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1  2, cytomegalovirus, Epstein-Barr virus, and varicella zoster virus.
2 erpes simplex virus and 8 patients (38%) had varicella zoster virus.
3 ese is also activated by the closely related varicella zoster virus.
4 ew mutations or recombination with wild-type Varicella zoster virus.
5 losely related to herpes simplex viruses and varicella-zoster virus.
6 G synthesis, and elevated antibody titers to varicella-zoster virus.
7 d, for example, in relation to pertussis and varicella-zoster virus.
8 pathogens herpes simplex viruses 1 and 2 and varicella-zoster virus.
9 ation were detected in OPV, mumps virus, and varicella-zoster virus.
10 uses herpes simplex virus type 1 (HSV-1) and varicella-zoster virus.
11 1), P. jirovecii pneumonia (1.77; .42-7.47), varicella-zoster virus (1.51; .71-3.22), as well as over
12 -Barr virus, 3%; herpes simplex virus 1, 3%; varicella zoster virus, 3%; HHV7, 2%; and herpes simplex
13 d 9 with herpes simplex virus (8.8%), 5 with varicella-zoster virus (4.9%), 27 with cytomegalovirus (
14 CF; human cytomegalovirus (HCMV) 11% in GCF; varicella zoster virus 6% in saliva and 3% in GCF; of hu
15                    For herpes simplex virus, varicella zoster virus and cytomegalovirus, these advanc
16 RN, PCR-positive for herpes simplex virus or varicella zoster virus and evaluated between January 200
17 genes of both herpes simplex virus (HSV) and varicella zoster virus and functions, in part, by coupli
18  beyond CMV to other herpes viruses, such as varicella zoster virus and possibly Epstein-Barr virus.
19 ld decrease external boosting of immunity to varicella zoster virus and thereby increase incidence of
20 cropsy of two monkeys inoculated with simian varicella-zoster virus and euthanized 117 days later.
21 y, other human-restricted viruses.IMPORTANCE Varicella-zoster virus and human cytomegalovirus infect
22 ia in adults includes common agents, such as varicella-zoster virus and influenza virus, as well as r
23 finding associated with uveitis secondary to varicella-zoster virus and Toxoplasma gondii coinfection
24 s of anterior uveitis in his left eye due to varicella-zoster virus and Toxoplasma gondii coinfection
25 ty of herpes simplex virus, cytomegalovirus, varicella zoster virus, and Epstein-Barr virus in our po
26                        Herpes simplex virus, varicella zoster virus, and pseudorabies virus are neuro
27  2, human herpesvirus 6, human parechovirus, varicella-zoster virus, and Cryptococcus neoformans/Cryp
28 Findings from skin biopsy, viral culture for varicella-zoster virus, and skin prick test to common fo
29                                              Varicella-zoster virus antigen was found in 45 of 70 GCA
30                                              Varicella-zoster virus antigen was frequently found in p
31 e whether herpes zoster antigen (also called varicella-zoster virus antigen) was detectable in tempor
32 gical boosting, through which reexposures to varicella-zoster virus are thought to reduce the individ
33 nstrate that childhood infections, including varicella zoster virus, are associated with an increased
34 h HLA-B27-associated (4460 [2465] pg/mL) and varicella-zoster virus-associated (5386 [1778] pg/mL) uv
35  for other infections (herpes simplex virus, varicella zoster virus, bacterial and fungal infections)
36 itis (AU), owing to either herpes simplex or varicella zoster virus, by using the Standardization of
37 clonal antibodies against a major antigen of varicella-zoster virus called gE.
38 MPORTANCE Herpes simplex viruses 1 and 2 and varicella-zoster virus cause significant morbidity and m
39                                  We compared varicella-zoster virus cell-mediated immunity (VZV-CMI)
40 se encephalitis virus, herpes simplex virus, varicella zoster virus, cytomegalovirus, dengue virus an
41  (HSV) and other alphaherpesviruses, such as varicella-zoster virus, depend upon the capacity to navi
42 ES by the corresponding region from ORF61 of varicella-zoster virus did not rescue ND10 fusion.
43  (as determined by testing lesions swabs for varicella zoster virus DNA by polymerase chain reaction)
44 ence of confirmed varicella (by detection of varicella zoster virus DNA or epidemiological link) from
45                                              Varicella zoster virus DNA was detected 2 months after t
46                                              Varicella zoster virus encephalitis was infrequent follo
47 th HSE (p.Leu297Val) and 1 in a patient with varicella-zoster virus encephalitis (p.Leu199Phe).
