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1 [31 of 73] vs 57.8% [52 of 90]; P = .053 by univariate analysis).
2 hat were significant at the 0.2 level in the univariate analysis.
3 herapy interventions that was not evident by univariate analysis.
4 different between both outcome groups in the univariate analysis.
5 their impact on biomarkers was identified by univariate analysis.
6 were predictors of successful downstaging on univariate analysis.
7 ssociated with a lower CMV infection rate on univariate analysis.
8 cally significant variables as determined by univariate analysis.
9 eatures predicted unfavourable survival in a univariate analysis.
10 Student t and chi tests were used for univariate analysis.
11 pervised learning method and by conventional univariate analysis.
12 performed by using parameters with P < .2 in univariate analysis.
13 were associated with fistula formation in a univariate analysis.
14 results predicted postoperative delirium on univariate analysis.
15 s with ESRD (73.9% versus 71.8%, P<0.001) on univariate analysis.
16 were associated with higher ABSITE scores on univariate analysis.
17 ) were associated with excellent outcomes in univariate analysis.
18 ginal multivariate phenotypic traits for the univariate analysis.
19 nization were significant predictors only by univariate analysis.
20 icantly associated with future events in the univariate analysis.
21 HR of CD49d for OS was 2.5 (2.3 for TFS) in univariate analysis.
22 associated with different LSF categories at univariate analysis.
23 sociated with longer hospitalization only on univariate analysis.
24 associated with reduced graft survival in a univariate analysis.
25 score is associated with shorter survival in univariate analysis.
26 roups of patients were compared using paired univariate analysis.
27 of postoperative double AC formation in the univariate analysis.
28 line predictors of more rapid progression on univariate analysis.
29 f biomarkers, and was in accordance with the univariate analysis.
30 hed a significance level of less than .05 in univariate analysis.
31 factors independently associated with CR by univariate analysis.
32 was associated with a low mortality rate on univariate analysis (0.7% vs 1.2%, P = 0.05), a shorter
33 ession model, the 2 covariates identified on univariate analysis (1 geometric and 1 stress) were foun
34 ars did not differ between the groups in the univariate analysis (31.2% [95% CI 26.8-35.5] with intra
35 roved survival to hospital discharge on both univariate analysis (52% vs 33%, p = 0.054) and after co
36 Of 2,781 CF patients, 2,100 were used for univariate analysis, 764 for Kaplan-Meier survival funct
37 ositive or HER2-positive disease, however on univariate analysis a high RUNX1 protein was significant
45 differences between groups were assessed by univariate analysis and confirmed using a multivariate m
47 ultivariate model, factors with p < 0.200 on univariate analysis and factors derived from the previou
51 , MetS, and periodontitis was tested through univariate analysis and multivariate logistic regression
53 he influence of risk variables was tested by univariate analysis and multivariate logistic regression
54 Outcomes were compared in these cohorts via univariate analysis and multivariate logistic regression
56 d complete data on all factors identified in univariate analysis and were included in multivariate an
57 antly associated with both PFS and OS in the univariate analysis and were still statistically signifi
59 portance in projection) and the results of a univariate analysis (ANOVA), allowed the identification
69 OP requiring treatment after keratoplasty in univariate analysis but not in multivariate analysis.
70 Baseline HOMA-IR was associated with SVR in univariate analysis, but not after adjustment for other
71 R = 1.66 [95% CI: 1.05-2.61]; p = 0.0291) at univariate analysis, but not at the multivariate analysi
72 greater odds of showing advanced glaucoma in univariate analysis, but not in multivariate analyses.
73 ) correlated with better seizure outcomes on univariate analysis, but only epilepsy duration remained
74 correlated with higher clinical LE rates on univariate analysis, but only excising more than 22 lymp
75 ne use was negatively associated with SVR in univariate analysis, but this association was not signif
76 = 1.72; 95% CI 1.28-2.32; p < 0.001) in the univariate analysis, but with no significance in the mul
83 horter in the transcutaneous biopsy group on univariate analysis compared to the other groups; howeve
92 ighty-one HTx recipients were included, with univariate analysis demonstrating peak hazards of mortal
104 can predict poor cancer specific survival on univariate analysis for locally advanced and metastatic
106 nomic, and clinical factors were analyzed by univariate analysis for their association with 30-day mo
107 ses compared with patients without CP in the univariate analysis for untreated patients (hazard ratio
114 as not significantly associated with tSCC in univariate analysis (hazard ratio = 1.48; 95% CI, .95-2.
