ライフサイエンスコーパス: urinary bladder

1 liver and kidney, and was excreted into the urinary bladder).

2 pump urine from the renal pelvis toward the urinary bladder.

3 rm the presence of c-kit positive ICs in pig urinary bladder.

4 and three distinct histological areas of the urinary bladder.

5 y and tumor tissue and clearance through the urinary bladder.

6 the C-PRO group were also diminished in the urinary bladder.

7 iosensitizing treatment, thereby sparing the urinary bladder.

8 on in isolated detrusor muscle of guinea pig urinary bladder.

9 ich result from developmental defects of the urinary bladder.

10 prognosis than urothelial carcinomas of the urinary bladder.

11 ediate different sensations arising from the urinary bladder.

12 IR) structures in the neonatal and adult rat urinary bladder.

13 bladder, left and right kidneys, spleen, and urinary bladder.

14 (TL) sensory neurons that innervate the rat urinary bladder.

15 Primary elimination was via kidneys to the urinary bladder.

16 cral spinal neurons receiving input from the urinary bladder.

17 s and mechanosensory transduction within the urinary bladder.

18 f visceral nociception between the colon and urinary bladder.

19 arkable permeability barrier function of the urinary bladder.

20 rience sensory and motor dysfunctions of the urinary bladder.

21 t to cause hyperplasia and carcinomas in the urinary bladder.

22 lusions in the epithelium of the urethra and urinary bladder.

23 scle (detrusor) and urothelium layers of the urinary bladder.

24 ubset of unmyelinated fibres innervating the urinary bladder.

25 ts on apoptotic cell death in the developing urinary bladder.

26 arances suggested chronic obstruction in the urinary bladder.

27 ging agent into the submucosa of the porcine urinary bladder.

28 inal afferent nerve endings in the mammalian urinary bladder.

29 V and radioactivity concentration within the urinary bladder.

30 e and aggressive neuroendocrine tumor of the urinary bladder.

31 l system with contiguous extension up to the urinary bladder.

32 nter the brain and was not excreted into the urinary bladder.

33 s critical for enhancing UPEC fitness in the urinary bladder.

34 ignaling of ketamine-induced cystitis in rat urinary bladder.

35 PERIOD 2 (PER2) in a contractile organ, the urinary bladder.

36 f the oesophagus, stomach, larynx, lung, and urinary bladder.

37 ignificant increases above that in wild-type urinary bladders.

38 fmol/mg of tissue, respectively), and murine urinary bladder (1.4 +/- 0.1 and 6.2 +/- 2.4 fmol/mg of

39 0.8 fmol/mg of tissue, respectively), canine urinary bladder (1.8 +/- 0.5 and 9.0 +/- 6.0 fmol/mg of

40 l (18)F-FDG administration were observed for urinary bladder (75.9 +/- 17.2), stomach (48.4 +/- 14.3)

41 invasive transitional cell carcinoma of the urinary bladder (a model of human invasive bladder cance

42 otransmission was characterized in the mouse urinary bladder, a model for the pathological or ageing

43 tracer administration, at which time minimal urinary bladder activity was present to interfere with t

44 Similar findings were also observed in the urinary bladder, although the inflammatory infiltrate wa

45 ents with transitional-cell carcinoma of the urinary bladder and 1149 controls in Spain; all the part

46 20 patients with small-cell carcinoma of the urinary bladder and concurrent urothelial carcinoma.

47 athy characterized by severe dilation of the urinary bladder and defective intestinal motility.

48 The sensory neurons innervating the urinary bladder and distal colon project to similar regi

49 lomeningocele typically possess a neurogenic urinary bladder and exhibit varying degrees of bladder d

50 ith a higher decorin expression, than in the urinary bladder and heart, two tissues with a lower deco

51 ole in the activation of Akt and MAPK in the urinary bladder and in bladder hypertrophy during cystit

52 role of mGluR5 in the neural control of the urinary bladder and in the inhibition of the micturition

53 exists for vagal innervation of the male rat urinary bladder and to assess whether those vagal affere

54 In primary tumors of lung, urinary bladder and tongue, induced by different carcino

55 eurally mediated plasma extravasation in the urinary bladder and urethra were examined in urethane-an

56 Urinary bladder and uterine guidelines did not have any

57 ucing the volume-induced contractions of rat urinary bladder and was devoid of cardiovascular effects

58 r the liver and kidneys, 1 for spleen, 1 for urinary bladder, and 1 generic compartment accounting fo

59 ssues and was highest in the salivary gland, urinary bladder, and distal intestinal tract.

