (CIs) comparing exposed and unexposed participants were estimated using Cox proporti

203 nancy outcome compared to 23 of 82 (23%) IPT-unexposed participants.

204 ent chemical and surface properties from the unexposed particles, while there were no significant dif

205 compared with a propensity score-matched ICM-unexposed patient group.

206 o be similar to propensity score-matched ICM-unexposed patient groups; similar data in pediatric pati

207 omparing with a propensity score-matched ICM-unexposed patient sample undergoing abdominal US.

208 Overall, 336 lymphoma cases occurred: 220 in unexposed patients (incidence rate [IR] per 1000 person-

209 significantly different between exposed and unexposed patients (median, 47 [IQR, 36-53] vs 54 [IQR,

210 Among unexposed patients (n = 17,060), there were 7,218 deaths

211 maltreatment-related concern) and up to two unexposed patients (those without such read codes) from

212 ignificantly smaller EF and SF declines than unexposed patients across courses and a lower risk for L

213 rate of C difficile infection in exposed and unexposed patients and factors associated with transmiss

214 posed to psychological distress, compared to unexposed patients, controlling for socioeconomic charac

215 In a multivariable Cox model, compared with unexposed patients, the risk of lymphoma was higher amon

216 ng were similar for dexrazoxane-exposed and -unexposed patients.

217 se events at baseline, in the exposed versus unexposed patients.

218 eactivity against SARS-CoV-2 was observed in unexposed people; however, the source and clinical relev

219 g exposed individuals (64.0 exposed vs. 96.2 unexposed per 100,000 person-years, P < 0.001).

220 asaki disease in the risk interval versus in unexposed person-time were 0.84 (95% CI 0.65-1.08; p = 0

221 is the "dry-run" analysis, which divides the unexposed population into "pseudo-exposed" and "pseudo-u

222 ing strains were introduced into the therapy-unexposed population via at least 6 independent transmis

223 divided by cumulative risk of stroke in the unexposed population, were determined, and stroke risk f

224 finches (Haemorhous mexicanus) from disease-unexposed populations, which have not evolved protective

225 ns exposed to P. vivax malaria compared with unexposed populations.

226 e differences between the actual exposed and unexposed populations.

227 The reference group consisted of 1,744,447 unexposed pregnancies (24 years; 40% white).

228 ccording to endoscopy status using 1,589,173 unexposed pregnancies as reference.

229 Gabapentin-unexposed pregnancies served as the reference.

230 Comparing lithium-exposed with unexposed pregnancies, significance remained for any mal

231 exposed pregnancies and 131 cases among 1852 unexposed pregnancies; hazard ratio, 0.71; 95% CI, 0.45

232 1 exposed pregnancies and 4 cases among 2004 unexposed pregnancies; hazard ratio, 2.43; 95% CI, 0.45

233 exposed pregnancies and 783 cases among 7072 unexposed pregnancies; prevalence odds ratio, 0.86; 95%

234 exposed pregnancies and 277 cases among 7072 unexposed pregnancies; prevalence odds ratio, 1.10; 95%

235 exposed pregnancies and 407 cases among 7096 unexposed pregnancies; prevalence odds ratio, 1.15; 95%

236 exposed pregnancies and 220 cases among 6660 unexposed pregnancies; prevalence odds ratio, 1.19; 95%

237 l carbamazepine exposure history impacts the unexposed progeny up to the F(4) generations and that pa

238 = 4.68/1,000 person-years) compared with the unexposed (rate = 7.21/1,000 person-years).

239 person-years, 203.6 for exposed vs 219.3 for unexposed; rate ratio, 0.93; 95% CI, 0.87-0.99).

240 person-years, 300.6 for exposed vs 257.5 for unexposed; rate ratio, 1.17; 95% CI, 0.94-1.45).

241 0%-0%) to 74% (95% CI 72%-76%); (3) conflict-unexposed regions had a pooled prevalence of probable PT

242 ce: [Formula: see text] comparing exposed to unexposed) replicated ([Formula: see text]) in an EPIC (

243 no significance was observed in the nicotine-unexposed sample (n = 2,548) of the replication cohort.

244 ethod was applied to a convenience set of 96 unexposed serum samples and a blinded set of 80 samples

245 nomas >/=10 mm was higher among exposed than unexposed siblings (10.5% vs 1.8%; mOR = 8.59; 95% CI: 3

246 ant when exposed children were compared with unexposed siblings (incidence of autism spectrum disorde

247 (OR = 1.05, 95% CI 0.96-1.14, p = 0.27), and unexposed siblings (OR = 0.99, 95% CI 0.85-1.14, p = 0.9

248 Corresponding risks in exposed versus unexposed siblings were also estimated.

249 ders) and by comparing exposed children with unexposed siblings.

250 causes these effects and stress responses at unexposed sites distal to the irradiated tissue.

