ライフサイエンスコーパス: ureteral

1 ered to prevent stone migration and maintain ureteral access.

2 revious renal transplantations; technique of ureteral anastomosis; use of ureteral stent; total ische

3 by one of 11 readers for renal vascular and ureteral anatomic variants.

4 f both the donor and recipient; arterial and ureteral anatomy; procurement by transplant surgeon vers

5 ponses to TGF-beta and Bmp signaling, in the ureteral and bladder mesenchyme during embryogenesis.

6 depends at least in part on intercalation of ureteral and bladder muscle.

7 en attributed to impaired development of the ureteral and pelvic smooth muscle.

8 Successful outcomes can be obtained for ureteral and renal calculi that are similar to the adult

9 s, and can result in severe bladder, kidney, ureteral, and genital pathologies.

10 One hundred eight calculi (85 renal, 21 ureteral, and two in the bladder) were present in 32 of

11 ing consequences, such as mesenteric, bowel, ureteral, and/or bladder obstruction.

12 P1 mutant mice also had improper ureteral bladder insertion sites and shortened intravesi

13 Normal morphogenesis of the kidney, ureteral bud differentiation, ureteropelvic junction for

14 icantly in the spontaneous passage of distal ureteral calculi, thereby reducing the need for surgical

15 ogy pertaining to the treatment of renal and ureteral calculi.

16 y, idiopathic urethritis, and the passage of ureteral calculi.

17 Kidney stones and ureteral stents can cause ureteral colic and pain.

18 There was a small increase in ureteral complication (8.3% vs. 2.3% P=0.06) and a signi

19 Ureteral complication rate was 1.9% in stent versus 5.8%

20 ys were excluded, unless already developed a ureteral complication.

21 s independently associated with reduction in ureteral complications (incidence rate ratios [IRR], 0.4

22 sed to examine the association of stent with ureteral complications (leak or stricture) and urinary t

23 We determined the incidence of ureteral complications and assessed the association with

24 se of a ureteral stent is protective against ureteral complications and increased donor age is associ

25 e associated with a significant reduction in ureteral complications but increases UTI risk.

26 Stents were associated with a decrease in ureteral complications in deceased donor recipients (IRR

27 splantations after living donor nephrectomy, ureteral complications occurred in 18 (3.7%), including

28 Ureteral complications of renal transplants after living

29 o factors were significantly associated with ureteral complications on multivariate analysis: increas

30 Given that the existing literature on ureteral complications pertains mostly to deceased as op

31 ors, we aimed to assess the risk factors for ureteral complications solely after living donor nephrec

32 amine the association of stents with risk of ureteral complications, particularly in relationship wit

33 is associated with an increased incidence of ureteral complications.

34 s circulation and produced urine through the ureteral conduit in vivo.

35 loyable formulation of the two drugs reduced ureteral contraction amplitude and frequency by 90% and

36 ongenital anomaly frequently associated with ureteral defects.

37 study the molecular mechanisms that modulate ureteral development, we inactivated Smad4, the common S

38 Tbx18 has been shown to be essential for ureteral development.

39 ty and specificity was achieved by using the ureteral dilatation criterion, which had 73% sensitivity

40 Ureteral dilatation may yield the best accuracy for the

41 Only ureteral dilatation was significantly related to all-gra

42 Renal length, ureteral dilatation, pelvic dilatation, and corticomedul

43 tract developmental defects, including renal/ureteral duplication, hydroureter, and hydronephrosis, w

44 sterior urethral valves, neurogenic bladder, ureteral ectopy, or bladder exstrophy.

45 , and sonic hedgehog (Shh) expression in the ureteral epithelium.

46 procedure: midline incisional hernia, repair ureteral fistula, and repair enterocutaneous fistula.

47 Unobstructed antegrade ureteral flow was defined by the presence of contrast ma

48 the same surgeon with a previously described ureteral implantation, and a 7F ureteral stent attached

49 erse events, with one related to the study a ureteral injury incurred during sentinel-lymph-node diss

50 rs ( approximately 100 cells/cluster) at the ureteral insertion and along thick bundles of nerve fibe

51 al, E15.5 Fgfr2(ST-/-) mice exhibit improper ureteral insertion sites into the bladder, consistent wi

52 rostral migration of CARTp-IR cells from the ureteral insertion toward the bladder body during postna

53 my groups included mesh exposure (8% vs 0%), ureteral kinking managed intraoperatively (0% vs 7%), gr

54 Urological complications, namely ureteral leak and obstruction, remain a major source of

55 mplications occurred in 18 (3.7%), including ureteral leak in 10 (2.1%) and ureteral stricture in 8 (

56 Eleven patients (21 kidneys) had ureteral leaks or fistulas, one patient (one kidney) had

57 fication was quantified as the percentage of ureteral length that contained enhanced urine.