48 the phenotypic spectrum of TLR3 mutations to varicella-zoster virus encephalitis and support the role
49                                              Varicella zoster virus encodes an immediate-early (IE) p
50 deficiency virus (HIV)-herpes simplex virus, varicella zoster virus, Epstein-Barr virus (EBV), and cy
51  ZVL and, together with baseline immunity to varicella-zoster virus, explains the effect of age on th
52 rpes zoster is a common late complication of varicella-zoster virus exposure and can be further compl
53 hus, a 30-h delay after death did not affect varicella-zoster virus expression in latently infected g
54                                   Studies of varicella-zoster virus gene expression during latency re
55                                          The varicella-zoster virus geometric mean titer (GMT) and ge
56 were randomized 1:1 to receive either HZ/su (varicella zoster virus glycoprotein E; AS01B Adjuvant Sy
57 ve (at months 0, 1, 3) three doses of 50 mug varicella-zoster virus glycoprotein E (gE) adjuvanted wi
58                                An adjuvanted varicella-zoster virus glycoprotein E (gE) subunit vacci
59 ubjects received 3 doses of HZ/su (50 microg varicella-zoster virus glycoprotein E [gE] combined with
60 g older adults, a subunit vaccine containing varicella-zoster virus glycoprotein E and the AS01B adju
61  zoster vaccine showed a greater increase in varicella-zoster virus gpELISA antibody compared with su
62 equences of wild-type and vaccine strains of varicella-zoster virus have been published and listed in
63 erritin levels were highest in patients with varicella-zoster virus, hepatitis, or malaria (median, 1
64 megalovirus (HR, 3.98 [95% CI, 1.40-11.26]), varicella zoster virus (HR, 1.49 [95% CI, 1.18-1.89]), h
65 ytomegalovirus [CMV], herpes simplex I/II or varicella zoster virus [HSV/VZV], blood stream infection
66 gnificant members of the herpesvirus family: varicella zoster virus, human cytomegalovirus, and Epste
67 rveillance, combined with information from a Varicella Zoster Virus Identification Program, which use
68 ogic modulators restores immune responses to varicella-zoster virus in vaccinees.
69 highly dependent on the host cell, we tested varicella zoster virus-infected cell lysates and clinica
70 cation) were associated with protection from varicella zoster virus infection (hazard ratio, 0.43; 95
71 contact dermatitis, infectious folliculitis, varicella zoster virus infection, fixed drug eruption, a
72 processes, including ubiquitin clearance and Varicella Zoster Virus infection.
73 or who had resided in a country with endemic varicella-zoster virus infection for 30 years or more we
74 erties that may favor reactivation of latent varicella-zoster virus infection.
75 nd increased susceptibility to bacterial and varicella zoster virus infections.
76                                        While varicella-zoster virus is also insensitive to interferon
77  regulate infection of host cells.IMPORTANCE Varicella-zoster virus is an important human pathogen, w
78                          Because features of varicella-zoster virus latency are similar in primate an
79                                          The varicella-zoster virus major transactivator, IE62, conta
80                             One patient with varicella zoster virus meningitis and acute GVHD had iC9
81                 Mumps, measles, rubella, and varicella-zoster viruses (MMRV) may cause severe infecti
82                                    Available varicella-zoster virus models can be classified in 3 mai
83 es (parechovirus, dengue virus, Nipah virus, varicella-zoster virus, mumps virus, measles virus, lyss
84  = 60), followed by tuberculosis (n = 8) and varicella zoster virus (n = 7).
85 e fills a notable gap in our knowledge about varicella zoster virus neuronal transportation.
