ライフサイエンスコーパス: uric acid

1 cal pathways (e.g., involving bile acids and uric acid).

2 ukocytes, immunoglobulin G, haptoglobin, and uric acid.

3 endent modulation in optical response toward uric acid.

4 acid and 100% of glycine, glutamic acid and uric acid.

5 pro-oxidant and pro-inflammatory effects of uric acid.

6 accounted for 4.3% of the variance of serum uric acid.

7 thine oxidase of hypoxanthine to xanthine to uric acid.

8 I 1.11, 1.30) per 59.48 micromol/L (1 mg/dL) uric acid.

9 , apolipoprotein B, apolipoprotein CIII, and uric acid.

10 C2A9), polymorphisms with variation in serum uric acid.

11 xanthine into xanthine and then further into uric acid.

12 for calcium oxalate, calcium phosphate, and uric acid.

13 or vitamin D, oxalate, cysteine, purines and uric acid.

14 shold, mostly due to renal underexcretion of uric acid.

15 nity of immobilized enzyme (uricase) towards uric acid.

16 approach to identify loci influencing serum uric acid.

17 vity C-reactive protein) 2.8 (1.3-6.1) mg/L, uric acid 7.2 (5.8-8.7) mg/dL, and urine protein-creatin

18 natriuretic peptide (482 +/- 337 pg/mL) and uric acid (8.2 +/- 2.6 mg/dL), decreased left ventricula

19 principle, this biosensor was used to detect uric acid, a biomarker for wound severity and healing, i

20 Excess circulating uric acid, a product of hepatic glycolysis and purine me

21 While uric acid alone was unable to trigger a chloride secreto

22 Uric acid also amplified the effects of elevated glucose

23 Uric acid also proved superior to placebo in reducing in

24 Improvement Amendments (CLIA) criterion for uric acid analysis (+/-17%).

25 MDA, uric acid and ALT levels also increased, whereas GSH and

26 matography (HPLC), were compared to quantify uric acid and antioxidant reducing agents in 36 milk sam

27 l for electrochemical detection of dopamine, uric acid and ascorbic acid.

28 ly interfering physiological species such as uric acid and ascorbic acid.

29 positive genetic correlations between serum uric acid and BMI z score (rhoG = 0.45, P = 0.002), perc

30 laboratory analysis for the readily detected uric acid and for the clozapine which is present at 100-

31 erence observed in the presence of dopamine, uric acid and fructose.

32 blood was taken for the measurement of serum uric acid and glycosylated haemoglobin (HbA1C).

33 exclusion of the main interfering substances uric acid and hydrogen peroxide.

34 inflammation, where there is large amount of uric acid and inflammatory peroxidases.

35 At the bedside in the last 5 years, uric acid and nerinetide are the only compounds tested f

36 Any interference of uric acid and other electroactive AAs was noticed.

37 levels of various small molecules, including uric acid and p-hydroxybenzoic acid among their structur

38 Uric acid and the GRS were both associated with cardiova

39 acetyl-D-hexosamine and fumaric acid (2 mo); uric acid and tyrosine (6 mo).

40 , respiration rate and low concentrations of uric acid and tyrosine, analytes associated with disease

41 homeostasis (potassium), purine degradation (uric acid), and oxidative stress as regulated by glutath

42 eading to de novo lipogenesis, production of uric acid, and accumulation of visceral and ectopic fat.

43 ine N-oxide, beta-hydroxybutyrate, trimethyl uric acid, and alanine.

44 tose (dietary or endogenous), its metabolite uric acid, and aldose reductase (AR, the only endogenous

45 um(III) chloride, ferricyanide/ferrocyanide, uric acid, and ascorbic acid.

46 Fasting glucose, triglycerides, uric acid, and bilirubin levels were decreased in the sa

47 eart rates, respiratory rates, and sweat pH, uric acid, and glucose, as well as deliver programmed th

48 serum urea nitrogen, serum creatinine, serum uric acid, and serum phosphorus; and faster rate of decl

49 ells, macrophages, and neutrophils; elevated uric acid; and increased NLRP3, a major inflammasome com

50 Elevated concentrations of serum uric acid are associated with increased risk of gout and

51 quent elevation in fructose, sorbitol and/or uric acid, are important factors contributing to alcohol

52 iated with any biochemical marker except for uric acid, arguing against pleiotropy.

