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1 and pneumonia, and an increase observed for varicella zoster.
2 prophylaxis against Pneumocystis carinii and varicella zoster.
4 varicella-zoster virus vaccine in preventing varicella-zoster and herpes zoster is well documented, a
5 bacterial and viral (due to herpes simplex, varicella zoster, and enteroviruses) meningitis/encephal
6 smosis, other infections (such as syphillis, varicella-zoster, and parvovirus B19), cytomegalovirus,
7 reaction (PCR) analysis for Herpes simplex, varicella zoster, cytomegalovirus, Epstein-Barr virus an
9 number and critical immunization coverage of varicella-zoster infection in Belgium, Italy, Poland, an
10 virus (OPV), rubella, measles, yellow fever, varicella-zoster, multivalent measles/mumps/rubella, and
11 of herpes zoster disease, which is caused by Varicella zoster Nevertheless, the pathophysiological me
12 roidism, hypercholesterolemia, hypertension, varicella zoster, peripheral vascular disease, and autoi
13 clonal expansions in response to attenuated varicella-zoster vaccination in four pairs of adult iden
14 laria vaccine and the subunit glycoprotein E varicella zoster vaccine (both currently in phase III).
16 thy, such as prior herpes simplex keratitis, varicella-zoster viral keratitis, the linear form of Thy
18 megalovirus (HR, 3.98 [95% CI, 1.40-11.26]), varicella zoster virus (HR, 1.49 [95% CI, 1.18-1.89]), h
20 s HSV1 and HSV2 (also termed HHV1 and HHV2), varicella zoster virus (VZV or HHV3), EBV (HHV4), cytome
21 tients showed a decreased ability to control varicella zoster virus (VZV) and Epstein-Barr virus (EBV
27 response biomarkers measuring antibodies to varicella zoster virus (VZV) by glycoprotein-based enzym
28 lex virus types 1 (HSV-1) and 2 (HSV-2), and varicella zoster virus (VZV) by weekly polymerase chain
29 se of herpes zoster caused by the attenuated varicella zoster virus (VZV) contained in Zostavax in a
30 , or no history of zoster (group 3) revealed varicella zoster virus (VZV) DNA in saliva samples from
31 rs who were immunized with Zostavax revealed varicella zoster virus (VZV) DNA in swabs of skin inocul
34 as an alternative to sampling of rashes for varicella zoster virus (VZV) genotyping and further char
35 s positive for herpes simplex virus (HSV) or varicella zoster virus (VZV) in 79% to 100% of cases of
36 Clinical reports observe the reactivation of varicella zoster virus (VZV) in people who have recovere
39 virus (CMV), herpes simplex virus (HSV), and varicella zoster virus (VZV) infections were monitored i
42 As) from patients with giant cell arteritis, varicella zoster virus (VZV) is seen in perineurial cell
46 s" postulates that reexposure to circulating varicella zoster virus (VZV) over the life span inhibits
51 ne responses to a high-titer live attenuated varicella zoster virus (VZV) vaccine (zoster vaccine), w
52 te the efficacy and safety of an inactivated varicella zoster virus (VZV) vaccine for herpes zoster p
53 Since the introduction of live attenuated varicella zoster virus (VZV) vaccine in 1995 there has b
55 lovirus (CMV), Epstein-Barr virus (EBV), and varicella zoster virus (VZV) was determined in crewmembe
58 portion of HZ cases caused by vaccine-strain varicella zoster virus (VZV), assessed the positive pred
59 c primers to detect DNA from JC virus (JCV), varicella zoster virus (VZV), cytomegalovirus (CMV), Eps
62 erpesviruses, herpes simplex virus (HSV) and varicella zoster virus (VZV), results in the rapid accum
63 immunogenicity of live-attenuated Oka/Merck varicella zoster virus (VZV)-containing vaccine (hereaft
65 We investigated the relationship between varicella zoster virus (VZV)-specific memory CD4(+) T ce
66 virus type 1 (HSV-1) and type 2 (HSV-2) and varicella zoster virus (VZV)-was determined in autonomic
71 CF; human cytomegalovirus (HCMV) 11% in GCF; varicella zoster virus 6% in saliva and 3% in GCF; of hu
72 ytomegalovirus [CMV], herpes simplex I/II or varicella zoster virus [HSV/VZV], blood stream infection
74 RN, PCR-positive for herpes simplex virus or varicella zoster virus and evaluated between January 200
75 genes of both herpes simplex virus (HSV) and varicella zoster virus and functions, in part, by coupli
76 beyond CMV to other herpes viruses, such as varicella zoster virus and possibly Epstein-Barr virus.
