ライフサイエンスコーパス: unresponsiveness

1 treatment (referred to as possible sustained unresponsiveness).

2 tact despite structural injury and prolonged unresponsiveness.

3 ited their activation and resulted in T-cell unresponsiveness.

4 nuation, indicating development of beta-cell unresponsiveness.

5 le of Treg cells in allergen-specific T-cell unresponsiveness.

6 owder and a cooked egg to test for sustained unresponsiveness.

7 months and were considered to have sustained unresponsiveness.

8 gesting a role for CD4+ T cells in enforcing unresponsiveness.

9 reas the loss of consciousness is defined by unresponsiveness.

10 imal T cell signaling events from functional unresponsiveness.

11 t may function to mediate TNF-induced T cell unresponsiveness.

12 M cells via psigma1 could induce a state of unresponsiveness.

13 and resulted in the induction of Ag-specific unresponsiveness.

14 transactivation activity, leading to T cell unresponsiveness.

15 se 3 is required for the induction of T cell unresponsiveness.

16 is a dominant hallmark that mandates T cell unresponsiveness.

17 ive either propofol or dexmedetomidine until unresponsiveness.

18 o examine the mechanism of in vivo rituximab unresponsiveness.

19 CAV and demonstrated in vitro donor-specific unresponsiveness.

20 T (T reg) cells to sites where they maintain unresponsiveness.

21 cyte reaction was used as standard to detect unresponsiveness.

22 n 30% desensitization, and 13% had sustained unresponsiveness.

23 pe and associates with poor prognosis and RA unresponsiveness.

24 e system employs to produce antigen-specific unresponsiveness.

25 l-established method of inducing immunologic unresponsiveness.

26 ipients demonstrated in vitro donor-specific unresponsiveness.

27 ion responses were associated with sustained unresponsiveness.

28 receptor blockade partially reversed T cell unresponsiveness.

29 nical trials and, in some studies, sustained unresponsiveness.

30 lly associated with acquisition of sustained unresponsiveness.

31 delta power increased from responsiveness to unresponsiveness.

32 , and occurs through the induction of B cell unresponsiveness.

33 d with the likelihood of achieving sustained unresponsiveness.

34 tokine signal pathways and leading to T cell unresponsiveness.

35 cing febrile illness that leads to IFN-gamma unresponsiveness.

36 030 children [0.2%]) and not associated with unresponsiveness.

37 in T-cell exhaustion, a state of functional unresponsiveness.

38 feeding of peanut butter to assess sustained unresponsiveness.

39 domain-only protein (Hopx) to mediate T cell unresponsiveness.

40 sive leg raising test cases and thus preload unresponsiveness.

41 peanut powder, and all 4 achieved sustained unresponsiveness.

42 d reports about conscious experiences during unresponsiveness.

43 nd only 10.8% of subjects achieved sustained unresponsiveness.

44 th after stopping OIT and achieved sustained unresponsiveness.

45 e primary outcome was induction of sustained unresponsiveness 2 to 5 weeks after discontinuation of t

46 of 7443 mg of WP; 3 (13%) achieved sustained unresponsiveness 8 to 10 weeks off therapy.

47 ial-precoeruleus complex produced behavioral unresponsiveness, a monotonous sub-1-Hz cortical EEG, an

48 allenge with alloantigen failed to break the unresponsiveness, a transient recovery from tolerance wa

49 with 10 of these 12 demonstrating sustained unresponsiveness after 2 to 4 weeks.

50 3 years, (3) percentage attaining sustained unresponsiveness after 3 years, (4) immunologic end poin

51 Sustained unresponsiveness after 4 weeks of avoidance was seen in

52 urther work is required to confirm sustained unresponsiveness after a longer period of secondary pean

53 The majority of children with sustained unresponsiveness after extended egg OIT are able to succ

54 e transient desensitization versus sustained unresponsiveness after OIT.

55 This is the first demonstration of sustained unresponsiveness after peanut OIT, occurring in half of

56 mine whether peanut OIT can induce sustained unresponsiveness after withdrawal of OIT.

57 To identify a mechanism for T cell unresponsiveness against mycobacterial lipid Ags in lepr

58 The Full Outline of UnResponsiveness, an emerging alternative, is more sensi

59 was associated with attainment of sustained unresponsiveness and a reduction in adverse reactions.