60 ive cultures were obtained from the urethra, urinary bladder, and epididimydes, and the ID(50) was de

61 ically significant higher uptake in kidneys, urinary bladder, and lacrimal gland.

62 ormin was primarily taken up by the kidneys, urinary bladder, and liver but also to a lesser extent i

63 s after injection were found in the kidneys, urinary bladder, and liver.

64 FS of canine mesenteric artery (CMA), canine urinary bladder, and murine urinary bladder in the amoun

65 eased risk of human cancers, including skin, urinary bladder, and respiratory tract cancers.

66 rest (ROIs, 50 pixels) were placed in liver, urinary bladder, and tumor tissue in both image sets.

67 Papillary and invasive cancers of the urinary bladder appear to evolve and progress through di

68 The sensory components of the urinary bladder are responsible for the transduction of

69 small bowel, lumbar vertebra, psoas muscle, urinary bladder) as well as the noise-equivalent countin

70 the neurons processing information from the urinary bladder at this level of the neural axis are lik

71 lays a role in the development of TCC of the urinary bladder by acting as a bona fide tumor suppresso

72 ale rat urinary bladder tumors arise through urinary bladder calculi formation but is insufficient to

73 at desensitization of TRPV1 receptors in the urinary bladder can minimize the effects of cross-sensit

74 SIR, 9.78; 95% CI, 1.18 to 35.30; P = .009), urinary bladder cancer (SIR, 9.51; 95% CI, 1.15 to 34.37

75 Genome-wide association studies (GWAS) of urinary bladder cancer (UBC) have yielded common variant

76 Urinary bladder cancer (UBC) patients at muscle invasive

77 reast cancer (BC), prostate cancer (PC), and urinary bladder cancer (UBC), and is therefore an import

78 Most deaths from urinary bladder cancer are owing to metastatic disease.

79 50% of candidate TumiD targets in T24 human urinary bladder cancer cells is augmented by UPF1.

80 ortant as the effect of NAT2 polymorphism on urinary bladder cancer differs dramatically between mono

81 are continued efforts in the development of urinary bladder cancer markers.

82 Efforts continue in the development of urinary bladder cancer markers.

83 hough efforts continue in the development of urinary bladder cancer markers.

84 COX-2 has also been strongly implicated in urinary bladder cancer primarily by studies with nonsele

85 s) infer a consistent and robust increase in urinary bladder cancer risk following exposures to aroma

86 d for various cancers, this paper focuses on urinary bladder cancer, a cancer in which a role for NAT

87 However, for some malignancies such as urinary bladder cancer, the ability to accurately assess

88 d sensitive telomerase activity detection in urinary bladder cancer.

89 developing various cancer types, especially urinary bladder cancer.

90 frequently observed halo artifact around the urinary bladder caused by inaccurate scatter correction

91 ns, 5.8 mm vs 11.9 mm; P < .001), and in the urinary bladder compared with PET/CT (means, 5.9 mm vs 1

92 Afferent pathways innervating the urinary bladder consist of myelinated Adelta- and unmyel

93 Afferent pathways innervating the urinary bladder consist of myelinated Adelta-fibers and

94 Liver, spleen, urinary bladder contents, and total-body activities were

95 comes primarily (97.2%) from activity in the urinary bladder contents.

96 Consistent with increased urinary bladder contractility caused by the absence of B

97 Here, the contributions of each pathway to urinary bladder contraction and the underlying electrica

98 We propose a concept of urinary bladder control involving a previously unidentif

99 The urinary bladder (critical organ), liver, kidney, and spl

100 Results: The urinary bladder (critical organ), liver, kidney, and spl

101 The urinary bladder demonstrated the largest peak uptake (18

102 t 6 to 8 weeks, either 4-layer multilaminate urinary bladder-derived extracellular matrix or expanded

103 t, galanin expression in nerve fibers in the urinary bladder detrusor and urothelium was decreased or

104 Transgenic expression of survivin in the urinary bladder did not cause histologic abnormalities o

105 rom interstitial cystitis, a chronic painful urinary bladder disorder characterized by thinning or ul

106 sponsive MRF neurons that did not respond to urinary bladder distension (i.e. out of the 46 remaining

107 r neurons was recorded in response to graded urinary bladder distension (UBD) in rats pretreated with

108 ims of this study were to examine effects of urinary bladder distension (UBD) on T(3)-T(4) spinal neu

109 of 283 spinal dorsal horn neurons (DHNs) to urinary bladder distension (UBD).

