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1 difficile diarrhea; vulvovaginal candidiasis/vaginitis).
2 nfirmed a nonprotective role for PMNs during vaginitis.
3 onses previously characterized during murine vaginitis.
4 tions with various prevalences of infectious vaginitis.
5 sexually transmitted infection (STI) causing vaginitis.
6 n microscopy was associated with Trichomonas vaginitis.
7 very, low birth weight, cervical cancer, and vaginitis.
8 ntibodies are protective in a mouse model of vaginitis.
9 tic NAATs that can detect the main causes of vaginitis.
10 tory role for gamma/delta T cells in Candida vaginitis.
11 one-associated susceptibility to C. albicans vaginitis.
12 ques as an alternative model of experimental vaginitis.
13 sis and immunity associated with C. albicans vaginitis.
14 ant than systemic CMI for protection against vaginitis.
15 en at the time that they were suffering from vaginitis.
16 culation does not mediate protection against vaginitis.
17 biotic treatment or in patients with aerobic vaginitis.
18 nal candidiasis suffering from acute Candida vaginitis.
19 us acidophilus in the prevention of candidal vaginitis.
20 ispose women to recurring attacks of Candida vaginitis.
21  to the characteristic signs and symptoms of vaginitis.
22 study examining the intersection of STIs and vaginitis.
23 women having a laboratory-diagnosed cause of vaginitis, 81 (47%) received 1 or more inappropriate pre
24 (STIs) in women seeking care for symptoms of vaginitis and bacterial vaginosis, revealing highly comp
25                         Women diagnosed with vaginitis and cervicitis had a higher prevalence of M. g
26 where it may be asymptomatic or cause severe vaginitis and cervicitis.
27 bacillus species isolated per patient in the vaginitis and control groups, respectively.
28 olates obtained from patients suffering from vaginitis and four isolates from commercial products in
29 croflora in women with signs and symptoms of vaginitis and is comparable in diagnostic accuracy to th
30 rable effects by systemic CMI or PMN against vaginitis and suggest that if local T cells are importan
31 ssociations of STIs with two major causes of vaginitis and underscore the importance of STI testing i
32 tment of Candida vulvovaginitis, Trichomonas vaginitis, and bacterial vaginosis.
33 lis and M. hominis bacterial counts, Candida vaginitis, and herpes simplex virus (HSV) were positivel
34                        Resistance to Candida vaginitis appears additive in CD10 x B6F1 animals and do
35                    MRMs were associated with vaginitis (aPR: 1.8; 1.14-2.85), cervicitis (aPR: 3.5; 1
36  (qPCR) assay, including testing for aerobic vaginitis (AV), Candida, sexually transmitted infections
37 iae is a ubiquitous, ascomycetous yeast, and vaginitis caused by this organism has been reported only
38 6.8%, 21.1%, 11.8%, and 15.4% of urethritis, vaginitis, cervicitis, and pelvic inflammatory disease (
39 the Aptima BV and Aptima Candida/Trichomonas vaginitis (CV/TV) assays.
40                   Accurate and comprehensive vaginitis diagnostics are needed to direct treatment and
41 all three with two episodes of S. cerevisiae vaginitis, different strains were isolated during the re
42                                   Infectious vaginitis due to bacterial vaginosis (BV), vulvovaginal
43 of yeast culture plus exclusion of alternate vaginitis etiologies.
44  years of age who presented with symptoms of vaginitis, exposure to T. vaginalis, or multiple sexual
45  this was statistically significant only for vaginitis (for vaginitis, OR = 1.88 [95% CI = 1.37 to 2.
46 %); samples from women who were negative for vaginitis had significantly lower positivity rates (7.9%
47  candidiasis (VVC) by the Affirm and Candida vaginitis Hologic TMA ASR assays was assessed using micr
48         Armed with new criteria for defining vaginitis immunopathology, the purpose of this study was
49 extent, the Bcr1 pathways in contributing to vaginitis immunopathology.
50 based assays for determining the etiology of vaginitis in a cohort of 323 symptomatic women.