60 Although anesthesia undoubtedly induces unresponsiveness and amnesia, the extent to which it cau

61 nating from these MSCs retain their TGF-beta unresponsiveness and become inflammatory.

62 Parasite Ag-specific T cell unresponsiveness and diminished IFN-gamma production are

63 podocytes of diabetic mice, causing insulin unresponsiveness and DN.

64 of key inhibitory molecules reversed T cell unresponsiveness and enabled maximal T cell functions, e

65 was effective in inducing possible sustained unresponsiveness and immune changes that suggest modulat

66 ents, tested in vitro, showed donor-specific unresponsiveness and in specimens from allograft biopsie

67 In animal models, EPIT induces sustained unresponsiveness and prevents further sensitization medi

68 halitogenic determinant induced PLP-specific unresponsiveness and protected mice from induction of EA

69 h anti-idiotype 3H1 or CEA could reverse CEA unresponsiveness and result in the induction of CEA-spec

70 to those with well-documented growth hormone unresponsiveness and severe short stature.

71 ne animal maintained donor-specific cellular unresponsiveness and stable anti-alphaGal antibody level

72 o baboons can induce donor-specific cellular unresponsiveness and stable anti-alphaGal antibody level

73 with severe COVID-19 who, despite prolonged unresponsiveness and structural brain abnormalities, dem

74 mechanisms, PD-L1-dependent T cell-intrinsic unresponsiveness and the activation of T regulatory cell

75 t partial TCR signaling that leads to T cell unresponsiveness and tolerance in vivo.

76 ed by drug effects, by confusing behavioral "unresponsiveness" and internally generated consciousness

77 tures of the human disease, including T cell unresponsiveness, and thus represent an appropriate mode

78 tress, erythema, decreased body temperature, unresponsiveness, and, often, death.

79 The PLA2G1B/gp41 pair induced CD4+ T cell unresponsiveness (anergy).

80 Immunological unresponsiveness-anergy- of CD4(+) T cells is characteri

81 p27-differentiated macrophages induce severe unresponsiveness/anergy in T cells.

82 Antiestrogen unresponsiveness appears to be the major acquired resist

83 ugh the molecular mechanisms underlying this unresponsiveness are unknown, some models of B cell aner

84 respond and/or relapse and the mechanisms of unresponsiveness are unknown.

85 spectively) and thus did not predict preload unresponsiveness (area under the receiver-operating char

86 l patients (0 of 8) displayed donor specific unresponsiveness as gauged by IFN-gamma expression and T

87 hallenge with an optional month-38 sustained unresponsiveness assessment.

88 een subjects underwent an optional sustained unresponsiveness assessment; 14 of those (77.8%) maintai

89 were evaluated for continuation of sustained unresponsiveness at 30 months and 36 months.

90 The primary end point, sustained unresponsiveness at 4 weeks after stopping early interve

91 Four subjects had sustained unresponsiveness at study completion.

92 olerance is manifest as a bias toward immune unresponsiveness, both in the context of a major histoco

93 ne is remarkable in that it induces profound unresponsiveness, but subjects often report "ketamine dr

94 ted lympholysis assays showed donor-specific unresponsiveness by day 30 across MHC class I barriers.

95 ote the induction of antigen-specific T-cell unresponsiveness by DC.

96 f B7/CD28 may facilitate induction of T-cell unresponsiveness by generating AAMphi.

97 animals demonstrated in vitro donor-specific unresponsiveness by MLR and CML and did not demonstrate

98 then slowly induced longer lasting mast cell unresponsiveness by removing membrane FcepsilonRI.

99 cells persist in the periphery in a state of unresponsiveness called anergy.

100 tive at old than at young age, due to T cell unresponsiveness, caused by various age-related changes

101 (PPOIT) was effective at inducing sustained unresponsiveness compared with placebo in a double-blind

102 g rapid eye movement (REM) sleep, behavioral unresponsiveness contrasts strongly with intense brain-w

103 Anergic features and chemokine unresponsiveness could be simultaneously reversed by cul

104 toms or memory problems; 76 had seizures, 88 unresponsiveness (decreased consciousness), 86 dyskinesi

105 entation to hospital, low weight for height, unresponsiveness, deep breathing, hypoxemia, grunting, a

106 In unresponsiveness, delta waves propagated from frontal to

107 ring passive leg raising can predict preload unresponsiveness diagnosed by the absence of increase in

108 This IL-2 unresponsiveness did not require the continuous presence

109 lts unveil a fundamental mechanism of T cell unresponsiveness different from anergy or exhaustion, dr

110 ediates this dynamic antigen-specific T cell unresponsiveness differs from previously described forms

111 ect thalamocortical activity contributing to unresponsiveness during sleep.

