ライフサイエンスコーパス: vagus

1 marker of sensory nerve activation) of human vagus.

2 the gut and ascend to the brainstem via the vagus.

3 itary tract, and dorsal motor nucleus of the vagus.

4 ptin and electrical activity of the afferent vagus.

5 salis of Meynert and dorsal motor nucleus of vagus.

6 s solitaries and dorsal motor nucleus of the vagus.

7 s located in the dorsal motor nucleus of the vagus.

8 neural fulcrum, can be elicited from either vagus.

9 ch-sensitive ion channel, was present at the vagus afferent nerve endings innervating the aortic arch

10 y volunteers were assessed and compared with vagus/airway nerve and cough responses in a cigarette sm

11 identified received indirect central inputs (vagus alone: 25%; stellate ganglion alone: 27%; both: 48

12 in response to electrical stimulation of the vagus and activation of cardiopulmonary, chemo- and baro

13 ct (NTS) and the dorsal motor nucleus of the vagus and affected within-meal satiation.

14 olarization of murine, guinea pig, and human vagus and firing of Adelta-fibers (not C-fibers), which

15 e of pharmacological studies on the isolated vagus and patch clamp and single-channel inside-out expe

16 ed to the reticuloendothelial system via the vagus and splenic nerves.

17 voked depolarization of guinea pig and human vagus, and this was inhibited by a transient receptor po

18 t procedures such as deep brain stimulation, vagus, and trigeminal nerve stimulation are effective on

19 s present in the dorsal motor nucleus of the vagus, as well as the olfactory bulb and anterior olfact

20 us coeruleus and dorsal motor nucleus of the vagus, but not in the nucleus basalis of Meynert, raphe

21 itarius, and the dorsal motor nucleus of the vagus, but not the area postrema.

22 ted no obvious disruption and lesions of the vagus caused some alterations to song behavior.

23 Dissection of the right vagus decreased peritoneal group 3 innate lymphoid cell

24 y nucleus [NST], dorsal motor nucleus of the vagus [DMN] and catecholaminergic neurons of the ventrol

25 tract (nTS) and dorsal motor nucleus of the vagus (DMNV).

26 mbiguus (NA) and dorsal motor nucleus of the vagus (DMNX).

27 solitarius (NTS) dorsal motor nucleus of the vagus (DMV) and area postrema) decrease gastric tone and

28 f neurons in the dorsal motor nucleus of the vagus (DMV) and nucleus ambiguus (nAmb) that express the

29 The dorsal motor nucleus of the vagus (DMV) contains preganglionic motor neurons that co

30 The dorsal motor nucleus of the vagus (DMV) in the brainstem consists primarily of vagal

31 alamus (PVN) and dorsal motor nucleus of the vagus (DMV) in the mouse brain as well as expression of

32 The Dorsal Motor Nucleus of Vagus (DMV) is degenerated in many patients with early s

33 re made from rat dorsal motor nucleus of the vagus (DMV) neurones in thin brainstem slices.

34 FD) dysregulated dorsal motor nucleus of the vagus (DMV) neurones, prior to the development of obesit

35 are modulated by dorsal motor nucleus of the vagus (DMV) neurones, whose activity is finely tuned by

36 orpus-projecting dorsal motor nucleus of the vagus (DMV) neurones.

37 eactivity in rat dorsal motor nucleus of the vagus (DMV) neurones.

38 orneurons of the dorsal motor nucleus of the vagus (DMV) that is capable of modulating systemic blood

39 neurones of the dorsal motor nucleus of the vagus (DMV) to GLP-1 and its analogues; (2) the effects

40 terneuron in the dorsal motor nucleus of the vagus (DMV), a nucleus that is intimately involved in re

41 d neurons in the dorsal motor nucleus of the vagus (DMV), a population of cholinergic neurons that sh

42 olitarius (NTS), dorsal motor nucleus of the vagus (DMV), and ventrolateral medulla (VLM).

43 ansmitter in the dorsal motor nucleus of the vagus (DMV).

44 ) neurons in the dorsal motor nucleus of the vagus (DMV).

45 lease in the rat dorsal motor nucleus of the vagus (DMV).

46 m neurons in the dorsal motor nucleus of the vagus (DMV).

47 c neurons of the dorsal motor nucleus of the vagus (DMV).

48 t the level of the rostral medulla where the vagus fibers intersect with the Sp5 and sp5.

