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1 slower pacing cycle lengths, akin to reverse use dependence.
2 ents, shifted voltage dependence and reduced use dependence.
3 red for channel activation and showed strong use dependence.
4 was increased, and repetitive pulsing showed use dependence.
5 agonists, (ii) voltage dependence, and (iii) use dependence.
6 id in onset, reversible, and did not display use dependence.
7 at activation of the channel exhibits strong use dependence.
8 ndence and nonalcohol psychoactive substance use dependence.
9 heat and agonist activations differ in cross use-dependence.
10 )3.2 T currents exhibited little voltage- or use-dependence.
11 11.2%; and nonalcohol psychoactive substance use dependence, 15.2%.
12 inactivation could be partly responsible for use dependence, a more stringent test would require that
13 s (1 s Vh = -46 mV), showed substantial 1 Hz use dependence and had inactivation (60 ms pulse) recove
14 standing factors that determine specificity, use dependence and other properties of K(+) channel drug
15                           The drug exhibited use-dependence and significantly slowed the apparent rec
16 is common, often leading to prolonged opioid use, dependence, and neuropathic pain.
17 t in IVS5 did not mimic the complete loss of use dependence as observed for the replacement of the wh
18 s had a 1 s Vh of -29 mV, showed little 1 Hz use dependence at -67 mV and recovered from the inactiva
19 cited by short depolarizations showed strong use dependence at frequencies as low as 1 Hz, although r
20 y critically shape the dynamic character and use dependence for cranial afferent transmission at the
21 role of inactivated channel conformation and use dependence for diltiazem, a specific benzothiazepine
22 ion attenuate lidocaine (lignocaine)-induced use dependence; however, the pharmacological role of slo
23      Quantitative analyses indicate that the use dependence in heat sensitivity is pertinent to the p
24 ines conduction in peripheral nerves and its use dependence in tetrodotoxin-resistant (TTXr) sodium c
25             We assayed individual-level tool-use dependence in wild New Caledonian crows by analyzing
26 fects of lacosamide on slow inactivation and use-dependence in Nav1.7 variants from non-responsive pa
27                                        Their use dependence is modest and this results from being neu
28            We propose that lidocaine-induced use dependence may involve an allosteric conformational
29                                This "reverse use dependence" may reduce inhibition during physiologic
30 eived a primary (DSM-IV) diagnosis of heroin use/dependence (n = 2797) and (2) data from unstructured
31  recovery from inactivation and decrease the use dependence of channel activity with and without the
32 pendent homologs could largely eliminate the use dependence of heat sensitivity of TRPV3.
33                                         This use dependence of heat sensitivity thus provides a mecha
34 versal of AMPA-eEPSC block, verifying strict use dependence of philanthotoxin.
35 SEP amplitude) state (r = 0.5), suggesting a use dependence of prior functional state period.
36 mined the molecular basis that underlies the use dependence of temperature sensitivity of TRPV3.
37 o be permeable to Ca(2+), accounting for the use dependence of the run down.
38 s implications for gating, drug affinity and use dependence of their respective channel complexes.
39                         Physiologically, the use dependence of TRPV2 confers nociceptors with a hyper
40                                          The use dependence of TRPV2 reveals that the channel can hav
41 er (~11 nM), conferring to lubeluzole a huge use-dependence of great therapeutic value.
42             The resulting enhancement in its use dependence reduces channel availability, which expla
43        However, how the channels acquire the use dependence remains unknown.
44 ocked sodium channels efficiently but lacked use dependence, similar to R-like.
45                F1586C fully abolished A-core use dependence, suggesting that A-core binds to the loca
46  for the 2 binding conformations can control use-dependence, the hallmark of successful antiarrhythmi
47 ngle channel properties of mutants that lost use dependence toward diltiazem were characterized by dr
48 temperatures, inhibition had strong "reverse use dependence", whereby inhibition was relieved by repe
49  VX-548 had the unusual property of "reverse use-dependence," whereby inhibition could be relieved by