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1 he ECM and altered formation of the coronary vasa vasorum.
2 use ECs line the vascular epithelium and the vasa vasorum.
3  from the development of immature neointimal vasa vasorum.
4 complete tissue integration and formation of vasa vasorum.
5 osclerotic plaque and associated adventitial vasa vasorum.
6 s the proliferation and intimal extension of vasa vasorum.
7 atherogenic diet, suggestive of newly formed vasa vasorum.
8 ural changes, including the formation of new vasa vasorum.
9 ssels, with proportionally more second-order vasa vasorum.
10 etween VEGF/VPF immunostaining and extent of vasa vasorum.
11 the function and dysfunction of the arterial vasa vasorum.
12 espectively, P < 0.01) and in the density of vasa vasorum (1.84+/-0.05/mm2 vs. 4.73+/-0.24/mm2; respe
13 ular adipose tissue promote expansion of the vasa vasorum, activation of fibroblasts, and differentia
14 ro-computed tomography techniques that image vasa vasorum anatomy in relation to the atheroma.
15 ed especially by an increase of second-order vasa vasorum and disorientation of normal vasa vasorum s
16 ansplanted islets received blood supply from vasa vasorum and had access to drainage through venous t
17 populations comprise and cohabitate with the vasa vasorum and how they might contribute to physiologi
18                   We observed more extensive vasa vasorum and intimal neovascularization in knockout
19 ain direct access to the artery wall via the vasa vasorum and may initiate, promote, and destabilize
20 imal graft manipulation with preservation of vasa vasorum and nerves) reduces the risk of SVG failure
21 cytes and T lymphocytes, and the role of the vasa vasorum and surrounding perivascular adipose tissue
22       IP NCs arose directly from adventitial vasa vasorum and were anatomically and quantitatively re
23 f helping detect and even grade intracranial vasa vasorum, and this may provide new insights into our
24                                          The vasa vasorum are a vital microvascular network supportin
25                      The cellular origins of vasa vasorum are ill-defined and may involve circulating
26 nd with the development of vascular disease, vasa vasorum are known to develop.
27         Macrophages in the plaque and around vasa vasorum are reduced, but we detect no direct effect
28 onstrate that rPAI-1(23) treatment decreased vasa vasorum area and length, which was supported by mic
29           Contrary to the prevailing view of vasa vasorum as solely adverse, this study underscores t
30 hat the coronary vessel wall, especially the vasa vasorum, as well as bone marrow-derived endothelial
31 In conclusion, rPAI-1(23) inhibits growth of vasa vasorum, as well as vessels within the adjacent pla
32 dventitia, particularly within microvessels (vasa vasorum) but not in cells of the intima or media.
33  rPAI-1(23)-stimulated mechanisms that cause vasa vasorum collapse.
34  reconstructed confocal microscopy images of vasa vasorum demonstrate that rPAI-1(23) treatment decre
35                                   We observe vasa vasorum density correlates highly with the extent o
36 prevents the increase in VEGF expression and vasa vasorum density of coronary arteries in experimenta
37                                              Vasa vasorum density was higher in the HC group compared
38 lloon-injured coronary arteries, adventitial vasa vasorum density was increased (3.16+/-0.17/mm2 vs.
39 nned, and reconstructed, and quantitation of vasa vasorum density was performed.
40                              Plaque size and vasa vasorum density were compared to 2 controls: mice f
41  from the artery lumen and outer adventitial vasa vasorum, deposit proatherogenic plasma molecules, r
42 a and disarray, and stenotic arteries in the vasa vasorum due to medial SMC proliferation.
43 are also vasculogenic and may be a source of vasa vasorum during atherogenesis.
44 ccount for the relative lack of intracranial vasa vasorum early in life.
45 culture adventitial fibroblasts (AdvFBs) and vasa vasorum endothelial cells (VVECs) from the adventit
46 ic cells and macrophages), progenitor cells, vasa vasorum endothelial cells and pericytes, and adrene
47 Myddosome signaling complex, proinflammatory vasa vasorum endothelial cells, and hyperactivated fibro
48                                              Vasa vasorum endothelium and contractile mural cells reg
49 ctivity was found in luminal and adventitial vasa vasorum endothelium.
50                                          The vasa vasorum function both as a master integrator of art
51                           Recent advances in vasa vasorum imaging for understanding cardiovascular di
52                         To inhibit growth of vasa vasorum in atherogenic mice and assess its effect o
53  Recent attention has focused on the role of vasa vasorum in atherosclerotic and restenotic coronary
54         The three-dimensional anatomy of the vasa vasorum in early coronary atherosclerosis is unknow
55 causes regression or collapse of adventitial vasa vasorum in hypercholesterolemic mice by stimulating
56 titate three-dimensional spatial patterns of vasa vasorum in normal and balloon injured porcine coron
57 ate the three-dimensional spatial pattern of vasa vasorum in normal and experimental hypercholesterol
58 ssessment of the therapeutic response of the vasa vasorum in patients with atherosclerotic plaque.