58 adic multicellular clusters after unilateral ureteral ligation and migrated into the parietal Bowman'

59 vely constant in the obstructed kidney after ureteral ligation.

60 CT/PET showed that the right ureteral mass and all lung nodules had regressed or disa

61 In Six1(-/-) mice, the ureteral mesenchymal precursors failed to condense and d

62 e absence of canonical Smad signaling in the ureteral mesenchyme, but not in the urothelium itself, l

63 ranscription factor selectively expressed in ureteral mesenchyme, regulates smooth muscle differentia

64 gical steps for robotic pyeloplasty, robotic ureteral neocystostomy with ureteral reimplantation and

65 Compared with controls, ureteral obstructed animals displayed increased tubular

66 uropathy (19%), bowel obstruction (14%), and ureteral obstruction (12%).

67 the protective effects in a 3-day unilateral ureteral obstruction (3dUUO) mouse model.

68 3 hours after reperfusion; after unilateral ureteral obstruction (day 7) in mice; and after gentamic

69 s, we subjected the hypomorphs to unilateral ureteral obstruction (UUO) and again found significant p

70 mice over a time course following unilateral ureteral obstruction (UUO) and compared to sham controls

71 , in two models of kidney injury: unilateral ureteral obstruction (UUO) and ischemia reperfusion.

72 After unilateral ureteral obstruction (UUO) and ischemia/reperfusion, Nog

73 thelial cells of a mouse model of unilateral ureteral obstruction (UUO) and related cell models using

74 ction in renal fibrosis following unilateral ureteral obstruction (UUO) in mice, a model of progressi

75 Unilateral ureteral obstruction (UUO) is a well-characterized murin

76 e kidney, muscle, and exosomes of unilateral ureteral obstruction (UUO) mice.

77 Using the established unilateral ureteral obstruction (UUO) model of kidney fibrosis as o

78 The unilateral ureteral obstruction (UUO) model of kidney injury induce

79 d JNK targets were activated in a unilateral ureteral obstruction (UUO) model of renal fibrosis in vi

80 In a mouse unilateral ureteral obstruction (UUO) model of renal fibrosis, inju

81 tubular fibrosis by using a mouse unilateral ureteral obstruction (UUO) model.

82 We used the unilateral ureteral obstruction (UUO) mouse model to investigate th

83 Using the unilateral ureteral obstruction (UUO) mouse model, we examined whet

84 renal fibrosis-mice subjected to unilateral ureteral obstruction (UUO) or fed an adenine-rich diet-a

85 ic kidney from mice 14 days after unilateral ureteral obstruction (UUO) surgery.

86 l tubules of kidneys subjected to unilateral ureteral obstruction (UUO) using Cre-loxP-mediated gene

87 injury in vivo, a mouse model of unilateral ureteral obstruction (UUO) was employed.

88 y fibrosis and inflammation after unilateral ureteral obstruction (UUO) were analyzed.

89 g this type of injury, modeled by unilateral ureteral obstruction (UUO), cells undergo epithelial-to-

90 odel of renal fibrosis induced by unilateral ureteral obstruction (UUO), HDAC8 was primarily expresse

91 urine fibrotic kidneys, following unilateral ureteral obstruction (UUO), resulting in an increase in

92 interstitial fibrosis induced by unilateral ureteral obstruction (UUO), the levels of renal ATX prot

93 rbates kidney fibrosis induced by unilateral ureteral obstruction (UUO).

94 interstitial fibrosis induced by unilateral ureteral obstruction (UUO).

95 acrophages upregulated Mrc2 after unilateral ureteral obstruction (UUO).

96 the tubulointerstitium (TI) after unilateral ureteral obstruction (UUO).

97 II (Ang II) and mice subjected to unilateral ureteral obstruction (UUO).

98 the fate of these cells following unilateral ureteral obstruction (UUO).

99 ar epithelial injury initiated by unilateral ureteral obstruction (UUO).

