ライフサイエンスコーパス: varicosity

1 DCS (this ratio is 0.50 for axons, 0.55 for varicosities).

2 2 weeks without changing the total number of varicosities.

3 and a slight decrease in the total number of varicosities.

4 within EYFP-positive or EYFP-negative axonal varicosities.

5 , largely because of reduced addition of new varicosities.

6 were clustered opposite synapsin-containing varicosities.

7 vidence for functional NMDARs in basket cell varicosities.

8 ability and causing calcium influx at axonal varicosities.

9 tic specializations opposite to their axonal varicosities.

10 as well as contorted axons with Tau-positive varicosities.

11 ong fibers characterized by regularly spaced varicosities.

12 n, vamp2, was present in a fraction of those varicosities.

13 d axon terminals exhibited numerous terminal varicosities.

14 GABA(B) receptors in a subpopulation of its varicosities.

15 ication of SIV adjacent to catecholaminergic varicosities.

16 ning-related formation of new sensory neuron varicosities.

17 s in retinal arbors with smaller than normal varicosities.

18 totagmin and SNAP-25 were localized to nerve varicosities.

19 of excitatory-like, presumably glutamatergic varicosities.

20 h or sparsely spiny, but never bear distinct varicosities.

21 r and these depolarizations propagate to the varicosities.

22 ific clusters of dense terminal branches and varicosities.

23 d convergent synapses from TH-immunoreactive varicosities.

24 he numerical density of the nLOT cholinergic varicosities.

25 idine that measure cardiac sympathetic nerve varicosities.

26 rons and vasoactive intestinal peptide (VIP) varicosities.

27 retracting synaptic branches and presynaptic varicosities.

28 re accompanied by the emergence of dendritic varicosities.

29 n the superficial layers of area TE and 8.74 varicosities/100 microm in the deep layers.

30 Labeled axons demonstrated 11.67 varicosities/100 microm on average in the superficial la

31 In area V1 we observed 8.24 varicosities/100 microm.

32 ENK colocalized with VGLUT2 in up to 77% of varicosities (17 +/- 21%, n = 21 neurons).

33 7% of somatostatin (SOM)-immunoreactive (IR) varicosities; 20 +/- 4.3% of substance P (SP)-IR varicos

34 holine transporter-immunoreactive (VAChT-IR) varicosities (80% +/- 1.7%, n = 4, P < 0.001) contained

35 sicular acetylcholine transporter (VAChT)-IR varicosities (88 +/- 3%, P < 0.001).

36 Among alpha-synuclein-IR varicosities, 96% +/- 0.9%, 99% +/- 0.6%, 83% +/- 1.9%,

37 tagmin-1-, synaptobrevin-2-, and CSPalpha-IR varicosities, 98% +/- 0.7%, 96% +/- 0.7%, 88% +/- 1.6%,

38 in (5HT) triggered growth of new presynaptic varicosities, a synaptic mechanism of long-term sensitiz

39 ter fibers giving rise to numerous spherical varicosities abutting type II hair cells and afferent pr

40 arget-dependent increases in axon growth and varicosities accompany the formation of functional synap

41 ould be differences in the density of axonal varicosities across regions, and that these differences,

42 es in synaptic structure between cholinergic varicosities activating alpha7 and non-alpha7 classes of

43 ities along dendrites, decreases the size of varicosities after sublethal NMDA exposure, and protects

44 neurons with varicosities and the number of varicosities along dendrites, decreases the size of vari

45 More than half of presynaptic varicosities along its cholinergic axons make traditiona

46 he spinal cord, an increase in 5-HT-positive varicosities along motor-associated cholinergic neurons

47 imes upon entering the bladder and developed varicosities along their axon terminal endings.

48 toward the outer retina, exhibiting numerous varicosities along their course.

49 ns and the relative distribution of efferent varicosities among hair cells and afferents are also int

50 cosities; 20 +/- 4.3% of substance P (SP)-IR varicosities and 9 +/- 1.3% vasoactive intestinal polype

51 n of the newly synthesized sensorin from the varicosities and activation of the autocrine responses o

52 osis of the dense-cored vesicles from axonal varicosities and acts by diffusion-a localized volume tr

53 have a high number of densely packed, large varicosities and an extensive rostrocaudal (two or three

54 Multifocal axonal varicosities and axonal loss were found in deep cortex a

55 ng extensive process outgrowth with numerous varicosities and expression of neuronal and synaptic mar

56 and close appositions between PNMT-ir axonal varicosities and orexin-ir profiles were observed.

