ライフサイエンスコーパス: vagus nerve

1 nucleus of the solitary tract (NTS) via the vagus nerve.

2 (LXRalpha/beta) in the nodose ganglia of the vagus nerve.

3 and cells, which are closely related to the vagus nerve.

4 on, with these effects being mediated by the vagus nerve.

5 ubunit (CTb) and isolectin B4 (IB4) into the vagus nerve.

6 roadmap for internal organ sensation by the vagus nerve.

7 by parasympathetic signals delivered by the vagus nerve.

8 ry neurons traveling to the brainstem in the vagus nerve.

9 e ganglionic structure situated alone in the vagus nerve.

10 he gut and recruiting local afferents of the vagus nerve.

11 erent parasympathetic fibres of the cervical vagus nerve.

12 ount of direct viscerosensory input from the vagus nerve.

13 ytokines in sepsis through activation of the vagus nerve.

14 iple functions, including stimulation of the vagus nerve.

15 of ghrelin is mediated by activation of the vagus nerve.

16 ting through hepatic branch afferents of the vagus nerve.

17 n is regulated by neural signals through the vagus nerve.

18 d wired to the spleen via this branch of the vagus nerve.

19 using particularly on the involvement of the vagus nerve.

20 nt signaling at the central processes of the vagus nerve.

21 e brain from visceral regions is through the vagus nerve.

22 inimum onset latency from stimulation of the vagus nerve.

23 y could spread from the gut to brain via the vagus nerve.

24 uterine afferent innervation provided by the vagus nerve.

25 while fat and satiation signals require the vagus nerve.

26 eneral visceral information largely from the vagus nerve.

27 diated signaling to the hypothalamus via the vagus nerve.

28 olarization of guinea pig and human isolated vagus nerve.

29 mission onto neurons, including those of the vagus nerve.

30 brain UGN augments fecal output through the vagus nerve.

31 rfusion or acidification are mediated by the vagus nerves.

32 e stimulation of the left and right cervical vagus nerves.

33 , glossopharyngeus, and pharyngeal branch of vagus nerves.

34 e autonomic nerves: the glossopharyngeal and vagus nerves.

35 the proximal end of the transected cervical vagus nerve (3 pulses, 100 Hz, 1 ms, 100-500 muA).

36 em regulates innate immune responses via the vagus nerve, a mechanism termed the cholinergic antiinfl

37 iorates gut barrier dysfunction in sepsis by vagus nerve activation via central ghrelin receptors.

38 -inflammatory influences but also may reduce vagus nerve activity.

39 trally administered methoctramine stimulated vagus-nerve activity measured by changes in instantaneou

40 Here, we identify two populations of mouse vagus nerve afferents (P2ry1, Npy2r), each a few hundred

41 eus (Pf) in monkeys is strongly activated by vagus nerve afferents.

42 ulating the rostral cut ends of the cervical vagus nerves also evoked a sympathetic adrenergic nerve-

43 Thus, PrP(Sc) in CJD affects the vagus nerve analogously to alpha-synuclein in Parkinson

44 or in 12 dogs to stimulate the left cervical vagus nerve and a radiotransmitter for continuous record

45 ishes the anti-inflammatory potential of the vagus nerve and beta2-agonists to control inflammation i

46 application of PBG potentiated responses to vagus nerve and cardiopulmonary afferent stimulation, an

47 This study indicates that vagus nerve and cholinergic agonists activate the sympat

48 Likewise, vagus nerve and cholinergic agonists fail to control inf

49 blish the anti-inflammatory potential of the vagus nerve and cholinergic agonists in immunocompromise

50 Thus, vagus nerve and cholinergic agonists inhibit systemic in

51 the anti-inflammatory potential of both the vagus nerve and cholinergic agonists, and abrogate their

52 n, and stimulation both indirectly using the vagus nerve and directly using various intracranial targ

53 e (ACh) is the major neurotransmitter of the vagus nerve and exerts its parasympathetic actions via a

54 a neural mechanism that is controlled by the vagus nerve and inhibits local cytokine release, thereby

55 region receives autonomic afferents via the vagus nerve and is an important site of AngII actions.

56 tiation during normal drinking, involves the vagus nerve and is transmitted to key forebrain neurons

57 n anti-inflammatory reflex arc involving the vagus nerve and memory T cells is necessary for resoluti

58 increased responses were observed in ex vivo vagus nerve and neuron cell bodies in the vagal ganglia.