49 dition, PLI images showed that a minority of vagus fibers separated from the vagus trajectory and joi

50 ent fibres was connected to the intact right vagus in chronically instrumented dogs.

51 the afferent (sensory) and motor (efferent) vagus in regulation of appetite, mood, and the immune sy

52 o and depolarization of human and guinea pig vagus in vitro, and inhibited calcium influx in airway-s

53 ignaling between Hgf and Met is required for vagus innervation of the pharyngeal arches.

54 nder control conditions stopped breathing in vagus-intact or vagotomized, anesthetized, spontaneously

55 The effect in vagus-intact rats was to slow the rhythm to a pace equiv

56 alpha,beta-meATP also evoked CSR response in vagus-intact rats.

57 nerve fibres by stimulation of the cervical vagus is associated with NO production and release in th

58 from ectopic lipid accumulation via a brain-vagus-liver axis and may be a therapeutic strategy to am

59 (including the parasympathetic dorsal motor vagus) mediated improved glucose homeostasis independent

60 These data establish a necessary role of vagus-mediated dopamine neuron activity in postingestive

61 on between postingestive sucrose sensing and vagus-mediated dopamine neuron activity in the ventral t

62 educe meal size and increase the efficacy of vagus-mediated satiation signals.

63 T1-weighted images showed hyperintense vagus medullar striae, coursing towards the dorsomedial

64 xamine the mechanism underlying targeting of vagus motor axons to the pharyngeal arches in zebrafish.

65 The vagus motor neuron topographic map is therefore determin

66 that axon initiation is delayed in posterior vagus motor neurons independent of neuron birth time.

67 We focus on the vagus motor neurons, which are topographically arranged

68 re expressed in posterior, but not anterior, vagus motor neurons.

69 by specifying anterior-posterior identity in vagus motor neurons.

70 odels indicate various lesions affecting the vagus, muscle, enteric neurons, interstitial cells of Ca

71 1)R in afferent and efferent branches of the vagus nerve (Cnr1(flox/flox); Phox2b-Cre mice).

72 Efferent activation of the cervical vagus nerve (cVN) dampens systemic inflammatory processe

73 ients by closed-loop stimulation of the left vagus nerve (LVN).

74 to small multifunctional nerves such as the vagus nerve (VN) which is the main multimodal autonomic

75 their anatomical relation to branches of the vagus nerve (Xth cranial nerve).

76 iorates gut barrier dysfunction in sepsis by vagus nerve activation via central ghrelin receptors.

77 -inflammatory influences but also may reduce vagus nerve activity.

78 Here, we identify two populations of mouse vagus nerve afferents (P2ry1, Npy2r), each a few hundred

79 Thus, PrP(Sc) in CJD affects the vagus nerve analogously to alpha-synuclein in Parkinson

80 or in 12 dogs to stimulate the left cervical vagus nerve and a radiotransmitter for continuous record

81 ishes the anti-inflammatory potential of the vagus nerve and beta2-agonists to control inflammation i

82 This study indicates that vagus nerve and cholinergic agonists activate the sympat

83 Likewise, vagus nerve and cholinergic agonists fail to control inf

84 blish the anti-inflammatory potential of the vagus nerve and cholinergic agonists in immunocompromise

85 Thus, vagus nerve and cholinergic agonists inhibit systemic in

86 the anti-inflammatory potential of both the vagus nerve and cholinergic agonists, and abrogate their

87 e (ACh) is the major neurotransmitter of the vagus nerve and exerts its parasympathetic actions via a

88 tiation during normal drinking, involves the vagus nerve and is transmitted to key forebrain neurons

89 n anti-inflammatory reflex arc involving the vagus nerve and memory T cells is necessary for resoluti

90 increased responses were observed in ex vivo vagus nerve and neuron cell bodies in the vagal ganglia.

91 c mechanism that involves recruitment of the vagus nerve and subsequent activation of endocannabinoid

92 many types of neuroimmune circuits, with the vagus nerve and sympathetic innervation of numerous orga

93 core the anti-inflammatory properties of the vagus nerve and the potential of neuro-immune interactio

94 they also initiate activity in the afferent vagus nerve and thereby signal directly to the brainstem

95 was abolished by surgical transection of the vagus nerve and was not observed in other peripheral org

96 These results unveil CB(1)R in the vagus nerve as a key component underlying normal gastroi

97 omyography input and use it to stimulate the vagus nerve at specific time points of the respiratory c

98 ion that targets the auricular branch of the vagus nerve at the cymba conchae of the ear.