86 ilar function in human herpesviruses such as varicella-zoster virus or herpes simplex viruses.
87 -coinfected children and were independent of varicella-zoster virus or herpes-simplex virus 1 coinfec
88 derate quality showed an association between varicella zoster virus reactivation (ophthalmic zoster)
89     Because there is no good animal model of varicella zoster virus reactivation from latency, this e
90 genes, Treponema pallidium, parvovirus, HIV, varicella zoster virus, Rubella, Cytomegalovirus, and He
91                 Among the 131 live births to varicella-zoster virus-seronegative women, there was no
92 nation that elicited an exceptionally strong varicella zoster virus-specific B-cell and CD8 T-cell re
93 in 50-59-year-old subjects were examined for varicella-zoster virus-specific antibody responses to va
94 1, CTLA-4, and TIM-3, whereas <2% of CMV- or varicella-zoster virus-specific CD4(+) T cells expressed
95  the change from baseline in IgG antibody to varicella-zoster virus-specific glycoproteins (gpELISA)
96 ng heat-inactivated or replication-defective varicella-zoster virus to prevent HZ in immunocompromise
97 The continued success of the live attenuated varicella-zoster virus vaccine in preventing varicella-z
98                The licensed live, attenuated varicella-zoster virus vaccine prevents herpes zoster in
99 s HSV1 and HSV2 (also termed HHV1 and HHV2), varicella zoster virus (VZV or HHV3), EBV (HHV4), cytome
100 tients showed a decreased ability to control varicella zoster virus (VZV) and Epstein-Barr virus (EBV
101                                              Varicella zoster virus (VZV) and the two herpes simplex
102                                              Varicella zoster virus (VZV) antibody titers (measured b
103                                              Varicella zoster virus (VZV) antigen was found in all of
104                     Vasculopathies caused by varicella zoster virus (VZV) are indicative of a product
105                                      CMV and varicella zoster virus (VZV) are significant causes of m
106  response biomarkers measuring antibodies to varicella zoster virus (VZV) by glycoprotein-based enzym
107 lex virus types 1 (HSV-1) and 2 (HSV-2), and varicella zoster virus (VZV) by weekly polymerase chain
108 se of herpes zoster caused by the attenuated varicella zoster virus (VZV) contained in Zostavax in a
109 , or no history of zoster (group 3) revealed varicella zoster virus (VZV) DNA in saliva samples from
110 rs who were immunized with Zostavax revealed varicella zoster virus (VZV) DNA in swabs of skin inocul
111                            Herpesvirions and varicella zoster virus (VZV) DNA were recently reported
112                                              Varicella zoster virus (VZV) establishes latency in dors
113                                              Varicella zoster virus (VZV) establishes lifelong persis
114  as an alternative to sampling of rashes for varicella zoster virus (VZV) genotyping and further char
115 s positive for herpes simplex virus (HSV) or varicella zoster virus (VZV) in 79% to 100% of cases of
116 Clinical reports observe the reactivation of varicella zoster virus (VZV) in people who have recovere
117                          Reactivation of the varicella zoster virus (VZV) increases during aging.
118                                              Varicella zoster virus (VZV) infections are a relevant c
119 virus (CMV), herpes simplex virus (HSV), and varicella zoster virus (VZV) infections were monitored i
120                                              Varicella zoster virus (VZV) is a neurotropic alphaherpe
121                                              Varicella zoster virus (VZV) is a skin-tropic virus that
122 As) from patients with giant cell arteritis, varicella zoster virus (VZV) is seen in perineurial cell
123                                              Varicella zoster virus (VZV) is the causative agent of c
124                                              Varicella Zoster Virus (VZV) is the causative agent of v
125                                              Varicella zoster virus (VZV) is the etiological agent of
126 s" postulates that reexposure to circulating varicella zoster virus (VZV) over the life span inhibits
127                                              Varicella zoster virus (VZV) reactivation results in zos
128                    An adjuvanted recombinant varicella zoster virus (VZV) subunit vaccine is being de
129  zoster (HZ) cases may play a larger role in varicella zoster virus (VZV) transmission.