100 f tubulointerstitial fibrosis via unilateral ureteral obstruction (UUO).

101 odel of renal fibrosis induced by unilateral ureteral obstruction (UUO).

102 f CKD, and in mouse kidneys after unilateral ureteral obstruction (UUO).

103 al tubular autophagy in mice with unilateral ureteral obstruction (UUO).

104 us angiotensin-(1-7) in mice with unilateral ureteral obstruction (UUO).

105 mia-reperfusion (I/R, days 1-56), unilateral ureteral obstruction (UUO, days 1-10), and Alport mice (

106 f a single dose of suramin immediately after ureteral obstruction abolished the expression of fibrone

107 Endotoxemia and ureteral obstruction also increased NGAL and MCP-1 gene

108 lary atrophy in mice subjected to unilateral ureteral obstruction and a complete reversal of obesity

109 n1alpha1-eGFPL10a mice subject to unilateral ureteral obstruction and analyzed and validated the resu

110 18 induces TLR4 expression during unilateral ureteral obstruction and induces TLR4 expression in HK-2

111 reatment of idiopathic RPF aims at relieving ureteral obstruction and inducing disease regression, an

112 ow that renal fibrosis induced by unilateral ureteral obstruction and metastasis of human cancer xeno

113 retroperitoneum, where it frequently causes ureteral obstruction and renal failure.

114 nt macrophages limited kidney fibrosis after ureteral obstruction by driving extracellular matrix deg

115 cantly more fibrosis after 7 d of unilateral ureteral obstruction compared with wild-type mice, despi

116 er ischemia-reperfusion injury or unilateral ureteral obstruction demonstrates that amphiregulin was

117 ys of diabetic rats and mice with unilateral ureteral obstruction depicted significant loss of SCAI e

118 at day 21, relative to untreated unilateral ureteral obstruction disease model.

119 is, we subjected VDR-null mice to unilateral ureteral obstruction for 7 days.

120 ed ischemia-reperfusion injury or unilateral ureteral obstruction in mice with proximal tubule cell-s

121 n a kidney injury model caused by unilateral ureteral obstruction in mice.

122 voluted tubule cells and those of unilateral ureteral obstruction in the afflicted mouse kidney sugge

123 n highly selected children with intraluminal ureteral obstruction in the hands of a very experienced

124 ed inflammation and fibrosis associated with ureteral obstruction in vivo Therefore, domain 4 of CTGF

125 After unilateral ureteral obstruction in wild-type mice, we observed a pr

126 Ureteral obstruction induced Shh, predominantly in the r

127 Unilateral ureteral obstruction injury was induced in Snai1 knockou

128 ession in the afflicted kidney by unilateral ureteral obstruction is accompanied by changes in Usf1 a

129 we report that renal nerve stimulation after ureteral obstruction is the primary profibrotic signal a

130 Unilateral ureteral obstruction kidney Mphis form three distinct su

131 hen subjected to the normotensive unilateral ureteral obstruction model of endogenous RAS activation,

132 T) controls were subjected to the unilateral ureteral obstruction model of kidney fibrosis.

133 We used the unilateral ureteral obstruction model of progressive kidney fibrosi

134 In the unilateral ureteral obstruction model of renal fibrosis, let-7c upr

135 Here, in the unilateral ureteral obstruction model of renal fibrosis, tubular ep

136 that led to severe fibrosis in a unilateral ureteral obstruction model of renal fibrosis.

137 ers of disease progression in the unilateral ureteral obstruction model of renal interstitial fibrosi

138 In the unilateral ureteral obstruction model, ICG-001 ameliorated renal in

139 ism was confirmed in vivo using a unilateral ureteral obstruction mouse model.

140 he normal and injured kidneys, we found that ureteral obstruction not just blocked the NP elimination

141 , compound 8, starting the day of unilateral ureteral obstruction operation, inhibited collagen depos

142 +) C57BL/6 mice were subjected to unilateral ureteral obstruction or sham surgery (n = 8/group; sham,

143 Finally, in vivo model of unilateral ureteral obstruction revealed that matrix stiffness-regu

144 elayed administration of MC1568 at 3 d after ureteral obstruction reversed the expression of alpha-SM

145 abetic rat and mouse kidneys with unilateral ureteral obstruction showed SMA expression, as evidenced

146 in treatment of mice subjected to unilateral ureteral obstruction similarly reduced YAP/TAZ levels an

147 injecting oleic acid followed by unilateral ureteral obstruction surgery in mice resulted in enhance

148 We performed unilateral ureteral obstruction surgery on four male and three fema

149 and then performed either sham or unilateral ureteral obstruction surgery.

