ライフサイエンスコーパス: urine output

1 racterize longitudinal creatinine trends and urine output.

2 higher proteinuria and a marked reduction in urine output.

3 es associated with a significant increase in urine output.

4 , AKI was defined using serum creatinine and urine output.

5 xtraction ratio; hematocrit; hemoglobin; and urine output.

6 on (APACHE) III score, serum creatinine, and urine output.

7 , serum and urine sodium and osmolality, and urine output.

8 increased metabolic acidosis, and decreased urine output.

9 pectively, with abdominal pain and decreased urine output.

10 er and had significantly higher minimum 4-hr urine output.

11 fferences in donor age and lowest total 4-hr urine output.

12 d flow, blood urea nitrogen, creatinine, and urine output.

13 with reduced filling pressures and increased urine output.

14 nd-diastolic area correlated negatively with urine output.

15 sing creatinine and narrowed with increasing urine output.

16 and poor performers based on blood flow and urine output.

17 ney injury manifested by serum creatinine or urine output.

18 tion of decongestive therapy because of poor urine output.

19 macroscopic appearance, renal blood flow and urine output.

20 ney injury in 67.2% of the patients with low urine output.

21 bitors stimulate AQP2 trafficking and reduce urine output.

22 lated, PDSinh-C01 produced a 60% increase in urine output.

23 nd classified AKI using serum creatinine and urine output.

24 ase in serum creatinine level or decrease in urine output.

25 Diuretic dose was a strong predictor of urine output.

26 miR-192 blunted the adaptational increase of urine output.

27 The addition of nesiritide did not change urine output.

28 output and the factors associated with high urine output.

29 c) Pearson correlations were quantified with urine output.

30 inine (1.91 [1.06 to 3.44] per 1 mg/dl), and urine output (0.12 [0.03 to 0.44] L/d) were independentl

31 using the NRPE to direct therapy mean daily urine output (1.8 0.9 l vs. 3.0 0.8 l), net fluid output

32 pital stay or by study day 14, and a greater urine output (2,926 +/- 1,179 ml vs. 2,350 +/- 1,066 ml;

33 7 years, respectively) and lowest total 4-hr urine output (327+/-208 versus 507+/-437 cc, respectivel

34 atients by levels of serum creatinine and/or urine output according to Kidney Disease Improving Globa

35 se: Improving Global Outcomes creatinine and urine output acute kidney injury criteria were applied s

36 In early AKI, urine output after a furosemide stress test (FST), which

37 AUC)+/-SEM of 0.87+/-0.09 (P<0.0001), 2-hour urine output after FST was significantly better than eac

38 sily detected in the urine in the very first urine output after ischemia in both mouse and rat models

39 patients who developed acute kidney injury, urine output alone was a better mortality predictor than

40 alGuard Therapy, which aims to maintain high urine output; alpha-melanocyte-stimulating hormone, with

41 eir tissue levels negatively correlated with urine output among all kidneys at six hours.

42 deletion resulted in a threefold increase in urine output and a 500-fold increase in glucosuria, as w

43 Urine output and absolute Na(+) excretion decreased by 6

44 ived an FST and evaluated the ability of FST urine output and biomarkers to predict the development o

45 Urine output and creatinine clearance progressively incr

46 1) to characterize the baseline agreement of urine output and creatinine definitions of AKI; (2) to r

47 Monitoring bladder catheters for urine output and early signs of urinary tract infections

48 In CoCl2-treated animals, acute urine output and endogenous lithium clearance increased

49 on provoked a three- to fourfold increase in urine output and endogenous lithium clearance, 33% inhib

50 of 13 different commonly consumed drinks on urine output and fluid balance when ingested in a euhydr

51 in-2 knockdown in the hypothalamus decreases urine output and fluid intake, and increases urine osmol

52 luding ACE inhibitors, BG9719 increased both urine output and GFR.

