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1 ifically enriched according to the size of a uterine fibroid.
2 medical history was notable only for a large uterine fibroid.
3 preserved bone mineral density in women with uterine fibroids.
4 among women reporting both endometriosis and uterine fibroids.
5 with birth weight, gestational duration and uterine fibroids.
6 possibly pose a health hazard for women with uterine fibroids.
7 e observed clinical efficacy of EGCG against uterine fibroids.
8 ith placebo for the treatment of symptomatic uterine fibroids.
9 s regarding the molecular characteristics of uterine fibroids.
10 shows a modestly enlarged uterus with three uterine fibroids.
11 HIFU provides an excellent option to treat uterine fibroids.
12 s effective and safe in treating symptomatic uterine fibroids.
13 stream regulator REST in the pathogenesis of uterine fibroids.
14 KT-mTOR) pathway] is aberrantly expressed in uterine fibroids.
15 use model for a gene that is misexpressed in uterine fibroids.
16 tracellular matrix deposition, a hallmark of uterine fibroids.
17 ucing heavy menstrual bleeding in women with uterine fibroids.
18 cretion of the 2 collections and the risk of uterine fibroids.
19 tissue after focused ultrasound treatment of uterine fibroids.
20 heparin might be useful in the treatment of uterine fibroids.
21 tanding of the molecular basis of the common uterine fibroids.
22 mmon non-Mendelian manifestation of isolated uterine fibroids.
23 t properties, is an investigational drug for uterine fibroids.
24 racellular matrix, which is a key feature of uterine fibroids.
25 we implemented GDEC on the scRNA-seq data of uterine fibroids.
26 hes for prevention and medical management of uterine fibroids.
27 icipants were disproportionately affected by uterine fibroids.
28 elopment of long-term hormonal therapies for uterine fibroids.
29 higher positive predictive values (PPVs) for uterine fibroids (0.86 and 0.87 vs. 0.78) and for endome
30 o-controlled, 6-month phase 3 trials (Elaris Uterine Fibroids 1 and 2 [UF-1 and UF-2]) to evaluate th
31 this study was to identify risk factors for uterine fibroids among women undergoing tubal sterilizat
32 fant formula feeding and ultrasound-detected uterine fibroids among young African-American women with
35 y embolization, in women who had symptomatic uterine fibroids and did not want to undergo hysterectom
37 diagnostic information) to identify incident uterine fibroids and endometriosis patients among n=750
38 once daily, may have efficacy in women with uterine fibroids and heavy bleeding while avoiding hypoe
39 t of women aged 40 years with a diagnosis of uterine fibroids and no desire for future pregnancy was
40 ctively controlled excessive bleeding due to uterine fibroids and reduced the size of the fibroids.
42 59 women (mean age, 35.9 years +/- 4.8) with uterine fibroids and/or adenomyosis who were unable to c
43 justed for age, BMI, race, family history of uterine fibroids, and isoflavone excretion, this trend r
44 I 25-29.9 kg/m(2), asthma, thrombocytopenia, uterine fibroids, antidepressant use, induction of labou
59 esized to be risk factors for development of uterine fibroids, but few studies have investigated the
60 ight urine collections (48 h apart) from 170 uterine fibroid cases and 173 controls were analyzed for
63 visualization at aortography performed after uterine fibroid embolization (UFE) and, using OA arterio
69 ers and diseases (polycystic ovary syndrome, uterine fibroids, endometriosis) as well as contraceptio
74 amycin (mTOR) pathway in the pathogenesis of uterine fibroids has been suggested in several studies.
75 ndometriosis, polycystic ovary syndrome, and uterine fibroids-have remained stubbornly understudied d
76 n was found between isoflavone excretion and uterine fibroids; however, the intake of soy foods, the
78 follicle, can be associated with the common uterine fibroids in a syndrome called multiple cutaneous
80 ion for the chronic treatment of symptomatic uterine fibroids in women who cannot or do not want to t
87 ose a unifying mechanism for pathogenesis of uterine fibroids mediated by H19 and identify a pathway
88 of Ms P, a 41-year-old woman with recurrent uterine fibroids, menorrhagia, anemia, and fatigue who w
89 Women with a history of endometriosis and uterine fibroids might have an increased long term risk
92 ptor site near a polypyrimidine tract, and a uterine fibroid obtained from a patient showed loss of h
97 o differences in breast fibrocystic disease, uterine fibroids, or endometrial lining thickness as ass
100 on (-$30,850; 95% CI, -$31,629 to -$30,091), uterine fibroid resection (-$1509; 95% CI, -$1754 to -$1
101 erence, -37.7 days; 95% CI, -41.1 to -34.3), uterine fibroid resection (mean difference, -11.7 days;
106 o-megabase interval spanning FH in the NIEHS Uterine fibroid study, a cross-sectional study of fibroi
107 effects on the molecular pathways linked to uterine fibroids, such as re-establishing normal ovarian
108 health-related quality-of-life domain of the Uterine Fibroid Symptom and Quality of Life (UFS-QOL) qu
117 ciation between atrial fibrillation (AF) and uterine fibroids (UF) is unclear, though UF are linked t
118 enetically (ORs = 1.06-2.09) associated with uterine fibroids (UF), PCOS, heavy menstrual bleeding (H
121 ecords (EHRs) present opportunities to study uterine fibroids uterine fibroids and endometriosis with
122 e of FH and the linked genes in nonsyndromic uterine fibroids, we explored a two-megabase interval sp
123 Cases comprised women aged 17-44 years whose uterine fibroids were first visualized at the time of tu
128 om normally cycling premenopausal women with uterine fibroids, who were not on hormonal treatment at
130 ing to joint categories of endometriosis and uterine fibroids, with an increased risk of all cause pr