112 cell engraftment and in vitro donor-specific unresponsiveness, enabling prolonged survival of subsequ

113 he induction of chimerism and donor-specific unresponsiveness following pig SpTx.

114 in addition to being markers for immunologic unresponsiveness, gene expression levels of TCL1A and CD

115 ildren with egg allergy and induce sustained unresponsiveness in a clinically significant subset.

116 n primates and induced long-term Ag-specific unresponsiveness in a memory T cell-mediated inflammator

117 Treatment may result in sustained unresponsiveness in a proportion of patients, whereas ot

118 f immunological tolerance (long-term antigen unresponsiveness in an immunocompetent host) presents th

119 e characterized a common mechanism of T cell unresponsiveness in cancer driven by the upregulation of

120 fe and effective method of inducing specific unresponsiveness in CD4+ T cells for the prevention and

121 (+)Ifngr1(f/f) mice with selective IFN-gamma unresponsiveness in CD8alpha(+) dendritic cells displaye

122 An initial assessment of IL-2 unresponsiveness in cells from selected HIV-infected ind

123 Attempts to identify growth hormone unresponsiveness in children with idiopathic short statu

124 iotic and peanut OIT and assessing sustained unresponsiveness in children with peanut allergy.

125 gether, our findings indicate that chemokine unresponsiveness in CLL lymphocytes results from failure

126 ergy, an acquired state of T cell functional unresponsiveness in Foxp3(-) cells, have both been impli

127 e to peripheral self-Ags is the induction of unresponsiveness in mature specific T cells.

128 de (alpha-GalCer) induces long-term NKT cell unresponsiveness in mice.

129 lood indicated donor-specific posttransplant unresponsiveness in micro-cell-mediated lympholysis (m-C

130 f the VTL grafts demonstrated donor-specific unresponsiveness in MLR assays, development of periphera

131 effective in achieving donor-specific immune unresponsiveness in models of organ transplantation.

132 otocol and evidence for early donor-specific unresponsiveness in one of these patients.

133 d accompany the acquisition of food allergen unresponsiveness in oral immunotherapy.

134 regulatory T cells, and through induction of unresponsiveness in precursors of T effector cells, beta

135 anti-CD3 directly induces a state of immune unresponsiveness in primed pathogenic autoreactive effec

136 All recipients showed donor-specific unresponsiveness in standard cell-mediated lympholysis a

137 gnaling induces a state of anergy or antigen unresponsiveness in T cells, mediated through calcineuri

138 e mediated through deletion and induction of unresponsiveness in targeted memory T-cell populations.

139 the induction of immunological tolerance (or unresponsiveness in the absence of exogenous immunosuppr

140 n the recipients and donor-specific cellular unresponsiveness in vitro.

141 one of the animals displayed donor-specific unresponsiveness in vitro.

142 DCs and the promotion of DC-mediated T cell unresponsiveness in vivo.

143 as Hopx) in iT(reg) cells to mediate T cell unresponsiveness in vivo.

144 novel category is characterized by their ABA unresponsiveness in Ws and activation in rop10-1 at 1 mi

145 ergy, an acquired state of T cell functional unresponsiveness, in natural peripheral tolerance remain

146 This MC unresponsiveness is antigen-specific and covers the seru

147 ss to irrelevant, nontolerizing Ag, and this unresponsiveness is associated with significant apoptosi

148 This unresponsiveness is associated with the constitutive act

149 ely inhibited in autoreactive cells in which unresponsiveness is maintained by anergy.

150 e presence and persistence of donor-specific unresponsiveness is not available.

151 ponse in newborns and the mechanism for this unresponsiveness is not clear.

