ライフサイエンスコーパス: ureteral obstruction

1 s, during normal aging, and after unilateral ureteral obstruction).

2 ry angiogenesis in the cornea and unilateral ureteral obstruction).

3 nce of prominent leukocyte infiltrates after ureteral obstruction.

4 d sterile inflammation induced by unilateral ureteral obstruction.

5 ion on kidney fibrosis induced by unilateral ureteral obstruction.

6 sis, cisplatin nephrotoxicity, and bilateral ureteral obstruction.

7 d levels of active TGF-beta after unilateral ureteral obstruction.

8 ed renal tubular epithelium after unilateral ureteral obstruction.

9 ding to tubular atrophy following unilateral ureteral obstruction.

10 elial cell (TEC) apoptosis during unilateral ureteral obstruction.

11 ent of fibrosis in a rat model of unilateral ureteral obstruction.

12 odel of renal fibrosis induced by unilateral ureteral obstruction.

13 the upper urinary tract following unilateral ureteral obstruction.

14 ntly found in patients with renal disease or ureteral obstruction.

15 was also increased in p27-/- mice following ureteral obstruction.

16 ve other common causes for cholecystitis and ureteral obstruction.

17 hemia/reperfusion injury (IRI) or unilateral ureteral obstruction.

18 ce had less renal fibrosis during unilateral ureteral obstruction.

19 otected against renal fibrosis in unilateral ureteral obstruction.

20 enal fibrosis in a mouse model of unilateral ureteral obstruction.

21 ontralateral kidneys of rats with unilateral ureteral obstruction.

22 nterstitial fibrosis in mice with unilateral ureteral obstruction.

23 ated gene expression in mice with unilateral ureteral obstruction.

24 s after subjecting the animals to unilateral ureteral obstruction.

25 ice with an MMP13 inhibitor after unilateral ureteral obstruction.

26 ofibroblasts in a murine model of unilateral ureteral obstruction.

27 enuated collagen deposition after unilateral ureteral obstruction.

28 exacerbated inflammation and fibrosis after ureteral obstruction.

29 beta-catenin, and expression of Snail after ureteral obstruction.

30 and macrophage infiltration after unilateral ureteral obstruction.

31 eys significantly increased after unilateral ureteral obstruction.

32 er in kidneys from CLOCK-deficient mice with ureteral obstruction.

33 to ischemia-reperfusion injury or unilateral ureteral obstruction.

34 chymal damage were significantly worse after ureteral obstruction.

35 ently available for the cancer patients with ureteral obstruction.

36 kidneys closely parallels that observed with ureteral obstruction.

37 ey injury induced by ischemia reperfusion or ureteral obstruction.

38 c cystinosis, in a mouse model of unilateral ureteral obstruction.

39 uropathy (19%), bowel obstruction (14%), and ureteral obstruction (12%).

40 the protective effects in a 3-day unilateral ureteral obstruction (3dUUO) mouse model.

41 We report a case of ureteral obstruction 5 years after cadaveric renal trans

42 shared several clinical features, including ureteral obstruction (5/7 patients), lymphocele (3/7), b

43 on also occurred in kidneys after unilateral ureteral obstruction, a model of tubulointerstitial fibr

44 f a single dose of suramin immediately after ureteral obstruction abolished the expression of fibrone

45 After unilateral ureteral obstruction, all members of the Wnt family exce

46 Endotoxemia and ureteral obstruction also increased NGAL and MCP-1 gene

47 lary atrophy in mice subjected to unilateral ureteral obstruction and a complete reversal of obesity

48 n1alpha1-eGFPL10a mice subject to unilateral ureteral obstruction and analyzed and validated the resu

49 nterstitial fibrosis in models of unilateral ureteral obstruction and aristolochic acid fibrosis asso

50 in renal fibrotic mice induced by unilateral ureteral obstruction and aristolochic acid nephropathy.