59 c mechanism that involves recruitment of the vagus nerve and subsequent activation of endocannabinoid

60 many types of neuroimmune circuits, with the vagus nerve and sympathetic innervation of numerous orga

61 core the anti-inflammatory properties of the vagus nerve and the potential of neuro-immune interactio

62 they also initiate activity in the afferent vagus nerve and thereby signal directly to the brainstem

63 was abolished by surgical transection of the vagus nerve and was not observed in other peripheral org

64 DAF-2 DA) during stimulation of the cervical vagus nerves and acetylcholine perfusion in the absence

65 (end-tidal CO(2), 4.3 +/- 0.2%) with intact vagus nerves and three vagotomized cats, AP-SND phase wa

66 stem of growth and metabolism acting via the vagus nerve, and is implicated in a variety of altered e

67 solitary tract, dorsal motor nucleus of the vagus nerve, and salivatory nuclei), gustatory system (r

68 iginating in the dorsal motor nucleus of the vagus nerve, and the second postganglionic, originating

69 cants that affect the brain-gut axis via the vagus nerve, and then travel to higher centers, compromi

70 nflammatory potential of the parasympathetic vagus nerve, and they represent a potential pharmacologi

71 Afferent drive in cough is carried by the vagus nerve, and vagal afferent nerve terminals have bee

72 rphology of the facial, glossopharyngeal and vagus nerves are abnormal in Spry1-/-;Spry2-/- embryos.

73 l pathways, with or without mediation by the vagus nerve, are known to participate in regulation of e

74 ependent of GLP-1 receptors (GLP-1Rs) in the vagus nerve, area postrema, and paraventricular nucleus.

75 These results unveil CB(1)R in the vagus nerve as a key component underlying normal gastroi

76 Damage to vagus nerves as well as evidence of esophagus dilation o

77 omyography input and use it to stimulate the vagus nerve at specific time points of the respiratory c

78 ion that targets the auricular branch of the vagus nerve at the cymba conchae of the ear.

79 hat low-level, transcutaneous stimulation of vagus nerve at the tragus (LLTS) reduces cardiac inflamm

80 ermine whether electrical stimulation of the vagus nerve attenuates kidney ischemia-reperfusion injur

81 rallel, neural reflex circuits including the vagus nerve-based inflammatory reflex are physiological

82 cal stimulation at the earlobe (far from the vagus nerve branch) to control for somatosensory stimula

83 blockade of the afferent transmission in the vagus nerve by topical application of local anaesthetic.

84 Furthermore neuromodulation of the vagus nerve can be used in the treatment of obesity.

85 us system, via an inflammatory reflex of the vagus nerve, can inhibit cytokine release and thereby pr

86 A prototypical vagus nerve circuit, the inflammatory reflex, inhibits t

87 1)R in afferent and efferent branches of the vagus nerve (Cnr1(flox/flox); Phox2b-Cre mice).

88 The vagus nerve contains afferent fibers that transmit senso

89 Thus, the vagus nerve contains intermingled sensory neurons consti

90 The vagus nerve contains primary visceral afferents that con

91 The vagus nerve controls inflammation in healthy, but not in

92 ays; we show that, among these pathways, the vagus nerve conveys stomach-distension signals to PB(Pdy

93 esponse to VCMS, providing evidence that the vagus nerve conveys VCMS directly from the genitalia to

94 tumor necrosis factor effect of the efferent vagus nerve could be a therapeutic target in IBD through

95 Efferent activation of the cervical vagus nerve (cVN) dampens systemic inflammatory processe

96 ugh it is known that peripheral axons of the vagus nerve degenerate and then regenerate to a limited

97 Instead, L. reuteri acts in a vagus nerve-dependent manner and rescues social interact

98 genetic versus electrical stimulation of the vagus nerve displays different temporal characteristics

99 ry tract (mNTS), dorsal motor nucleus of the vagus nerve (DMV) and the hypoglossal nucleus (HN).

100 tarius (NTS) and dorsal motor nucleus of the vagus nerve (DMV) constitute sensory and motor nuclei of

101 nsmission in the dorsal motor nucleus of the vagus nerve (DMV).

