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1 ction was masked by the prior isolation of a vancomycin-resistant enterococcus.
2 hicillin-resistant Staphylococcus aureus and vancomycin-resistant Enterococcus.
3 its parent compound, vancomycin (V), against vancomycin-resistant enterococcus.
4 gnificantly reduces fecal bacterial loads of Vancomycin-Resistant Enterococcus.
5 hicillin-resistant Staphylococcus aureus and vancomycin-resistant Enterococcus.
6 sistant Staphylococcus aureus infections and vancomycin-resistant enterococcus.
7 hicillin-resistant Staphylococcus aureus and vancomycin-resistant Enterococcus.
8 osa, 15 of 86 (17.4%; 95% CI 9.4% to 25.4%), vancomycin-resistant Enterococcus, 25 of 180 (13.9%, 95%
9 to appropriate antibiotic for patients with vancomycin-resistant Enterococcus (4.2 versus 43.7 h; P=
11 thicillin-resistant Staphylococcus aureus or vancomycin-resistant Enterococcus admission surveillance
12 hicillin-resistant Staphylococcus aureus and vancomycin-resistant Enterococcus and no increase in adv
13 methicillin-resistant Staphylococcus aureus, vancomycin-resistant Enterococcus, and carbapenem-resist
14 cus pneumoniae, beta-hemolytic streptococci, vancomycin-resistant Enterococcus, and Enterobacteriacea
15 ltidrug resistant organisms, including MRSA, vancomycin-resistant enterococcus, and Gram-negative org
16 extended spectrum beta-lactamase producers, vancomycin-resistant enterococcus, and highly-resistant
17 hylococcus aureus, Clostridioides difficile, vancomycin-resistant Enterococcus, and MDR gram-negative
18 hicillin-resistant Staphylococcus aureus and vancomycin-resistant enterococcus are inconsistent with
19 nterococcal domination increased the risk of Vancomycin-resistant Enterococcus bacteremia 9-fold, and
23 ancomycin on susceptibility to C. difficile, vancomycin-resistant Enterococcus, carbapenem-resistant
26 proof of concept, wild-type (ATCC 29212) and vancomycin-resistant Enterococcus cells were incubated a
28 methicillin-resistant Staphylococcus aureus, vancomycin-resistant enterococcus, Clostridium difficile
30 e detected, including increased incidence of vancomycin-resistant Enterococcus colonization or diseas
31 hicillin-resistant Staphylococcus aureus and vancomycin-resistant Enterococcus colonization were intr
33 relatedness, including the first known U.S. vancomycin-resistant enterococcus (E. faecalis strain V5
34 ~8-fold and 2~4-fold greater potency against vancomycin resistant Enterococcus faecalis and methicill
35 ses consisted of 1 patient with recalcitrant vancomycin-resistant Enterococcus faecalis (VRE) and 2 p
36 against vancomycin-susceptible organisms and vancomycin-resistant Enterococcus faecalis (VRE) of VanB
37 n-resistant Staphylococcus aureus (MRSA) and vancomycin-resistant Enterococcus faecalis (VRE), and an
44 A chromogenic medium for identification of vancomycin-resistant Enterococcus faecalis and Enterococ
45 investigate the changes to PG composition in vancomycin-resistant Enterococcus faecalis following the
46 n of the cell envelope of a clinical pair of vancomycin-resistant Enterococcus faecalis isolates from
47 and January 1995, we identified a cluster of vancomycin-resistant Enterococcus faecalis isolates invo
49 methicillin-resistant Staphylococcus aureus, vancomycin-resistant Enterococcus faecalis, extended-spe
50 rains carry transposon Tn1546, acquired from vancomycin-resistant Enterococcus faecalis, which is kno
55 llin-resistant Staphylococcus aureus (MRSA), vancomycin-resistant Enterococcus faecalis/faecium (VREf
58 stant S. aureus (1.3 +/- 0.4 microg/mL), and vancomycin-resistant Enterococcus faecium (MIC(50) 2.9 +
59 tbreaks involving three important pathogens: vancomycin-resistant Enterococcus faecium (n=19), methic
60 sus group (1), Streptococcus pneumoniae (6), vancomycin-resistant Enterococcus faecium (VRE FCM) (16)
61 onization of the gastrointestinal tract with vancomycin-resistant Enterococcus faecium (VRE) has beco
65 REA), and epidemiological data for typing 45 vancomycin-resistant Enterococcus faecium (VRE) isolates
68 and a clinical prediction rule to guide anti-vancomycin-resistant Enterococcus faecium (VRE) therapy.
69 can inhibit dense colonization of the gut by vancomycin-resistant Enterococcus faecium (VRE), a leadi
70 llin-resistant Staphylococcus aureus (MRSA), vancomycin-resistant Enterococcus faecium (VRE), and bet
71 llin-resistant Staphylococcus aureus (MRSA), vancomycin-resistant Enterococcus faecium (VRE), Escheri
72 n resistance can lead to marked expansion of vancomycin-resistant Enterococcus faecium (VRE), Klebsie
75 n-resistant Staphylococcus aureus (MRSA) and vancomycin-resistant Enterococcus faecium (VREF) with MI
77 inal colonization by the nosocomial pathogen vancomycin-resistant Enterococcus faecium (VREfm) can le
78 e is known about the population structure of vancomycin-resistant Enterococcus faecium (VREfm) in Lat
82 Nosocomial acquisition and transmission of vancomycin-resistant Enterococcus faecium (VREfm) is the
84 mia, 54 (19.6%) were due to rESKAPE strains (vancomycin-resistant Enterococcus faecium [0], methicill
85 Many of these resistant microbes, such as vancomycin-resistant Enterococcus faecium and carbapenem
86 medium designed to recover and differentiate vancomycin-resistant Enterococcus faecium and Enterococc
87 Gram-positive bacteria (high priority) were vancomycin-resistant Enterococcus faecium and meticillin
88 rointestinal colonization by ampicillin- and vancomycin-resistant Enterococcus faecium C68 in a mouse
89 lishment of gastrointestinal colonization by vancomycin-resistant Enterococcus faecium C68 in a mouse
90 OG1RF and against two serologically related, vancomycin-resistant Enterococcus faecium clinical isola
94 ons; lower respiratory tract infections; and vancomycin-resistant Enterococcus faecium infections, in
95 ted States in late 1999 for the treatment of vancomycin-resistant Enterococcus faecium infections.