51 The area under the curve diminished during ureteral obstruction and correlated well with mean corti

52 These changes occurred in the unilateral ureteral obstruction and folic acid models of fibrosis a

53 Patients were clinically stable with known ureteral obstruction and had been referred for antegrade

54 Ureteral obstruction and high ureteral pressure reduced

55 18 induces TLR4 expression during unilateral ureteral obstruction and induces TLR4 expression in HK-2

56 reatment of idiopathic RPF aims at relieving ureteral obstruction and inducing disease regression, an

57 d kidney inflammation in both the unilateral ureteral obstruction and ischemic-reperfusion injury-ind

58 ow that renal fibrosis induced by unilateral ureteral obstruction and metastasis of human cancer xeno

59 onic kidney injury (cisplatin and unilateral ureteral obstruction and reanalyzed previously published

60 retroperitoneum, where it frequently causes ureteral obstruction and renal failure.

61 en-induced mice were subjected to unilateral ureteral obstruction and thymidine analogue labeling and

62 s and may lead to serious complications like ureteral obstruction and unbearable pain.

63 s and renal fibrosis in mice with unilateral ureteral obstruction, and also attenuates ER stress, pro

64 rile renal inflammation following unilateral ureteral obstruction, and in experimental pyelonephritis

65 studied: Maleate nephrotoxicity, unilateral ureteral obstruction, and LPS preconditioning.

66 dney of renal fibrosis induced by unilateral ureteral obstruction, and primarily located in interstit

67 ificantly at 14 and 21 days after unilateral ureteral obstruction, and renal oxidized protein levels

68 endoureterotomy is useful for other types of ureteral obstruction, and we aimed to assess its long-te

69 inflammation in murine models of unilateral ureteral obstruction, antimembrane basal GN, and infusio

70 aradigm for immune mediated inflammation and ureteral obstruction as a model for non-immune mediated

71 interstitial fibrosis in mouse kidney after ureteral obstruction, as demonstrated by a reduced inter

72 nt macrophages limited kidney fibrosis after ureteral obstruction by driving extracellular matrix deg

73 Ureteral obstruction caused significant increases in int

74 cantly more fibrosis after 7 d of unilateral ureteral obstruction compared with wild-type mice, despi

75 Of 442 patients with ureteral obstruction confirmed at CT, 200 had documented

76 n in a mouse kidney injury model (unilateral ureteral obstruction), consisting of an initial increase

77 in the contralateral kidney after unilateral ureteral obstruction could promote lymphangiogenesis.

78 3 hours after reperfusion; after unilateral ureteral obstruction (day 7) in mice; and after gentamic

79 er ischemia-reperfusion injury or unilateral ureteral obstruction demonstrates that amphiregulin was

80 ys of diabetic rats and mice with unilateral ureteral obstruction depicted significant loss of SCAI e

81 diagnosis was established, and the degree of ureteral obstruction determined on urograms was compared

82 at day 21, relative to untreated unilateral ureteral obstruction disease model.

83 scriptomics (lithium-induced NDI, unilateral ureteral obstruction, endotoxin-induced NDI) or proteomi

84 ed to 3 models of renal fibrosis (unilateral ureteral obstruction, folic acid nephropathy [FAN], and

85 is, we subjected VDR-null mice to unilateral ureteral obstruction for 7 days.

86 In mice subjected to unilateral ureteral obstruction, genetic deletion or pharmacologic

87 enges, namely, fistulae, abscesses, bowel or ureteral obstruction, hemorrhage, cancer and thickened m

88 In animals exposed to unilateral ureteral obstruction, Hh pathway suppression by expressi

89 After unilateral ureteral obstruction, IL-27 deficiency resulted in incre

90 allowed accurate prediction of the degree of ureteral obstruction in 44 (94%) of 47 patients with acu

91 nowledge, this is the first reported case of ureteral obstruction in a transplant kidney caused by an

92 images can be used to predict the degree of ureteral obstruction in acute ureterolithiasis.

93 ed ischemia-reperfusion injury or unilateral ureteral obstruction in mice with proximal tubule cell-s

94 in the fibrotic kidney induced by unilateral ureteral obstruction in mice, whereas renal Smad abundan

95 n a kidney injury model caused by unilateral ureteral obstruction in mice.