152 The mechanism of rituximab unresponsiveness is not known.

153 This unresponsiveness is rapidly reversible, requiring contin

154 ss of UCB lymphocytes is attributable to pan-unresponsiveness, lymphocyte repressive or recipient-spe

155 a TLR agonist LPS, suggesting that NKT cell unresponsiveness may be a major mechanism of terminating

156 This serotype-specific unresponsiveness may reflect immune paralysis due to lar

157 early development of antigen-specific T-cell unresponsiveness mediated by BM-derived antigen-presenti

158 Preload unresponsiveness (negative passive leg raising test) was

159 eed to be treated for 7 to achieve sustained unresponsiveness (number needed to treat, 1.27; 95% CI,

160 These results suggest that the Ag unresponsiveness of anergic B cells can be overcome by c

161 Thus maintenance of the unresponsiveness of anergic cells is critical for preven

162 The sustained unresponsiveness of at least 16 weeks after vaccination

163 on, responsible for lipopolysaccharide (LPS) unresponsiveness of C3H/HeJ mice, into the TIR domain of

164 Unresponsiveness of COX-2 to IL-10 is due to the deficie

165 th CO(2)), based on previous studies showing unresponsiveness of Glu deprotonation to CO(2).

166 on and induced a variable period of relative unresponsiveness of IgG anti-dsDNA-producing B cells, as

167 The surprising unresponsiveness of Inhba expression to hypoxia was conf

168 tumor cells, which stands in contrast to the unresponsiveness of KRAS-mutant cancers to EGFR-directed

169 ssion of FoxP3N/NLS sufficiently induces the unresponsiveness of mouse primary CD4+ CD25- T cells, wh

170 These data indicate that there is an early unresponsiveness of neonatal alveolar macrophages to Pne

171 n vivo and in vitro, fully rescues the ppGpp-unresponsiveness of RNAP lacking omega, likely explainin

172 gehog pathway inhibitor, cyclopamine, and 4) unresponsiveness of Smoothened-/- mouse embryonic fibrob

173 Immunological unresponsiveness of T cells to alloantigen can be induce

174 the transcription factor NFATc2, as well as unresponsiveness of T cells.

175 on, our findings explain the profound immune unresponsiveness of the Aly mouse.

176 The unresponsiveness of the memory cells from the nonpersist

177 ch dramatically contrasted with the complete unresponsiveness of the MSG-derived hepatocytes, also as

178 Autoreactive B cells can maintain anergy via unresponsiveness of their BCRs to self-antigens.

179 The unresponsiveness of Thy-1 (+) cells is not because of de

180 In contrast to the in vitro unresponsiveness of Treg cells when cultured alone, subs

181 echanisms underlying how FoxP3 maintains the unresponsiveness of Tregs.

182 duced TACI expression is responsible for the unresponsiveness of XID mouse to TI-2 Ags and BCR activa

183 d (APRIL) or BAFF and their receptors in the unresponsiveness of XID mouse to TI-2 Ags.

184 highlighted the critical role of functional unresponsiveness or 'anergy'.

185 n the mature repertoire, but that functional unresponsiveness or anergy exists in the mature B-cell r

186 T cell unresponsiveness or anergy is one of the mechanisms that

187 jections of alpha-GalCer result in long-term unresponsiveness or anergy of iNKT cells, severely limit

188 l killer T (NKT) cells, results in long-term unresponsiveness or anergy, which severely limits its cl

189 ipheral T-cell tolerance, which manifests as unresponsiveness or death through apoptosis.

190 nce that DCs in situ induce antigen-specific unresponsiveness or tolerance in central lymphoid organs

191 not related to inadequate nutrition, insulin unresponsiveness, or growth hormone deficiency.

192 -positive periphery with no signs of anergy, unresponsiveness, or prior activation.

193 at the time of desensitization or sustained unresponsiveness oral food challenges.

194 Unresponsiveness rarely denoted unconsciousness, as the

195 ted specifically to acquisition of sustained unresponsiveness rather than to receiving PPOIT treatmen

196 The induction of alloantigen-specific unresponsiveness remains an elusive goal in organ transp

197 ytokines responsible as a mechanism for this unresponsiveness, restimulation assays revealed increase

198 evidence of heterogeneity in Full Outline of Unresponsiveness score agreement across hospitals.

199 This demonstrates that the Full Outline of Unresponsiveness score can be utilized reliably in criti

200 et of patients for which the Full Outline of Unresponsiveness score could be particularly beneficial

201 The Full Outline of Unresponsiveness score has emerged as an alternative to

202 ter-rater reliability of the Full Outline of Unresponsiveness score in five intensive care units.

203 linked to Glasgow Coma Scale/Full Outline of UnResponsiveness score information.