57 othalamic projections contain small synaptic varicosities and other features that resemble the modula

58 synapses between presynaptic climbing fiber varicosities and postsynaptic Purkinje cell spines and e

59 th of new neuritic processes and presynaptic varicosities and retracted growth at the highest LPA con

60 occur via a loose association between nerve varicosities and smooth muscle cells.

61 tion in the number of inhibitory perisomatic varicosities and synaptic GAD65/67 immunoreactivities in

62 ccompanied by the maintenance of presynaptic varicosities and target-dependent regulation of mRNA dis

63 c strength and enhanced the formation of new varicosities and target-dependent regulation of mRNA dis

64 We also found that the distribution of axon varicosities and terminal field locations show substanti

65 Axons from the medial CeA formed numerous varicosities and terminals enveloping the HSD2 neurons.

66 otably, capsids acquired envelopes at axonal varicosities and terminals where the sites forming synap

67 own increases the percentage of neurons with varicosities and the number of varicosities along dendri

68 laced in a region of the muscle containing a varicosity and held at a potential sufficient to oxidize

69 and notably, capsids are enveloped at axonal varicosity and terminals, which are the sites forming sy

70 dendritic damage (characterized by swellings/varicosities), and dysregulated neuronal excitability (d

71 sis-dependent LTF, growth of new presynaptic varicosities, and activation of MAPK and its translocati

72 s were associated with enkephalin-containing varicosities, and enkephalin-induced clathrin- and dynam

73 e neuronal activities including MIP/SIFamide varicosities, and leads the contraction of myoepithelial

74 aintenance of synaptic efficacy, presynaptic varicosities, and mRNA distributions.

75 immunoreactivity was observed in processes, varicosities, and neuronal cell bodies of the olfactory

76 c filopodia, elaboration of undifferentiated varicosities, and potentiation of spontaneous release fr

77 the growth and formation of new presynaptic varicosities, and the target-dependent regulation of mRN

78 ehavioral training), approximately 10% of PF varicosities appeared and disappeared over a period of 2

79 Importantly, EM2 and glutamate-containing varicosities appose spinal neurons that express MOR alon

80 d the nerve terminals change similarly to be varicosities apposed to these islands.

81 quired for PF-PC LTP, although noradrenergic varicosities are apposed in PF-PC contacts.

82 ansients evoked by release of ATP from nerve varicosities are elementary signals in the process of ne

83 We further show that axonal varicosities are induced by persistent Ca(2+) increase,

84 f long-term memory during which newly formed varicosities are labile and require sustained CPEB-depen

85 eflect changes in synaptic density, but axon varicosities are likely to be the most sensitive anatomi

86 These isolated varicosities are not found along particular topological

87 Some DLCST axon arbors with varicosities are seen near large neurons in the ventral

88 us but not in dendrites, indicating that the varicosities are sites of dopamine release.

89 These varicosities are thought to form largely nonjunctional-t

90 ermore, the number of synaptic-like terminal varicosities around PNs was decreased.

91 identical pathologic increases in dendritic varicosities as seen in Tat transgenic mice in vivo.

92 ar structures within axonal growth cones and varicosities as well as at axonal branch points in cultu

93 ine, were registered at noradrenergic axonal varicosities as well as at cell bodies.

94 onin appear to be selective for serotonergic varicosities, as dopaminergic and corazonergic varicosit

95 arkers accumulated abnormally in YFP-labeled varicosities associated with neuritic plaques.

96 rain, we find evidence for avoidance between varicosities at distances lower than 1.75 microm.

97 al neurons induced the formation of numerous varicosity-bearing tortuous processes, as well as the co

98 ellular injections of Neurobiotin to examine varicosities belonging to heart excitor (HE) neurons on

99 tic neuron, being present in spine-targeting varicosities but distinctly absent from those synapsing

100 ed by targeted retraction of the 5HT-induced varicosities but, rather, by an apparently arbitrary ret

101 presynaptic membrane were absent from DA-IPC varicosities, but the vesicular SNARE protein, vamp2, wa

102 rons from 6.0 +/- 0.9 to 2.3 +/- 0.4 CART-IR varicosities/cell.