96 voluted tubule cells and those of unilateral ureteral obstruction in the afflicted mouse kidney sugge

97 n highly selected children with intraluminal ureteral obstruction in the hands of a very experienced

98 Ureteral obstruction in the transplant recipient can pro

99 ed inflammation and fibrosis associated with ureteral obstruction in vivo Therefore, domain 4 of CTGF

100 After unilateral ureteral obstruction in wild-type mice, we observed a pr

101 We present evidence that: i) ureteral obstruction induced cell-specific but transient

102 Ureteral obstruction induced Shh, predominantly in the r

103 Reversing the unilateral ureteral obstruction-induced inflammatory microenvironme

104 In a novel mouse model, unilateral ureteral obstruction-induced inflammatory milieu activat

105 ed lung fibrosis and resistant to unilateral ureteral obstruction-induced kidney fibrosis.

106 ysyl oxidase inhibitor alleviated unilateral ureteral obstruction-induced tubular dilatation and prol

107 tubulointerstitial region of the unilateral ureteral obstruction-injured kidney in mice correlating

108 Unilateral ureteral obstruction injury was induced in Snai1 knockou

109 eate), prerenal (endotoxemia), or postrenal (ureteral obstruction) injury.

110 ession in the afflicted kidney by unilateral ureteral obstruction is accompanied by changes in Usf1 a

111 Ureteral obstruction is marked by disappearance of the v

112 we report that renal nerve stimulation after ureteral obstruction is the primary profibrotic signal a

113 erstitial fibrosis in response to unilateral ureteral obstruction, ischemia or nephrotoxins.

114 els of kidney fibrosis induced by unilateral ureteral obstruction, ischemia-reperfusion injury, or hi

115 Unilateral ureteral obstruction kidney Mphis form three distinct su

116 ependent murine fibrosis model of unilateral ureteral obstruction, kidney fibrosis was unexpectedly m

117 rmethylation of 94 genes in mouse unilateral ureteral obstruction kidneys, which was markedly reduced

118 ul division of the isthmus, and avoidance of ureteral obstruction, long-term data revealed good graft

119 hen subjected to the normotensive unilateral ureteral obstruction model of endogenous RAS activation,

120 T) controls were subjected to the unilateral ureteral obstruction model of kidney fibrosis.

121 We used the unilateral ureteral obstruction model of progressive kidney fibrosi

122 In the unilateral ureteral obstruction model of renal fibrosis, let-7c upr

123 Here, in the unilateral ureteral obstruction model of renal fibrosis, tubular ep

124 that led to severe fibrosis in a unilateral ureteral obstruction model of renal fibrosis.

125 ers of disease progression in the unilateral ureteral obstruction model of renal interstitial fibrosi

126 In the unilateral ureteral obstruction model, ICG-001 ameliorated renal in

127 ism was confirmed in vivo using a unilateral ureteral obstruction mouse model.

128 placement were performed in 45 patients with ureteral obstruction (n = 40), leak (n = 3), or both (n

129 he normal and injured kidneys, we found that ureteral obstruction not just blocked the NP elimination

130 In one additional control animal, ureteral obstruction occurred at day 10, and was associa

131 , compound 8, starting the day of unilateral ureteral obstruction operation, inhibited collagen depos

132 y disease and fibrosis induced by unilateral ureteral obstruction or adenine diet.

133 +) C57BL/6 mice were subjected to unilateral ureteral obstruction or sham surgery (n = 8/group; sham,

134 5 expression in fibrotic mouse kidneys after ureteral obstruction or unilateral ischemia-reperfusion

135 ed to 5/6 subtotal nephrectomy or unilateral ureteral obstruction, plasma levels of angiopoietin-2 al

136 A signature of unilateral ureteral obstruction, proximal tubular atrophy leads to

137 Finally, in vivo model of unilateral ureteral obstruction revealed that matrix stiffness-regu

138 elayed administration of MC1568 at 3 d after ureteral obstruction reversed the expression of alpha-SM

139 abetic rat and mouse kidneys with unilateral ureteral obstruction showed SMA expression, as evidenced

140 in treatment of mice subjected to unilateral ureteral obstruction similarly reduced YAP/TAZ levels an

141 injecting oleic acid followed by unilateral ureteral obstruction surgery in mice resulted in enhance

142 We performed unilateral ureteral obstruction surgery on four male and three fema

143 and then performed either sham or unilateral ureteral obstruction surgery.