102 n subdiaphragmatically vagotomized rats, the vagus nerve does not appear to be the only route by whic

103 regulates leukocyte trafficking because the vagus nerve does not innervate endothelial cells.

104 s well as the pathophysiological outcomes of vagus nerve dysfunction resulting in obesity, mood disor

105 An additional mechanism regulating vagus nerve effects on the pancreas involves Group II an

106 ell as rats, posttraining stimulation of the vagus nerve enhances memory consolidation.

107 IRF also caused vagus nerve, esophageal, and lung injury while PFA did n

108 ical stimulation of the central stump of the vagus nerve (ESV) transected at the spinal cervical leve

109 gical transection of the vagus nerve inhibit vagus nerve-evoked splenic nerve responses.

110 ons or electrical activation of the cervical vagus nerve evokes action potentials in the splenic nerv

111 Efferent vagus nerve fibers terminating in the celiac-superior me

112 velop future pharmacotherapy targeted to the vagus nerve for the treatment of obesity are proposed.

113 The vagus nerve forms a neuro-anatomical link between the ga

114 , phrenic (PN), hypoglossal (HN) and central vagus nerves from neonatal and juvenile rats in situ.

115 e demonstrated the importance of splenic and vagus nerve functions in the inflammatory process throug

116 The vagus nerve has been implicated in the regulation of sev

117 The vagus nerve has emerged as an important modulator of the

118 y, a severing of the peripheral axons of the vagus nerve, has been extensively utilized to determine

119 We screened the vagus nerve in 162 sporadic and 30 genetic CJD cases.

120 e-associated prion protein (PrP(Sc) ) in the vagus nerve in different forms and molecular subtypes of

121 These findings corroborate the role of the vagus nerve in regulating hippocampal activity and the i

122 review highlights evidence for a role of the vagus nerve in the development of obesity and how target

123 nce of the action of CCK at the level of the vagus nerve in the regulation of food intake, body weigh

124 Stimulation of the vagus nerve in the so-called cholinergic antiinflammator

125 einforcing, revealing a role for the hepatic vagus nerve in transforming sugar sensing by the gut int

126 Stimulation of the vagus nerve increases fibrogenic cytokines in humans, th

127 olish antiinflammatory signaling through the vagus nerve, indicating that peripheral muscarinic recep

128 e nucleus, medial lemniscus, pontine nuclei, vagus nerve, inferior olive, abducens nucleus, and motor

129 cal blockade and surgical transection of the vagus nerve inhibit vagus nerve-evoked splenic nerve res

130 The efferent vagus nerve inhibits cytokine release through alpha7-nic

131 The vagus nerve innervates the gut and can detect diverse in

132 The vagus nerve innervates visceral organs and may contribut

133 The vagus nerve innervating the gut plays an important role

134 The vagus nerve is a key body-brain connection that monitors

135 The vagus nerve is a major conduit between lung and brain re

136 The common hepatic branch of the vagus nerve is a two-way highway of communication betwee

137 The vagus nerve is an important source of afferent informati

138 conclusion there is strong evidence that the vagus nerve is involved in the development of obesity an

139 To what extent the vagus nerve is of importance in Th2 inflammatory respons

140 Ch activation of nicotinic receptors via the vagus nerve is the primary mediator of severe hypoglycem

141 The vagus nerve is thought to participate in signal transduc

142 gical resection of the hepatic branch of the vagus nerve led to a marked increase in liver glucose pr

143 ients by closed-loop stimulation of the left vagus nerve (LVN).

144 It indicated that the vagus nerve may influence PF by enhancing fibrogenic fac

145 Pharmacologic stimulation of the vagus nerve may offer a novel approach of anti-sepsis th

146 Vagus nerve-mediated reflexes also control immune system

147 ion of cytokine production, is essential for vagus nerve-mediated regulation of neutrophil activation

148 The therapeutic potential of vagus nerve modulation to attenuate or reverse these pat

149 gical resection of the hepatic branch of the vagus nerve, negates the effects of central insulin and

150 ing, whereas optogenetic stimulation of left vagus nerve neurons significantly increases VTA dopamine

151 o 16 recording carbon fibers in the cervical vagus nerve of 22 isoflurane-anesthetized rats.

