ライフサイエンスコーパス: uropathy

1 ty associated significantly with obstructive uropathy.

2 ary tract infection, trauma, and obstructive uropathy.

3 from the development of in utero obstructive uropathy.

4 ronic renal failure secondary to obstructive uropathy.

5 ied at 4-6 months of age from an obstructive uropathy.

6 g the pathobiology of congenital obstructive uropathy.

7 es ischemic hypoxic insults, and obstructive uropathy.

8 anding of the pathophysiology of obstructive uropathy.

9 use of medications for treating obstructive uropathy.

10 nal insufficiency as a result of obstructive uropathy.

11 ble insight into a wide range of obstructive uropathies.

12 ue insights into a wide range of obstructive uropathies and has been demonstrated to be useful in the

13 ease characterized by congenital obstructive uropathy and abnormal facial expression.

14 Obstructive uropathy and dysplasia were the cause of CRI in 92% of t

15 were stratified according to the underlying uropathy and the type of initial management during child

16 Urinary tract malformations, obstructive uropathy, and hypoplasia/dysplasia are extremely importa

17 rs, such as young recipient age, obstructive uropathy, and overall intensity of immunosuppressive the

18 al cell fate and nephron loss in obstructive uropathy are not fully understood.

19 emic bowel (75%); the lowest was obstructive uropathy-associated urinary tract infection (26%).

20 Chronic obstructive uropathy (COU) created by unilateral ureteric ligation i

21 Congenital obstructive uropathy (COU) is a common cause of developmental defect

22 Congenital obstructive uropathy (COU) is a prevalent human developmental defect

23 t animals exhibit seizures and/or obstuctive uropathy, each of unknown cause.

24 rvention has been done for fetal obstructive uropathy for over a decade, yet little is known about lo

25 n the fetus as it is affected by obstructive uropathy has had no significant advances in the past yea

26 urrent study, a model of chronic obstructive uropathy in the mouse is established and the role of lym

27 abdominal pain, abdominal mass, obstructive uropathy, infertility, menstrual irregularities and abno

28 Obstructive uropathy is the most common complication, although other

29 In a mouse obstructive uropathy model of chronic kidney disease, interstitial f

30 ute or chronic renal conditions, obstructive uropathies, neoplasia, or infectious processes between a

31 .1 per y younger; P < 0.001) and obstructive uropathy (OR, 12.4; P < 0.01) as primary renal disease.

32 ing GD-CNVs and novel deletions; obstructive uropathy (OU) had a lower CNV burden and an intermediate

33 ic CKD based on diagnosis: FSGS, obstructive uropathy (OU), aplasia/dysplasia/hypoplasia (A/D/H), and

34 postischemic renal fibrosis and obstructive uropathy, treatment with N-terminal Slit2 before or afte

35 d non-acute cystitis, obstructive and reflux uropathy, urolithiasis, or hypertension were less likely

36 iteria), exclusion criteria were obstructive uropathy, urothelial carcinoma, and metastatic cancer.

37 es with the most severe forms of obstructive uropathy, usually associated with a fatal neonatal cours

38 standing the mechanisms of fetal obstructive uropathy will be essential for the specific management o