110 Urinary bladder distention (0.5-2.0 mL; 20 seconds) was

111 ficantly increased for both viscerovisceral (urinary bladder distention and colorectal distention) co

112 55 of 152 (36%) spinal neurons responded to urinary bladder distention in dextran sulfate sodium-tre

113 an background activity of neurons excited by urinary bladder distention in rats with dextran sulfate

114 threshold volume for excitatory responses to urinary bladder distention in rats with inflamed colon (

115 nse curves of excitatory responses to graded urinary bladder distention were significantly increased

116 olorectal distention) convergent neurons and urinary bladder distention-receptive neurons in rats wit

117 rly abolished sensory nerve outflow from the urinary bladder during bladder filling.

118 These changes may contribute to urinary bladder dysfunction after SCI.

119 ecessive disease characterized by congenital urinary bladder dysfunction, associated with a significa

120 condition which characterizes many types of urinary bladder dysfunctions including urinary incontine

121 Positive cultures from the urethra, urinary bladder, epididymides, and testes were obtained

122 4) IFU had positive cultures of the urethra, urinary bladder, epididymides, and/or testes.

123 e to intestinal epithelial, lung epithelial, urinary bladder epithelial, and liver cells.

124 rvoirs (QIRs) in Lamp1+ endosomes within the urinary bladder epithelium.

125 ng revealed only low levels of F-Dapa in the urinary bladder, even after displacement of kidney bindi

126 PK uptake was highest in the gallbladder and urinary bladder, followed by the liver, kidney, bone mar

127 The highest uptake was in the urinary bladder, followed by the liver, kidneys, and spl

128 implications for treatment of dysfunctional urinary bladder, for which this atropine- and P2X1 antag

129 lts reveal a central role for BK channels in urinary bladder function and indicate that BK channel dy

130 hat K(V) 7 channel activation likely affects urinary bladder function by blocking afferent nerve outf

131 s indicate that retigabine primarily affects urinary bladder function by inhibiting TC generation and

132 The role of SK3 channels in urinary bladder function was assessed using cystometry i

133 To address the role of the BK channel in urinary bladder function, the gene mSlo1 for the pore-fo

134 ot of Homer2 or Homer3) resulted in impaired urinary bladder function, which was associated with high

135 The pons contains neurons that control urinary bladder function.

136 e of K(V) 7 channels as regulators of murine urinary bladder function.

137 ft ventricle; liver; spleen; kidneys; bowel; urinary bladder; gluteus muscle; and malignant lesions.

138 and treatment of small cell carcinoma of the urinary bladder has become evident.

139 Traditionally, sensory signaling in the urinary bladder has been attributed to activation of bla

140 ing frequency and altered sensation from the urinary bladder has been suggested.

141 The discovery of bacteria in the female urinary bladder has fundamentally changed current dogma

142 xplore the specific autoimmune mechanisms of urinary bladder has long been hindered due to a lack of

143 tic or unresectable urothelial cancer of the urinary bladder has traditionally been treated with syst

144 dings of spinal afferents that innervate the urinary bladder have never been unequivocally identified

145 st, B1R mRNA was not detected in control rat urinary bladder; however, following acute (24 h) and chr

146 PV1, is under investigation for treatment of urinary bladder hyper-reflexia and chronic pain conditio

147 rcinoma (TCC), the most common cancer of the urinary bladder in dogs, is usually diagnosed at an adva

148 and greater accumulation of (18)F-FLT in the urinary bladder in male than female mice.

149 pannexin channels into the lumen of the rat urinary bladder in response to distension or stimulation

150 ry (CMA), canine urinary bladder, and murine urinary bladder in the amounts of 7.1 +/- 0.7, 26.5 +/-

151 ion (UTI), manifested by inflammation of the urinary bladder, in humans and is a major global public

152 in human transitional cell carcinomas of the urinary bladder including 36 primary tumors and 1 recurr

153 rogenic contractions of guinea pig and mouse urinary bladder, indicating a second mode of action of n

154 t uptake of 18F-annexin V in the kidneys and urinary bladder, indicating rapid renal clearance of 18F

155 d bacterial adhesion to epithelial cells and urinary bladder infection in mice.

156 esistance, vasculitis, diabetic nephropathy, urinary bladder infections, prostatitis, gastric paresis

157 dder afferent nerve hyperexcitability during urinary bladder inflammation or irritation.

158 llular ATP during pathological conditions of urinary bladder inflammation or irritation.