152 Aplysia californica, and in axons within the vagus nerve of a mammal, the musk shrew Suncus murinus.

153 ed A- and Adelta-axons from excised cervical vagus nerves of young adult Kcna1-null mice and age-matc

154 the effects of electrical stimulation of the vagus nerve on learning and memory, mood, seizure suppre

155 st the relevance of CB(1)R in neurons of the vagus nerve on metabolic homeostasis and gastrointestina

156 fect of a unilateral section of the cervical vagus nerve on the distribution of P2X(1), P2X(2), P2X(3

157 s suggest an anti-fibrillatory action of the vagus nerve on the ventricle, although the exact mechani

158 Treatment with vagus nerve or sham stimulation was administered concurr

159 We therefore postulate that the vagus nerve participates in informing the brain about tu

160 Stimulation of the vagus nerve prevents the damaging effects of cytokine re

161 teral electrical stimulation of the cervical vagus nerve produced bradypnoea by exclusively extending

162 egion of the medulla oblongata, to which the Vagus nerves project, is activated by vaginal-cervical s

163 The vagus nerve projects directly to the HSD2 neurons and th

164 We conclude that the Vagus nerves provide a spinal cord-bypass pathway for va

165 The vagus nerves provide much of the capsaicin-sensitive noc

166 In particular, the vagus nerve provides the parasympathetic innervation to

167 Transection of the vagus nerve proximal to the ganglion results in loss of

168 Thus, action potentials originating in the vagus nerve regulate T cells, which in turn produce the

169 These results indicate that the vagus nerve regulates both netrin-1 and pro-resolving li

170 n-initiated peritoneal inflammation that the vagus nerve regulates local expression of netrin-1, an a

171 Recent studies have implicated vagus nerve regulation of splenic cholinergic nicotinic

172 tension events from the glossopharyngeal and vagus nerves, respectively.

173 injections of both CTb and IB4 into the same vagus nerve resulted in labeling of two exclusive popula

174 Vagal blockade, which inhibits the vagus nerve, results in significant weight loss.

175 Extracellular recordings in the isolated vagus nerve revealed that the conduction of action poten

176 In rats with intact vagus nerves, RTN neurons were inhibited, also to a vari

177 nvergence of the dorsal motor nucleus of the vagus nerve, sections of the medial retropharyngeal lymp

178 rate variability (markers of low cholinergic/vagus nerve sensitivity).

179 We have previously shown that efferent vagus nerve signals regulate cytokine production through

180 the "inflammatory reflex," is dependent upon vagus nerve signals that inhibit cytokine production and

181 ated (cholinergic) receptor that responds to vagus nerve signals was previously unknown.

182 ermore, lesions of the hepatic branch of the vagus nerve significantly impair postingestive-dependent

183 fluid percussion injury (FPI), FPI with sham Vagus Nerve Simulation (VNS), and FPI with chronic inter

184 Effects may be mediated via the vagus nerve, spinal cord, or neuroendocrine systems.

185 f fundoplication restored axial stretch- and vagus nerve-stimulated LES relaxation as well as LES cra

186 Here we show that an implantable vagus nerve-stimulating device in epilepsy patients inhi

187 Here, we use brief bursts of closed-loop vagus nerve stimulation (CL-VNS) delivered during rehabi

188 ement anti-inflammatory therapy via cervical vagus nerve stimulation (cVNS) one should selectively ac

189 We hypothesize that left-sided low-level vagus nerve stimulation (LL-VNS) can suppress sympatheti

190 Non-invasive vagus nerve stimulation (nVNS), single-transcranial magn

191 ared the effects of transcutaneous auricular vagus nerve stimulation (taVNS, Cerbomed Nemos) with sha

192 Transcutaneous vagus nerve stimulation (tVNS) has been proposed to stim

193 Vagus nerve stimulation (up to four times daily) in RA p

194 Specifically, the authors demonstrate that vagus nerve stimulation (VNS) activates the cholinergic

195 The hypothesis that left vagus nerve stimulation (VNS) at the cervical level resu

196 ctive of this study was to determine whether vagus nerve stimulation (VNS) can enhance the consolidat

197 Vagus nerve stimulation (VNS) has been shown to enhance

198 Vagus nerve stimulation (VNS) has been shown to exert ca

199 NCE STATEMENT Recent studies have implicated vagus nerve stimulation (VNS) in enhanced learning and m

200 Vagus nerve stimulation (VNS) is a bioelectronic therapy

201 Vagus nerve stimulation (VNS) is a common treatment for

202 ABSTRACT: Vagus nerve stimulation (VNS) is an emerging therapy for

203 Noninvasive, transcutaneous vagus nerve stimulation (VNS) is currently used as a tre

204 e immunity, and modulation of this reflex by vagus nerve stimulation (VNS) is effective in various in

