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1 2, cytomegalovirus, Epstein-Barr virus, and varicella zoster virus.
2 erpes simplex virus and 8 patients (38%) had varicella zoster virus.
3 ese is also activated by the closely related varicella zoster virus.
4 ew mutations or recombination with wild-type Varicella zoster virus.
5 losely related to herpes simplex viruses and varicella-zoster virus.
6 G synthesis, and elevated antibody titers to varicella-zoster virus.
7 d, for example, in relation to pertussis and varicella-zoster virus.
8 pathogens herpes simplex viruses 1 and 2 and varicella-zoster virus.
9 ation were detected in OPV, mumps virus, and varicella-zoster virus.
10 uses herpes simplex virus type 1 (HSV-1) and varicella-zoster virus.
11 1), P. jirovecii pneumonia (1.77; .42-7.47), varicella-zoster virus (1.51; .71-3.22), as well as over
12 -Barr virus, 3%; herpes simplex virus 1, 3%; varicella zoster virus, 3%; HHV7, 2%; and herpes simplex
13 d 9 with herpes simplex virus (8.8%), 5 with varicella-zoster virus (4.9%), 27 with cytomegalovirus (
14 CF; human cytomegalovirus (HCMV) 11% in GCF; varicella zoster virus 6% in saliva and 3% in GCF; of hu
16 RN, PCR-positive for herpes simplex virus or varicella zoster virus and evaluated between January 200
17 genes of both herpes simplex virus (HSV) and varicella zoster virus and functions, in part, by coupli
18 beyond CMV to other herpes viruses, such as varicella zoster virus and possibly Epstein-Barr virus.
19 ld decrease external boosting of immunity to varicella zoster virus and thereby increase incidence of
20 cropsy of two monkeys inoculated with simian varicella-zoster virus and euthanized 117 days later.
21 y, other human-restricted viruses.IMPORTANCE Varicella-zoster virus and human cytomegalovirus infect
22 ia in adults includes common agents, such as varicella-zoster virus and influenza virus, as well as r
23 finding associated with uveitis secondary to varicella-zoster virus and Toxoplasma gondii coinfection
24 s of anterior uveitis in his left eye due to varicella-zoster virus and Toxoplasma gondii coinfection
25 ty of herpes simplex virus, cytomegalovirus, varicella zoster virus, and Epstein-Barr virus in our po
27 2, human herpesvirus 6, human parechovirus, varicella-zoster virus, and Cryptococcus neoformans/Cryp
28 Findings from skin biopsy, viral culture for varicella-zoster virus, and skin prick test to common fo
31 e whether herpes zoster antigen (also called varicella-zoster virus antigen) was detectable in tempor
32 gical boosting, through which reexposures to varicella-zoster virus are thought to reduce the individ
33 nstrate that childhood infections, including varicella zoster virus, are associated with an increased
34 h HLA-B27-associated (4460 [2465] pg/mL) and varicella-zoster virus-associated (5386 [1778] pg/mL) uv
35 for other infections (herpes simplex virus, varicella zoster virus, bacterial and fungal infections)
36 itis (AU), owing to either herpes simplex or varicella zoster virus, by using the Standardization of
38 MPORTANCE Herpes simplex viruses 1 and 2 and varicella-zoster virus cause significant morbidity and m
40 se encephalitis virus, herpes simplex virus, varicella zoster virus, cytomegalovirus, dengue virus an
41 (HSV) and other alphaherpesviruses, such as varicella-zoster virus, depend upon the capacity to navi
43 (as determined by testing lesions swabs for varicella zoster virus DNA by polymerase chain reaction)
44 ence of confirmed varicella (by detection of varicella zoster virus DNA or epidemiological link) from
48 the phenotypic spectrum of TLR3 mutations to varicella-zoster virus encephalitis and support the role
50 deficiency virus (HIV)-herpes simplex virus, varicella zoster virus, Epstein-Barr virus (EBV), and cy
51 ZVL and, together with baseline immunity to varicella-zoster virus, explains the effect of age on th
52 rpes zoster is a common late complication of varicella-zoster virus exposure and can be further compl