99 hat low-level, transcutaneous stimulation of vagus nerve at the tragus (LLTS) reduces cardiac inflamm

100 ermine whether electrical stimulation of the vagus nerve attenuates kidney ischemia-reperfusion injur

101 cal stimulation at the earlobe (far from the vagus nerve branch) to control for somatosensory stimula

102 Furthermore neuromodulation of the vagus nerve can be used in the treatment of obesity.

103 A prototypical vagus nerve circuit, the inflammatory reflex, inhibits t

104 Thus, the vagus nerve contains intermingled sensory neurons consti

105 The vagus nerve contains primary visceral afferents that con

106 The vagus nerve controls inflammation in healthy, but not in

107 ays; we show that, among these pathways, the vagus nerve conveys stomach-distension signals to PB(Pdy

108 tumor necrosis factor effect of the efferent vagus nerve could be a therapeutic target in IBD through

109 ugh it is known that peripheral axons of the vagus nerve degenerate and then regenerate to a limited

110 genetic versus electrical stimulation of the vagus nerve displays different temporal characteristics

111 regulates leukocyte trafficking because the vagus nerve does not innervate endothelial cells.

112 s well as the pathophysiological outcomes of vagus nerve dysfunction resulting in obesity, mood disor

113 An additional mechanism regulating vagus nerve effects on the pancreas involves Group II an

114 ell as rats, posttraining stimulation of the vagus nerve enhances memory consolidation.

115 ons or electrical activation of the cervical vagus nerve evokes action potentials in the splenic nerv

116 Efferent vagus nerve fibers terminating in the celiac-superior me

117 velop future pharmacotherapy targeted to the vagus nerve for the treatment of obesity are proposed.

118 e demonstrated the importance of splenic and vagus nerve functions in the inflammatory process throug

119 The vagus nerve has been implicated in the regulation of sev

120 The vagus nerve has emerged as an important modulator of the

121 We screened the vagus nerve in 162 sporadic and 30 genetic CJD cases.

122 e-associated prion protein (PrP(Sc) ) in the vagus nerve in different forms and molecular subtypes of

123 review highlights evidence for a role of the vagus nerve in the development of obesity and how target

124 Stimulation of the vagus nerve in the so-called cholinergic antiinflammator

125 einforcing, revealing a role for the hepatic vagus nerve in transforming sugar sensing by the gut int

126 Stimulation of the vagus nerve increases fibrogenic cytokines in humans, th

127 cal blockade and surgical transection of the vagus nerve inhibit vagus nerve-evoked splenic nerve res

128 The vagus nerve innervates the gut and can detect diverse in

129 The vagus nerve innervates visceral organs and may contribut

130 The vagus nerve innervating the gut plays an important role

131 The vagus nerve is a key body-brain connection that monitors

132 The vagus nerve is a major conduit between lung and brain re

133 conclusion there is strong evidence that the vagus nerve is involved in the development of obesity an

134 To what extent the vagus nerve is of importance in Th2 inflammatory respons

135 Ch activation of nicotinic receptors via the vagus nerve is the primary mediator of severe hypoglycem

136 It indicated that the vagus nerve may influence PF by enhancing fibrogenic fac

137 The therapeutic potential of vagus nerve modulation to attenuate or reverse these pat

138 ing, whereas optogenetic stimulation of left vagus nerve neurons significantly increases VTA dopamine

139 o 16 recording carbon fibers in the cervical vagus nerve of 22 isoflurane-anesthetized rats.

140 Aplysia californica, and in axons within the vagus nerve of a mammal, the musk shrew Suncus murinus.

141 st the relevance of CB(1)R in neurons of the vagus nerve on metabolic homeostasis and gastrointestina

142 s suggest an anti-fibrillatory action of the vagus nerve on the ventricle, although the exact mechani

143 Treatment with vagus nerve or sham stimulation was administered concurr

144 The vagus nerve projects directly to the HSD2 neurons and th

145 In particular, the vagus nerve provides the parasympathetic innervation to

146 Thus, action potentials originating in the vagus nerve regulate T cells, which in turn produce the

147 These results indicate that the vagus nerve regulates both netrin-1 and pro-resolving li

148 n-initiated peritoneal inflammation that the vagus nerve regulates local expression of netrin-1, an a