103 The latter formed dense, basket-like varicosity clusters (CGRP+ baskets) that enveloped myent

104 itation in older kittens, corticospinal axon varicosities colocalize synaptophysin like adults, sugge

105 cantly higher proportions of CALR+ and CGRP+ varicosities colocalized in baskets than in circular mus

106 microscopic examination of a sample of these varicosities confirmed that labeled boutons formed synap

107 s that 5-HT activates ApCdc42 in a subset of varicosities contacting the postsynaptic motor neuron an

108 1 +/- 0.3% of biotinamide-labeled extrinsic varicosities contained alpha-synuclein-immunoreactivity

109 Within RTN, 51% of BDA-labelled axonal varicosities contained detectable levels of vesicular gl

110 d contorted or fragmented axons with swollen varicosities containing alphaSyn and Tau.

111 However, densities of terminal varicosities correlate with recovery of swimming behavio

112 icroscopy, we determined that mCherry-VGluT2 varicosities correspond to axon terminals, forming asymm

113 otential-evoked calcium transients in axonal varicosities, demonstrating the effectiveness of activat

114 Noticeably, the VGluT2+ and VIAAT+ varicosity density in Mo7 is 5-fold higher than in Mo5 a

115 excess exogenous serotonin decreases native varicosity density in older larvae, and these acute effe

116 aricosities increase substantially while the varicosity density remains relatively constant.

117 rotonin is an autoregulator for serotonergic varicosity density.

118 n terminal and preterminal branching, and in varicosity density.

119 mation is complete, motor neuron presynaptic varicosities develop into large bulbous protrusions that

120 Fewer varicosities developed in acutely treated mice while mor

121 rthermore, calcium indicators in basket cell varicosities did not report any change in intracellular

122 TRPCs) was similarly absent, although DA-IPC varicosities did show ryanodine receptor immunoreactivit

123 Histidine decarboxylase-containing varicosities diffusely innervate striatal and mesolimbic

124 These anterogradely labeled varicosities displayed synaptophysin immunoreactivity, i

125 y of substance P (Sub P) to induce dendritic varicosities (DVs) or beads in neurons of the rostral ve

126 hese afferent axons provide numerous swollen varicosities, each presynaptic to many small profiles, a

127 k LTF and the rapid synthesis of sensorin at varicosities even in the absence of sensory neuron cell

128 the largest number of anterogradely labeled varicosities followed by vesicular GABA transporter (VGA

129 Focal swelling or varicosity formation in dendrites and loss of dendritic

130 synapse maturation and curtailed growth and varicosity formation of sensory neurons contacting L7, b

131 own that microtubule dynamics play a role in varicosity formation, very little is known about the pro

132 env, gag, and nef RNA, and catecholaminergic varicosities from the ANS were mapped by sucrose phospha

133 Unlike mature boutons, new varicosities have synaptic vesicles which are distribute

134 tween the absolute density of axons and axon varicosities; however, the ratio measures are strongly c

135 Among 1,136 DbH-positive varicosities identified within the parvocellular PVN in

136 ddressed was whether distally located DA-IPC varicosities, identified by tyrosine hydroxylase (TH) im

137 mate the number of motoneurons as well as of varicosities immunopositive for glutamate (VGluT1+, VGlu

138 remarkably high density of axons and axonal varicosities immunoreactive for serotonin (5-HT) and ore

139 erve endings from mouse cortex and in intact varicosities in a neuronal cell line using fluorescence

140 se of NET in endosomes of single boutons and varicosities in a Rab11-dependent manner.

141 ined that at least 55% of the tracer-labeled varicosities in areas TE and V1 colocalized synaptophysi

142 s of the hypothalamus (PVH), with fibers and varicosities in close apposition to a subset of melanoco

143 at the release face of bouton-like synaptic varicosities in contact with muscle cells, and lowest in

144 the number and size of glutamatergic axonal varicosities in cortical cultures.

145 d stimulation of glutamate receptors induces varicosities in dendrites but not in axons.

146 % vasoactive intestinal polypeptide (VIP)-IR varicosities in guinea pig rectal myenteric ganglia.

147 We found more 5-HT-positive varicosities in lamina X adjacent to central canal clust

148 71 +/- 0.8% of VAChT-IR varicosities in myenteric ganglia of human colon were al

149 tic bouton number and the ability to bud new varicosities in response to acute neuronal stimulation.

150 versican promotes enlargement of presynaptic varicosities in retinal axons.

