ライフサイエンスコーパス: urine pH

1 o cystine stone formation, especially at low urine pH.

2 s intestinal and urine calcium and increases urine pH.

3 o be significantly related to differences in urine pH.

4 anifested by metabolic acidosis and alkaline urine pH.

5 is in patients with type 2 diabetes is a low urine pH.

6 nd demonstrated salutary release kinetics at urine pH.

7 suria, ketonuria, proteinuria, hematuria and urine pH.

8 urine adenosine concentration and decreased urine pH.

9 and inversely proportional to the change in urine pH; a regression equation was generated to relate

10 bonate (24.5+/-3.4 to 23.7+/-3.5 mmol/L) and urine pH (all P<0.001), but did not change urinary ammon

11 ctor is its urease enzyme which can increase urine pH and form urinary stones, causing catheter block

12 n wild-type mice, secretin acutely increased urine pH and pendrin function in isolated perfused corti

13 ncluding dissolution of uric acid by raising urine pH and the higher rates of brushite stone recurren

14 r retention of calcium and magnesium, higher urine pH, and mild hypermagnesemia.

15 ) the associations of body fatness with 24-h urine pH as the outcome variable, respectively.

16 It has recently been suggested that a low urine pH be added to the abnormalities linked to insulin

17 se findings, we question the view that a low urine pH be added to the abnormalities linked to insulin

18 ncreases in body fatness to be paralleled by urine pH decreases (P <= 0.0002).

19 of kidney function impairment with a lowered urine pH even at a young age.

20 pite the harsh chemical conditions of stored urine (pH > 9 and total ammonia nitrogen > 4000 mg N/L),

21 mia, no rise in urine PCO2 with alkaluria, a urine pH > 5.5, and urine potassium excretion rate not s

22 of Gpr116 caused a significant reduction in urine pH (i.e., acidification) accompanied by an increas

23 conclusion is based on the finding of a low urine pH in individuals with clinical syndromes associat

24 to but cannot entirely account for the lower urine pH in patients with type 2 diabetes.

25 on manifests as a disproportionally low 24-h urine pH in relation to the sum of actually excreted amm

26 Urine pH inversely correlated with both body weight and

27 Urine pH is more alkaline and metabolic acidosis is more

28 e is around pH 6, bacterial urease increases urine pH leading to the precipitation of calcium and mag

29 r in Proteus mirabilis, cause an increase in urine pH - leading to blockage.

30 rum creatinine, uric acid, calcium and lower urine pH level.

31 The urine pH of black women was 0.11 units higher (P = 0.03)

32 ne calcium levels; however, the increases in urine pH, oxalate, and phosphate levels lead to increase

33 IMGU in this population, with no relation to urine pH (r = 0.02).

34 urrogate estimate of insulin resistance, and urine pH (r = 0.06).

35 Urine pH remained significantly lower in patients with t

36 with type 2 diabetes and UASF had lower 24-h urine pH than NV.

37 fected with the two species exhibited higher urine pH values, urolithiasis, bacteremia, and more pron

38 Urine pH, volume, and 24-h urinary excretion of calcium,

39 eosinophils, monocytes, and cholesterol; the urine pH was also elevated.

40 Of the four signals associated with urine pH, we note that the pH-increasing alleles of two

41 Thiosulfate treatment lowered urine pH, which would lower calcium phosphate supersatur