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2 v-Abl Delta858-1080 was rescued by activated v-Akt and was also moderately rescued by activated v-H-R
4 into the medium by COMMA-1D/Akt1 or COMMA-1D/v-akt cells revealed elevated gelatinase activity that w
11 isome proliferator-activated receptor gamma, v-AKT murine thymoma oncogene homolog 2, zinc metallopro
12 led to an increase in the phosphorylation of v-AKT murine thymoma viral oncogene (AKT) and enhanced t
13 bination of oncogenic forms of activation of v-akt murine thymoma viral oncogene homolog (AKT) and NR
15 Concomitant expression of activated forms of v-akt murine thymoma viral oncogene homolog (AKT) and Ra
16 racellular signal-regulated kinase (ERK) and v-akt murine thymoma viral oncogene homolog (AKT) in a s
17 tes the phosphatidylinositol 3-kinase (PI3K)/v-AKT murine thymoma viral oncogene homolog (AKT) pathwa
19 I3K), phosphatase and tensin homolog (PTEN), v-akt murine thymoma viral oncogene homolog (AKT), and m
24 gulator of G-protein signalling 4 (RGS4) and V-AKT murine thymoma viral oncogene homolog 1 (AKT1) the
25 plex 1 (RAPTOR), the serine/threonine kinase V-Akt murine thymoma viral oncogene homolog 1 (AKT1), an
27 720 and induced hyperphosphorylation of AKT (v-akt murine thymoma viral oncogene homolog 1), a phenot
28 ouse model of primary liver cancer driven by v-akt murine thymoma viral oncogene homolog and neurobla
29 metinib indicated a compensatory increase in v-akt murine thymoma viral oncogene homolog signaling an
30 gh expression of miR-155 sensitizes cells to v-akt murine thymoma viral oncogene homolog-1 inhibitors
31 ery of tau kinases such as protein kinase A, v-Akt murine thymoma viral oncogene homolog-1, glycogen
33 ed the role of the phosphoinositide-3-kinase/v-akt murine thymoma viral oncogene homolog/mammalian ta
34 d the metabolic and growth properties of the v-akt murine thymoma viral oncogene homolog/mammalian ta
37 noma cells inhibits the HGF-induced MET-AKT (v-Akt murine thymoma viral oncogene) signaling pathway a
38 showed hypertrophic signaling (activation of v-akt murine thymoma viral oncogene/protein kinase B [AK
40 t agar; however, cells overexpressing either v-akt or Akt1 became highly invasive when grown on the E
41 cells, expression of activated forms of Akt (v-Akt or myristoylated Akt) results in enhanced survival
42 homologue of the transforming viral oncogene v-Akt, plays a central role in the regulation of cell su
45 tein-coupled receptors (GPCRs) activate PI3K/v-AKT thymoma viral oncoprotein (AKT) to regulate many c