204 The Full Outline of UnResponsiveness score may be a better predictor of mort

205 The Full Outline of UnResponsiveness score might be a better prognostic tool

206 tem reflex components of the Full Outline of UnResponsiveness score showed a much wider range of mort

207 The Full Outline of Unresponsiveness score showed excellent inter-rater agre

208 , 0.663-0.768) and using the Full Outline of UnResponsiveness score was 0.742 (95% CI, 0.694-0.790),

209 In multivariable models, the Full Outline of UnResponsiveness score was more useful than the Glasgow

210 Glasgow Coma Scale and Full Outline of UnResponsiveness score were recorded within 1 hour of ad

211 xes and respiration into the Full Outline of UnResponsiveness score.

212 a new coma score, the FOUR (Full Outline of UnResponsiveness) score.

213 ts that the successful induction of specific unresponsiveness secondary to intrathymic transplantatio

214 upon activation, and CD39-/- DCs showed ATP unresponsiveness (secondary to P2-receptor desensitizati

215 mediate some of the parasite-specific T cell unresponsiveness seen in patent filarial infection.

216 n which 27.5% of subjects achieved sustained unresponsiveness (SU) after 2 years.

217 23 of 55 subjects passed the 10-g sustained unresponsiveness (SU) challenge 8 weeks after discontinu

218 While desensitization and sustained unresponsiveness (SU) have been shown with egg oral immu

219 nduced desensitization in most and sustained unresponsiveness (SU) in a smaller subset.

220 h OIT; after avoidance, 9 achieved sustained unresponsiveness (SU), and 13 had transient desensitizat

221 eanut OIT, a limited remission, or sustained unresponsiveness (SU), has further been demonstrated.

222 rechallenge at month 32 to assess sustained unresponsiveness (SU).

223 ific Abs in external secretions and systemic unresponsiveness termed oral or mucosal tolerance.

224 or the maintenance of specific immunological unresponsiveness that can reduce the severity of the det

225 Furthermore, it induced a profound state of unresponsiveness that could be overcome only by prolonge

226 sponse rates, however, was tempered by tumor unresponsiveness through both intrinsic and acquired dru

227 ays and consider the potential for reversing unresponsiveness through stimulatory signals or replacem

228 rritin level <=100 ng/mL) and intolerance or unresponsiveness to 1 month or more of oral iron were re

229 memory T cells that was able to break T cell unresponsiveness to a nonmutated tumor Ag and provide pr

230 ucocorticoid deficiency (FGD), or hereditary unresponsiveness to adrenocorticotropin (ACTH; OMIM 2022

231 in seven of nine animals, including complete unresponsiveness to Ag challenges in two animals.

232 -cell activation was followed by full T-cell unresponsiveness to allergen after 1 year in the MAT-Fel

233 , can be a powerful way of inducing specific unresponsiveness to alloantigens in vivo.

234 w-dose UVB exposure induces antigen-specific unresponsiveness to antigen(s) introduced through UV-irr

235 to deletion of the corresponding T cells and unresponsiveness to antigenic rechallenge with strong ad

236 s in erroneous T cell receptor signaling and unresponsiveness to antigenic restimulation.

237 telet function testing and presumed clinical unresponsiveness to aspirin, has been previously reporte

238 ion arrest in STAT5b-RARalpha(+) APL and its unresponsiveness to ATRA, we examined the effect of STAT

239 leukaemia cells, which refers to a state of unresponsiveness to B cell receptor stimulation.

240 ved poorly after adoptive transfer, and were unresponsiveness to BCR stimulation in vitro.

241 ne profile provides a mechanism that ensures unresponsiveness to commensal bacteria while maintaining

242 PC unresponsiveness to CXCL12 results in a marked accumulat

243 localized in splenic follicles despite their unresponsiveness to CXCL13.

244 mice complemented these defects and reversed unresponsiveness to DEN-induced liver injury and maligna

245 T cells, and those off drugs showed specific unresponsiveness to donor alloantigens.

246 gely T cell mediated, that promotes specific unresponsiveness to donor alloantigens.

247 a key mechanism for inducing and maintaining unresponsiveness to donor alloantigens.

248 oping novel strategies for inducing specific unresponsiveness to donor alloantigens.

249 injection of TIMP-GLIA nanoparticles induced unresponsiveness to gliadin and reduced markers of infla

250 cterized by a more advanced presentation and unresponsiveness to H. pylori eradication therapy.