53 hus, a 30-h delay after death did not affect varicella-zoster virus expression in latently infected g
56 were randomized 1:1 to receive either HZ/su (varicella zoster virus glycoprotein E; AS01B Adjuvant Sy
57 ve (at months 0, 1, 3) three doses of 50 mug varicella-zoster virus glycoprotein E (gE) adjuvanted wi
59 ubjects received 3 doses of HZ/su (50 microg varicella-zoster virus glycoprotein E [gE] combined with
60 g older adults, a subunit vaccine containing varicella-zoster virus glycoprotein E and the AS01B adju
61 zoster vaccine showed a greater increase in varicella-zoster virus gpELISA antibody compared with su
62 equences of wild-type and vaccine strains of varicella-zoster virus have been published and listed in
63 erritin levels were highest in patients with varicella-zoster virus, hepatitis, or malaria (median, 1
64 megalovirus (HR, 3.98 [95% CI, 1.40-11.26]), varicella zoster virus (HR, 1.49 [95% CI, 1.18-1.89]), h
65 ytomegalovirus [CMV], herpes simplex I/II or varicella zoster virus [HSV/VZV], blood stream infection
66 gnificant members of the herpesvirus family: varicella zoster virus, human cytomegalovirus, and Epste
67 rveillance, combined with information from a Varicella Zoster Virus Identification Program, which use
69 highly dependent on the host cell, we tested varicella zoster virus-infected cell lysates and clinica
70 cation) were associated with protection from varicella zoster virus infection (hazard ratio, 0.43; 95
71 contact dermatitis, infectious folliculitis, varicella zoster virus infection, fixed drug eruption, a
73 or who had resided in a country with endemic varicella-zoster virus infection for 30 years or more we
77 regulate infection of host cells.IMPORTANCE Varicella-zoster virus is an important human pathogen, w
83 es (parechovirus, dengue virus, Nipah virus, varicella-zoster virus, mumps virus, measles virus, lyss
87 -coinfected children and were independent of varicella-zoster virus or herpes-simplex virus 1 coinfec
88 derate quality showed an association between varicella zoster virus reactivation (ophthalmic zoster)
89 Because there is no good animal model of varicella zoster virus reactivation from latency, this e
90 genes, Treponema pallidium, parvovirus, HIV, varicella zoster virus, Rubella, Cytomegalovirus, and He
92 nation that elicited an exceptionally strong varicella zoster virus-specific B-cell and CD8 T-cell re
93 in 50-59-year-old subjects were examined for varicella-zoster virus-specific antibody responses to va
94 1, CTLA-4, and TIM-3, whereas <2% of CMV- or varicella-zoster virus-specific CD4(+) T cells expressed
95 the change from baseline in IgG antibody to varicella-zoster virus-specific glycoproteins (gpELISA)
96 ng heat-inactivated or replication-defective varicella-zoster virus to prevent HZ in immunocompromise
97 The continued success of the live attenuated varicella-zoster virus vaccine in preventing varicella-z
99 s HSV1 and HSV2 (also termed HHV1 and HHV2), varicella zoster virus (VZV or HHV3), EBV (HHV4), cytome
100 tients showed a decreased ability to control varicella zoster virus (VZV) and Epstein-Barr virus (EBV
106 response biomarkers measuring antibodies to varicella zoster virus (VZV) by glycoprotein-based enzym
107 lex virus types 1 (HSV-1) and 2 (HSV-2), and varicella zoster virus (VZV) by weekly polymerase chain
108 se of herpes zoster caused by the attenuated varicella zoster virus (VZV) contained in Zostavax in a
109 , or no history of zoster (group 3) revealed varicella zoster virus (VZV) DNA in saliva samples from
110 rs who were immunized with Zostavax revealed varicella zoster virus (VZV) DNA in swabs of skin inocul
114 as an alternative to sampling of rashes for varicella zoster virus (VZV) genotyping and further char
115 s positive for herpes simplex virus (HSV) or varicella zoster virus (VZV) in 79% to 100% of cases of
116 Clinical reports observe the reactivation of varicella zoster virus (VZV) in people who have recovere
119 virus (CMV), herpes simplex virus (HSV), and varicella zoster virus (VZV) infections were monitored i