51        We compared susceptibility to Candida vaginitis in derived murine substrains differing in sens
52 ndidiasis and a murine model of experimental vaginitis indicate that systemic cell-mediated immunity
53 creased susceptibility to experimental yeast vaginitis, indicating a role for alpha(1,2)fucosylated c
54                                              Vaginitis is often diagnosed by microscopy and limited t
55     ALS gene expression patterns in a murine vaginitis model were identical to those from the clinica
56 allenge but had no effect in the C. albicans vaginitis model.
57                              For Trichomonas vaginitis (n=55), HIV-1 RNA decreased from 3.67 to 3.05
58 tion, genital ulcers, Trichomonas vaginalis, vaginitis or cervicitis, and male circumcision.
59 sted odds ratio, 18.34; 95% CI, 4.95-67.96), vaginitis or vulvovaginitis (adjusted odds ratio, 5.17;
60 nce, preterm labor during current pregnancy, vaginitis or vulvovaginitis, and sepsis.
61 stically significant only for vaginitis (for vaginitis, OR = 1.88 [95% CI = 1.37 to 2.58]; for cervic
62 nts (P=.0001; positive association), Candida vaginitis (P=.007), and HSV (P=.03) were significantly a
63         It was demonstrated that (i) in each vaginitis patient, there was only one colonizing strain;
64  that Ebola virus causes uterine cervicitis, vaginitis, posthitis, and medullary adrenalitis.
65                       Women with symptoms of vaginitis presenting to one of three clinical centers we
66             This animal model of C. glabrata vaginitis provides a means to study the genetics and pat
67               Among women without infectious vaginitis, return visits for vaginitis symptoms were mor
68                            Clinical disease (vaginitis), serum and vaginal washing antibody levels, a
69 VC) and from an animal model of experimental vaginitis suggest that deficiencies in immune function s
70 gest, as have other studies, that women with vaginitis symptoms may be at risk for an STI.
71    From May to December 2018, 111 women with vaginitis symptoms prompting the clinician to order an A
72 munity practice setting, 42% of women having vaginitis symptoms received inappropriate treatment.
73 hout infectious vaginitis, return visits for vaginitis symptoms were more common among women treated
74 ows to lower titers and produces less severe vaginitis than wild-type or gC rescued virus, indicating
75 acity to cause immunopathology during murine vaginitis to this avirulent hypofilamentous strain.
76 nt, 79% were of white race, and 38% reported vaginitis treatment within the past month.
77 ontaining the three common organisms causing vaginitis: Trichomonas vaginalis, Candida species, and G
78 dependent reference standard for trichomonal vaginitis (TV), a positive result in either assay was de
79 ansmitted infection (STI) that can result in vaginitis, urethritis, and preterm birth.
80 d in women from whom samples were tested for vaginitis using a molecular diagnostic assay.
81  the combined search terms of diagnosis with vaginitis, vaginal discharge, candidiasis, bacterial vag
82 acterium, Candida, gastrointestinal- system, vaginitis, vaginosis-bacterial, and related terms.
83 d nucleic acid amplification test (NAAT) for vaginitis/vaginosis diagnosis; Nugent scoring (BV); yeas
84  may be a valuable tool for the diagnosis of vaginitis/vaginosis in laboratory and POC settings.
85 = 14 years old with signs and/or symptoms of vaginitis/vaginosis.
86  difficile diarrhea, C. difficile infection, vaginitis/vulvovaginal candidiasis, and pneumonia.
87 he importance of fungal morphogenesis during vaginitis was addressed with a two-pronged approach.
88 of three patients suffering from C. glabrata vaginitis were clonally plated on agar containing CuSO(4
89 lcer disease, vaginal discharge, and Candida vaginitis were significantly associated with HIV-1 seroc
90 samples obtained from women with symptoms of vaginitis were tested with the Aptima BV and Aptima Cand
91            In females, trichomoniasis causes vaginitis, while in males, it is frequently asymptomatic
92 nt were evaluated for women with symptoms of vaginitis who were seeking care at community practice si
93 onents for the immunopathogenesis of Candida vaginitis, with implications for transition from benign
94  family practice clinic might only receive a vaginitis workup, whereas those seen at a sexually trans
95             This parasite, a common cause of vaginitis worldwide, is one of the earlier branching euk