150 In mice with ureteral obstruction that were treated with the pan anti

151 th bone marrow from coll-GFP mice, underwent ureteral obstruction to induce fibrosis.

152 In vivo, using ureteral obstruction to model renal fibrosis, we observe

153 mean age, 45 years; P = .54) with unilateral ureteral obstruction underwent contemporaneous urinalysi

154 ng protein were subjected to left unilateral ureteral obstruction versus sham operation.

155 Unilateral ureteral obstruction was performed on wild-type and IL-2

156 men, 137 women; mean age, 59 years) without ureteral obstruction who underwent unenhanced scanning a

157 significance to multiple painful episodes of ureteral obstruction with eventual kidney failure.

158 eate), prerenal (endotoxemia), or postrenal (ureteral obstruction) injury.

159 n in a mouse kidney injury model (unilateral ureteral obstruction), consisting of an initial increase

160 s, during normal aging, and after unilateral ureteral obstruction).

161 ry angiogenesis in the cornea and unilateral ureteral obstruction).

162 on also occurred in kidneys after unilateral ureteral obstruction, a model of tubulointerstitial fibr

163 After unilateral ureteral obstruction, all members of the Wnt family exce

164 s and renal fibrosis in mice with unilateral ureteral obstruction, and also attenuates ER stress, pro

165 rile renal inflammation following unilateral ureteral obstruction, and in experimental pyelonephritis

166 studied: Maleate nephrotoxicity, unilateral ureteral obstruction, and LPS preconditioning.

167 ificantly at 14 and 21 days after unilateral ureteral obstruction, and renal oxidized protein levels

168 endoureterotomy is useful for other types of ureteral obstruction, and we aimed to assess its long-te

169 inflammation in murine models of unilateral ureteral obstruction, antimembrane basal GN, and infusio

170 interstitial fibrosis in mouse kidney after ureteral obstruction, as demonstrated by a reduced inter

171 In mice subjected to unilateral ureteral obstruction, genetic deletion or pharmacologic

172 enges, namely, fistulae, abscesses, bowel or ureteral obstruction, hemorrhage, cancer and thickened m

173 In animals exposed to unilateral ureteral obstruction, Hh pathway suppression by expressi

174 After unilateral ureteral obstruction, IL-27 deficiency resulted in incre

175 ependent murine fibrosis model of unilateral ureteral obstruction, kidney fibrosis was unexpectedly m

176 ed to 5/6 subtotal nephrectomy or unilateral ureteral obstruction, plasma levels of angiopoietin-2 al

177 A signature of unilateral ureteral obstruction, proximal tubular atrophy leads to

178 tion of folic acid nephropathy or unilateral ureteral obstruction, TbetaRII(endo+/-) mice exhibited l

179 In the kidneys with unilateral ureteral obstruction, there were significantly more apop

180 After 21 d of unilateral ureteral obstruction, total kidney collagen was signific

181 ts or wild-type mice subjected to unilateral ureteral obstruction, TRPC3 expression increased in the

182 s of renal interstitial fibrosis, unilateral ureteral obstruction, unilateral ischemia-reperfusion, C

183 fibrogenesis in mice subjected to unilateral ureteral obstruction, whereas activation of Hif in myelo

184 antly in injured epithelium after unilateral ureteral obstruction, whereas downstream signaling from

185 Kidney hydronephrosis in C2RD was caused by ureteral obstruction, which was, in turn, induced by SCF

186 Reversing the unilateral ureteral obstruction-induced inflammatory microenvironme

187 In a novel mouse model, unilateral ureteral obstruction-induced inflammatory milieu activat

188 ysyl oxidase inhibitor alleviated unilateral ureteral obstruction-induced tubular dilatation and prol

189 tubulointerstitial region of the unilateral ureteral obstruction-injured kidney in mice correlating

190 chymal damage were significantly worse after ureteral obstruction.

191 beta-catenin, and expression of Snail after ureteral obstruction.

192 to ischemia-reperfusion injury or unilateral ureteral obstruction.

193 ently available for the cancer patients with ureteral obstruction.

194 kidneys closely parallels that observed with ureteral obstruction.

195 ey injury induced by ischemia reperfusion or ureteral obstruction.