53 Changes in urine output and glomerular filtration rate are therefor

54 on of fluctuation of vasopressin and nightly urine output and its role in patient selection for desmo

55 performed serial biopsies and monitored the urine output and kinetic estimated glomerular filtration

56 Both stimuli increased urine output and lithium clearance three- to four-fold a

57 These LUTS may be masked by low urine output and may pose risk to renal allografts after

58 atment was associated with higher cumulative urine output and natriuresis, findings consistent with b

59 Compared with pretreatment values, urine output and net fluid balance increased by 130% and

60 Other efficacy measures included urine output and patient-reported symptoms.

61 UTB(inh)-14 also increased urine output and reduced urine osmolality in mice given

62 etate (AOAA), respectively, caused increased urine output and reduced urine osmolality in mice that w

63 the frequency of AKI definition components (urine output and serum creatinine [sCr] concentration ch

64 inine were all significantly improved, while urine output and serum lactate had beneficial trends.

65 ly increased solute-free water clearance and urine output and significantly decreased urinary osmolal

66 BG9719 alone caused an increase in urine output and sodium excretion (P<0.05).

67 Water consumption, urine output and sodium excretion were higher in HS rats

68 l mortality and examine interactions between urine output and survival.

69 00032-0.01 mg/kg) dose-dependently increased urine output and the diuretic effect reached a plateau a

70 dels to identify the impact of nesiritide on urine output and the factors associated with high urine

71 and hydration to improve renal perfusion and urine output and to minimize uric acid or calcium phosph

72 Secondary compensation returned urine output and urinary Na(+) excretion to control leve

73 Volume Overload) trial with complete data on urine output and urine sodium concentration (UNa) were a

74 Decreased urine output and/or increased serum creatinine may heral

75 ) was seen for continuous items (e.g., 24-hr urine output) and coded items requiring judgment (e.g.,

76 (2) lower donor age, (3) higher minimum 4-hr urine output, and (4) more HLA matches in recipients of

77 itioned base excess, 2) maintained a greater urine output, and 3) received furosemide; and slower in

78 Heart rate, arterial blood pressure, urine output, and base deficit (as a reflection of sever

79 ents, arterial and mixed venous blood gases, urine output, and biochemical and hematologic analyses w

80 crit, improves mean systemic blood pressure, urine output, and cardiac function, and decreases the ne

81 e to the kidney graft, she produced adequate urine output, and creatinine and glomerular filtration r

82 dose escalation guided by symptoms, weight, urine output, and electrolyte/renal monitoring.

83 n, there were no differences in body weight, urine output, and fluid balance.

84 ted with deteriorating acid-base status, low urine output, and hyperkalemia.

85 mpt to normalize blood pressure, heart rate, urine output, and mental status, which are the tradition

86 owed a dramatic improvement in hemodynamics, urine output, and metabolic acidosis, as well as a perce

87 al congestion score, dyspnea assessment, net urine output, and net weight change.

88 etter preserved gas exchange, renal flow and urine output, and prolonged survival.

89 recipient and donor age, cold ischemia time, urine output, and Scr.

90 First, ensuing hypertension decreases urine output, and second, guanylyl cyclase-A (GC-A), the

91 ven when blood pressure, cardiac output, and urine output are within clinically acceptable ranges.

92 m creatinine concentration and a decrease in urine output, are considered tantamount to the injury of

93 de of circulatory pressures, cardiac output, urine output, arterial blood gases, ventilation:perfusio

94 Mean arterial blood pressure, heart rate, urine output, arterial blood oxygen, and PCO2 values, ar

95 eart rate, a lower troponin T, and a greater urine output as compared with norepinephrine (p < .05).

96 After volume expansion, urine output as well as urinary sodium and cyclic GMP ex

97 Cumulative urine output at 4 h after ingestion of cola, diet cola,

98 Urine output at 65 mm Hg was 49+/-18 mL/hr and was 43+/-

99 mated glomerular filtration rates, and daily urine output at days 1, 7, 15, and 30 after kidney trans

100 In these same patients, furosemide increased urine output at the expense of decreased GFR.

101 patients who were nonoliguric (>400 ml/d of urine output) at initiation of the study.