149 Recent studies have implicated vagus nerve regulation of splenic cholinergic nicotinic

150 injections of both CTb and IB4 into the same vagus nerve resulted in labeling of two exclusive popula

151 Extracellular recordings in the isolated vagus nerve revealed that the conduction of action poten

152 We have previously shown that efferent vagus nerve signals regulate cytokine production through

153 the "inflammatory reflex," is dependent upon vagus nerve signals that inhibit cytokine production and

154 ermore, lesions of the hepatic branch of the vagus nerve significantly impair postingestive-dependent

155 Here, we use brief bursts of closed-loop vagus nerve stimulation (CL-VNS) delivered during rehabi

156 ement anti-inflammatory therapy via cervical vagus nerve stimulation (cVNS) one should selectively ac

157 We hypothesize that left-sided low-level vagus nerve stimulation (LL-VNS) can suppress sympatheti

158 Non-invasive vagus nerve stimulation (nVNS), single-transcranial magn

159 ared the effects of transcutaneous auricular vagus nerve stimulation (taVNS, Cerbomed Nemos) with sha

160 Transcutaneous vagus nerve stimulation (tVNS) has been proposed to stim

161 Vagus nerve stimulation (up to four times daily) in RA p

162 Specifically, the authors demonstrate that vagus nerve stimulation (VNS) activates the cholinergic

163 ctive of this study was to determine whether vagus nerve stimulation (VNS) can enhance the consolidat

164 Vagus nerve stimulation (VNS) has been shown to enhance

165 Vagus nerve stimulation (VNS) has been shown to exert ca

166 NCE STATEMENT Recent studies have implicated vagus nerve stimulation (VNS) in enhanced learning and m

167 Vagus nerve stimulation (VNS) is a bioelectronic therapy

168 Vagus nerve stimulation (VNS) is a common treatment for

169 ABSTRACT: Vagus nerve stimulation (VNS) is an emerging therapy for

170 Noninvasive, transcutaneous vagus nerve stimulation (VNS) is currently used as a tre

171 e immunity, and modulation of this reflex by vagus nerve stimulation (VNS) is effective in various in

172 Vagus nerve stimulation (VNS) is widely used to treat dr

173 sought to investigate the effect of cervical vagus nerve stimulation (VNS) on cerebral blood flow (CB

174 he pilot study was to evaluate the effect of Vagus Nerve Stimulation (VNS) paired with sounds in chro

175 loped a novel strategy that uses closed-loop vagus nerve stimulation (VNS) paired with tactile rehabi

176 Recent research has shown that vagus nerve stimulation (VNS) paired with tones or with

177 eviously shown the safety and feasibility of vagus nerve stimulation (VNS) paired with upper-limb reh

178 evoked cardiac response to bipolar cervical vagus nerve stimulation (VNS) reflects a dynamic interac

179 Vagus nerve stimulation (VNS) therapy was shown to impro

180 ion Registry investigated whether adjunctive vagus nerve stimulation (VNS) with treatment as usual in

181 investigate the effect of vagotomy (VGX) and vagus nerve stimulation (VNS), on the development and se

182 Only vagus nerve stimulation (VNS), which continues to develo

183 ndotoxin to the same levels as implant-based vagus nerve stimulation (VNS).

184 The anti-inflammatory effects of vagus nerve stimulation are well known.

185 animals, could help to explain the effect of vagus nerve stimulation as a treatment for headache diso

186 Vagus nerve stimulation can ameliorate autoimmune diseas

187 epeatedly pairing tones with brief pulses of vagus nerve stimulation completely eliminated the physio

188 Left-sided low-level vagus nerve stimulation did not change vagal nerve activ

189 ntiinflammatory pathway." Here, we show that vagus nerve stimulation during endotoxemia specifically

190 Vagus nerve stimulation fails to control serum TNF level

191 s: 1) sepsis group (eight pigs), 2) sepsis + vagus nerve stimulation group (nine pigs), and 3) contro

192 Axial stretch and vagus nerve stimulation induced cranial displacement of

193 This suggests that vagus nerve stimulation might provide a significant ther

194 vidence that the anti-fibrillatory effect of vagus nerve stimulation on the ventricle is mediated by

195 Vagus nerve stimulation paired with exposure to conditio

196 Vagus nerve stimulation partially or completely prevente

197 It is unclear how vagus nerve stimulation regulates leukocyte trafficking

198 Vagus nerve stimulation remains a promising, yet unprove

199 Similarly, vagus nerve stimulation significantly attenuates neutrop

200 Left-sided low-level vagus nerve stimulation suppresses stellate ganglion ner

201 Together, these results establish that vagus nerve stimulation targeting the inflammatory refle

202 hase studies have supported potential use of vagus nerve stimulation to deliver autonomic regulation

203 nt large animal model of progressive sepsis, vagus nerve stimulation was associated with a number of

204 therapies are available, including surgery, vagus nerve stimulation, and deep brain stimulation.