122 As) from patients with giant cell arteritis, varicella zoster virus (VZV) is seen in perineurial cell
126 s" postulates that reexposure to circulating varicella zoster virus (VZV) over the life span inhibits
131 ne responses to a high-titer live attenuated varicella zoster virus (VZV) vaccine (zoster vaccine), w
132 te the efficacy and safety of an inactivated varicella zoster virus (VZV) vaccine for herpes zoster p
133 Since the introduction of live attenuated varicella zoster virus (VZV) vaccine in 1995 there has b
135 lovirus (CMV), Epstein-Barr virus (EBV), and varicella zoster virus (VZV) was determined in crewmembe
138 portion of HZ cases caused by vaccine-strain varicella zoster virus (VZV), assessed the positive pred
139 c primers to detect DNA from JC virus (JCV), varicella zoster virus (VZV), cytomegalovirus (CMV), Eps
142 erpesviruses, herpes simplex virus (HSV) and varicella zoster virus (VZV), results in the rapid accum
143 immunogenicity of live-attenuated Oka/Merck varicella zoster virus (VZV)-containing vaccine (hereaft
145 We investigated the relationship between varicella zoster virus (VZV)-specific memory CD4(+) T ce
146 virus type 1 (HSV-1) and type 2 (HSV-2) and varicella zoster virus (VZV)-was determined in autonomic
152 SV functioned as a monopartite NLS, while in varicella-zoster virus (VZV) activity required an adjace
153 are the main architectural contrasts between varicella-zoster virus (VZV) and herpes simplex virus (H
154 d the Us9 homologs from two human pathogens, varicella-zoster virus (VZV) and herpes simplex virus ty
157 herpes simpex virus 1 and 2 (HSV-1, HSV-2), varicella-zoster virus (VZV) and human herpesvirus 8 (HH
158 reactivation of herpesviruses, most commonly varicella-zoster virus (VZV) and pseudorabies virus (PRV
159 simplex virus type 1 (HSV-1) is conserved in varicella-zoster virus (VZV) and pseudorabies virus (PRV
160 ype 1 (EHV-1), pseudorabies virus (PRV), and varicella-zoster virus (VZV) and their subsequent functi
162 gument proteins encoded by ORF11 and ORF9 of varicella-zoster virus (VZV) are conserved among all alp
163 Infection of human neurons in vitro with varicella-zoster virus (VZV) at a low multiplicity of in
167 highly infectious, human-restricted pathogen varicella-zoster virus (VZV) causes chickenpox and shing
168 ating VZV from clinical specimens.IMPORTANCE Varicella-zoster virus (VZV) causes chickenpox and shing
170 ies for treatment of VZV diseases.IMPORTANCE Varicella-zoster virus (VZV) causes herpes zoster, a maj
174 y throughout the study and were analyzed for varicella-zoster virus (VZV) DNA by use of both qualitat
181 ts had similar magnitude memory responses to varicella-zoster virus (VZV) ex vivo restimulation measu
184 tive target for antiviral therapy.IMPORTANCE Varicella-zoster virus (VZV) has infected over 90% of pe
186 (EBV) EB2, herpes simplex virus (HSV) ICP27, varicella-zoster virus (VZV) IE4/ORF4, and cytomegalovir
189 nses in the bone marrow.IMPORTANCE Childhood varicella-zoster virus (VZV) immunization induces immune
190 ects immediate-early protein IE63 encoded by varicella-zoster virus (VZV) in the cytoplasm of product
191 sensitivity to detect antibody responses to varicella-zoster virus (VZV) in vaccinated individuals,
193 Previous studies have demonstrated that varicella-zoster virus (VZV) infection activates ERK1/2,
198 acaques (RMs) recapitulates the hallmarks of varicella-zoster virus (VZV) infection of humans, includ
200 extensively studied the role of autophagy in varicella-zoster virus (VZV) infection, and have observe
201 ent infant with concurrent primary wild-type varicella-zoster virus (VZV) infection, in whom chickenp
209 The immediate early 62 protein (IE62) of varicella-zoster virus (VZV) is a major viral trans-acti
219 e major immediate early 62 (IE62) protein of varicella-zoster virus (VZV) is delivered to newly infec
221 mary infection, latency, and reactivation by varicella-zoster virus (VZV) is incompletely understood.