151 We found that CHT-immunoreactive axon varicosities in the AVN displayed a smaller cross-sectio

152 that mechanical stress specifically induces varicosities in the axons but not the dendrites of centr

153 be the neuropil distribution of serotonergic varicosities in the brain and ventral nerve cord (VNC) o

154 studies of individual central vagal afferent varicosities in the caudal brainstem slice preparation s

155 nch contains large numbers of vesicles, with varicosities in the close vicinity of Purkinje dendrites

156 dritic arborization, and extensive dendritic varicosities in the cortical neurons of CM-infected brai

157 fixatives induce the formation of bead-like varicosities in the dendrites and axons of brain and spi

158 release from exocytosis events at individual varicosities in the Drosophila larval system by amperome

159 een in fiber staining which may reflect true varicosities in the fiber or simply varying densities of

160 CPA resulted in numerous labeled fibers with varicosities in the ipsilateral subnucleus reticularis d

161 m to examine the development of serotonergic varicosities in the larval CNS.

162 We found that TH immunoreactive varicosities in the outer retina possessed vesicular mon

163 ced the appearance of NGF-receptor-IR axonal varicosities in the rat medial septum.

164 escence revealed a mixture of fine and thick varicosities in the SAP + IG but only fine fibers in con

165 the turnover of dendritic spines and axonal varicosities in the somatosensory cortex of mice lacking

166 findings, the density of 5-HT-immunoreactive varicosities in the superficial dorsal horn of the spina

167 OP) on substance P (SP)-immunoreactive (-ir) varicosities in the superficial dorsal horn of the spina

168 eurites/terminal swellings and vesicle-laden varicosities in the synaptic neuropil.

169 on between aggression and the number of axon varicosities in the telencephalic region proposed to be

170 Moreover, the number of new mossy fiber varicosities in these parts of the cerebellar nuclei is

171 ble, consistent with the formation of axonal varicosities in vivo induced by mechanical impact in a m

172 e located close to the plasmalemma of axonal varicosities in which no electron-lucent microvesicles o

173 real density of cholinergic terminals (fiber varicosities) in the dentate gyrus.

174 Terminal varicosities increase in density rostral to the lesion s

175 e of the neuropil and number of serotonergic varicosities increase substantially while the varicosity

176 ound that the production of peptide-positive varicosities increased considerably during the last week

177 ppearance, a few fibers with en passant type varicosities (indicating synapses) were observed in the

178 fibers exhibited excitatory, vGLUT2-positive varicosities, indicating their synaptic integration into

179 ve retraction of newly formed sensory neuron varicosities induced by 5-HT.

180 the associated growth of new sensory neuron varicosities induced by repeated pulses of serotonin (5-

181 mitter from vesicles that accumulate in axon varicosities induces a local rise in cytoplasmic calcium

182 Ca(2+) transients in other varicosities initiated intracellular Ca(2+) waves in adj

183 Finally, axonal varicosity initiation can trigger action potentials to a

184 ere assessed at 6 weeks for need for further varicosity intervention, which was completed with either

185 ly via the conversion of some preexisting PF varicosities into multisynaptic terminals.

186 e that, in the retina, dopamine synthesis in varicosities is affected by the spiking activity of reti

187 rite extension, and formation of presynaptic varicosities is displayed by photoreceptors after retina

188 The density of DbetaH varicosities is greater in the prefrontal cortex than in

189 The ratio of varicosities is stable over a greater than seven-fold ra

190 onfocal imaging of individual sensory neuron varicosities labeled with three different fluorescent ma

191 ncluding cosmetic spider veins, asymptomatic varicosities, large painful varicose veins, edema, hyper

192 Presynaptic axonal varicosities, like postsynaptic spines, are dynamically

193 re characterized by contorted processes with varicosity-like structures, some containing both alpha-s

194 r in striatal dopaminergic presynaptic nerve varicosities, making them unusually susceptible to inhib

195 apse, with larger varicosity size but normal varicosity number, indicating that these synaptic parame

196 Few NOS+ varicosities occurred in baskets.

197 agonist-evoked influx of Ca(2+) in dendritic varicosities of A17 amacrine cells from diabetic compare

198 ARs elicited small Ca(2+) transients in axon varicosities of cerebellar stellate cell interneurons.

199 estine (ICC-DMP) are closely associated with varicosities of enteric motor neurons and generate respo

200 lpha(+) ) cells, are closely associated with varicosities of enteric motor neurons and suggested to m

201 was localized in restricted fashion to axon varicosities of neurons recorded from laminae II-V, alth

202 Moreover, ATRP is present in peripheral varicosities of protraction motoneurons and enhances per

203 14 was used to stain thin axons and terminal varicosities of the crayfish neuromuscular junction.

