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1 capacity of cells contributing to the future upper lip.
2 al maxillary excess and hypermobility of the upper lip.
3 al maxillary excess and hypermobility of the upper lip.
4 dge, a broad and smooth philtrum, and a thin upper lip.
5 ferents, was activated by stimulation of the upper lip (5-30 V; 10 Hz), and BFcrb was recorded at the
6 ease of IQR, P-value 0.0371), and wrinkle on upper lip (7.7% more wrinkle on upper lip per increase o
7 minent eyes, a narrow nasal bridge, a tented upper lip, a high palate, an open mouth, tightly adheren
8 al examination revealed hypermobility of the upper lip and absence of generalized altered passive eru
9 ilateral swellings, and midline notch in the upper lip and alveolus.
10 istinct anatomic malformations affecting the upper lip and hard and soft palate.
11 activity in the primary somatosensory cortex upper lip and jaw (S1ULp-S1J) region of the somatosensor
12  face was represented from chin/lower lip to upper lip and neck/upper face in a rostrocaudal sequence
13 rther understand how retinoic acid regulates upper lip and palate morphogenesis we searched for genes
14 hat retinoic acid is a critical regulator of upper lip and primary palate development in Xenopus laev
15  better understand the complex nature of the upper lip and primary palate development which will lead
16                                          The upper lip and primary palate form an essential separatio
17 growth and differentiation necessary for the upper lip and primary palate morphogenesis.
18 nction also results in a median cleft in the upper lip and primary palate.
19 and RARgamma result in a median cleft in the upper lip and primary palate.
20 that fate mapping revealed contribute to the upper lip and primary palate.
21 ormalin (1.5%, 50 mul) was injected into the upper lip and the animal's behaviors recorded for 45 min
22 representation correspond to the upper face, upper lip, and chin plus lower lip, whereas three or fou
23 ntubation included a grade of class 3 on the upper lip bite test (lower incisors cannot extend to rea
24                                  An abnormal upper lip bite test, which is easily assessed by clinici
25 crest cell (cNCC) mesenchyme is required for upper lip closure.
26                  In the fetal specimens, the upper lip (coronal plane), alveolar ridge, tooth sockets
27 y during the cellular zippering required for upper lip formation in mammals.
28 olved in regulating mid-face development and upper lip fusion and are therefore strong candidates for
29               We examine cellular drivers of upper lip fusion in the mammalian embryo by establishing
30 l adhesion threshold requirement specific to upper lip fusion.
31    Somatosensory stimuli were applied to the upper lip (glabrous skin) and the chin (hairy skin).
32 tered passive eruption (APE) and hypermobile upper lip (HUL), has not been reported.
33 ngival display (EGD) etiologies (hypermobile upper lip [HUL], altered passive eruption [APE], and sho
34 ed in response to tactile stimulation of the upper lip in 69% of cases, the lower lip in 85% of cases
35 res reorganizes after deafferentation of the upper lip in neonatal rats (postnatal day [PND] 1-30).
36 displacement) to the glabrous surface of the upper lip in order to index the modulation and specifici
37 rtently affect facial structures such as the upper lip, lateral aspect of the nose, and lower eyelid.
38                     Stimulation of the nose, upper lip, lower lip, and chin caused a somatotopic late
39  drives cNCC mesenchyme proliferation during upper lip morphogenesis, and that disruption of this seq
40                                          The upper lip of the chick embryo forms by confluence of pri
41 reflects maxillary versus mandibular origin, upper lip origin and whisker rostrocaudal arc.
42 d wrinkle on upper lip (7.7% more wrinkle on upper lip per increase of IQR, P-value 0.0218).
43 t (lower incisors cannot extend to reach the upper lip; positive likelihood ratio, 14 [95% CI, 8.9-22
44  bitemporal narrowing, broad nasal tip, thin upper lip, posteriorly rotated or low-set ears, and micr
45 izing enzyme is ectopically expressed in the upper lip primordia of Lrp6-deficient embryos, indicatin
46                                          The upper lip region has the most significant change in HDX
47  ectoderm of the medial nasal processes, the upper lip remained intact in mutant mice.
48    The field potentials evoked in crus II by upper lip stimulation did not differ between wild-type a
49                           In wild-type mice, upper lip stimulation increased BFcrb in crus II by 32 +
50  somatosensory cortex blood flow produced by upper lip stimulation was not attenuated in D2-null mice
51 ], altered passive eruption [APE], and short upper lip [SUL]) in a nondental adult population and to
52 eatures that include a prominent maxilla and upper lip that readily reveal the upper gingiva, widely
53  This cross-sectional study aimed to examine upper lip (UL) and smile characteristics and soft tissue
54                                              Upper lip vermilion length (ULVL), total UL length, inte
55 such as hypertrichosis, thick eyebrows, thin upper lip vermilion, and upturned nose.
56                                           2) Upper lip whisker axons had more boutons than those from