130                                              Varicella zoster virus (VZV) typically causes chickenpox
131 ne responses to a high-titer live attenuated varicella zoster virus (VZV) vaccine (zoster vaccine), w
132 te the efficacy and safety of an inactivated varicella zoster virus (VZV) vaccine for herpes zoster p
133    Since the introduction of live attenuated varicella zoster virus (VZV) vaccine in 1995 there has b
134 s, granulomatous aortitis, and intracerebral varicella zoster virus (VZV) vasculopathy.
135 lovirus (CMV), Epstein-Barr virus (EBV), and varicella zoster virus (VZV) was determined in crewmembe
136                        IL-10 and immunity to varicella zoster virus (VZV) were measured at baseline a
137 cytomegalovirus, herpes simplex virus (HSV), varicella zoster virus (VZV), and rubella.
138 portion of HZ cases caused by vaccine-strain varicella zoster virus (VZV), assessed the positive pred
139 c primers to detect DNA from JC virus (JCV), varicella zoster virus (VZV), cytomegalovirus (CMV), Eps
140 d were hepatitis A (HAV), hepatitis B (HBV), varicella zoster virus (VZV), measles, and mumps.
141 on childhood disease, chicken pox, caused by varicella zoster virus (VZV), over an 11-y period.
142 erpesviruses, herpes simplex virus (HSV) and varicella zoster virus (VZV), results in the rapid accum
143  immunogenicity of live-attenuated Oka/Merck varicella zoster virus (VZV)-containing vaccine (hereaft
144                                     Boost of varicella zoster virus (VZV)-specific cellular immunity
145     We investigated the relationship between varicella zoster virus (VZV)-specific memory CD4(+) T ce
146  virus type 1 (HSV-1) and type 2 (HSV-2) and varicella zoster virus (VZV)-was determined in autonomic
147  1 and 2 and the sequence-divergent pathogen varicella zoster virus (VZV).
148 ompromised individuals after reactivation of varicella zoster virus (VZV).
149 n pathogens herpes simplex viruses (HSV) and varicella zoster virus (VZV).
150 ses with age, which leads to reactivation of varicella zoster virus (VZV).
151                                              Varicella-zoster virus (VZV) activates the phosphatidyli
152 SV functioned as a monopartite NLS, while in varicella-zoster virus (VZV) activity required an adjace
153 are the main architectural contrasts between varicella-zoster virus (VZV) and herpes simplex virus (H
154 d the Us9 homologs from two human pathogens, varicella-zoster virus (VZV) and herpes simplex virus ty
155                                  Intraocular varicella-zoster virus (VZV) and HSV type 1 (HSV-1) infe
156                            The herpesviruses varicella-zoster virus (VZV) and human cytomegalovirus (
157  herpes simpex virus 1 and 2 (HSV-1, HSV-2), varicella-zoster virus (VZV) and human herpesvirus 8 (HH
158 reactivation of herpesviruses, most commonly varicella-zoster virus (VZV) and pseudorabies virus (PRV
159 simplex virus type 1 (HSV-1) is conserved in varicella-zoster virus (VZV) and pseudorabies virus (PRV
160 ype 1 (EHV-1), pseudorabies virus (PRV), and varicella-zoster virus (VZV) and their subsequent functi
161                              Infections with varicella-zoster virus (VZV) are associated with a range
162 gument proteins encoded by ORF11 and ORF9 of varicella-zoster virus (VZV) are conserved among all alp
163     Infection of human neurons in vitro with varicella-zoster virus (VZV) at a low multiplicity of in
164 y, IDE has been proposed as the receptor for varicella-zoster virus (VZV) attachment.