251 Unresponsiveness to harmless antigens is established thr

252 of intracellular signaling may contribute to unresponsiveness to IFNalpha/beta in HIV-1 disease.

253 finding that was associated with lymphocyte unresponsiveness to IL-12 stimulation in vitro.

254 emagglutinin fusion peptide along with their unresponsiveness to inducers of IFN as measured in vitro

255 ting as disturbed lymph node homeostasis and unresponsiveness to inflammatory stimuli.

256 Accordingly, cured mice showed T-cell unresponsiveness to InsB9-23 stimulation and increased T

257 itamin A deficiency could be associated with unresponsiveness to interferon-based antiviral therapy.

258 onse to antiviral therapy are due to greater unresponsiveness to intracellular actions of interferon

259 These CD8+ T cells transferred unresponsiveness to naive recipients.

260 The observed unresponsiveness to OVA-psigma1 could be adoptively tran

261 tagenesis led to constitutive activation and unresponsiveness to PIP3 in vitro or insulin in vivo.

262 her the manipulation of PKB can overcome the unresponsiveness to protection of the diabetic myocardiu

263 myocardium and, importantly, it reversed the unresponsiveness to protection of the diabetic myocardiu

264 x, proliferated and failed to mediate T cell unresponsiveness to rechallenge with antigen.

265 al dendritic cells (DCs) that mediate T cell unresponsiveness to rechallenge with antigen.

266 kdown in any of the mechanisms that maintain unresponsiveness to self (a state known as self-toleranc

267 ndritic cells may play a role in maintaining unresponsiveness to self-Ag during chronic inflammation.

268 series of discrete checkpoints that maintain unresponsiveness to self.

269 es, a critical process in the maintenance of unresponsiveness to self.

270 These data suggest that the unresponsiveness to T-dependent Ags displayed by hCR2-po

271 mechanisms; apoptotic contraction and marked unresponsiveness to TCR stimulation, as a synchronized h

272 At day 120, MLR demonstrated unresponsiveness to the host and donor antigens but stro

273 veloping in SW thymus grafts showed specific unresponsiveness to the major histocompatibility complex

274 ns are effective immunogens to overcome host unresponsiveness to the nominal antigen, the structural

275 lating altered peptide ligand, L144, induced unresponsiveness to the self peptide, proteolipid protei

276 To overcome unresponsiveness to the self-high molecular weight melan

277 the SW but not the HU grafts showed specific unresponsiveness to the SW donor.

278 ocorticoid resistance (GCR) is defined as an unresponsiveness to the therapeutic effects, including t

279 s associated with the development of in vivo unresponsiveness to the transplanted spleen.

280 However, costs and unresponsiveness to therapy in a sizeable proportion of

281 patients at higher risk of GVHD progression, unresponsiveness to therapy, or death.

282 tolerance can be harnessed to induce humoral unresponsiveness to transplanted alloantigens.

283 One mechanism contributing to immunologic unresponsiveness toward tumors may be presentation of tu

284 ested by newborn mice, stimulates a state of unresponsiveness toward viral antigens.

285 Additionally, immunological 'unresponsiveness' towards the resident commensal microfl

286 baseline, transition into unresponsiveness, unresponsiveness, transition into responsiveness, and re

287 Mechanisms underlying this acquired unresponsiveness, typified by diminished functional resp

288 into five states: baseline, transition into unresponsiveness, unresponsiveness, transition into resp

289 Possible sustained unresponsiveness was achieved in 82.1% receiving PPOIT a

290 T cell unresponsiveness was associated with defects in TCR prox

291 Sustained unresponsiveness was further assessed by using identical

292 Unresponsiveness was partially overcome using high-dose,

293 CD19 unresponsiveness was partially reversible, where nonresp

294 The tolerant T-cell unresponsiveness was reversed by the addition of IL-2.

295 This unresponsiveness was reversible by treatment with anti-C

296 ndpoint of general anesthetics is behavioral unresponsiveness, which is commonly associated with loss

297 Three animals demonstrated donor-specific unresponsiveness, while maintaining normal alloresponses

298 ffects are seen that can distinguish between unresponsiveness with and without consciousness.

299 and language disintegration into a state of unresponsiveness with catatonic features often associate

300 is characterized by antigen-specific T-cell unresponsiveness with diminished IFN-gamma and IL-2 prod