205 mulation, (3) surgical approaches, including vagus nerve stimulation, epidural electrical stimulation

206 timulation frequency-dependent manner during vagus nerve stimulation, with comparable increases seen

207 red for inhibition of cytokine production by vagus nerve stimulation.

208 r requirements significantly decreased after vagus nerve stimulation.

209 ugh neurostimulation techniques, such as the vagus nerve stimulator.

210 The vagus nerve supplies low-threshold chemo- and mechanosen

211 em dorsal motor nucleus (DMN) project in the vagus nerve to communicate with the lungs, liver, gastro

212 ction or injury travels through the afferent vagus nerve to integrative regions in the brainstem, and

213 AchR) has been shown to be necessary for the vagus nerve to modulate the systemic inflammatory respon

214 Recent observations linking the vagus nerve to plasticity in the central nervous system

215 other to provide rhythmic motor drive to the vagus nerve to slow the heart.

216 ti-inflammatory pathway that consists of the vagus nerve to spleen circuit, which has been stimulated

217 by anorexigenic hormones, which act via the vagus nerve to stimulate feeding, are unknown.

218 Electrical stimulation of the cervical vagus nerve was initiated 6 hours after the induction of

219 s to exert axial stretch on the LES, and the vagus nerve was isolated in the neck for electrical stim

220 Human vagus nerve was used to confirm key observations in anim

221 perexcitability in large myelinated axons in vagus nerve which could contribute to autonomic dysfunct

222 ctively transected the hepatic branch of the vagus nerve while infusing resveratrol centrally.

223 development of obesity and how targeting the vagus nerve with neuromodulation or pharmacology can be

224 iginating in the dorsal motor nucleus of the vagus nerve, and the second postganglionic, originating

225 cants that affect the brain-gut axis via the vagus nerve, and then travel to higher centers, compromi

226 nflammatory potential of the parasympathetic vagus nerve, and they represent a potential pharmacologi

227 Afferent drive in cough is carried by the vagus nerve, and vagal afferent nerve terminals have bee

228 ependent of GLP-1 receptors (GLP-1Rs) in the vagus nerve, area postrema, and paraventricular nucleus.

229 IRF also caused vagus nerve, esophageal, and lung injury while PFA did n

230 y, a severing of the peripheral axons of the vagus nerve, has been extensively utilized to determine

231 e nucleus, medial lemniscus, pontine nuclei, vagus nerve, inferior olive, abducens nucleus, and motor

232 Vagal blockade, which inhibits the vagus nerve, results in significant weight loss.

233 nvergence of the dorsal motor nucleus of the vagus nerve, sections of the medial retropharyngeal lymp

234 Effects may be mediated via the vagus nerve, spinal cord, or neuroendocrine systems.

235 Defining the interactions among the vagus nerve, the enteric nervous system, and the intesti

236 at juxtaparanodes of myelinated axons in the vagus nerve, the primary conduit for parasympathetic inn

237 xpression in juxtaparanodes of the wild-type vagus nerve, the primary source of parasympathetic input

238 nervated by the gastroduodenal branch of the vagus nerve, the transection of which influences food in

239 vascular areas innervated by branches of the vagus nerve, whereas only cells in the carotid labyrinth

240 ervation from a nonspinal source through the vagus nerve, which innervates the distal colon as well.

241 uring sterile endotoxemia independent of the vagus nerve, without affecting innate immune cell activa

242 rallel, neural reflex circuits including the vagus nerve-based inflammatory reflex are physiological

243 Instead, L. reuteri acts in a vagus nerve-dependent manner and rescues social interact

244 gical transection of the vagus nerve inhibit vagus nerve-evoked splenic nerve responses.

