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1 QDs fluorescence, which was proportional to uric acid concentration.
2 between the EC-SERS signal intensity and the uric acid concentration.
3 ciated with a significant reduction in serum uric acid concentrations.
4 ffects of vitamin C supplementation on serum uric acid concentrations.
5 hiasis, type 2 diabetes mellitus, and higher uric acid concentrations.
6 .9 +/- 3.1 mg/dL, P < 0.0001), and 24-h mean uric acid concentrations (0%: -0.13 +/- 0.07; 10%: 0.15
7 ase activity, leading to uniquely high serum uric acid concentrations (200-500 microM) compared with
8 lyses adjusted for age and smoking, men with uric acid concentrations above the median at enrollment
10 oducts, but only a few studies have compared uric acid concentrations among individuals who exclude s
11 variation in SLC2A9 is associated with serum uric acid concentrations, an important biomarker of rena
12 ing response in the range from 0 to 1.0mM of uric acid concentration and the apparent Michaelis-Mente
13 opinion on the nature of link between serum uric acid concentration and the risk for cardiovascular
15 servational studies have identified elevated uric acid concentration as a risk factor for diabetes, w
16 usality arguments, one can start using serum uric acid concentration as an inexpensive cardiovascular
17 ated a weighted genetic risk score (GRS) for uric acid concentration based on eight uric acid-regulat
18 tudy was to investigate differences in serum uric acid concentrations between meat eaters, fish eater
19 to compare the area under the serial plasma uric acid concentration curves during the first 96 hours
20 ect, we found that a 59.48 micromol/L higher uric acid concentration did not have a causal effect on
22 at explain 1.7-5.3% of the variance in serum uric acid concentrations, following a genome-wide associ
23 ailable in 169 subjects (85%); the change in uric acid concentration from baseline to the end of the
25 ers and their strong associations with serum uric acid concentrations, GLUT9 and ABCG2 appeared to be
26 te that our CaT-SMelor directly measured the uric acid concentration in clinical human blood samples,
30 nosine, inosine, hypoxanthine, xanthine, and uric acid concentrations in an in vivo canine model.
31 GFR and decreases BUN, serum phosphorus, and uric acid concentrations in patients with moderate to se
33 1 diabetes and smokers, suggesting that high uric acid concentrations in vivo might serve a protectiv
34 t our hypothesis that the reduction in serum uric acid concentration induced by sevelamer would be co
36 ty, 0.99; LR, 13 [95% CI, 3.1-53]) and serum uric acid concentration of 5.5 mg/dL or lower (sensitivi
40 Despite cytoreductive chemotherapy, plasma uric acid concentrations remained low throughout the tre
42 competitive inhibitor of XO, reduced plasma uric acid concentrations similarly in both groups (P < 0
45 ch 1-mg/dl increase in genetically predicted uric acid concentration were significant for cardiovascu
49 ine clinical characteristics, including mean uric acid concentrations, were similar in subjects rando
50 ood linearity over a wide range of 0-700 muM uric acid concentration with a limit of detection (LOD)
51 earity over a wide range of 0.05 mM to 1.0mM uric acid concentration with enhanced response of 2.7 mA