59 origin of plaque vasculature and the role of vasa vasorum in plaque growth.
60        We also studied the spatial growth of vasa vasorum in regions of neointimal formation.
61       Coronary arteries contain a network of vasa vasorum in the adventitia.
62 iew offers insight into the possible role of vasa vasorum in the development of intracranial vascular
63 These data demonstrate that formation of new vasa vasorum in vasculitis is regulated by inflammatory
64  arteries suggests that the formation of new vasa vasorum is determined by the nature of the immune r
65  coronary artery bypass grafting, injure the vasa vasorum, leading to treatment failures.
66 educed macrophage infiltration alongside neo vasa vasorum-like structure formation, reduced calcifica
67 ionally, neovascularization arising from the vasa vasorum may promote atherosclerotic plaque progress
68  and vasculogenesis potentiate mitigation of vasa vasorum-mediated contributions to cardiovascular di
69 es is accompanied by neovascularization from vasa vasorum microvessels extending through the tunica m
70 mplex layer of the vessel wall consisting of vasa vasorum microvessels, nerves, fibroblasts, immune c
71 mplex layer of the vessel wall consisting of vasa vasorum microvessels, nerves, fibroblasts, immune c
72                                              Vasa vasorum, microvessels supplying arterial walls, are
73 al nitric oxide synthase (due to ingrowth of vasa vasorum), neointima formation, and loss of smooth m
74 pport a role for the endogenous ET system in vasa vasorum neovascularization in early coronary athero
75 in (ET) receptor antagonism reduces coronary vasa vasorum neovascularization in experimental hypercho
76 , a promoter of adventitial inflammation and vasa vasorum neovascularization in experimental models o
77 hanistic role of the endogenous ET system in vasa vasorum neovascularization in hypercholesterolemia
78 Cs, in a process involving ET-1, to regulate vasa vasorum neovascularization occurring in the adventi
79 ed phases, the role of eccentric remodeling, vasa vasorum neovascularization, and mechanisms of plaqu
80  molecular mechanisms regulating adventitial vasa vasorum neovascularization, which occurs in the pul
81 ede "macrovascular endothelial dysfunction." Vasa vasorum neovascularization, with endothelial leakag
82 increased, suggesting a mechanistic role for vasa vasorum neovascularization.
83 n the coronary arterial wall as well as with vasa vasorum neovascularization.
84  the endothelial cell barrier of adventitial vasa vasorum networks marks Checkpoint 2; the invasion o
85                     Activation and growth of vasa vasorum occur in all blood vessel layers primarily
86 gests that adventitial neovascularization of vasa vasorum occurs in experimental hypercholesterolemic
87                                       Normal vasa vasorum originated from the coronary artery lumen,
88 ix can be imaged, as can angiogenesis of the vasa vasorum, plaque inflammation, and fibrin deposits o
89 act IA specimens, we identified an extensive vasa vasorum plexus in the IA wall.
90 growth factor-2 as mediators associated with vasa vasorum proliferation.
91 es of vasa vasorum were defined: first-order vasa vasorum ran longitudinally parallel to the vessel a
92                          Evidence of whether vasa vasorum remodeling incites or governs disease progr
93 on," which is composed of dysfunction of the vasa vasorum's endothelium as well as "microcellular end
94       CML was localized to aortic and aortic vasa vasorum smooth muscle but not to collagen or elasti
95 er vasa vasorum and disorientation of normal vasa vasorum spatial pattern.
96                     After 12 weeks, coronary vasa vasorum structure was assessed by three-dimensional
97 e findings highlight a more nuanced role for vasa vasorum, suggesting both adverse and protective rem
98 the quantitative response of the adventitial vasa vasorum to balloon-induced coronary injury.
99 -vessel vasculitis affecting branches of the vasa vasorum tree.
100                                          The vasa vasorum (vessels of vessels) are a dynamic microvas
101 to neovascularization in the coronary artery vasa vasorum (VV).
102                  The density of newly formed vasa vasorum was proportional to vessel stenosis (r = 0.
103 ntiangiogenic effect of TSP-1, the number of vasa vasorum was reduced in aortas from diabetic rats.
104                       Two different types of vasa vasorum were defined: first-order vasa vasorum ran
105                          In normal arteries, vasa vasorum were restricted to the adventitia, but in i
106                          Angiogenesis of the vasa vasorum, which are branches of bronchial arteries,
107 lammation; and to stimulate expansion of the vasa vasorum, which can act as a conduit for continued i
108  analyses show significant colocalization of vasa vasorum with calcifications, a strong correlation w
109 to support development and maturation of the vasa vasorum within varying plaque types.

 
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