77 ld decrease external boosting of immunity to varicella zoster virus and thereby increase incidence of
78 (as determined by testing lesions swabs for varicella zoster virus DNA by polymerase chain reaction)
79 ence of confirmed varicella (by detection of varicella zoster virus DNA or epidemiological link) from
83 were randomized 1:1 to receive either HZ/su (varicella zoster virus glycoprotein E; AS01B Adjuvant Sy
84 cation) were associated with protection from varicella zoster virus infection (hazard ratio, 0.43; 95
85 contact dermatitis, infectious folliculitis, varicella zoster virus infection, fixed drug eruption, a
90 derate quality showed an association between varicella zoster virus reactivation (ophthalmic zoster)
91 Because there is no good animal model of varicella zoster virus reactivation from latency, this e
92 ion (PCR) for herpes simplex virus (HSV) and varicella zoster virus was done in 237 (69%) and 82 (24%
93 -Barr virus, 3%; herpes simplex virus 1, 3%; varicella zoster virus, 3%; HHV7, 2%; and herpes simplex
94 ty of herpes simplex virus, cytomegalovirus, varicella zoster virus, and Epstein-Barr virus in our po
96 nstrate that childhood infections, including varicella zoster virus, are associated with an increased
97 for other infections (herpes simplex virus, varicella zoster virus, bacterial and fungal infections)
98 itis (AU), owing to either herpes simplex or varicella zoster virus, by using the Standardization of
99 se encephalitis virus, herpes simplex virus, varicella zoster virus, cytomegalovirus, dengue virus an
100 deficiency virus (HIV)-herpes simplex virus, varicella zoster virus, Epstein-Barr virus (EBV), and cy
101 gnificant members of the herpesvirus family: varicella zoster virus, human cytomegalovirus, and Epste
102 genes, Treponema pallidium, parvovirus, HIV, varicella zoster virus, Rubella, Cytomegalovirus, and He
103 highly dependent on the host cell, we tested varicella zoster virus-infected cell lysates and clinica
104 nation that elicited an exceptionally strong varicella zoster virus-specific B-cell and CD8 T-cell re
109 1), P. jirovecii pneumonia (1.77; .42-7.47), varicella-zoster virus (1.51; .71-3.22), as well as over
110 d 9 with herpes simplex virus (8.8%), 5 with varicella-zoster virus (4.9%), 27 with cytomegalovirus (
112 SV functioned as a monopartite NLS, while in varicella-zoster virus (VZV) activity required an adjace
113 are the main architectural contrasts between varicella-zoster virus (VZV) and herpes simplex virus (H
114 d the Us9 homologs from two human pathogens, varicella-zoster virus (VZV) and herpes simplex virus ty
117 herpes simpex virus 1 and 2 (HSV-1, HSV-2), varicella-zoster virus (VZV) and human herpesvirus 8 (HH
118 reactivation of herpesviruses, most commonly varicella-zoster virus (VZV) and pseudorabies virus (PRV
119 simplex virus type 1 (HSV-1) is conserved in varicella-zoster virus (VZV) and pseudorabies virus (PRV
120 ype 1 (EHV-1), pseudorabies virus (PRV), and varicella-zoster virus (VZV) and their subsequent functi
122 gument proteins encoded by ORF11 and ORF9 of varicella-zoster virus (VZV) are conserved among all alp