204 light or GABA/glycine inhibitors but only in varicosities of the DA axon plexus, not in perikarya or

205 as shown by immunocytochemistry to reside in varicosities of the DA plexus but not in dendrites, indi

206 ularity is mirrored in the spacing of axonal varicosities of the stratified bipolar cells, which have

207 Outer retinal varicosities of this putative GABAergic IPC did colocali

208 ited by NT-3, without affecting the synaptic varicosity of the presynaptic terminals.

209 correlated with ongoing Ca(2+) transients in varicosities on the axons of presumably inhibitory motor

210 lesioned side, as well as the number of CART varicosities on the surface of TRH neurons from 6.0 +/-

211 15 +/- 1.4% of biotinamide-labeled extrinsic varicosities; only 1 +/- 0.3% of biotinamide-labeled ext

212 ributed upon stimulation toward the sites of varicosity outgrowth.

213 s capable of rapidly budding new presynaptic varicosities over the course of minutes in response to e

214 .9-fold in the vicinity of catecholaminergic varicosities (P < 0.0001).

215 es at 9 months further reduced the number of varicosities/PN.

216 to quantify the density of axons and axonal varicosities present in DCS (the latter represent presyn

217 to quantify the density of axons and axonal varicosities present in the FDLS.

218 E neurons still resulted in peptide-positive varicosity production.

219 ricosities, as dopaminergic and corazonergic varicosities remain qualitatively intact following serot

220 That these ChAT-positive varicosities represent presynaptic release sites were de

221 se axons always exhibited varying numbers of varicosities representing axon terminals.

222 acquire the more stable properties of mature varicosities required for the persistence of LTF.

223 developed in acutely treated mice while more varicosities resolved in mice with delayed treatment.

224 VGluT1+, VGluT2+, and VIAAT+ varicosities respectively represent: 28%, 41%, and 31% i

225 are composed of a sequence of many beads or varicosities separated by intervaricose segments.

226 ndergo degeneration with formation of axonal varicosities sequestering transported proteins and progr

227 The fraction of dynamic PF varicosities significantly diminished during training on

228 ructural defects at the synapse, with larger varicosity size but normal varicosity number, indicating

229 ontrast, VGluT2 expression was restricted to varicosities, some of them coexpressing mCherry.

230 ry and higher-order dendrites exhibited rich varicosities, sometimes with dendritic spines.

231 l characteristics of CHT-immunoreactive axon varicosities specifically within the anteroventral thala

232 jal (ICC-IM) are associated with cholinergic varicosities, suggesting a role in mediating excitatory

233 matched by activity in closely apposed nerve varicosities, suggesting EGCs were not only innervated b

234 Newly formed branches presented varicosities, suggesting that new axons were more than j

235 Also, somatic versus neuropil location of varicosities suggests that most of these afferents are i

236 enteric neurons, extensive OT-immunoreactive varicosities surrounded many others.

237 d in nerve trunks attached to the IMG and in varicosities surrounding IMG neurones.

238 mber of delta-opioid receptor-immunoreactive varicosities that appose the postsynaptic membrane of th

239 This pattern produces many varicosities that are clustered together but also includ

240 re clustered together but also includes some varicosities that are very isolated.

241 s formed "axonal cartridges," with clustered varicosities that contacted the axon initial segments of

242 in contact with muscle cells, and lowest in varicosities that did not contact muscle cells.

243 Of SOM-IR, SP-IR, and VIP-IR varicosities that lacked VAChT-immunoreactivity, only 1

244 nt neurons give rise to numerous presynaptic varicosities that target hair cells and afferent process

245 itative changes in nerves including enlarged varicosities that were also seen following capsaicin and

246 ely localized to a limited population of its varicosities, the majority of likely synaptic-release si

247 examine the spatial pattern of serotonergic varicosities, the synaptic sites of serotonin release in

248 al layers and extends long processes without varicosities to cortical layers 3 and 4.

249 ion of PKM--caused the number of presynaptic varicosities to revert to the original, pretraining valu

250 We tested the sensitivity of serotonergic varicosities to serotonin by adding neurotransmitter at

251 expressed by genetic manipulation in Type II varicosities to study octopamine release in Drosophila.