53 eptor 4 (TLR-4), interleukin-18 (IL-18), and uric acid as markers of the inflammatory host response i

54 dehydrogenase, resulting in the formation of uric acid as well as ROS.

55 on Resonance (LRSPR) biosensor for detecting uric acid, as a model analyte, has been developed in thi

56 omavirus, dopamine, glutamic acid, IgG, IgE, uric acid, ascorbic acid, acetlycholine, cortisol, cytos

57 ng compounds in biological fluids, including uric acid, ascorbic acid, glucose and acetaminophen.

58 was performed using a genetic score with 24 uric acid-associated loci.

59 (SERS) approach for the routine analysis of uric acid at clinically relevant levels in urine patient

60 been utilized in the nanomolar detection of uric acid at physiological pH in water.

61 from anionic analytes like ascorbic acid and uric acid at physiological pH.

62 correlated to BMI, whereas higher levels of uric acid (beta=0.164; P<0.001), proadrenomedullin (beta

63 The prepared LRSPR based uric acid bio-sensor gives good response characteristics

64 The genetic score raised uric acid by 17 micromol/L (95% CI 15, 18) per SD increa

65 droperoxide is a product of the oxidation of uric acid by inflammatory heme peroxidases.

66 lobin (HbA1c), insulin resistance (HOMA-IR), uric acid, C-reactive protein (CRP), alanine transaminas

67 ated with higher levels of serum creatinine, uric acid, calcium and lower urine pH level.

68 itive relationship with the concentration of uric acid, cholesterol, and amylase.

69 e increasing lysophosphatidylcholine (18:2), uric acid, citrulline, and inosine levels, which are gen

70 ower autophagic activity in cells exposed to uric acid compared with control conditions.

71 Human serum uric acid concentration (SUA) is a complex trait.

72 ated a weighted genetic risk score (GRS) for uric acid concentration based on eight uric acid-regulat

73 ect, we found that a 59.48 micromol/L higher uric acid concentration did not have a causal effect on

74 te that our CaT-SMelor directly measured the uric acid concentration in clinical human blood samples,

75 ch 1-mg/dl increase in genetically predicted uric acid concentration were significant for cardiovascu

76 ood linearity over a wide range of 0-700 muM uric acid concentration with a limit of detection (LOD)

77 between the EC-SERS signal intensity and the uric acid concentration.

78 QDs fluorescence, which was proportional to uric acid concentration.

79 .9 +/- 3.1 mg/dL, P < 0.0001), and 24-h mean uric acid concentrations (0%: -0.13 +/- 0.07; 10%: 0.15

80 investigate the causal effect of circulating uric acid concentrations on type 2 diabetes risk.

81 Insulin resistance and uric acid concentrations were beneficially affected by r

82 variation in SLC2A9 is associated with serum uric acid concentrations, an important biomarker of rena

83 eking, were associated with higher levels of uric acid concurrently and when uric acid was measured 3

84 rain samples was determined by measuring the uric acid content of their aqueous extracts.

85 icted versus reference values of protein and uric acid content were found to be fitting with R(2) of

86 idated with reference methods of protein and uric acid content.

87 The potential mechanisms by which uric acid could cause vasoconstriction and a progressive

88 , the authors believe that TLR-4, IL-18, and uric acid could have a role in the inflammatory patholog

89 predictors of disease progression including uric acid, creatinine and surprisingly, blood pressure,

90 Clec12A detects uric acid crystals and limits proinflammatory pathways b

91 ng EPEC and STEC infections, we noticed that uric acid crystals became enmeshed in the neutrophilic e

92 While trying to visualize uric acid crystals formed during EPEC and STEC infection

93 Uric acid crystals were formed in vivo in the lumen of t

94 activated by stimuli that include nigericin, uric acid crystals, amyloid-beta fibrils and extracellul

95 well as other sterile particulates, such as uric acid crystals, induces DCs to produce IL-2 followin

96 errets, that form calcium oxalate, struvite, uric acid, cystine and other stone types.

97 Elevated brain natriuretic peptide and uric acid, decreased left ventricular ejection fraction,

98 rs placental function via a xanthine oxidase/uric acid-dependent mechanism, and similar effects may o

99 sitivity, selectivity, and stability towards uric acid detection in human saliva, covering the concen

100 ient samples have been used for quantitative uric acid detection using a simple, rapid colloidal SERS

101 ase, an efficient and sensitive approach for uric acid determination was also established.