159 eceptors (Trks) in micturition reflexes with urinary bladder inflammation.

160 termine the pathway involved in transmitting urinary bladder inputs to thoracic spinal segments.

161 Small cell carcinoma of the urinary bladder is a rare and aggressive neuroendocrine

162 In most cases, small-cell carcinoma of the urinary bladder is admixed with other histological types

163 The urinary bladder is innervated by small afferent neurons

164 An abnormal urinary bladder is no longer a contra-indication to rena

165 2110 to guinea pig cardiac (KD = 5.8 nM) and urinary bladder (KD = 4.9 nM) membranes were of high aff

166 en found in all colorectal, ovarian, breast, urinary bladder, kidney, lung, and pancreatic tumors stu

167 ed in the prostate, breast, colon, pancreas, urinary bladder, kidney, lung, liver, stomach, testis, a

168 , which confirmed the conclusion that in the urinary bladder, low P(CO2) is due to the lack of CA.

169 Urinary bladder malformations associated with bladder ou

170 muscle actin-positive cells were present in urinary bladder matrix (UBM) than in ePTFE (22.2+/-3.3%

171 Urinary bladder matrix-BM (UBM-BM) was found to be a rig

172 Both male and female 5-HT3A mutant mice had urinary bladder mucosal and smooth muscle hyperplasia an

173 The epithelium of the urinary bladder must maintain a highly impermeable barri

174 sual case of a foreign body removed from the urinary bladder of a 63-year-old male which mimicked a p

175 ateral sprouting in the descending colon and urinary bladder of adult transgenic mice (i.e., those ti

176 yA-producing, uropathogenic E. coli into the urinary bladder of BALB/cJ mice.

177 veral years because of the presence in their urinary bladder of large, yellowish stones.

178 ng water reduces E. coli colonization in the urinary bladder of mice.

179 axonal densities in the descending colon and urinary bladder of NGF transgenic mice with and without

180 root ganglion (DRG) neurons innervating the urinary bladder of rats.

181 cells of Cajal (ICC) have been identified in urinary bladder of several species, but their presence i

182 cognitive impairment, nystagmus, and spastic urinary bladder of varying severity.

183 e fiber immunoreactivity was observed in the urinary bladders of 5-HT3Avs/vs compared with 5-HT3A wil

184 rogrammed paths, in a water bath, and in the urinary bladders of live pigs.

185 The urinary bladders of SK3T/T had significantly greater bla

186 for calcitonin gene-related peptide) in the urinary bladders of transgenic mice, fibers undergo spro

187 is disease manifested as a massively dilated urinary bladder, or megabladder, with disrupted SM in it

188 determined that the epithelial lining of the urinary bladder, or urothelium, expresses two subtypes o

189 g- or obesity-associated SPCs, such as lung, urinary bladder, oral cavity/pharynx, colorectal, pancre

190 d effective dose was 0.017 mSv/MBq, with the urinary bladder, osteogenic cells, and red marrow receiv

191 r types were analyzed: stomach, small bowel, urinary bladder, other urothelial, breast, ovarian, and

192 thrography evaluation of the urethra and the urinary bladder plays a very important role in the diagn

193 ildren with small capacity, defunctionalized urinary bladders present unique operative challenges.

194 owever, activation of TRPV1 receptors in the urinary bladder prior to acute colitis increased the num

195 ed ovarian, uterine, and vaginal tumors, and urinary bladder proliferative lesions, including three t

196 uracy to within approximately 20% of in vivo urinary bladder radiotracer concentrations.

197 e users analyzed SPECT/CT images for in vivo urinary bladder radiotracer uptake using quantitative so

198 The urinary bladder received the highest radiation dose with

199 The urinary bladder receives the highest OD in all investiga

200 2-/-, and P2X2/P2X3(Dbl-/-) mice had reduced urinary bladder reflexes and decreased pelvic afferent n

201 endogenous nerve growth factor (NGF) in the urinary bladder regulated type I collagen expression bec

202 days postsurgery, mice were euthanized, the urinary bladder removed, then fresh-fixed and stained fo

203 nal cell death and functional changes in the urinary bladder, resulting in bladder hyperdistension, u

204 1.3 cm) and a fistula between the appendix, urinary bladder, right scrotum, and right groin.

205 erative, other organs, such as the mammalian urinary bladder, shift from near-quiescence to a highly

206 The synergy between intrinsic urinary bladder signalling mechanisms and an inflammator

207 le synovium and perisynovial adipose tissue, urinary bladder, skeletal muscle, myocardium, and viscer

208 let obstruction (PBOO) induces remodeling of urinary bladder smooth muscle (detrusor).