97 an also identify isolated colonies as either vancomycin-resistant Enterococcus faecium or Enterococcu
99 methicillin-resistant Staphylococcus aureus, vancomycin-resistant Enterococcus faecium, and beta-lact
100 fied that both inhibited and dispersed MRSA, vancomycin-resistant Enterococcus faecium, and Staphyloc
101 e may find future use as antibiotics against vancomycin-resistant Enterococcus faecium, methicillin-r
109 iridae and Siphoviridae families that target Vancomycin-Resistant Enterococcus gut colonization.
110 ately controlled and powered, infection with vancomycin-resistant enterococcus has had more of an eff
111 hicillin-resistant Staphylococcus aureus and vancomycin-resistant Enterococcus in a neurocritical car
112 k we analyzed data observed on the spread of vancomycin-resistant Enterococcus in an intensive care u
113 hicillin-resistant Staphylococcus aureus and vancomycin-resistant Enterococcus in neurocritical care
114 46 has only been described in human clinical vancomycin-resistant enterococcus isolates unique to the
115 ifficile infection and dense colonization by vancomycin-resistant Enterococcus, K. pneumoniae, and E.
117 im-sulfamethoxazole-resistant MRSA (n = 10), vancomycin-resistant Enterococcus (n = 37), high-level g
118 s aureus: RRRR, 0.87; 95% CI, 0.75-1.00; and vancomycin-resistant Enterococcus: RRRR, 0.82; 95% CI, 0
119 ria monocytogenes, Bacillus anthracis, and a vancomycin-resistant Enterococcus sp. with MIC or IC50 v
120 d for detection of Staphylococcus aureus and vancomycin-resistant Enterococcus species (VRE) using a
123 ation of methicillin resistant S. aureus and vancomycin resistant Enterococcus spp. was completed an
124 llin-resistant Staphylococcus aureus (MRSA), vancomycin-resistant Enterococcus spp. (VRE), extended-s
125 methicillin-resistant Staphylococcus aureus, vancomycin-resistant Enterococcus spp. and several multi
126 y detecting and discriminating genotypes for vancomycin-resistant Enterococcus spp. in the clinical l
127 icillin-resistant Staphylococcus aureus, and vancomycin-resistant Enterococcus spp. in the environmen
128 ncept, amplification of a gene specific to a vancomycin-resistant Enterococcus strain was performed o
129 hicillin-resistant Staphylococcus aureus and vancomycin-resistant Enterococcus surveillance cultures.
131 n-resistant Staphylococcus aureus (MRSA) and vancomycin-resistant Enterococcus (VRE) among ICU patien
133 cus aureus (MRSA), Clostridium difficile, or vancomycin-resistant Enterococcus (VRE) and severity and
136 by the highly antibiotic-resistant bacterium vancomycin-resistant Enterococcus (VRE) can exceed 10(9)
137 lin azide agar plus vancomycin to screen for vancomycin-resistant enterococcus (VRE) colonization.
139 n-resistant Staphylococcus aureus (MRSA) and vancomycin-resistant enterococcus (VRE) have achieved si
140 -resistant Staphylococcus aureus (MRSA), and vancomycin-resistant Enterococcus (VRE) in patients with
145 n-resistant Staphylococcus aureus (MRSA) and vancomycin-resistant Enterococcus (VRE) is driving the d
147 ecovered from perirectal swabs collected for vancomycin-resistant Enterococcus (VRE) surveillance as
149 llin-resistant Staphylococcus aureus (MRSA), vancomycin-resistant Enterococcus (VRE), and MDR Enterob
150 erior to direct plating for the detection of vancomycin-resistant enterococcus (VRE), but vancomycin
151 llin-resistant Staphylococcus aureus (MRSA), vancomycin-resistant enterococcus (VRE), extended-spectr
152 with antibiotic-resistant bacteria, such as vancomycin-resistant Enterococcus (VRE), is a dangerous
153 ighly antibiotic-resistant bacteria, such as vancomycin-resistant Enterococcus (VRE), is a growing cl
154 nce can result in intestinal domination with vancomycin-resistant Enterococcus (VRE), leading to bloo
155 parent drug, including the ESKAPE pathogens, vancomycin-resistant Enterococcus (VRE), methicillin-res
156 llin-resistant Staphylococcus aureus (MRSA), vancomycin-resistant Enterococcus (VRE), Pseudomonas aer
157 n-resistant Staphylococcus aureus (MRSA) and vancomycin-resistant Enterococcus (VRE), while also exhi
164 thicillin-resistant Staphylococcus aureus or vancomycin-resistant Enterococcus) was constructed.