102 er normal values of systolic blood pressure, urine output, base deficit, hemoglobin, and cardiac inde

103 ore reliable but less expedient, whereas the urine output based definition is rapid but less reliable

104 We also assessed 10 creatinine and urine output-based SGF definitions relative to 12-month

105 pressure (CPP), central venous pressure, and urine output before and after the administration of hype

106 Validated questionnaires and low urine output before transplantation may be used to ident

107 (serum creatinine 132-354 umol/L or minimum urine output between 0.3 and 0.5mL/kg/hr), or 3) severe

108 ently with HBOC, but consistent patterns for urine output, blood urea nitrogen, and creatinine, were

109 espectively, when serum creatinine level and urine output both indicated stage 3 AKI.

110 reversed the changes in renal blood flow and urine output, but impaired creatinine clearance persiste

111 uretic dose was a strong predictor of higher urine output, but neurohormonal activation (as evidenced

112 Tolvaptan increased urine output by 3 h in a dose-dependent manner (p < 0.00

113 sultambenzosulfonamide at 20 mg/kg increased urine output by 3-5-fold and reduced urine osmolality by

114 ral venous pressure, mean arterial pressure, urine output, central venous (or mixed venous) oxygen sa

115 We sought to evaluate an automated urine output collections system and compare it to nursin

116 hanges in kidney blood flow, creatinine, and urine output compared to vehicle.

117 intake, and decreased water consumption and urine output compared with Akita mice.

118 A treatment results in a further increase in urine output compared with diabetes alone, suggesting a

119 For all 15 animals, physiologic data (urine output, core temperature, arterial pressure, heart

120 Urine output correlated with cortical ADC with furosemid

121 Urine output, creatinine clearance, and sodium and potas

122 was defined according to serum creatinine or urine output criteria based on the contemporary definiti

123 Cumulative application of the creatinine and urine output criteria characterizes renal excretory and

124 he Kidney Disease: Improving Global Outcomes urine output criteria for acute kidney injury lack speci

125 ned the significance of oliguria meeting the urine output criteria for AKI (AKI-UO) and examined its

126 nts meet both the serum creatinine level and urine output criteria for AKI and when these abnormaliti

127 tinine definitions of AKI; (2) to refine the urine output criteria to identify the thresholds that be

128 ition of AKI used and whether AKI defined by urine output criteria was included; the incidence was lo

129 AKI is often defined using creatinine and urine output criteria.

130 ury is diagnosed according to creatinine and urine output criteria.

131 s normalization of both serum creatinine and urine output criteria.

132 We found that the conventional urine output criterion (6 hr, 0.5 ml/kg/h) has specifici

133 Our goal is to examine the urine output criterion and augment it with physiological

134 he Kidney Disease: Improving Global Outcomes urine output criterion for acute kidney injury.

135 he Kidney Disease: Improving Global Outcomes urine output criterion.

136 he Kidney Disease: Improving Global Outcomes urine output criterion.

137 a standardization for the processing of big urine output datasets to improve consistency in AKI diag

138 Thus, their hyperglycemia, hyperphagia and urine output declined significantly.

139 0% versus 69% versus 74%, respectively), and urine output (decreased discharge dose from OOD dose in

140 se in his serum creatinine without change in urine output; dialysis was not required.

141 AP), systemic vascular resistance index, and urine output did not demonstrate any significant changes

142 severity and duration across creatinine and urine output domains with the risk for RRT and likelihoo

143 Urine output down trended from 37 L to 6 L per day with

144 arrhythmias, peak lactate, inotropic scores, urine output, duration of mechanical ventilation, intens

145 ht provide useful information in addition to urine output during decongestive treatment in heart fail

146 On day 1, KW-3902 monotherapy increased urine output during the first 6 h (445, 531, 631, and 57

147 nnaire detailing their fluid consumption and urine output during the race.

