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1  myocardial infarction; 90 patients [42.1%], unstable angina pectoris).
2 ents with acute coronary syndrome (82 MI, 44 unstable angina pectoris).
3 ed in patients with myocardial infarction or unstable angina pectoris.
4  infarct of an age less than the duration of unstable angina pectoris.
5 ocardial lesions develop in association with unstable angina pectoris.
6 ial ischemic necroses in other patients with unstable angina pectoris.
7 also produced the clinical manifestations of unstable angina pectoris.
8 ant prognostic significance in patients with unstable angina pectoris.
9  of recurrent myocardial infarction (MI) and unstable angina pectoris.
10  specifically and significantly activated in unstable angina pectoris and is not affected by severity
11 ents presenting with stable angina pectoris, unstable angina pectoris,and ST-segment elevation myocar
12                         Patients with MI and unstable angina pectoris had higher VEGF levels compared
13 tomy within 72 h of presentation with either unstable angina pectoris or acute myocardial infarction.
14 ischarge in 444 of 1473 patients with either unstable angina pectoris or non-Q-wave myocardial infarc
15 ients aged 18 years or older, with stable or unstable angina pectoris or patients who had a myocardia
16 ligible patients had stable angina pectoris, unstable angina pectoris, or non-ST-elevation myocardial
17 ute myocardial infarction (MI) (n = 205) and unstable angina pectoris (UAP) (n = 185).
18  acute myocardial infarction (MI) (n =57) or unstable angina pectoris (UAP) (n =60) were consecutivel
19 nsistent with the duration of the episode of unstable angina pectoris were observed in 6 of 21 patien
20                   We observed a patient with unstable angina pectoris who developed foci of ischemic