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1  EMG high-frequency activity in the external urethral sphincter.
2 traction of the pelvic floor and the anal or urethral sphincter.
3 ile also recording the EMG from the external urethral sphincter.
4  peripheral effects directly on the external urethral sphincter.
5  the bladder and the other into the external urethral sphincter.
6 could be used to deliver nitric oxide to the urethral sphincter.
7 ated control of the bladder and the external urethral sphincter.
8 abies virus (PRV) injected into the external urethral sphincter.
9 ve) that inserted directly into the external urethral sphincter.
10  while also directly inhibiting the external urethral sphincter, a pattern necessary to overcome detr
11 nal cystometry, with acquisition of external urethral sphincter activity and bladder pressure, was us
12 irmed electrophysiologically by the measured urethral sphincter activity evoked in response to select
13 d coordination between detrusor and external urethral sphincter activity that has been observed clini
14 ic floor electromyogram (EMG) as a proxy for urethral sphincter activity.
15                    The EMG from the external urethral sphincter also showed marked decreases after cl
16  measured simultaneously at the level of the urethral sphincter and at the level of the penis using a
17 ladder pressure to facilitate urination, and urethral sphincters and pelvic floor muscles, which cont
18 tudy, leak point pressure (LPP) and external urethral sphincter electromyography (EMG) were performed
19                LPP and simultaneous external urethral sphincter electromyography (EUS EMG) were recor
20 amic testing with electromyogram of external urethral sphincter (EUS) activity in both female and mal
21 eflexes relax the bladder and evoke external urethral sphincter (EUS) contraction (guarding reflex) t
22 anisms of injury from VD leading to external urethral sphincter (EUS) dysfunction, and assess if tran
23 absence of bladder contractions and external urethral sphincter (EUS) electromyographic (EMG) activat
24                                     External urethral sphincter (EUS) electromyography (EMG) was typi
25 the changes in bladder pressure and external urethral sphincter (EUS) electroneurogram (ENG) evoked b
26 accompanied by rhythmic bursting of external urethral sphincter (EUS) EMG and expulsion of urine from
27 enter in rats that is involved with external urethral sphincter (EUS) functionality during micturitio
28                                     External urethral sphincter (EUS) injections resulted in labellin
29                                 The external urethral sphincter (EUS) plays a crucial role in maintai
30 etrusor) and striated muscle of the external urethral sphincter (EUS) that is controlled by spinal an
31 stometry) and coordination with the external urethral sphincter (EUS) were assessed 5 d post-SCI unde
32 neurovascular bundles (NVB) and the external urethral sphincters (EUS), can enable physicians to perf
33 incter (URS), commonly known as the external urethral sphincter, facilitates urinary continence by co
34  reflex pathways that coordinate bladder and urethral sphincter function.
35 rs and inhibits the activity of the external urethral sphincter in anesthetized rats.
36 rameters and on the activity of the external urethral sphincter in anesthetized rats.
37 s are thought to functionally regenerate the urethral sphincter in patients with suspected intrinsic
38 ells (MPC) injected into a model of striated urethral sphincter injury that reproduces the histopatho
39 ing age, BMI, membranous urethral length and urethral sphincter insufficiency.
40  muscle precursor cells (MPCs) into striated urethral sphincter irreversibly damaged by electrocoagul
41                            Relaxation of the urethral sphincter is abolished by inhibitors of nitric
42                     In the medulla, external urethral sphincter neurons and bladder neurons and doubl
43  the pontine micturition center and external urethral sphincter neurons were found in the locus coeru
44                                 The striated urethral sphincter of older male rats was damaged by ele
45  been identified in very high density in the urethral sphincters of a variety of animals and in human
46 g for individuals with neurologically driven urethral sphincter overactivity.
47 in young women, associated with a failure of urethral sphincter relaxation, and a treatment option th
48 n studies suggest that nitric oxide mediates urethral sphincter relaxation.
49 rom the rat urinary bladder and the external urethral sphincter simultaneously within the same tissue
50  of the gene, for neuronal nitric oxide have urethral sphincters that do not relax in response to ele
51 train, was injected into either the external urethral sphincter, the bladder base, or the bladder bod
52 , neurons labeled by virus from the external urethral sphincter were found in Onuf's nucleus, the dor
53 rethral length and elevation of the external urethral sphincter, whereas a recent large series demons
54 lts from chronic denervation of the striated urethral sphincter with associated fibrosis.