245 Vagus nerve-mediated reflexes also control immune system

246 ion of cytokine production, is essential for vagus nerve-mediated regulation of neutrophil activation

247 f fundoplication restored axial stretch- and vagus nerve-stimulated LES relaxation as well as LES cra

248 Here we show that an implantable vagus nerve-stimulating device in epilepsy patients inhi

249 diated signaling to the hypothalamus via the vagus nerve.

250 olarization of guinea pig and human isolated vagus nerve.

251 brain UGN augments fecal output through the vagus nerve.

252 nucleus of the solitary tract (NTS) via the vagus nerve.

253 (LXRalpha/beta) in the nodose ganglia of the vagus nerve.

254 and cells, which are closely related to the vagus nerve.

255 on, with these effects being mediated by the vagus nerve.

256 ubunit (CTb) and isolectin B4 (IB4) into the vagus nerve.

257 roadmap for internal organ sensation by the vagus nerve.

258 by parasympathetic signals delivered by the vagus nerve.

259 ry neurons traveling to the brainstem in the vagus nerve.

260 e ganglionic structure situated alone in the vagus nerve.

261 he gut and recruiting local afferents of the vagus nerve.

262 erent parasympathetic fibres of the cervical vagus nerve.

263 ount of direct viscerosensory input from the vagus nerve.

264 using particularly on the involvement of the vagus nerve.

265 uterine afferent innervation provided by the vagus nerve.

266 y could spread from the gut to brain via the vagus nerve.

267 while fat and satiation signals require the vagus nerve.

268 mission onto neurons, including those of the vagus nerve.

269 eneral visceral information largely from the vagus nerve.

270 fibrillation ablation on the function of the vagus nerve/UGI system.

271 DAF-2 DA) during stimulation of the cervical vagus nerves and acetylcholine perfusion in the absence

272 rphology of the facial, glossopharyngeal and vagus nerves are abnormal in Spry1-/-;Spry2-/- embryos.

273 Damage to vagus nerves as well as evidence of esophagus dilation o

274 , phrenic (PN), hypoglossal (HN) and central vagus nerves from neonatal and juvenile rats in situ.

275 ed A- and Adelta-axons from excised cervical vagus nerves of young adult Kcna1-null mice and age-matc

276 The vagus nerves provide much of the capsaicin-sensitive noc

277 matory mechanism mediated by the sciatic and vagus nerves that modulates the production of catecholam

278 After the C-fibre conduction in both vagus nerves was blocked, right-atrial injection of caps

279 Electrical stimulation of the right and left vagus nerves was initiated 30min after the induction of

280 tension events from the glossopharyngeal and vagus nerves, respectively.

281 acial nerve and not the glossopharyngeal and vagus nerves, suggesting that the facial nerve is most s

282 rfusion or acidification are mediated by the vagus nerves.

283 e stimulation of the left and right cervical vagus nerves.

284 e autonomic nerves: the glossopharyngeal and vagus nerves.

285 n neurons located in the dorsal motor of the vagus or indirectly by acting on pancreatic stellate cel

286 In addition, unilateral lesion of the vagus reduced Fos expression in the ipsilateral nucleus

287 rase in the CPEB2 KO dorsal motor nucleus of vagus resulted in hyperactivation of parasympathetic sig

288 This study shows that the trigeminal- and vagus systems interconnect anatomically at the level of

289 n neurons in the dorsal motor nucleus of the vagus that project to the stomach.

290 roscopy, we observed a sparse input from the vagus to most HSD2 neurons.

291 The course of the vagus tract in the rostral medulla was demonstrated in t

292 minority of vagus fibers separated from the vagus trajectory and joined the trigeminal spinal nucleu

293 n's disease, the dorsal motor nucleus of the vagus undergoes severe degeneration and pathological alp

294 ostrema, and the dorsal motor nucleus of the vagus), ventrolateral periaqueductal gray, dorsal parabr

295 e left stellate ganglion (SNA), left cardiac vagus (VNA), and arterial blood pressure.

296 Lastly, the vagus was de-afferented with capsaicin, and 5HT3-recepto

297 ansmission in the KO dorsal motor nucleus of vagus was overactivated because KO mice lack CPEB2-suppr

298 r neurons in the dorsal motor nucleus of the vagus were found only in mice.

299 stimulation of the abdominal branches of the vagus, which innervates portions of the female reproduct

300 Anterograde tracing from the vagus with CTb or IB4 was combined with immunohistochemi