196 c cystinosis, in a mouse model of unilateral ureteral obstruction.

197 d sterile inflammation induced by unilateral ureteral obstruction.

198 ion on kidney fibrosis induced by unilateral ureteral obstruction.

199 sis, cisplatin nephrotoxicity, and bilateral ureteral obstruction.

200 nce of prominent leukocyte infiltrates after ureteral obstruction.

201 d levels of active TGF-beta after unilateral ureteral obstruction.

202 ed renal tubular epithelium after unilateral ureteral obstruction.

203 ding to tubular atrophy following unilateral ureteral obstruction.

204 elial cell (TEC) apoptosis during unilateral ureteral obstruction.

205 enal fibrosis in a mouse model of unilateral ureteral obstruction.

206 ated gene expression in mice with unilateral ureteral obstruction.

207 s after subjecting the animals to unilateral ureteral obstruction.

208 ice with an MMP13 inhibitor after unilateral ureteral obstruction.

209 ofibroblasts in a murine model of unilateral ureteral obstruction.

210 enuated collagen deposition after unilateral ureteral obstruction.

211 exacerbated inflammation and fibrosis after ureteral obstruction.

212 and macrophage infiltration after unilateral ureteral obstruction.

213 eys significantly increased after unilateral ureteral obstruction.

214 er in kidneys from CLOCK-deficient mice with ureteral obstruction.

215 fusion-induced injury, diabetic nephropathy, ureteral obstructive disease, and kidney allograft rejec

216 ls of chronic kidney disease: (1) Unilateral ureteral obstructive nephropathy, (2) streptozotocin-ind

217 ation and log rolling or between bladder and ureteral opacification (P > .05).

218 There was no significant difference between ureteral opacification and log rolling or between bladde

219 No difference in ureteral opacification was observed between the log-roll

220 Ureteral opacification was quantified as the percentage

221 ed to the recipient, whose bladder above the ureteral opening level was removed.

222 y and biopsy showed a 4-cm mass at the right ureteral orifice positive for a high-grade papillary tra

223 lrasonographic (US) nephrostograms to assess ureteral patency after percutaneous nephrolithotomy (PCN

224 Contrast-enhanced US demonstrated ureteral patency in eight studies and obstruction in two

225 ide a safer, more convenient way to evaluate ureteral patency than fluoroscopy.

226 e between US and fluoroscopic assessments of ureteral patency was evaluated by using a Clopper-Pearso

227 us urologic reconstruction and pretransplant ureteral pathologic conditions increased the risk of uro

228 for surgeon-controlled robotic management of ureteral pathology and evaluate the developments in the

229 Ureterolysis and other mid-ureteral pathology have been treated with durable functi

230 w the role of robotics for the management of ureteral pathology, in particular, ureteropelvic junctio

231 Ureteral, pelvic, and urinary tract dilatations were sig

232 ion to congenital defects in humans, such as ureteral-pelvic obstructions, may be related to the comp

233 Moreover, mutant mice showed abnormal ureteral peristalsis.

234 unction obstruction, ureteral strictures, or ureteral polyps.

235 Open ureteral reconstruction continues to have durable long-t

236 nced hands, minimally invasive approaches to ureteral reconstruction have proven to be feasible with

237 Surgeon-controlled, robotic-assisted, ureteral reconstructive and ablative surgery is being pe

238 Seven patients had no posttransplant ureteral reflux, three had grade 1 reflux, and two had g

239 oplasty, robotic ureteral neocystostomy with ureteral reimplantation and robotic ureteroureterostomy/

240 tomy, ureteroureterostomy, ureterolysis, and ureteral reimplantation with and without psoas hitch.

241 c urological procedures such as pyeloplasty, ureteral reimplantation, complete and partial nephrectom

242 Locally delivered ureteral relaxants therefore may improve ureter-related

243 We hypothesized that ureteral relaxation can be improved via the local admini

244 d screening assay to determine the extent of ureteral relaxation, we show that the calcium channel bl

245 Nevertheless, open ureteral repair remains a viable option with durable lon

246 wever, there were fewer nonvisualized distal ureteral segments with the longer imaging delay.

247 he middle (P </= .008) and distal (P < .001) ureteral segments.

248 e widths of the proximal, middle, and distal ureteral segments.

249 ency reduces the number and contractility of ureteral smooth muscle cells, leading to abnormal pyelou

250 f urothelial cells, and proper investment of ureteral smooth muscle cells.

