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1 capacity of cells contributing to the future upper lip.
2 al maxillary excess and hypermobility of the upper lip.
3 al maxillary excess and hypermobility of the upper lip.
4 dge, a broad and smooth philtrum, and a thin upper lip.
5 ferents, was activated by stimulation of the upper lip (5-30 V; 10 Hz), and BFcrb was recorded at the
6 ease of IQR, P-value 0.0371), and wrinkle on upper lip (7.7% more wrinkle on upper lip per increase o
7 minent eyes, a narrow nasal bridge, a tented upper lip, a high palate, an open mouth, tightly adheren
8 al examination revealed hypermobility of the upper lip and absence of generalized altered passive eru
11 activity in the primary somatosensory cortex upper lip and jaw (S1ULp-S1J) region of the somatosensor
12 face was represented from chin/lower lip to upper lip and neck/upper face in a rostrocaudal sequence
13 rther understand how retinoic acid regulates upper lip and palate morphogenesis we searched for genes
14 hat retinoic acid is a critical regulator of upper lip and primary palate development in Xenopus laev
15 better understand the complex nature of the upper lip and primary palate development which will lead
21 ormalin (1.5%, 50 mul) was injected into the upper lip and the animal's behaviors recorded for 45 min
22 representation correspond to the upper face, upper lip, and chin plus lower lip, whereas three or fou
23 ntubation included a grade of class 3 on the upper lip bite test (lower incisors cannot extend to rea
28 olved in regulating mid-face development and upper lip fusion and are therefore strong candidates for
33 ngival display (EGD) etiologies (hypermobile upper lip [HUL], altered passive eruption [APE], and sho
34 ed in response to tactile stimulation of the upper lip in 69% of cases, the lower lip in 85% of cases
35 res reorganizes after deafferentation of the upper lip in neonatal rats (postnatal day [PND] 1-30).
36 displacement) to the glabrous surface of the upper lip in order to index the modulation and specifici
37 rtently affect facial structures such as the upper lip, lateral aspect of the nose, and lower eyelid.
39 drives cNCC mesenchyme proliferation during upper lip morphogenesis, and that disruption of this seq
43 t (lower incisors cannot extend to reach the upper lip; positive likelihood ratio, 14 [95% CI, 8.9-22
44 bitemporal narrowing, broad nasal tip, thin upper lip, posteriorly rotated or low-set ears, and micr
45 izing enzyme is ectopically expressed in the upper lip primordia of Lrp6-deficient embryos, indicatin
48 The field potentials evoked in crus II by upper lip stimulation did not differ between wild-type a
50 somatosensory cortex blood flow produced by upper lip stimulation was not attenuated in D2-null mice
51 ], altered passive eruption [APE], and short upper lip [SUL]) in a nondental adult population and to
52 eatures that include a prominent maxilla and upper lip that readily reveal the upper gingiva, widely
53 This cross-sectional study aimed to examine upper lip (UL) and smile characteristics and soft tissue