165                                              Varicella-zoster virus (VZV) causes chicken pox and shin
166                                              Varicella-zoster virus (VZV) causes chickenpox and react
167 highly infectious, human-restricted pathogen varicella-zoster virus (VZV) causes chickenpox and shing
168 ating VZV from clinical specimens.IMPORTANCE Varicella-zoster virus (VZV) causes chickenpox and shing
169                                              Varicella-zoster virus (VZV) causes chickenpox upon prim
170 ies for treatment of VZV diseases.IMPORTANCE Varicella-zoster virus (VZV) causes herpes zoster, a maj
171                                              Varicella-zoster virus (VZV) causes varicella and establ
172                                              Varicella-zoster virus (VZV) characteristically forms mu
173                    We studied a patient with varicella-zoster virus (VZV) CNS vasculopathy and as par
174 y throughout the study and were analyzed for varicella-zoster virus (VZV) DNA by use of both qualitat
175                                    Wild-type varicella-zoster virus (VZV) DNA was identified in all 3
176              In this report, we investigated varicella-zoster virus (VZV) egress in a cell line from
177       Herein we describe an episode of focal varicella-zoster virus (VZV) encephalitis in a healthy y
178                  The neurotropic herpesvirus varicella-zoster virus (VZV) establishes a lifelong late
179                                              Varicella-zoster virus (VZV) establishes latency in huma
180                                              Varicella-zoster virus (VZV) establishes lifelong neuron
181 ts had similar magnitude memory responses to varicella-zoster virus (VZV) ex vivo restimulation measu
182                When grown in cultured cells, varicella-zoster virus (VZV) forms many aberrant light p
183                            Information about varicella-zoster virus (VZV) gB is limited, but homology
184 tive target for antiviral therapy.IMPORTANCE Varicella-zoster virus (VZV) has infected over 90% of pe
185          Mechanisms of neuronal infection by varicella-zoster virus (VZV) have been challenging to st
186 (EBV) EB2, herpes simplex virus (HSV) ICP27, varicella-zoster virus (VZV) IE4/ORF4, and cytomegalovir
187                                          The varicella-zoster virus (VZV) IE62 protein is the major t
188                                              Varicella-zoster virus (VZV) immediate-early 63 protein
189 nses in the bone marrow.IMPORTANCE Childhood varicella-zoster virus (VZV) immunization induces immune
190 ects immediate-early protein IE63 encoded by varicella-zoster virus (VZV) in the cytoplasm of product
191  sensitivity to detect antibody responses to varicella-zoster virus (VZV) in vaccinated individuals,
192                                              Varicella-zoster virus (VZV) induces apoptosis in human
193      Previous studies have demonstrated that varicella-zoster virus (VZV) infection activates ERK1/2,
194                                              Varicella-zoster virus (VZV) infection causes varicella,
195                                      Primary varicella-zoster virus (VZV) infection in humans produce
196                                              Varicella-zoster virus (VZV) infection is usually mild i
197            Transcriptional changes following varicella-zoster virus (VZV) infection of cultured human
198 acaques (RMs) recapitulates the hallmarks of varicella-zoster virus (VZV) infection of humans, includ
199                                              Varicella-zoster virus (VZV) infection provides a valuab
200 extensively studied the role of autophagy in varicella-zoster virus (VZV) infection, and have observe
201 ent infant with concurrent primary wild-type varicella-zoster virus (VZV) infection, in whom chickenp
202                                              Varicella-zoster virus (VZV) infections increasingly are
203                                              Varicella-zoster virus (VZV) is a common pathogen that c
204                                              Varicella-zoster virus (VZV) is a highly contagious agen
205                                              Varicella-zoster virus (VZV) is a highly neurotropic vir
206                                              Varicella-zoster virus (VZV) is a human alpha-herpesviru
207                                              Varicella-zoster virus (VZV) is a human alphaherpesvirus
208                                              Varicella-zoster virus (VZV) is a human neurotropic alph
209     The immediate early 62 protein (IE62) of varicella-zoster virus (VZV) is a major viral trans-acti
210                                              Varicella-zoster virus (VZV) is a medically important hu
211                                              Varicella-zoster virus (VZV) is a neurotropic alphaherpe
212                                              Varicella-zoster virus (VZV) is a ubiquitous pathogen th
213                                              Varicella-zoster virus (VZV) is a ubiquitous, highly cel
214                                              Varicella-zoster virus (VZV) is an alphaherpesvirus that
215                                              Varicella-zoster virus (VZV) is an alphaherpesvirus that
216                                              Varicella-zoster virus (VZV) is an alphaherpesvirus that
217                                              Varicella-zoster virus (VZV) is an alphaherpesvirus that
218                                              Varicella-zoster virus (VZV) is an extremely cell-associ
219 e major immediate early 62 (IE62) protein of varicella-zoster virus (VZV) is delivered to newly infec
220                                              Varicella-zoster virus (VZV) is highly cell associated w
221 mary infection, latency, and reactivation by varicella-zoster virus (VZV) is incompletely understood.