102 Peroxynitrite scavenger uric acid did not affect the first phase but ameliorated

103 ), allows for the absolute quantification of uric acid directly in a complex matrix such as that from

104 We find that uric acid directly inhibits uridine monophosphate syntha

105 Uric acid dose-dependently stimulated aldose reductase e

106 ve dietary sodium intake and increased serum uric acid during follow-up despite pharmacological contr

107 0 years before diagnosis and lower levels of uric acid during the 20 years before diagnosis, compared

108 identified parameters, including fractional uric acid excretion and plasma copeptin concentration, m

109 glycosuria, increased urinary phosphate, and uric acid excretion.

110 ixture of graphene oxide, copper nitrate and uric acid, followed by thermal annealing at 900 degrees

111 om healthy volunteers were first primed with uric acid for 24 h and then subjected to stimulation wit

112 The mean level of serum uric acid for both the groups was within normal range an

113 Quantitative detection of uric acid for rapid and routine diagnosis of early preec

114 Although blocking uric acid formation by allopurinol did not affect outcom

115 ), foods (fructose), and metabolic products (uric acid) function as survival signals to help reduce w

116 We wondered, however, if uric acid generated by XO also had biological effects in

117 These results suggest that uric acid generated during fructose metabolism may act a

118 Uric acid, generated from the metabolism of purines, has

119 selectivity against ascorbic acid, dopamine, uric acid, glucose and bovine serum albumin.

120 iopotential) sensors, sweat biochemical (pH, uric acid, glucose) sensors, thermal stimulators, and hu

121 eruricemia was defined as a concentration of uric acid >=7.0 mg/dL in men and >=5.7 mg/dL in women.

122 g metabolic and signaling pathways involving uric acid, gut microbiome products, and so-called uremic

123 gical and experimental evidence suggest that uric acid has a role in the etiology of type 2 diabetes

124 Because uric acid has been implicated as a mediator of adjuvant

125 Uric acid has been linked to the progression of native k

126 ng of epithelial absorption and secretion of uric acid has recently emerged, aided in particular by t

127 Baseline sputum uric acid, high mobility group box-1, CXCL8 mRNA, sputum

128 etic and regulatory networks with effects on uric acid homeostasis have also emerged.

129 toxic effect when combined with cyanuric and uric acids; however, it is unknown whether such effect c

130 ion end-products, high-mobility group box 1, uric acid, IL-33, or inflammasome activation.

131 scopy and used for quantitative detection of uric acid in 0.1 M NaF and synthetic urine at clinically

132 ng process and used to measure pyocyanin and uric acid in a wound fluid simulant at 37 degrees C.

133 g lipid/lipoprotein risk factors for CVD and uric acid in adults [age: 18-40 y; body mass index (in k

134 tocol is then employed for the estimation of uric acid in blood serum samples of healthy individuals.

135 etween RNFL and macular thickness with serum uric acid in both the groups.

136 This first GWAS of serum uric acid in continental Africans identified three assoc

137 Here, we performed the first GWAS for serum uric acid in continental Africans, with replication in A

138 Our results show that variation in serum uric acid in Hispanic children is under considerable gen

139 ited the expected pharmacodynamics to remove uric acid in hyperuricemic blood in vitro and multiple f

140 ricemic blood in vitro and multiple forms of uric acid in hyperuricemic geese.

141 d simple method for the reliable analysis of uric acid in milk.

142 wed significantly higher level of mean serum uric acid in no diabetic retinopathy group (p = 0.004 re

143 ed LRSPR sensor was also evaluated to detect uric acid in real serum samples.

144 Levels of uric acid in sweat were higher in patients with gout tha

145 n blood digestion, a massive accumulation of uric acid in the midgut posterior region, and a signific

146 The detection figures of merit for uric acid in the simulant at 37 degrees C were as follow

147 ayer (RNFL) and macular thickness with serum uric acid in type 2 diabetic patients.

148 High levels of uric acid in urine and serum can be indicative of hypert

149 quantum dots (QDs) for the determination of uric acid in urine sample is described.

150 sent study, we sought to investigate whether uric acid, in the soluble form, could also activate the

151 Uric acid induced the phenotype transition of vascular e

152 This stimulatory mechanism was mediated by uric acid-induced oxidative stress and stimulation of th

153 ich inhibits autophagy and recapitulates the uric acid-induced proinflammatory cytokine phenotype.

