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1 teral subdiaphragmatic vagotomy or were sham-vagotomized.
2 ed in the following groups of anaesthetized, vagotomized adult Sprague-Dawley rats (age 4 months), tr
7 e was also no significant difference between vagotomized and sham-vagotomized rats in the number of c
8 of the previously acquired CTA, because both vagotomized and sham-vagotomized rats rejected all of th
9 Experiments were conducted in anesthetized, vagotomized and spontaneously breathing rats while monit
10 prived) rats were anaesthetized, bilaterally vagotomized and underwent acute PVN inhibition by bilate
13 istered intrathecally (C4) to anaesthetized, vagotomized and ventilated male Sprague-Dawley rats befo
14 10 muM, 5 mul; 3 x 5 min), in anaesthetized, vagotomized and ventilated male Sprague-Dawley rats elic
16 c motor output was recorded in anesthetized, vagotomized, and mechanically ventilated rats with dual
18 fter intermittent hypoxia from anesthetized, vagotomized, and pump-ventilated control and C2 spinally
19 ditions stopped breathing in vagus-intact or vagotomized, anesthetized, spontaneously breathing adult
20 onses evoked by these stimuli were absent in vagotomized animals or in animals pretreated with the ga
21 njection of capsaicin in vagus-intact and in vagotomized animals was also abolished or attenuated aft
22 ose IL-1 beta, stimulation of plasma ACTH in vagotomized animals was also markedly diminished compare
23 s observed in sino-aortically denervated and vagotomized animals, we believe any contribution of the
26 riments were done in urethane-anaesthetized, vagotomized, aortic deafferented, ventilated rats except
28 +/- 0.2%) with intact vagus nerves and three vagotomized cats, AP-SND phase walk was characterized by
32 , additional experiments were performed in 5 vagotomized dogs to investigate vagally mediated mechani
33 ded phrenic activity in seven anaesthetized, vagotomized, glomectomized, paralysed and servo-ventilat
34 iled to increase preoptic PGE2 levels in the vagotomized guinea pigs (n = 10), whereas in their sham-
39 erve activity was measured in anaesthetized, vagotomized, neuromuscularly blocked and artificially ve
40 microA) stimulus intensity in anaesthetized, vagotomized, neuromuscularly blocked and artificially ve
41 (14-15 months old) which were anaesthetized, vagotomized, neuromuscularly blocked and ventilated: (1)
42 1 microM tetrodotoxin (TTX-R) was halved in vagotomized NGNs (21 +/- 8 vs. 56 +/- 8 % of total I(Na)
43 ecovery from inactivation was also slower in vagotomized NGNs (fast time constant, 2.8 +/- 0.4 vs. 1.
46 produced large, repetitive TTX-R I(Na) while vagotomized NGNs produced smaller TTX-S I(Na) that rapid
47 However, steady-state I(Na) inactivation in vagotomized NGNs was shifted -9 mV relative to control v
49 ial waveforms was increased by over 250 % in vagotomized NGNs with DAPs (19.0 +/- 2.1 pC) compared to
50 significantly different among control NGNs, vagotomized NGNs with DAPs and vagotomized NGNs without
53 control NGNs, vagotomized NGNs with DAPs and vagotomized NGNs without DAPs, averaging 54 +/- 7.9 (n =
58 dialysis) of conscious, subdiaphragmatically vagotomized or sham-operated guinea pigs following LPS a
60 not different from baseline levels either in vagotomized (P = 0.18) or intact (P > 0.05) animals.
61 eft C2 spinal cord hemisected, anesthetized, vagotomized, pancuronium paralyzed, and artificially ven
66 ing adult rats (n = 4) and in anaesthetized, vagotomized, paralysed and ventilated animals (n = 14).
67 isocapnic hypoxia in urethane-anaesthetized, vagotomized, paralysed and ventilated rats at different
68 vity was recorded in urethane-anaesthetized, vagotomized, paralysed and ventilated rats exposed to: (
69 ing aorta in 14 Dial-urethane anaesthetized, vagotomized, paralysed, artificially ventilated cats.
70 a in thirty-six Dial-urethane-anaesthetized, vagotomized, paralysed, artificially ventilated cats.
71 were from fifteen anaesthetized, bilaterally vagotomized, paralysed, artificially ventilated cats.
72 on (FI,O1), 0.1-0.12) was measured in twelve vagotomized, paralysed, artificially ventilated young ra
73 airway negative pressure in 15 decerebrated, vagotomized, paralyzed and artificially ventilated cats.
74 neurons in dl-pons in urethane-anesthetized, vagotomized, paralyzed, and servo-ventilated adult Sprag
75 Experiments were performed on anesthetized, vagotomized, paralyzed, ventilated, and spinally injured
76 irus (PRV) was injected into the pancreas of vagotomized rats and after 6 days survival, the pattern
78 cant difference between vagotomized and sham-vagotomized rats in the number of c-FLI-positive cells i
79 uired CTA, because both vagotomized and sham-vagotomized rats rejected all of the test intraoral infu
81 ne), artificially ventilated (FIO2=0.50) and vagotomized rats were presented with two or three, 5 min
82 In chloralose-anaesthetized, ventilated, vagotomized rats, acute hypoxia (10% O2, 60 s) evoked an
84 We used normal rats, subdiaphragmatically vagotomized rats, rats with denervated adrenal medullae
86 mited HPA activation in subdiaphragmatically vagotomized rats, the vagus nerve does not appear to be
97 e-anaesthetized, neuromuscularly blocked and vagotomized Sprague-Dawley rats, arterial blood pressure
100 ter exposure to CIH, urethane-anaesthetized, vagotomized, ventilated, paralysed rats had significantl