222                                              Varicella-zoster virus (VZV) is one of the most common a
223                                              Varicella-zoster virus (VZV) is renowned for its low tit
224                                              Varicella-zoster virus (VZV) is renowned for its very lo
225                                              Varicella-zoster virus (VZV) is the alphaherpesvirus tha
226                                              Varicella-zoster virus (VZV) is the causative agent of b
227                                              Varicella-zoster virus (VZV) is the causative agent of c
228                                              Varicella-zoster virus (VZV) is the etiological agent of
229                                              Varicella-zoster virus (VZV) is under consideration as a
230       We report a case of AIDS presenting as varicella-zoster virus (VZV) meningomyeloradiculitis ass
231 f herpes simplex virus 1 (HSV-1), HSV-2, and varicella-zoster virus (VZV) on 695 consecutive cutaneou
232                                              Varicella-zoster virus (VZV) open reading frame (ORF) 63
233                                          The varicella-zoster virus (VZV) open reading frame 54 (ORF5
234                                              Varicella-zoster virus (VZV) open reading frame 61 (ORF6
235                                Three loci in varicella-zoster virus (VZV) open reading frame 62 (ORF6
236 f transcripts corresponding to all 68 unique varicella-zoster virus (VZV) open reading frames (ORFs)
237                                          The varicella-zoster virus (VZV) ORF61 protein is necessary
238                      The architecture of the varicella-zoster virus (VZV) origin of DNA replication (
239                                          The varicella-zoster virus (VZV) origin of DNA replication (
240     In this report, we show that ORF61p, the varicella-zoster virus (VZV) ortholog of ICP0, does not
241 ced syncytium formation, a characteristic of varicella-zoster virus (VZV) pathology in skin and senso
242 this minireview is to provide an overview of varicella-zoster virus (VZV) phylogenetics and phylogeog
243                                  Analyses of varicella-zoster virus (VZV) protein expression during l
244                  ORF66p, a virion-associated varicella-zoster virus (VZV) protein, is a member of a c
245 IMPORTANCE The neurological damage caused by varicella-zoster virus (VZV) reactivation is commonly ma
246                                              Varicella-zoster virus (VZV) T-cell responses by interfe
247                                              Varicella-zoster virus (VZV) T-cell-mediated immunity (V
248                                          The varicella-zoster virus (VZV) terminase components (pORF2
249 r herpes simplex virus 1 (HSV-1), HSV-2, and varicella-zoster virus (VZV) to the BD Max system by usi
250                                   Studies of varicella-zoster virus (VZV) tropism for T cells support
251                                              Varicella-zoster virus (VZV) vaccine appears to be safe
252                                   Although a varicella-zoster virus (VZV) vaccine has been used for m
253 ldhood immunization with the live-attenuated varicella-zoster virus (VZV) vaccine induces protective
254  of a high-potency live-attenuated Oka/Merck varicella-zoster virus (VZV) vaccine.
255 75 years) immunized with the live-attenuated varicella-zoster virus (VZV) vaccine.
256                                              Varicella-zoster virus (VZV) vasculopathy produces strok
257 ent of active lesions (e.g. HSV-1, HSV-2 and varicella-zoster virus (VZV)).
258                                              Varicella-zoster virus (VZV), a double-stranded DNA alph
259 igated during the entire infectious cycle of varicella-zoster virus (VZV), a human herpesvirus.