251 KH mouse model skin samples as compared with unexposed skin tissue.

252 osed staff (281 males; 44+/-9 years) and 280 unexposed subjects (179 males; 43+/-7years).

253 mong the exposed subjects and 2.5% among the unexposed subjects (matched odds ratio [mOR] = 6.05; 95%

254 survivors, 55.7% (95% CI, 53.2% to 58.2%) of unexposed survivors, and 26.3% (95% CI, 24.0% to 28.3%)

255 y, with at least a 1-fold change relative to unexposed thrombocytes.

256 s assessed in 2 groups: children exposed and unexposed to a household member with tuberculosis.

257 (WWOE) was compared with that among infants unexposed to AEDs and born to WWOE.

258 Mortality for the 662 infants (14.4%) unexposed to ANS was 20.6% (136 of 661).

259 xposed to antenatal chemotherapy vs 12 [35%] unexposed to antenatal chemotherapy).

260 o antidepressants and in 819 children (0.5%) unexposed to antidepressants.

261 We included 323 isolates; 212 were unexposed to azithromycin, 14 were exposed </=30 days, a

262 We included 323 isolates: 212 were unexposed to azithromycin, 14 were exposed </=30 days, a

263 exposed to IFX as the only biologic vs those unexposed to biologics (malignancy: 1.12/1000 patient-ye

264 ed p53, PAI-1, and apoptosis in AECs of mice unexposed to bleomycin.

265 ercise capacity between survivors exposed or unexposed to cardiotoxic therapy and control subjects, a

266 ssification of individuals as exposed versus unexposed to early-life adversity may dilute observed ef

267 3 women exposed to FHT and 928 408 women not unexposed to FHT.

268 Compared with patients who were unexposed to ganapentinoids, gabapentinoid exposure was

269 HIV-exposed but not infected, and 1,478 were unexposed to HIV.

270 on, 8 years +/- 6; 484 female patients]; 925 unexposed to ICM [mean age, 7 years +/- 6; 484 female pa

271 eningioma cases among men were classified as unexposed to lead.

272 use of high-risk breast cancer screening) or unexposed to only gadoterate meglumine underwent 3.0-T b

273 4 and 31 weeks, without severe anomalies and unexposed to repeated courses of ANS.

274 of 61359 pregnancies, offspring exposed and unexposed to the influenza A(H1N1) vaccine during pregna

275 Compared with patients who were unexposed to thrombotic agents, the rates of hematuria-r

276 and closed workplaces with a reference group unexposed to unemployment.

277 Unexposed UK controls were also recruited.

278 differ between AED-exposed children and AED-unexposed unaffected children (median dnSNV/indel number

279 hout BDs not exposed prenatally to AEDs (AED-unexposed unaffected children), and AED-exposed children

280 exposed unaffected children, and 11 with AED-unexposed unaffected children.

281 morbidity and mortality, as compared to HIV-unexposed uninfected (HUU) infants.

282 the first 2 years of life among HEU and HIV-unexposed uninfected children (HUU) in the U.S.

283 the first 2 years of life among HEU and HIV-unexposed uninfected children (HUU) in the United States

284 istribution of syphilis-exposed and syphilis-unexposed uninfected children, and World Health Organiza

285 ore susceptible to severe infection than HIV-unexposed, uninfected (HUU) children, with altered innat

286 pregnant women were participants in the HIV-Unexposed-Uninfected (HU2) study.

287 f intra-arrest transport) at that same time (unexposed), using a time-dependent propensity score.

288 15 years, and 12.63% of exposed vs 5.46% of unexposed were diagnosed with attention-deficit/hyperact

289 estational age, 5.28% of exposed vs 2.14% of unexposed were diagnosed with autism spectrum disorder b

290 g were preterm, 2.54% of exposed vs 2.19% of unexposed were small for gestational age, 5.28% of expos

291 ative VTS children (332 HIV exposed, 574 HIV unexposed) were assessed in terms of cognition (Kaufman

292 xillary hair, and acne development comparing unexposed with those prenatally exposed for more than 12

293 5%CI: 1.0, 6.5; p=0.048) times higher in IPT-unexposed women compared to IPT-exposed women after cont

294 rval, 1.0-6.5; P = .048) times higher in IPT-unexposed women compared with IPT-exposed women after co

295 formula as infants have larger fibroids than unexposed women provides further support for persistent

296 fibroid size between soy formula-exposed and unexposed women using multivariable linear regression.

297 roids had significantly larger fibroids than unexposed women with fibroids.

298 ol/L; -1.8, 0.2 mmol/L, nonsignificant) than unexposed women.

299 pregnancy outcome compared with 23 (28%) IPT-unexposed women.

300 s (HIV)-exposed uninfected (HEU) and 100 HIV-unexposed Zimbabwean infants aged 6 weeks.