123 Infection of human neurons in vitro with varicella-zoster virus (VZV) at a low multiplicity of in
127 highly infectious, human-restricted pathogen varicella-zoster virus (VZV) causes chickenpox and shing
128 ating VZV from clinical specimens.IMPORTANCE Varicella-zoster virus (VZV) causes chickenpox and shing
130 ies for treatment of VZV diseases.IMPORTANCE Varicella-zoster virus (VZV) causes herpes zoster, a maj
134 y throughout the study and were analyzed for varicella-zoster virus (VZV) DNA by use of both qualitat
140 ts had similar magnitude memory responses to varicella-zoster virus (VZV) ex vivo restimulation measu
143 tive target for antiviral therapy.IMPORTANCE Varicella-zoster virus (VZV) has infected over 90% of pe
145 (EBV) EB2, herpes simplex virus (HSV) ICP27, varicella-zoster virus (VZV) IE4/ORF4, and cytomegalovir
147 nses in the bone marrow.IMPORTANCE Childhood varicella-zoster virus (VZV) immunization induces immune
148 ects immediate-early protein IE63 encoded by varicella-zoster virus (VZV) in the cytoplasm of product
149 sensitivity to detect antibody responses to varicella-zoster virus (VZV) in vaccinated individuals,
151 Previous studies have demonstrated that varicella-zoster virus (VZV) infection activates ERK1/2,
155 acaques (RMs) recapitulates the hallmarks of varicella-zoster virus (VZV) infection of humans, includ
157 extensively studied the role of autophagy in varicella-zoster virus (VZV) infection, and have observe
158 ent infant with concurrent primary wild-type varicella-zoster virus (VZV) infection, in whom chickenp
166 The immediate early 62 protein (IE62) of varicella-zoster virus (VZV) is a major viral trans-acti
176 e major immediate early 62 (IE62) protein of varicella-zoster virus (VZV) is delivered to newly infec
178 mary infection, latency, and reactivation by varicella-zoster virus (VZV) is incompletely understood.
187 f herpes simplex virus 1 (HSV-1), HSV-2, and varicella-zoster virus (VZV) on 695 consecutive cutaneou
192 f transcripts corresponding to all 68 unique varicella-zoster virus (VZV) open reading frames (ORFs)
195 In this report, we show that ORF61p, the varicella-zoster virus (VZV) ortholog of ICP0, does not
196 ced syncytium formation, a characteristic of varicella-zoster virus (VZV) pathology in skin and senso
197 this minireview is to provide an overview of varicella-zoster virus (VZV) phylogenetics and phylogeog
200 IMPORTANCE The neurological damage caused by varicella-zoster virus (VZV) reactivation is commonly ma
204 r herpes simplex virus 1 (HSV-1), HSV-2, and varicella-zoster virus (VZV) to the BD Max system by usi
208 ldhood immunization with the live-attenuated varicella-zoster virus (VZV) vaccine induces protective
214 The immediate early 62 protein (IE62) of varicella-zoster virus (VZV), a major viral trans-activa
217 1) and pseudorabies virus (PRV) and ORF66 in varicella-zoster virus (VZV), affects several viral and
219 pear healthy at 2 weeks after infection with varicella-zoster virus (VZV), and the cell culture mediu
220 ovirus, herpes simplex virus type 1 (HSV-1), varicella-zoster virus (VZV), and West Nile virus (WNV).