260     The immediate early 62 protein (IE62) of varicella-zoster virus (VZV), a major viral trans-activa
261                       Primary infection with varicella-zoster virus (VZV), a neurotropic alphaherpesv
262          Regulation of gene transcription in varicella-zoster virus (VZV), a ubiquitous human neurotr
263 1) and pseudorabies virus (PRV) and ORF66 in varicella-zoster virus (VZV), affects several viral and
264                          Here we report that varicella-zoster virus (VZV), an alphaherpesvirus that i
265 pear healthy at 2 weeks after infection with varicella-zoster virus (VZV), and the cell culture mediu
266 ovirus, herpes simplex virus type 1 (HSV-1), varicella-zoster virus (VZV), and West Nile virus (WNV).
267 sviruses, herpes simplex virus 1 (HSV-1) and varicella-zoster virus (VZV), confirmed the expression o
268 ults for herpes simplex virus 1/2 (HSV-1/2), varicella-zoster virus (VZV), cytomegalovirus (CMV), or
269 icella virus (SVV), the counterpart of human varicella-zoster virus (VZV), developed primary infectio
270     In this study, quantitative PCR detected varicella-zoster virus (VZV), herpes simplex virus 1 (HS
271                                              Varicella-zoster virus (VZV), in both wild-type and live
272 ing the medically important alphaherpesvirus varicella-zoster virus (VZV), induce fusion of the virio
273                                              Varicella-zoster virus (VZV), of the family Alphaherpesv
274 showed cytopathic changes, but HSV-1, unlike varicella-zoster virus (VZV), only rarely infected satel
275                                         Like varicella-zoster virus (VZV), simian varicella virus (SV
276 nation was more likely to identify wild-type varicella-zoster virus (VZV), whereas the presence of Ok
277                    Thus, we examined whether varicella-zoster virus (VZV)-infected cells produce amyl
278                                           In varicella-zoster virus (VZV)-infected primary human brai
279  the risk of herpes zoster (HZ), we compared varicella-zoster virus (VZV)-specific and nonspecific T-
280                                              Varicella-zoster virus (VZV)-specific cell-mediated immu
281 n association with an age-related decline in varicella-zoster virus (VZV)-specific cell-mediated immu
282 udy were to evaluate the association between varicella-zoster virus (VZV)-specific humoral and cell-m
283                                   To measure varicella-zoster virus (VZV)-specific immune responses u
284 ssays and flow cytometry, we determined that varicella-zoster virus (VZV)-specific peak T helper 1 (V
285 itive GCA is associated with TA infection by varicella-zoster virus (VZV).
286 rus (CMV), herpes simplex viruses (HSV), and varicella-zoster virus (VZV).
287 rpes zoster caused by reactivation of latent Varicella-Zoster virus (VZV).
288 ly recognized component of the life cycle of varicella-zoster virus (VZV).
289 lication that can occur with reactivation of varicella-zoster virus (VZV).
290  potency against hepatitis B virus (HBV) and varicella-zoster virus (VZV).
291                      Our work has shown that varicella-zoster virus (VZV; also called human herpesvir
292               None have yet been reported in varicella-zoster virus (VZV; also known as human herpesv
293     Immunity to measles, mumps, rubella, and varicella-zoster viruses (VZV; MMRV) is a common conditi
294 onal and pathogen-specific stimulation (with varicella-zoster virus [VZV] and cytomegalovirus [CMV]).
295  virus 1 [HSV-1], HSV-2, JC virus [JCV], and varicella-zoster virus [VZV]).
296 ion (PCR) for herpes simplex virus (HSV) and varicella zoster virus was done in 237 (69%) and 82 (24%
297                                    HHV-1 and Varicella-Zoster virus were detected only twice and HHV-
298 ty for CMV, EBV, herpes-simplex virus 1, and varicella-zoster virus were studied in 1079 6-year-old c
299 ncing to identify nosocomial transmission of varicella-zoster virus with fatal outcome.
300 nate gene activation by live yellow-fever or varicella-zoster virus (YFV/VZV) vaccines was more suspe

 
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