209 gical recordings from freshly isolated mouse urinary bladder smooth muscle (UBSM) cells, isometric co

210 Contraction of urinary bladder smooth muscle (UBSM) is caused by the re

211 ion of nerve transmission to Ca2+ signals in urinary bladder smooth muscle (UBSM) is incompletely und

212 tant role in controlling the excitability of urinary bladder smooth muscle (UBSM).

213 Urinary bladder smooth muscle cells of Slo(-/-) mice lac

214 Urinary bladder smooth muscle contraction is triggered b

215 ght to have a particularly prominent role in urinary bladder smooth muscle function and therefore are

216 A 50% decrease in MLCK in urinary bladder smooth muscle had no effect on RLC phosp

217 Urinary bladder smooth muscle had no measurable K(V) 7 c

218 s, increased phasic contractions of isolated urinary bladder smooth muscle strips and exposed high af

219 a2+](i), MLCK activation, and contraction in urinary bladder smooth muscle strips neurally stimulated

220 mining the excitability and contractility of urinary bladder smooth muscle.

221 s of NGF protein in the descending colon and urinary bladder, so these tissues display increased dens

222 In the absence of BK channels, urinary bladder spontaneous and nerve-evoked contraction

223 In contrast, an abnormal urinary bladder stores urine at high pressure, leaks, or

224 A normal urinary bladder stores urine at low pressure, does not l

225 xponential fitting of activity overlying the urinary bladder suggested that approximately 12% of acti

226 n introduced for the treatment of overactive urinary bladder syndrome.

227 -invasive transitional cell carcinoma of the urinary bladder (TCC-UB) and identified a spectrum of ge

228 s investigated the sensory components of the urinary bladder that may underlie the pathophysiology of

229 organs, and more accurate than PET/CT in the urinary bladder; the alignment of thoracic MR/PET with e

230 n were primarily inhibited by input from the urinary bladder through either supraspinal structures or

231 d UPEC attenuation of PMN trafficking to the urinary bladder through pathogen-specific local inductio

232 the peak neurogenic contraction of the mouse urinary bladder to 30-40% of control.

233 when attempting to smile and failure of the urinary bladder to void completely despite a lack of ana

234 ectional area of DRG neurons innervating the urinary bladder trigone (UBT) were evaluated by coupling

235 In the urinary bladder, TRPV4 is not only abundantly expressed

236 idence supports the hypothesis that male rat urinary bladder tumors arise through urinary bladder cal

237 s, based on increased incidences of male rat urinary bladder tumors at high exposure levels and on fe

238 Urinary bladder undergoes dramatic volume changes during

239 Surprisingly, sAPs in the mouse urinary bladder, unlike those from other species, are tr

240 y transduction can initiate visceral pain in urinary bladder, ureter, gut and uterus.

241 r responses to mechanical stimulation of the urinary bladder, urethra, colon and penis, and electrica

242 TNP-ATP) on pelvic afferents innervating the urinary bladder using an in vitro mouse bladder-pelvic n

243 investigation of small cell carcinoma of the urinary bladder using novel methods to understand the ge

244 Hodgkin lymphoma, ovary, pancreas, prostate, urinary bladder, uterus, and thyroid) were estimated, wh

245 ively active in lymphatic endothelium in the urinary bladder, uterus, intestine, heart, and airways.

246 4 muSv/MBq for an adult male with a 1.5 hour urinary bladder voiding interval.

247 1.4 muSv/MBq for an adult male, with a 1.5-h urinary bladder voiding interval.

248 During filling, urinary bladder volume increases dramatically with littl

249 (0.094 +/- 0.03), pancreas (0.066 +/- 0.01), urinary bladder wall (0.047 +/- 0.02), and adrenals (0.0

250 upper large intestinal wall (0.08 +/- 0.07), urinary bladder wall (0.08 +/- 0.02), and liver (0.07 +/

251 e human reference adult were received by the urinary bladder wall (0.262 mGy/MBq), kidneys (0.0296 mG

252 ans with the highest absorbed doses were the urinary bladder wall (0.38 mSv/MBq) and kidneys (0.054 m

253 ans with the highest absorbed doses were the urinary bladder wall (0.62 mSv/MBq) and the pancreas (0.