154 uvant in CD4KO mice might be associated with uric acid, inflammatory cytokines, and the recruitment o

155 filtering out signals from ascorbic acid and uric acid interferents.

156 pite its role in converting highly insoluble uric acid into 5-hydroxyisourate.

157 Uric acid is a damage-associated molecular pattern (DAMP

158 Uric acid is a final breakdown product of purine catabol

159 Uric acid is a potential important biomarker in urine an

160 d Mendelian randomization to examine whether uric acid is an independent and causal cardiovascular ri

161 These results suggest that high uric acid is causally related to adverse cardiovascular

162 Serum uric acid is determined by production and the net balanc

163 However, uric acid is not responsible for brain dysfunction, and

164 reanalysis of the URICO-ICTUS trial whether uric acid is superior to placebo in improving the functi

165 Serum uric acid is the end-product of purine metabolism and at

166 as to investigate whether variation in serum uric acid is under genetic influence and whether the ass

167 these damage-associated molecular patterns, uric acid, is increased in the maternal circulation in p

168 We show that XO-derived ROS, but not uric acid, is the trigger for IL1beta release and that X

169 e (35.2 [33.0-38.0] vs 33.0 [30.0-35.5] cm), uric acid level (4.9 [4.0-5.8] vs 4.5 [3.7-5.5] mg/dL),

170 height, family history of ESRD, higher serum uric acid level, and lower measured GFR.

171 eys, reduced oxidative stress, lowered serum uric acid level, reduced glomerular hyperfiltration and

172 sma AST, salivary AST, and salivary ALT with uric acid level.

173 tricular ejection fraction </=40%, and serum uric acid levels >/=9.5 mg/dL to receive allopurinol (ta

174 difference = 1.7, 95% CI: -0.1, 3.4), plasma uric acid levels (for those born early preterm, differen

175 , whereas both serum magnesium (P<0.001) and uric acid levels (P=0.008) improved.

176 nd the allopurinol groups had baseline serum uric acid levels (SDs) of 8.7 (1.6) mg/dl and 8.3 (1.4)

177 osodium urate crystals in joints when plasma uric acid levels are chronically elevated beyond the sat

178 These results indicate that, although uric acid levels are elevated in the airways of NO2-expo

179 A modest rise in uric acid levels beginning early after donation, and a s

180 purinol effectively and safely lowered serum uric acid levels in adults with stage 3 CKD and asymptom

181 etween maternal serum fructose and placental uric acid levels in humans.

182 Moreover, plasma uric acid levels in mice fed the WD were decreased after

183 ed cell systems and in the intestine in vivo Uric acid levels in the gut lumen increased in response

184 and non-human primates and normalized serum uric acid levels in uricase-deficient mice.

185 Uric acid levels were significantly reduced with allopur

186 We evaluated whether lowering serum uric acid levels with allopurinol improves endothelial d

187 d hemoglobin, albuminuria, triglycerides and uric acid levels, and worse measured glomerular filtrati

188 dase inhibitor allopurinol reduced placental uric acid levels, prevented placental inefficiency, and

189 with reduced ejection fraction and elevated uric acid levels, xanthine oxidase inhibition with allop

190 vels significantly correlated with placental uric acid levels.

191 prevent glucose reabsorption and lower serum uric acid levels.

192 enal function plays a critical role in serum uric acid levels.

193 Uric acid-lowering therapies may therefore not be benefi

194 ot be used as an argument against the use of uric acid-lowering therapy.

195 tioxidant molecules [taurine (m/z 124.0068), uric acid (m/z 167.0210), ascorbic acid (m/z 175.0241),

196 95% CI: -0.71, -0.02; SUCRAglucose: 74%) and uric acid (MD: -23.77 umol/L; 95% CI: -44.21, -3.32 umol

197 d renal inflammation phenotypes, reprogramed uric acid metabolism pathways, inhibited the activation

198 Next, we tested whether uric acid might mediate inflammasome activation in cells

199 We found that uric acid (monosodium urate [MSU]) crystals induce a pro

200 The levels of uric acid obtained by CV compared well to those determin

201 s not support a causal effect of circulating uric acid on diabetes risk.

202 ascorbic acid, 1.3 mM acetaminophen, 1.4 mM uric acid or 20 mM glucose.