251 se inhibitor ROCKi significantly relax human ureteral smooth muscle cells.

252 uggests that at least some Bmp4 functions in ureteral smooth muscle may be Smad-independent.

253 Despite a reduction in quantity of ureteral smooth muscle, differentiation proceeded withou

254 rcent of patients had a UOC (most frequently ureteral stenosis) close to biopsy.

255 theter (HR, 3.9; 95% CI, 2.8-5.4; P <0.001), ureteral stent (HR, 1.4; 95% CI, 1.1-1.8; P=0.01), age (

256 patients in this series after placement of a ureteral stent and instilment of diluted contrast into t

257 d exposure to antibodies, the placement of a ureteral stent at the time of kidney transplantation was

258 ly described ureteral implantation, and a 7F ureteral stent attached to a large diameter suprapubic c

259 Our findings reveal that the presence of a ureteral stent is associated with an increase in the ris

260 The use of a ureteral stent is protective against ureteral complicati

261 Ureteral stent placement at kidney transplantation may r

262 n of a temporizing nature: generally, either ureteral stent placement or nephrostomy drainage.

263 ale gender, prolonged use of Foley catheter, ureteral stent, age, and delayed graft function are inde

264 sis: increased donor age and no placement of ureteral stent.

265 s; technique of ureteral anastomosis; use of ureteral stent; total ischemia time; serum creatinine on

266 erpreting follow-up imaging, and the role of ureteral stenting and other interventions in management.

267 Immediate or delayed ureteral stenting is the most common procedure used to t

268 passage rates and reduce the pain caused by ureteral stenting.

269 Ureteral stents are associated with a significant reduct

270 Kidney stones and ureteral stents can cause ureteral colic and pain.

271 Controversy exists regarding the benefit of ureteral stents in kidney transplantation.

272 After removal of suprapubic catheters and ureteral stents, all patients were able to void spontane

273 CT, while those with moderate likelihood of ureteral stone (moderate STONE score, 6-9) underwent red

274 patients with moderate to high likelihood of ureteral stone to safely and effectively identify patien

275 n the ED with moderate to high likelihood of ureteral stone undergoing CT imaging.

276 Objective likelihood of ureteral stone was determined by using the previously de

277 All three ureteral stones (3, 4, and 8 mm) were correctly identifi

278 Stone migration during the treatment of ureteral stones can prove frustrating and increases both

279 ials and meta-analysis, patients with distal ureteral stones measuring less than 1 cm who are candida

280 ronic acid (Deflux) implants to mimic distal ureteral stones on computed tomography (CT) scan.

281 Medical expulsive therapy for ureteral stones provides a nonsurgical option for patien

282 lockers or alpha blockers were used to treat ureteral stones were eligible for inclusion in our analy

283 rs; male-female ratio, 14:8) with kidney and ureteral stones who underwent CT with z-axis modulation

284 vides a nonsurgical option for patients with ureteral stones.

285 Ureteral stricture disease commonly affects the cancer p

286 %), including ureteral leak in 10 (2.1%) and ureteral stricture in 8 (1.6%).

287 Proximally, ureteral strictures and symptomatic retrocaval ureters h

288 The management of ureteral strictures in transplanted kidney is challengin

289 erotomy should be a first line treatment for ureteral strictures of length 10 mm or shorter in kidney

290 mations, ureteropelvic junction obstruction, ureteral strictures, or ureteral polyps.

291 Surgeon-controlled, robotic-assisted ureteral surgery is well tolerated, feasible, and effect

292 mitant kidney injury by performing bilateral ureteral transection in mice.

293 We investigated the hypothesis that ureteral trauma caused by placement of indwelling stents

294 tient (one kidney) was suspected of having a ureteral tumor, and the final two patients (three kidney

295 anning at CT urography yielded no additional ureteral tumors and resulted in additional radiation exp

296 of EHT 1864 in mice dramatically attenuated ureteral unilateral obstruction-driven EGFR, p53, Rac1b,

297 igone are crucial for proper function of the ureteral valve mechanism; however, the developmental eve

298 Once in the bladder, the ureteral valve, a mechanism that is not well understood,

299 inoma infiltrating the full thickness of the ureteral wall.

300 acification scores for each segment and mean ureteral width measurements for each technique were comp

301 Similarly, mean ureteral widths were significantly higher with furosemid