254 ses (for 150 MBq injected) were found in the urinary bladder wall (12.2 mGy), spleen (8.1 mGy), kidne

255 the kidneys (14.3 +/- 3.6 muSv/MBq) and the urinary bladder wall (13.5 +/- 3.7 muSv/MBq).

256 e from the (11)C-nicotine injection were the urinary bladder wall (14.68 +/- 8.70 muSv/MBq), kidneys

257 estimate of radiation dose equivalent to the urinary bladder wall (ca. 180 +/- 30 mSv/MBq or 0.7 rem/

258 10 studies, 0.0151 cGy/MBq, occurred for the urinary bladder wall (with hydration and 1- to 2-h voidi

259 tracer, the absorbed dose was highest in the urinary bladder wall and kidneys, followed by the pancre

260 the critical organ (33 mGy), followed by the urinary bladder wall and spleen (10 mGy each), salivary

261 Human radiation dose estimates indicated urinary bladder wall as the dose-limiting organ (0.200 m

262 the highest absorbed radiation dose, was the urinary bladder wall at 7.96E-02 mSv/MBq.

263 voiding interval to 60 or 90 min lowered the urinary bladder wall dose to 0.0885 mGy/MBq (0.327 rad/m

264 ffective dose was 31 +/- 1 muSv/MBq, and the urinary bladder wall had the highest absorbed dose at 37

265 fective dose was 38 +/- 4 muSv/MBq, with the urinary bladder wall having the highest absorbed dose at

266 The urinary bladder wall is the critical organ for 11C-WAY10

267 The dose to the urinary bladder wall of the reference patient varies bet

268 e in children younger than 12 y, whereas the urinary bladder wall received the highest dose in older

269 The urinary bladder wall received the highest radiation dose

270 he urine (biological half-life ca. 4 h), the urinary bladder wall received the highest radiation dose

271 Hypertrophy occurs in urinary bladder wall smooth muscle (BSM) in men with par

272 The urinary bladder wall was the organ with the highest esti

273 The critical organ for dosimetry is the urinary bladder wall with a dose of 0.0586 +/- 0.0164 mG

274 doses (mSv/MBq) were 0.40 +/- 0.058 for the urinary bladder wall, 0.11 +/- 0.011 for the kidneys, 0.

275 Peak organ doses were to the urinary bladder wall, 0.258 mGy/MBq (0.955 rad/mCi), and

276 he highest absorbed radiation doses, was the urinary bladder wall, at 0.047 +/- 0.008 and 0.067 +/- 0

277 rbed organ doses were seen in the spleen and urinary bladder wall, followed by kidney, adrenals, and

278 the highest mean dose coefficients were the urinary bladder wall, kidneys, and spleen.

279 upper large intestine wall, small intestine, urinary bladder wall, kidneys, and thyroid had the highe

280 th high radiation burden included the lungs, urinary bladder wall, kidneys, gallbladder wall, heart w

281 6.6 and 60.6 times higher in the kidneys and urinary bladder wall, respectively, than estimates from

282 pt two--the excretory organs gallbladder and urinary bladder wall, which had peak activities at 32 an

283 The critical organ was the urinary bladder wall, with a dose of 0.406 mGy/MBq.

284 +/- 4 muGy/MBq for the absorbed dose in the urinary bladder wall.

285 allbladder wall, small intestine, liver, and urinary bladder wall.

286 the gallbladder, liver, lungs, kidneys, and urinary bladder wall.

287 duced only modest effects on the dose to the urinary bladder wall.

288 The critical organ was the urinary bladder wall.

289 ddition, the radioactivity signal within the urinary bladder was lower at 3 h after injection, especi

290 can be derived from the kidneys, ureter, or urinary bladder, we evaluated whether measurement of int

291 n and radioactivity concentration within the urinary bladder were analyzed in both scans.

292 ident cancer of liver, colorectal, lung, and urinary bladder were included as cases and up to four ag

293 Dynamic PET scans of the urinary bladder were obtained in 6 subjects; 2 subjects

294 se rate at 1 m (EDR-1m) from the sternum and urinary bladder were obtained.

295 Kidney pairs and urinary bladders were cultured 48 h after infection.

296 Urinary bladders were excised from C57BL6/J mice and the

297 ysiologic accumulation of radiotracer in the urinary bladder which may cause some lesions in its vici

298 nal gland resembles a kidney, connected to a urinary bladder with a nephropore (exit opening) and a c

299 wed a markedly-contracted and small-capacity urinary bladder with a thickened, irregular and edematou

300 including pain and discomfort related to the urinary bladder, without infection or other identifiable