148 one did not significantly affect the rate of urine output, endogenous lithium clearance (an inverse m

149 e downstream advantages, including increased urine output, enhanced plasma volume, reduced weight los

150 ns exist regarding accurate ascertainment of urine output even within the intensive care unit.

151 significantly greater bladder capacity, and urine output exceeded the infused saline volume.

152 ht loss had occurred and the patient's daily urine output exceeded their daily fluid intake.

153 flow doubled, blood urea nitrogen rose, and urine output fell.

154 dergoing renal transplantation with <50 cc/h urine output for 8 consecutive hours over the first 24 h

155 The relative importance of urine output for predicting survival varies with admissi

156 envalues [lambda(i)]) imaging parameters and urine output from serial bladder volumes were calculated

157 ney injury (serum creatinine < 132 umol/L or urine output >= 0.5 mL/kg/hr), 2) mild-moderate acute ki

158 Conivaptan significantly increased urine output in a dose-dependent manner (-11+/-17, 68+/-

159 te clearance), urinary sodium excretion, and urine output in a double-blind, placebo-controlled, cros

160 Daily urine output in AQP1/AQP3 double knockout mice (15 ml) w

161 e significant changes in sodium excretion or urine output in GC-A-deficient mice.

162 ia of isolated mouse collecting ducts and on urine output in mice treated with tolvaptan, a VR2 block

163 Nesiritide did not increase urine output in patients with ADHF.

164 Urine output in the diuretic group was only higher on th

165 lowed by the mesenteric and celiac arteries, urine output increased 4.2-fold in wild-type mice compar

166 Urine output increased by >80% (p < .01) during the firs

167 The urine output increased from 70.7 (IQR: 70) ml/day to 1,2

168 n be precipitated by dehydration and reduced urine output, increased protein intake, heavy physical e

169 tegorized based on worst serum creatinine or urine output into: 1) no acute kidney injury (serum crea

170 m(2); P(trend)=0.032) but greater cumulative urine output (IRF, 8780 [7025 to 11 208] mL; worsening r

171 The change in urine output is consistent with the pharmacokinetics of

172 Cumulative urine output, kidney blood flow, blood urea nitrogen, an

173 A urine output less than 0.47 mL/kg/hr was associated with

174 dent on dialysis treatments, oliguric with a urine output less than 650 ml per day, and uremic with a

175 w hemoglobin levels were associated with low urine output, low serum albumin, high parathyroid hormon

176 in the ANG-3777 arm had larger increases in urine output; lower serum creatinine; greater reduction

177 ol/L or renal replacement therapy or minimum urine output < 0.3 mL/kg/hr).

178 mg/dL or 50% from baseline within 48 hrs or urine output < 0.5 mL/kg/hr for > 6 hrs despite fluid re

179 ary analysis with 108 having a negative FST (urine output < 200 mL in 2 hr following FST).

180 2) hyponatremia; or 3) diuretic resistance (urine output </=125 ml/h following intravenous furosemid

181 nd Cl(-)/Cr ratios were both associated with urine output </=1500 mL (area under the curve, 0.830 and

182 inine level >/=2 times the baseline level or urine output <0.5 ml per kilogram of body weight per hou

183 DGF was defined by urine output <30 cc/hour, decline in serum creatinine of

184 Accurate urine output measurements as well as serum creatinine va

185 Hourly urine output measurements were used to guide the infusio

186 Volumes of study fluid administered, 24-hour urine output, measures of resource utilization, and mort

187 e net iron excretion (measured by faecal and urine output minus food input).

188 The time of temperature and urine output nadir was used as a marker of circadian rhy

189 ameters of perfusion (pulse, blood pressure, urine output, normal base deficit).

190 ache, dizziness, constipation, and increased urine output occurred more frequently in the difelikefal

191 ality was associated independently with 24-h urine output (odds ratio [OR], 5.6; 95% confidence inter

192 /dL in serum creatinine within 48 hrs and/or urine output of < or = .5 mL/kg/hr for > 6 hrs.