231 f herpes simplex virus 1 (HSV-1), HSV-2, and varicella-zoster virus (VZV) on 695 consecutive cutaneou
236 f transcripts corresponding to all 68 unique varicella-zoster virus (VZV) open reading frames (ORFs)
240 In this report, we show that ORF61p, the varicella-zoster virus (VZV) ortholog of ICP0, does not
241 ced syncytium formation, a characteristic of varicella-zoster virus (VZV) pathology in skin and senso
242 this minireview is to provide an overview of varicella-zoster virus (VZV) phylogenetics and phylogeog
245 IMPORTANCE The neurological damage caused by varicella-zoster virus (VZV) reactivation is commonly ma
249 r herpes simplex virus 1 (HSV-1), HSV-2, and varicella-zoster virus (VZV) to the BD Max system by usi
253 ldhood immunization with the live-attenuated varicella-zoster virus (VZV) vaccine induces protective
260 The immediate early 62 protein (IE62) of varicella-zoster virus (VZV), a major viral trans-activa
263 1) and pseudorabies virus (PRV) and ORF66 in varicella-zoster virus (VZV), affects several viral and
265 pear healthy at 2 weeks after infection with varicella-zoster virus (VZV), and the cell culture mediu
266 ovirus, herpes simplex virus type 1 (HSV-1), varicella-zoster virus (VZV), and West Nile virus (WNV).
267 sviruses, herpes simplex virus 1 (HSV-1) and varicella-zoster virus (VZV), confirmed the expression o
268 ults for herpes simplex virus 1/2 (HSV-1/2), varicella-zoster virus (VZV), cytomegalovirus (CMV), or
269 icella virus (SVV), the counterpart of human varicella-zoster virus (VZV), developed primary infectio
270 In this study, quantitative PCR detected varicella-zoster virus (VZV), herpes simplex virus 1 (HS
272 ing the medically important alphaherpesvirus varicella-zoster virus (VZV), induce fusion of the virio
274 showed cytopathic changes, but HSV-1, unlike varicella-zoster virus (VZV), only rarely infected satel
276 nation was more likely to identify wild-type varicella-zoster virus (VZV), whereas the presence of Ok
279 the risk of herpes zoster (HZ), we compared varicella-zoster virus (VZV)-specific and nonspecific T-
281 n association with an age-related decline in varicella-zoster virus (VZV)-specific cell-mediated immu
282 udy were to evaluate the association between varicella-zoster virus (VZV)-specific humoral and cell-m
284 ssays and flow cytometry, we determined that varicella-zoster virus (VZV)-specific peak T helper 1 (V
293 Immunity to measles, mumps, rubella, and varicella-zoster viruses (VZV; MMRV) is a common conditi
294 onal and pathogen-specific stimulation (with varicella-zoster virus [VZV] and cytomegalovirus [CMV]).
296 ion (PCR) for herpes simplex virus (HSV) and varicella zoster virus was done in 237 (69%) and 82 (24%
298 ty for CMV, EBV, herpes-simplex virus 1, and varicella-zoster virus were studied in 1079 6-year-old c
300 nate gene activation by live yellow-fever or varicella-zoster virus (YFV/VZV) vaccines was more suspe