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1  sleep disruption that occurs in response to upper airway obstruction.
2 stridor, and extubation failure secondary to upper airway obstruction.
3 hemoglobin concentration, and hypoxemia from upper airway obstruction.
4 e when these children develop postextubation upper airway obstruction.
5 ithin 48 hrs of extubation in the absence of upper airway obstruction.
6 attacks, and the risk of asphyxiation due to upper airway obstruction.
7 disorder characterized by recurrent episodic upper airway obstruction.
8 e for reintubation was classified as airway (upper airway obstruction, 11; aspiration/excess pulmonar
9            Common reasons for exclusion were upper airway obstruction (13.5%) and cyanotic congenital
10             The model differentiates between upper airway obstruction and complications like bronchos
11 (OSA), characterized by recurrent periods of upper airway obstruction and intermittent hypoxaemia, is
12 rength, assessment of risk of postextubation upper airway obstruction and its prevention, use of post
13 resolution of some complications, especially upper airway obstruction and possibly immune-mediated an
14 2) assess the relationship between degree of upper airway obstruction and VB, and 3) assess the relat
15 ionale: Obstructive sleep apnea is recurrent upper airway obstruction caused by a loss of upper airwa
16 60 increased pharyngeal patency and relieved upper airway obstruction during non-REM sleep.Conclusion
17 ural drive to breathe in the pathogenesis of upper airway obstruction during sleep (OSA), based on th
18 re measurements correlate with the degree of upper airway obstruction during sleep and may have a rol
19 order characterized by recurrent episodes of upper airway obstruction during sleep resulting in oxyge
20 t sleep disorder, characterized by recurrent upper airway obstruction during sleep, resulting in inte
21 ep apnea (OSA) is characterized by recurrent upper airway obstruction during sleep.
22 t in many neuromuscular disorders mechanical upper airway obstruction from oropharyngeal weakness con
23 longer length of ventilation, postextubation upper airway obstruction, high respiratory effort postex
24 ologic disease, lower aPiMax, postextubation upper airway obstruction, higher preextubation positive
25         VB was associated with the degree of upper airway obstruction in a dose-response manner (VB(u
26 nces made in the prevention and treatment of upper airway obstruction in infancy and childhood.
27                        We determined whether upper airway obstruction in normal individuals with inta
28                                  Even though upper airway obstruction is potentially life-threatening
29  recapitulate craniofacial abnormalities and upper airway obstruction of human DS and can serve as an
30       When children developed postextubation upper airway obstruction, reintubation rates were 47.4%
31 when these children developed postextubation upper airway obstruction, reintubation rates were greate
32                  Despite all these advances, upper airway obstruction remains an important source of
33                                     However, upper airway obstruction secondary to severe bleeding in
34 latory pressures, pulmonary dysfunction, and upper airway obstruction that occur after combined smoke
35 xaemia owing to acute and temporary (12 min) upper-airway obstruction, the microbubble-mediated deliv
36 ntify clinically significant post-extubation upper airway obstruction (UAO) and differentiate subglot
37  upper airway resistance, using hallmarks of upper airway obstruction visible on clinical sleep studi
38                                              Upper airway obstruction was produced in 12 normal indiv
39                                Two levels of upper airway obstruction were induced in ten dogs by par