203 sociated molecular patterns (DAMPs), such as uric acid or ATP, via NLRP3, which leads to caspase-1-de

204 5% CI 1.06, 1.76; p = 0.015), elevated serum uric acid (OR 3.55; 95% CI 1.03, 12.27; p = 0.045), elev

205 rotein (OR, 0.99; 95% CI, 0.98-0.99); higher uric acid (OR, 1.13; 95% CI, 1.04-1.22); working in inst

206 roup differences in changes in hs-cTnI, CRP, uric acid, or urine protein-creatinine ratio were observ

207 ed by severe neurological manifestations and uric acid overproduction.

208 jury associated with ATP depletion, elevated uric acid, oxidative stress and inflammation.

209 In multivariate analysis, serum uric acid (P = 0.001), estimated glomerular filtration r

210 sizes, expressed as the percentage change in uric acid per deleted copy, are most pronounced among wo

211 enic factors, including uremic toxins (i.e., uric acid, phosphates, endothelin-1, advanced glycation

212 f lipid/lipoprotein risk factors for CVD and uric acid: postprandial triglyceride (0%: 0 +/- 4; 10%:

213 ctivated protein kinase phosphorylation, but uric acid priming induced phosphorylation of AKT and pro

214 broad inflammatory pathways associated with uric acid priming, with NF-kappaB and mammalian target o

215 oxygen species production was diminished by uric acid priming.

216 etion/export with a concomitant reduction of uric acid production.

217 tively related to blood urea nitrogen, serum uric acid, proteinuria, and supernatant IL-4; whereas po

218 Higher plasma uric acid (PUA) levels are associated with lower glomeru

219 Obesity and diets rich in uric acid-raising components appear to account for the i

220 sma allantoin levels, including allantoin-to-uric acid ratio and high xanthine-to-hypoxanthine ratio

221 renal secretion of d-lactate in exchange for uric acid reabsorption culminated in hyperuricemia and g

222 abolites whose renal excretion is coupled to uric acid reabsorption.

223 We evaluated whether soluble uric acid regulates aldose reductase expression both in

224 rescence microscopy, we investigated whether uric acid regulates aldose reductase, a key enzyme in th

225 ) for uric acid concentration based on eight uric acid-regulating single nucleotide polymorphisms.

226 In addition, ORMDL3 mediated uric acid release, another marker of cellular stress.

227 we investigated the mechanisms through which uric acid selectively lowers human blood monocyte produc

228 status, body mass index, haemoglobin, serum uric acid, serum albumin, albuminuria, and C reactive pr

229 waist-to-height ratio, alanine transaminase, uric acid, serum triglycerides, and blood pressure.

230 Serum uric acid showed a poor correlation with RNFL and macula

231 ared with placebo, allopurinol lowered serum uric acid significantly but did not improve endothelial

232 male gender, higher body mass index, higher uric acid, smoking, alcohol drinking, and hiatal hernia

233 gger inflammation, published data on soluble uric acid (sUA) in this context are discrepant.

234 Soluble uric acid (sUA) is found in high concentrations in the s

235 Increased serum uric acid (SUA) levels cause gout and are associated wit

236 apable of non-invasively monitoring salivary uric acid (SUA) levels.

237 Elevated levels of serum uric acid (SUA) were considered to be risk factors for c

238 The linear regression equations for uric acid substrate was stated as A = 0.0039C + 0.0519 w

239 were higher with potassium citrate; however, uric acid supersaturation was lower.

240 In the placenta, fructose induced de novo uric acid synthesis by activating the activities of the

241 used for ammonia detoxification and [(13)C] uric acid synthesis through multiple metabolic pathways,

242 rmediate of the electrochemical oxidation of uric acid that has a lifetime in solution of 23 ms as we

243 Uric acid therapy was associated with reduced infarct gr

244 Uric acid therapy was more effective than placebo in lim

245 sporter and sterically hinder the transit of uric acid through the substrate channel, albeit with vas

246 peroxidase), which is involved in oxidizing uric acid to allaintoin and hydrogen peroxide.

247 We found serum uric acid to be significantly heritable [h(2) +/- SD = 0

248 High affinity inhibition of uric acid transport requires URAT1 residues Cys-32, Ser-

249 which can be treated using inhibitors of the uric acid transporter, URAT1.