115 val in univariable and multivariable models (univariate analysis, hazard ratio [HR] for a one-fold in
116 ith an increased significant LVEF drop risk (univariate analysis: hazard ratio, 4.52; P < .001 and ha
117 owed a 58% reduction in risk of death in Cox univariate analysis (hazards ratio-HR = 0.42 confidence
118 for all variables showing the association in univariate analysis, HCM itself remained a robust predic
124 r rates of seizure recurrence (p = 0.004) in univariate analysis; however, its predictive value did n
138 st and powerful alternatives to the standard univariate analysis in genome-wide association studies.
145 associated with different LSF categories at univariate analysis; infiltrative morphologic structure,
149 r risk of distant metastases at follow-up in univariate analysis (Log-rank P = 0.0084) but not in mul
157 = 0.07), patients with PNI had worse DFS at univariate analysis (median DFS: 20 vs 15 months, P < 0.
169 s significantly associated with mortality in univariate analysis (odds ratio = 1.08 per mmol/L glucos
170 nificantly associated with mortality both in univariate analysis (odds ratio = 1.09 per 0.1 stress hy
171 associated with noninfectious uveitis in the univariate analysis (odds ratio, 2.53; 95% CI, 1.42-4.51
184 ong animal- and plant-based samples, one-way univariate analysis of variance followed by pair-wise co
188 CI, 0.28-0.71; p = 0.0007 and interleukin-6 univariate analysis only: odds ratio, 0.55; 95% CI, 0.36
189 en compared with premenopausal status in the univariate analysis (OR = 1.314, P = 0.043), and such re
190 with susceptibility to secondary DHF in the univariate analysis (OR = 1.60, 95% CI, 1.05-2.46), wher
193 ively, and the difference was significant in univariate analysis (P = .004) and in multivariate analy
194 ted with more perioperative complications on univariate analysis (P = .05), but multivariate regressi
196 FR-1 (chi(2)=45.5) were highly predictive in univariate analysis (P<0.0001) and in multivariable anal
198 was found to be statistically significant on univariate analysis (P=0.034), with white patients havin
203 n differences in levels of activation (i.e., univariate analysis), patterns of activity (i.e., multiv
211 < .001) and those with BRCA wild-type HGSOC (univariate analysis: reader 1, HR = 2.42, P < .001; read
225 ivariate adjustment compared to studies with univariate analysis (RR 2.15 [1.27-3.64] vs. 1.15 [0.88-
226 of intention in both univariate and adjusted univariate analysis (salience/coherence beta = 0.59, 95%
231 205 listings (27 393 pre-PAS; 24 439 T2DM), univariate analysis showed reduced percentages for SPK p
252 s predictors of PFS and OS by using log-rank univariate analysis, the multivariate Cox proportional h
264 PEEP did not have a large enough effect in univariate analysis to warrant inclusion in the multivar
267 mine, two methodologies have been developed; univariate analysis using CN emission band and multivari
269 chnique (P = 0.008) were risk factors in the univariate analysis using Cox regression models, whereas
270 ensitivity troponin-T predicted mortality in univariate analysis (Wald = 8.4; p = 0.004), but not whe
271 tive incidence of grade III-IV acute GVHD on univariate analysis was 8%, 12%, and 17% in the haploide
273 OS, and relapse-free survival (RFS) seen in univariate analysis was even greater in the multivariate
278 significant prognostic factors identified by univariate analysis was performed using step-up and step
285 orresponding 1-year rates of chronic GVHD on univariate analysis were 13%, 51%, and 33%, respectively
286 nificant overall survival prognosticators on univariate analysis were albumin, bilirubin, ascites, tu
287 Oral factors associated with death in the univariate analysis were decreased frequency of dental v
288 iables reaching the statistical threshold in univariate analysis were entered into a multivariable Co
289 ning set, variables found significant in the univariate analysis were fed into a multivariate regress
290 ning set, variables found significant in the univariate analysis were fed into a multivariate regress
291 ssion, covariates with a P value < 0.20 from univariate analysis were included in multivariate models
293 se (regression coefficient: 0.7; P = .04) on univariate analysis, whereas age (<70 years old) was the
294 f values were found for SUVmax or SUVmean at univariate analysis, whereas MTV60 was confirmed as an i
295 icting melanoma specific survival (MSS) in a univariate analysis, which was also consistent with AJCC
297 ous coronary intervention were associated in univariate analysis with occurrence of heart failure.