144 tion of folic acid nephropathy or unilateral ureteral obstruction, TbetaRII(endo+/-) mice exhibited l

145 In mice with ureteral obstruction that were treated with the pan anti

146 In the kidneys with unilateral ureteral obstruction, there were significantly more apop

147 th bone marrow from coll-GFP mice, underwent ureteral obstruction to induce fibrosis.

148 s of ischemia/reperfusion AKI and unilateral ureteral obstruction to investigate the role of IL-33 (i

149 In vivo, using ureteral obstruction to model renal fibrosis, we observe

150 After 21 d of unilateral ureteral obstruction, total kidney collagen was signific

151 renal fibrosis disease induced by unilateral ureteral obstruction, transforming growth factor-beta ty

152 ts or wild-type mice subjected to unilateral ureteral obstruction, TRPC3 expression increased in the

153 mean age, 45 years; P = .54) with unilateral ureteral obstruction underwent contemporaneous urinalysi

154 s of renal interstitial fibrosis, unilateral ureteral obstruction, unilateral ischemia-reperfusion, C

155 nancy should not be interpreted as a sign of ureteral obstruction unless the finding persists in the

156 schemic kidney injury is afforded by 24 h of ureteral obstruction (UO) applied 6 or 8 days prior to t

157 is in aging mice and in mice with unilateral ureteral obstruction (UUO) a known model of progressive

158 s, we subjected the hypomorphs to unilateral ureteral obstruction (UUO) and again found significant p

159 mice over a time course following unilateral ureteral obstruction (UUO) and compared to sham controls

160 s increased in mouse kidneys with unilateral ureteral obstruction (UUO) and in cultured NRK-52E cells

161 , in two models of kidney injury: unilateral ureteral obstruction (UUO) and ischemia reperfusion.

162 After unilateral ureteral obstruction (UUO) and ischemia/reperfusion, Nog

163 thelial cells of a mouse model of unilateral ureteral obstruction (UUO) and related cell models using

164 beta1 transgenic mice, and in the unilateral ureteral obstruction (UUO) and subtotal nephrectomy (SNX

165 xamined renal fibrosis induced by unilateral ureteral obstruction (UUO) in diabetic mouse models.

166 ction in renal fibrosis following unilateral ureteral obstruction (UUO) in mice, a model of progressi

167 Unilateral ureteral obstruction (UUO) is a well-characterized murin

168 y polarization and migration into unilateral ureteral obstruction (UUO) kidneys and unilateral ischem

169 e kidney, muscle, and exosomes of unilateral ureteral obstruction (UUO) mice.

170 aling and renal fibrosis in a rat unilateral ureteral obstruction (UUO) model by transferring a doxyc

171 Using the established unilateral ureteral obstruction (UUO) model of kidney fibrosis as o

172 The unilateral ureteral obstruction (UUO) model of kidney injury induce

173 d JNK targets were activated in a unilateral ureteral obstruction (UUO) model of renal fibrosis in vi

174 In a mouse unilateral ureteral obstruction (UUO) model of renal fibrosis, inju

175 tivation in renal fibrosis in the unilateral ureteral obstruction (UUO) model.

176 tubular fibrosis by using a mouse unilateral ureteral obstruction (UUO) model.

177 Most studies of unilateral ureteral obstruction (UUO) models have examined late tim

178 We used the unilateral ureteral obstruction (UUO) mouse model to investigate th

179 Using the unilateral ureteral obstruction (UUO) mouse model, we examined whet

180 renal fibrosis-mice subjected to unilateral ureteral obstruction (UUO) or fed an adenine-rich diet-a

181 9a-2 in renal fibrosis induced by unilateral ureteral obstruction (UUO) or renal ischemia/reperfusion

182 Two-day-old mice underwent unilateral ureteral obstruction (UUO) or sham operation; 28 days la

183 stigated between 3 and 14 d after unilateral ureteral obstruction (UUO) or sham surgery (n = 8 mice p

184 stigated between 3 and 14 d after unilateral ureteral obstruction (UUO) or sham surgery.

