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1 refluxate with the upper airway or by a vago-vagal reflex.
2  and pancreatic secretion evoked by the vago-vagal reflex.
3 inar cells and not indirectly through a vago-vagal reflex.
4 re applied to investigate involvement of the vagal reflex.
5  of CCK to control gastric function via vago-vagal reflexes.
6 pacts the gain or even the direction of vago-vagal reflexes.
7 estinal motility, which are mediated by vago-vagal reflexes.
8 tion and modulation of gastrointestinal vago-vagal reflexes.
9 ed pyloric relaxation is mediated via a vago-vagal reflex and NO release.
10                We explored the ITR impact on vagal reflex apnea elicited by intravenuously injected 5
11 ry afferent nerves and the subsequent airway vagal reflex arcs.
12  vagal complex, where upper gastrointestinal vagal reflexes are integrated.
13 ecent reports which have suggested that vago-vagal reflexes are inverted by the metabolic status of t
14 ion are more sensitive in amplifying gastric vagal reflexes; (b) in the periphery, CCK8 is more poten
15  animals survived for several months and the vagal reflex behaviour, exemplified by citric acid-induc
16 ions as a consequence of its actions on vago-vagal reflex circuit elements.
17 e hindbrain is also the location of the vago-vagal reflex circuitry regulating gastric motility.
18 haps permanent changes in the sensitivity of vagal-reflex circuitry.
19 ests that TNF(alpha) action directly on vago-vagal reflex control circuits causes the autonomic misre
20 lucose on the central components of the vago-vagal reflex control of gastric function, we performed b
21  (CRF) on the central components of the vago-vagal reflex control of gastric function.
22 ion procedure targeted on the elimination of vagal reflex evoked by high frequency stimulation or an
23 central neural pathways involved in the vago-vagal reflex in pancreatic secretion; the demonstration
24 uit exerts descending regulation over airway vagal reflexes in male and female rats using a range of
25 ts and to alter the function of gastric-vago-vagal reflexes in response to these stimuli.
26 bre terminals, which in turn initiate a vago-vagal reflex inhibition of gastric motor function.
27 ytin injections into the motor layer labeled vagal reflex interneurons that have radially directed de
28                 We hypothesize that the vago-vagal reflex mediating pancreatic secretion in the rat i
29                        No evidence of a vago-vagal reflex mediating the effects of CCK was observed.
30 t (ExStrT), an easily quantifiable marker of vagal reflexes, might identify high- and low-risk long Q
31                                     The vago-vagal reflex plays an important role in mediating pancre
32                It is suggested that the vago-vagal reflex plays an important role in mediating the ac
33 ensitivity, an established marker of reduced vagal reflexes, predicts low probability of symptoms amo
34 to control NST neurones responsible for vago-vagal reflex regulation of the stomach.
35 gestive process can powerfully regulate vago-vagal reflex sensitivity.
36 Intense exercise training, which potentiates vagal reflexes, should probably be avoided by LQT1 patie
37 obesity, suggesting that attenuation of vago-vagal reflex signalling may precede the development of o
38                                              Vagal reflexes slow heart rate and can change where the
39 ts from our parallel studies on gastric vago-vagal reflexes suggest that noradrenergic neurons in the
40 re, we report a role of delta9-THC in a vago-vagal reflex that can result in gastro-oesophageal reflu
41 , we report a role of Delta(9)-THC in a vago-vagal reflex that can result in gastro-oesophageal reflu