205 Vagus nerve stimulation (VNS) is widely used to treat dr

206 sought to investigate the effect of cervical vagus nerve stimulation (VNS) on cerebral blood flow (CB

207 he pilot study was to evaluate the effect of Vagus Nerve Stimulation (VNS) paired with sounds in chro

208 loped a novel strategy that uses closed-loop vagus nerve stimulation (VNS) paired with tactile rehabi

209 Recent research has shown that vagus nerve stimulation (VNS) paired with tones or with

210 eviously shown the safety and feasibility of vagus nerve stimulation (VNS) paired with upper-limb reh

211 We have previously shown that direct vagus nerve stimulation (VNS) reduces the slope of actio

212 evoked cardiac response to bipolar cervical vagus nerve stimulation (VNS) reflects a dynamic interac

213 Vagus nerve stimulation (VNS) therapy was shown to impro

214 ion Registry investigated whether adjunctive vagus nerve stimulation (VNS) with treatment as usual in

215 We hypothesized that electrical vagus nerve stimulation (VNS) would suppress harmaline t

216 investigate the effect of vagotomy (VGX) and vagus nerve stimulation (VNS), on the development and se

217 Only vagus nerve stimulation (VNS), which continues to develo

218 ndotoxin to the same levels as implant-based vagus nerve stimulation (VNS).

219 , with the carrageenan air pouch model, both vagus nerve stimulation and cholinergic agonists signifi

220 eloped a method of transcutaneous mechanical vagus nerve stimulation and then investigated whether th

221 erve stimulation (SNS) and right and/or left vagus nerve stimulation and was compared with DOR during

222 receive electrical, transcutaneous, or sham vagus nerve stimulation and were followed for survival o

223 The anti-inflammatory effects of vagus nerve stimulation are well known.

224 animals, could help to explain the effect of vagus nerve stimulation as a treatment for headache diso

225 Vagus nerve stimulation can ameliorate autoimmune diseas

226 epeatedly pairing tones with brief pulses of vagus nerve stimulation completely eliminated the physio

227 Left-sided low-level vagus nerve stimulation did not change vagal nerve activ

228 Transcutaneous vagus nerve stimulation dose-dependently reduced systemi

229 ntiinflammatory pathway." Here, we show that vagus nerve stimulation during endotoxemia specifically

230 Vagus nerve stimulation fails to control serum TNF level

231 Here we show that vagus nerve stimulation fails to inhibit tumor necrosis

232 US Food and Drug Administration approval of vagus nerve stimulation for recurrent treatment-resistan

233 s: 1) sepsis group (eight pigs), 2) sepsis + vagus nerve stimulation group (nine pigs), and 3) contro

234 hange in the direction of DOR, but bilateral vagus nerve stimulation increased and reversed DOR to ba

235 Axial stretch and vagus nerve stimulation induced cranial displacement of

236 Treatment with transcutaneous vagus nerve stimulation inhibited HMGB1 levels and impro

237 Electrical vagus nerve stimulation inhibits proinflammatory cytokin

238 Vagus nerve stimulation inhibits proinflammatory cytokin

239 Transcutaneous vagus nerve stimulation is an efficacious treatment for

240 This suggests that vagus nerve stimulation might provide a significant ther

241 vidence that the anti-fibrillatory effect of vagus nerve stimulation on the ventricle is mediated by

242 t may be exploited through either electrical vagus nerve stimulation or administration of alpha7 agon

243 Vagus nerve stimulation paired with exposure to conditio

244 Vagus nerve stimulation partially or completely prevente

245 It is unclear how vagus nerve stimulation regulates leukocyte trafficking

246 Vagus nerve stimulation remains a promising, yet unprove

247 Vagus nerve stimulation significantly (P<0.01) decreased

248 Similarly, vagus nerve stimulation significantly attenuates neutrop

249 Cervical vagus nerve stimulation suppressed power in the 8-12-Hz

250 Left-sided low-level vagus nerve stimulation suppresses stellate ganglion ner

251 Together, these results establish that vagus nerve stimulation targeting the inflammatory refle

252 hase studies have supported potential use of vagus nerve stimulation to deliver autonomic regulation

253 nt large animal model of progressive sepsis, vagus nerve stimulation was associated with a number of

254 te that bilateral autonomic activity (SNS or vagus nerve stimulation) cause reversible shifts of apex

255 therapies are available, including surgery, vagus nerve stimulation, and deep brain stimulation.