252 haft atrophy, and nearby axons develop large varicosities, together leading to neurite breakage and l

253 Timing of varicosity treatment is controversial with delayed and s

254 o either simultaneous phlebectomy or delayed varicosity treatment.

255 hibiting rapid elongation and retraction and varicosity turnover.

256 s, characterized by an overextended synaptic varicosity, underdeveloped synaptic morphology and disru

257 e occurrence of immunolabeling in individual varicosities varied widely between cells (39 +/- 36%, n

258 The numerical density of cholinergic varicosities was also unchanged in aged rats.

259 Formation of varicosities was frequently preceded or accompanied by h

260 found that the density of cholinergic fiber varicosities was higher in epileptic rats versus control

261 The density of labeled varicosities was highest in the pons and lowest in MI.

262 mine beta-hydroxylase (DbetaH)-immunostained varicosities was performed on several cortical regions a

263 ed tonic, and production of peptide-positive varicosities was substantially reduced as well.

264 get region, a large percentage of the BDA-ir varicosities was VGLUT2-ir (41-83%).

265 naptotagmin and SNAP-25-immunopositive nerve varicosities were concentrated in varicose regions of mo

266 ive synaptic contacts evident as bouton-like varicosities were detected in close apposition to latera

267 Although some BDA-labeled axons with varicosities were found bilaterally at all cervical leve

268 BDA-ir varicosities were found in the solitary tract nucleus (N

269 Serotonin fibers and varicosities were found throughout the spinal cord and w

270 cells, and most of the ChR2-positive axonal varicosities were immunoreactive for vesicular glutamate

271 Quantitative analysis revealed CGRP+ varicosities were most abundant in baskets, followed by

272 V1-derived axonal varicosities were mostly (>80%) glycinergic and a third

273 Numerous VGAT and VGLUT2 labeled varicosities were observed apposed to dDpMe-labeled axon

274 The highest densities of C3 axon varicosities were observed in Lamina X and the intermedi

275 rve fibers until P14, when nerve fibers with varicosities were observed in the urethra and bladder ne

276 The less common Orx varicosities were occasionally apposed to TH- or GABA-la

277 opulation, nitrergic subpopulation and (VIP) varicosities were reduced.

278 opulation, nitrergic subpopulation and (VIP) varicosities were reduced.

279 part of the nucleus, orexin-A-immunopositive varicosities were relatively more abundant, located in c

280 Some double-labeled BDA/VGAT varicosities were seen apposed to small somata labeled f

281 Varicosities were seen in fiber staining which may refle

282 e DMV >95% mCherry-immunoreactive(ir) axonal varicosities were tyrosine hydroxylase (TH)-ir and the s

283 NA varicosities were visualized by immunoperoxidase labelin

284 Efferent fibers and varicosities were visualized in the semicircular canal o

285 and internalized mostly in growth cones and varicosities, where synaptic vesicle markers were also c

286 cells significantly increased the number of varicosities, whereas cone cells increased process growt

287 bindin, are colocalized with the presynaptic varicosities, whereas gephyrin, Na-K-2Cl cotransporter 1

288 ated via the release of dopamine from nearby varicosities, which in turn leads to potentiation of the

289 ated via the release of dopamine from nearby varicosities, which in turn led to potentiation of the s

290 ncrease in sensorin synthesis, especially at varicosities, which precedes the secretion of sensorin.

291 hine treatment accelerated Tat-induced focal varicosities, which were accompanied by localized increa

292 butyric acid membrane transporter 1-positive varicosities, whose appearance resembles chandelier cart

293 GAC dendritic tree and endowed all dendritic varicosities with a small-center, strong-surround recept

294 in axon growth, the number of sensory neuron varicosities with release sites contacting the target, a

295 tion of delta-opioid receptor-immunoreactive varicosities with the labeling of the GABA-synthesizing

296 the ultrastructural relationship of NA axon varicosities with the somata and dendrites of identified

297 most abundant in baskets, followed by CALR+ varicosities, with a high degree of colocalization betwe

298 Sh) and the presence of orexin receptors and varicosities within the AccSh, we hypothesized that orex

299 can modulate the development of serotonergic varicosities within the fly central nervous system.

300 igantocellular preoptic area (gPOA) and axon varicosities within the ventral nucleus of the ventral t