193 A minimum hourly urine output of 0.5 mL/kg is a key target guiding periop

194 tated Ringer's solution, infused to a target urine output of 1 mL x kg(-1) x hr(-1) throughout the 24

195 red forty patients were included with a mean urine output of 1.05 mL/kg/hr and an overall in-hospital

196 increase in blood flow to 200 to 250 mL/min, urine output of 40 to 260 mL/h and increasing creatinine

197 Low urine output of less than 250 mL per day was also predic

198 yesian model was developed to predict future urine output on an hourly basis.

199 When subtle reductions in urine output or a rising creatinine are observed postope

200 oup received dialysis 3 times per week until urine output or creatinine clearance criteria were met.

201 lood urea nitrogen (OR, 1.02 per mg/dl), log urine output (OR, 0.64 per log ml/d), and heart rate (OR

202 id increase in serum creatinine, decrease in urine output, or both.

203 on of 1400W did not change renal blood flow, urine output, or creatinine clearance, whereas infusion

204 erences in GFR, effective renal plasma flow, urine output, or sodium excretion for any time interval

205 etabolism, skin microcirculatory blood flow, urine output, or splanchnic perfusion.

206 m and urine pharmacokinetics, did not affect urine output, osmolality, salt excretion, or acid-base b

207 Overall, in the setting of early AKI, FST urine output outperformed biochemical biomarkers for pre

208 ure resulted in a 25% increase in cumulative urine output over 5 days (median 10.8 versus 8.7 L mL in

209 The primary end point was cumulative urine output over 5 days.

210 sorption, and urine osmolality and increased urine output (P </= 0.03 except for urine output with ra

211 sorption, and urine osmolality and decreased urine output (P </= 0.04, except for sodium reabsorption

212 MAPC-treated kidneys demonstrated improved urine output (P = .009), decreased expression of injury

213 MAPC-treated kidneys demonstrated improved urine output (P = .009), decreased expression of injury

214 ed median 24-hour natriuresis (P = 0.03) and urine output (P = 0.005), expediting hospital discharge

215 After 6 hrs, intestinal wet/dry ratios, urine output, peritoneal fluid, and intraluminal fluid w

216 The treated dogs had excellent urine output posttransplant, with peak serum creatinine

217 tween nesiritide and predictors affected the urine output prediction.

218 y high blood pressure, enhanced thirst, high urine output, proteinuria, and kidney damage.

219 ody treatment: blood pressure; C(3a) levels; urine output; proteinuria; blood urea nitrogen; and kidn

220 ted with arterial pH (r2 = .28; p = .01) and urine output (r2 = .21; p = .03) when analyzed by log-li

221 validated an algorithm for computing hourly urine output rates and identifying oliguric AKI across i

222 Renin correlated significantly with urine output (repeated-measures correlation coefficient

223 ient's sCr value decreased to 1.4 mg/dl, and urine output returned to greater than 2000 ml per day.

224 for renal replacement therapy or changes in urine output, serum creatinine or both) or death.

225 patients with stage 1 acute kidney injury by urine output, serum creatinine or both, with risk increa

226 We measured stroke work, urine output, serum creatinine, among other parameters,

227 Graft function was monitored by urine output, serum creatinine, and renal biopsy.

228 Stage 1 urine output, serum creatinine, and urinary [TIMP-2]*[IG

229 Body weight, water intake, urine output, solute and urea excretion, serum and urine

230 etermine whether a low perioperative minimum urine output target is safe and fluid sparing when compa

231 y undergoing elective colectomy to a minimum urine output target of 0.2 mL/kg/h (low group) or 0.5 mL

232 A perioperative urine output target of 0.2 mL/kg/h is noninferior to the

233 nd higher final pump flow while having lower urine output than both the treatment groups.