250 sults demonstrate the association of SU with uric acid transporters in a minority population of Ameri

251 Uric acid triggered inflammatory responses in the gut, i

252 te structural elucidation of niacinamide and uric acid, two compounds potentially involved in the pat

253 electrochemical detection of dopamine (DA), uric acid (UA) and ascorbic acid (AA) on three dimension

254 low-density lipoprotein cholesterol (LDL-C), uric acid (UA) and diabetes-related traits such as fasti

255 ve interferences such as ascorbic acid (AA), uric acid (UA) and dopamine (DA).

256 The relationship between serum uric acid (UA) and outcomes after acute ischemic stroke

257 ration solutions and found that solubilizing uric acid (UA) by prewarming created erroneous results b

258 gen peroxide is the oxidative end product of uric acid (UA) by uricase, an efficient and sensitive ap

259 Uric acid (UA) can scavenge the peroxynitrite to avoid t

260 The roles of asymptomatic hyperuricemia or uric acid (UA) crystals in CKD progression are unknown.

261 Excessive production of uric acid (UA) in blood may lead to gout, hyperuricaemia

262 reductase (XOR), which is the sole source of uric acid (UA) in mammals, has been proposed to contribu

263 Recent data suggested a causative role of uric acid (UA) in the development of renal disease, in w

264 pression of xanthine oxidase and thereby the uric acid (UA) pathway of purine catabolism in macrophag

265 Elevated levels of uric acid (UA) were detected in mice undergoing sensitiz

266 d excellent photo-catalytic activity towards uric acid (UA) which served as the base for the Electroc

267 trochemical detection of ascorbic acid (AA), uric acid (UA), and dopamine (DA).

268 ection of ascorbic acid (AA), dopamine (DA), uric acid (UA), and serotonin (5-HT) in 0.1 M PBS (pH =

269 i-interference properties in the presence of uric acid (UA), ascorbic acid (AA) and glucose.

270 e common interferents ascorbic acid (AA) and uric acid (UA).

271 , Ascorbic Acid (AA), Acetaminophen (Ac) and Uric Acid (UA).

272 15, 18) per SD increase and explained 4% of uric acid variation.

273 nd study that compared the administration of uric acid versus placebo in stroke patients treated with

274 nary citrate, magnesium, oxalate, phosphate, uric acid, volume, and pH, and lower urinary sodium, res

275 159.4 ng/mL; P = 0.028), and increased serum uric acid (WA: 341.4 mumol/L; CB: 330 mumol/L; P = 0.020

276 Continuous sensing of uric acid was also performed using this biosensor which

277 Higher uric acid was associated with a higher diabetes risk aft

278 Uric acid was associated with a range of prevalent disea

279 Uric acid was associated with an increased rate of excel

280 Higher uric acid was associated with impulsivity in both humans

281 ant activity, we sought to determine whether uric acid was elevated and participated in a mouse model

282 The level of plasma uric acid was found to be highly significantly increased

283 The linear range for uric acid was from 4.0 to 70 muM, and the LOD was measur

284 Uric acid was higher in participants with an SSB intake

285 The linearity of the system toward uric acid was in the concentration range of 125-1000 mic

286 We found that uric acid was increased in the airways of mice exposed t

287 er levels of uric acid concurrently and when uric acid was measured 3 to 5 years later.

288 Serum uric acid was measured in 3315 patients of the Ludwigsha

289 The mean serum uric acid was significantly higher in patient with HbA1C

290 terferants (ascorbic acid, acetaminophen and uric acid), was revealed to be negligible.

291 ncentrations of leukocytes, haptoglobin, and uric acid were associated with a lower risk of Parkinson

292 lesterol high-density lipoprotein (HDL), and uric acid were measured.

293 ated AR metabolites (sorbitol, fructose, and uric acid), which correlated significantly with (1) incr

294 Thus, the Akt-PRAS40 pathway is activated by uric acid, which inhibits autophagy and recapitulates th

295 ovo pyrimidine synthesis-an effect traced to uric acid, which is 10-fold higher in the blood of human

296 Moreover, fructose metabolism yields uric acid, which is highly associated with NAFLD.

297 s not known whether the association of serum uric acid with SLC2A9 polymorphisms manifests in childre

298 Further, uric acid with the concentration of 0.1 muM is detected

299 g lipid/lipoprotein risk factors for CVD and uric acid within 2 wk.

300 rips were developed for on-site detection of uric acid without involving any sophisticated instrument

301 The uric acid/xanthine H(+) symporter, UapA, is a high-affin