221 sviruses, herpes simplex virus 1 (HSV-1) and varicella-zoster virus (VZV), confirmed the expression o
222 ults for herpes simplex virus 1/2 (HSV-1/2), varicella-zoster virus (VZV), cytomegalovirus (CMV), or
223 icella virus (SVV), the counterpart of human varicella-zoster virus (VZV), developed primary infectio
224 In this study, quantitative PCR detected varicella-zoster virus (VZV), herpes simplex virus 1 (HS
225 ing the medically important alphaherpesvirus varicella-zoster virus (VZV), induce fusion of the virio
227 showed cytopathic changes, but HSV-1, unlike varicella-zoster virus (VZV), only rarely infected satel
231 the risk of herpes zoster (HZ), we compared varicella-zoster virus (VZV)-specific and nonspecific T-
234 ssays and flow cytometry, we determined that varicella-zoster virus (VZV)-specific peak T helper 1 (V
243 nate gene activation by live yellow-fever or varicella-zoster virus (YFV/VZV) vaccines was more suspe
244 onal and pathogen-specific stimulation (with varicella-zoster virus [VZV] and cytomegalovirus [CMV]).
246 cropsy of two monkeys inoculated with simian varicella-zoster virus and euthanized 117 days later.
247 y, other human-restricted viruses.IMPORTANCE Varicella-zoster virus and human cytomegalovirus infect
248 ia in adults includes common agents, such as varicella-zoster virus and influenza virus, as well as r
249 finding associated with uveitis secondary to varicella-zoster virus and Toxoplasma gondii coinfection
250 s of anterior uveitis in his left eye due to varicella-zoster virus and Toxoplasma gondii coinfection
253 e whether herpes zoster antigen (also called varicella-zoster virus antigen) was detectable in tempor
254 gical boosting, through which reexposures to varicella-zoster virus are thought to reduce the individ
256 MPORTANCE Herpes simplex viruses 1 and 2 and varicella-zoster virus cause significant morbidity and m
260 the phenotypic spectrum of TLR3 mutations to varicella-zoster virus encephalitis and support the role
261 rpes zoster is a common late complication of varicella-zoster virus exposure and can be further compl
262 hus, a 30-h delay after death did not affect varicella-zoster virus expression in latently infected g
265 ve (at months 0, 1, 3) three doses of 50 mug varicella-zoster virus glycoprotein E (gE) adjuvanted wi
267 ubjects received 3 doses of HZ/su (50 microg varicella-zoster virus glycoprotein E [gE] combined with
268 g older adults, a subunit vaccine containing varicella-zoster virus glycoprotein E and the AS01B adju
269 zoster vaccine showed a greater increase in varicella-zoster virus gpELISA antibody compared with su
271 or who had resided in a country with endemic varicella-zoster virus infection for 30 years or more we
274 regulate infection of host cells.IMPORTANCE Varicella-zoster virus is an important human pathogen, w
279 -coinfected children and were independent of varicella-zoster virus or herpes-simplex virus 1 coinfec
280 The continued success of the live attenuated varicella-zoster virus vaccine in preventing varicella-z
282 ty for CMV, EBV, herpes-simplex virus 1, and varicella-zoster virus were studied in 1079 6-year-old c
284 2, human herpesvirus 6, human parechovirus, varicella-zoster virus, and Cryptococcus neoformans/Cryp
285 Findings from skin biopsy, viral culture for varicella-zoster virus, and skin prick test to common fo
286 (HSV) and other alphaherpesviruses, such as varicella-zoster virus, depend upon the capacity to navi
287 ZVL and, together with baseline immunity to varicella-zoster virus, explains the effect of age on th
288 erritin levels were highest in patients with varicella-zoster virus, hepatitis, or malaria (median, 1
289 es (parechovirus, dengue virus, Nipah virus, varicella-zoster virus, mumps virus, measles virus, lyss
290 h HLA-B27-associated (4460 [2465] pg/mL) and varicella-zoster virus-associated (5386 [1778] pg/mL) uv
291 in 50-59-year-old subjects were examined for varicella-zoster virus-specific antibody responses to va
292 1, CTLA-4, and TIM-3, whereas <2% of CMV- or varicella-zoster virus-specific CD4(+) T cells expressed
293 the change from baseline in IgG antibody to varicella-zoster virus-specific glycoproteins (gpELISA)
300 Immunity to measles, mumps, rubella, and varicella-zoster viruses (VZV; MMRV) is a common conditi