234 strated favorable macroscopic appearance and urine output, the kidney was transplanted into a 61-year

235 Rather than the absolute dose of diuretic or urine output, the primary signal of interest when evalua

236 urrent definitions of acute kidney injury, a urine output threshold of less than 0.5 mL/kg/hr is mode

237 ent definitions of acute kidney injury use a urine output threshold of less than 0.5 mL/kg/hr, which

238 sitivity of 0.58 and 0.48 for MIMIC-III; and urine output thresholds of 10 hours and 0.6 ml/kg/h have

239 Secondly, urine output thresholds of 12 hours and 0.6 ml/kg/h have

240 ed severe salt wasting and sharp increase in urine output under basal conditions.

241 he kidney via the renal artery would restore urine output (UO) and glomerular filtration rate (GFR) i

242 Concerns have been raised about the use of urine output (UO) criteria in CLD.

243 s defined by changes in serum creatinine and urine output (UO).

244 Patients with a preserved urine output (UOP) greater than 0.3 mL/kg/hr over the st

245 Daily fluid balance, urine output (UOP), and creatinine for days 1-7 of ARDS

246 ypertension, systemic hypotension, decreased urine output (UOP), and metabolic acidosis.

247 pro B-type natriuretic peptide (NT-proBNP), urine output (UOP), and plasma creatinine.

248 Total urine output, urinary protein, albumin/creatinine ratio,

249 We prospectively collected urine output using an electronic urine monitoring system

250 was to develop a model that analyses hourly urine output values in real time to identify those at ri

251 >/=50% from previous lowest value, and/or if urine output was <1 mL/kg/h on postnatal days 2 to 7.

252 Daily urine output was 1.5-fold greater in UT-B- deficient mic

253 For 24 hours, urine output was 113+/-51 mL/h with placebo and 110+/-56

254 Total urine output was 1294+/-1024 mL and 3492+/-1613 mL durin

255 Median (25th, 75th percentiles) 24-h urine output was 2,280 (1,550, 3,280) ml with nesiritide

256 tions were near normal; lactate was cleared; urine output was adequate.

257 When FST urine output was assessed in patients with increased bio

258 All urine output was collected daily.

259 Urine output was higher during the first when compared w

260 Urine output was higher for groups 2 and 3 than for grou

261 In treated animals, urine output was higher, metabolic acidosis was attenuat

262 rature, altered consciousness, and decreased urine output was independently associated with 90-day mo

263 Urine output was measured daily in the 75 patients who r

264 Urine output was measured in 5,864 subjects; of these, 5

265 Blood was sampled, total urine output was measured, and the animal was then kille

266 Low urine output was most strongly associated with mortality

267 FST urine output was the only biomarker to significantly pre

268 Urine output was the only clinical index of cardiac func

269 Urine output was then collected for the subsequent 4 h.

270 The mean renal blood flow and total urine output were 68.0 mL/min/100 g and 560 mL in the le

271 Arterial pressure and urine output were also attenuated in recombinant human a

272 Hourly temperature and urine output were ascertained from the patient records.

273 f age, serum creatinine, and intra-operative urine output were compared.

274 ons, serum biochemistries, fluid intake, and urine output were evaluated.

275 Flow, resistance, and urine output were measured serially for 4 hr.

276 Blood pressure, heart rate, and urine output were recorded, as well as blood urea nitrog

277 Urine outputs were collected over 3 h subsequent to i.m.

278 lary blood flow and red blood cell velocity, urine output) were measured.

279 sorption, and urine osmolality and increased urine output, while raxibacumab, a PA-directed monoclona

280 Regardless of the end point, combining FST urine output with individual biomarkers using logistic r

281 ncreased urine output (P </= 0.03 except for urine output with raxibacumab [P = 0.17]).

282 Hourly urine output with the xenograft was more than double the

283 ion of PDSinh-C01 produced a 30% increase in urine output, with increased Na(+) and Cl(-) excretion.

284 ed to determine the prognostic importance of urine output within the first 24 hours of admission to t

285 ted in favorable changes in hemodynamics and urine output without affecting blood pressure or heart r

286 lozin to standard diuretic therapy increases urine output without affecting renal function in patient

287 sopressin, and decreases in H(2)O intake and urine output without any effects on mean arterial pressu