185 of impaired renal growth: chronic unilateral ureteral obstruction (UUO) results in greater injury to

186 ne expression in a mouse model of unilateral ureteral obstruction (UUO) revealed significant inductio

187 ic kidney from mice 14 days after unilateral ureteral obstruction (UUO) surgery.

188 Unilateral ureteral obstruction (UUO) time-dependently induced Nupr

189 l tubules of kidneys subjected to unilateral ureteral obstruction (UUO) using Cre-loxP-mediated gene

190 injury in vivo, a mouse model of unilateral ureteral obstruction (UUO) was employed.

191 y fibrosis and inflammation after unilateral ureteral obstruction (UUO) were analyzed.

192 g this type of injury, modeled by unilateral ureteral obstruction (UUO), cells undergo epithelial-to-

193 odel of renal fibrosis induced by unilateral ureteral obstruction (UUO), HDAC8 was primarily expresse

194 odel of renal fibrosis induced by unilateral ureteral obstruction (UUO), in cultured mouse proximal t

195 urine fibrotic kidneys, following unilateral ureteral obstruction (UUO), resulting in an increase in

196 interstitial fibrosis induced by unilateral ureteral obstruction (UUO), the levels of renal ATX prot

197 rteen days after the induction of unilateral ureteral obstruction (UUO), wild-type mice reconstituted

198 at target myofibroblasts in mouse unilateral ureteral obstruction (UUO)-induced fibrotic kidneys.

199 acrophages upregulated Mrc2 after unilateral ureteral obstruction (UUO).

200 the tubulointerstitium (TI) after unilateral ureteral obstruction (UUO).

201 II (Ang II) and mice subjected to unilateral ureteral obstruction (UUO).

202 the fate of these cells following unilateral ureteral obstruction (UUO).

203 ar epithelial injury initiated by unilateral ureteral obstruction (UUO).

204 murine renal fibrosis induced by unilateral ureteral obstruction (UUO).

205 d in C57BL/6 mice with a model of unilateral ureteral obstruction (UUO).

206 e interstitial fibrosis caused by unilateral ureteral obstruction (UUO).

207 stered to rats that had undergone unilateral ureteral obstruction (UUO).

208 c kidney disease model induced by unilateral ureteral obstruction (UUO).

209 in lipidome in a mouse model with unilateral ureteral obstruction (UUO).

210 f tubulointerstitial fibrosis via unilateral ureteral obstruction (UUO).

211 odel of renal fibrosis induced by unilateral ureteral obstruction (UUO).

212 f CKD, and in mouse kidneys after unilateral ureteral obstruction (UUO).

213 al tubular autophagy in mice with unilateral ureteral obstruction (UUO).

214 us angiotensin-(1-7) in mice with unilateral ureteral obstruction (UUO).

215 rbates kidney fibrosis induced by unilateral ureteral obstruction (UUO).

216 interstitial fibrosis induced by unilateral ureteral obstruction (UUO).

217 mia-reperfusion (I/R, days 1-56), unilateral ureteral obstruction (UUO, days 1-10), and Alport mice (

218 chemia-reperfusion injury-(IRI-), unilateral ureteral obstruction-(UUO-), and folic acid- (FA-) induc

219 ng protein were subjected to left unilateral ureteral obstruction versus sham operation.

220 Unilateral ureteral obstruction was performed on wild-type and IL-2

221 arterial pressure, and in six other piglets ureteral obstruction was released.

222 ssue collagen in the kidneys after sustained ureteral obstruction, when compared with their wild-type

223 fibrogenesis in mice subjected to unilateral ureteral obstruction, whereas activation of Hif in myelo

224 monstrated no evidence of ureteral reflux or ureteral obstruction, whereas an immediate prior cohort

225 antly in injured epithelium after unilateral ureteral obstruction, whereas downstream signaling from

226 Kidney hydronephrosis in C2RD was caused by ureteral obstruction, which was, in turn, induced by SCF

227 men, 137 women; mean age, 59 years) without ureteral obstruction who underwent unenhanced scanning a

228 significance to multiple painful episodes of ureteral obstruction with eventual kidney failure.

229 tion may present with acute cholecystitis or ureteral obstruction without its classical clinical symp

230 DI, hypercalcaemia-associated NDI, bilateral ureteral obstruction), yielding contrasting views.