256 mulation, (3) surgical approaches, including vagus nerve stimulation, epidural electrical stimulation

257 n of nicotine, an alpha7 agonist that mimics vagus nerve stimulation, increases proinflammatory cytok

258 on celiac nerve abolishes TNF suppression by vagus nerve stimulation, suggesting that the cholinergic

259 During right or left vagus nerve stimulation, there was no change in the dire

260 timulation frequency-dependent manner during vagus nerve stimulation, with comparable increases seen

261 red for inhibition of cytokine production by vagus nerve stimulation.

262 r functional inhibition of TNF production by vagus nerve stimulation.

263 r requirements significantly decreased after vagus nerve stimulation.

264 Vagus-nerve stimulation (VNS) is licensed in several cou

265 ugh neurostimulation techniques, such as the vagus nerve stimulator.

266 a region which is densely innervated by the vagus nerve, suggesting that they could receive direct v

267 acial nerve and not the glossopharyngeal and vagus nerves, suggesting that the facial nerve is most s

268 nerve depolarization of guinea-pig and human vagus nerve suggestive of an inhibitory effect on affere

269 The vagus nerve supplies low-threshold chemo- and mechanosen

270 We conclude that the vagus nerves supply the guinea-pig oesophagus with nocic

271 ophagus, we show that in the guinea-pig, the vagus nerves supply the oesophagus with a large populati

272 matory mechanism mediated by the sciatic and vagus nerves that modulates the production of catecholam

273 Second, in three cats with intact vagus nerves that were hyperventilated (end tidal CO(2),

274 Defining the interactions among the vagus nerve, the enteric nervous system, and the intesti

275 at juxtaparanodes of myelinated axons in the vagus nerve, the primary conduit for parasympathetic inn

276 xpression in juxtaparanodes of the wild-type vagus nerve, the primary source of parasympathetic input

277 nervated by the gastroduodenal branch of the vagus nerve, the transection of which influences food in

278 em dorsal motor nucleus (DMN) project in the vagus nerve to communicate with the lungs, liver, gastro

279 expansion via electrical stimulation of the vagus nerve to identify the brain circuits responsible f

280 ction or injury travels through the afferent vagus nerve to integrative regions in the brainstem, and

281 AchR) has been shown to be necessary for the vagus nerve to modulate the systemic inflammatory respon

282 Recent observations linking the vagus nerve to plasticity in the central nervous system

283 other to provide rhythmic motor drive to the vagus nerve to slow the heart.

284 ti-inflammatory pathway that consists of the vagus nerve to spleen circuit, which has been stimulated

285 by anorexigenic hormones, which act via the vagus nerve to stimulate feeding, are unknown.

286 fibrillation ablation on the function of the vagus nerve/UGI system.

287 to small multifunctional nerves such as the vagus nerve (VN) which is the main multimodal autonomic

288 Electrical stimulation of the cervical vagus nerve was initiated 6 hours after the induction of

289 s to exert axial stretch on the LES, and the vagus nerve was isolated in the neck for electrical stim

290 Human vagus nerve was used to confirm key observations in anim

291 After the C-fibre conduction in both vagus nerves was blocked, right-atrial injection of caps

292 Electrical stimulation of the right and left vagus nerves was initiated 30min after the induction of

293 vascular areas innervated by branches of the vagus nerve, whereas only cells in the carotid labyrinth

294 perexcitability in large myelinated axons in vagus nerve which could contribute to autonomic dysfunct

295 ervation from a nonspinal source through the vagus nerve, which innervates the distal colon as well.

296 To test whether the Vagus nerves, which bypass the spinal cord, provide the

297 ctively transected the hepatic branch of the vagus nerve while infusing resveratrol centrally.

298 development of obesity and how targeting the vagus nerve with neuromodulation or pharmacology can be

299 uring sterile endotoxemia independent of the vagus nerve, without affecting innate immune cell activa

300 their anatomical relation to branches of the vagus nerve (Xth cranial nerve).