ライフサイエンスコーパス: vascular lumen

1 tly regulate the exit of leukocytes from the vascular lumen.

2 tin sulfate, and hyaluronic acid) lining the vascular lumen.

3 n the perivascular space, but not within the vascular lumen.

4 space with occasional processes probing the vascular lumen.

5 eliver antithrombotic drugs to the pulmonary vascular lumen.

6 om the circulation and from the walls of the vascular lumen.

7 helium and no localization to the irradiated vascular lumen.

8 mor thrombi that had completely occluded the vascular lumen.

9 glycosylated molecules that project into the vascular lumen.

10 lear phagocytes and exclude cells within the vascular lumen.

11 ll viability and establishment of perfusable vascular lumens.

12 ulates angiogenic genes and the formation of vascular lumens.

13 esion, which together drive the emergence of vascular lumens.

14 me-dependent increase in staining within the vascular lumen after irradiation.

15 P-selectin is rapidly translocated to the vascular lumen after tissue injury to initiate the adhes

16 helial cell shape, and formation of a patent vascular lumen all require defined endothelial cell pola

17 are non-atherosclerotic projections into the vascular lumen and have been linked to up to one-third o

18 utrophils shed extracellular vesicles in the vascular lumen and that inhibition of extracellular vesi

19 xtracellular matrix component that coats the vascular lumen and, under normal conditions, restricts a

20 etween the endothelial cells, which line the vascular lumen, and associated mural cells, namely vascu

21 sed by 47 fold in the tissue surrounding the vascular lumen, as compared with non-targeted liposomes.

22 P-selectin localization to the vascular lumen at 6-24 h was, in part, associated with p

23 nging from focal intimal thickening to total vascular lumen blockade due to smooth muscle cell prolif

24 ve eicosanoids that are generated within the vascular lumen by leukocytes and transcellular biosynthe

25 his purpose if they could be anchored to the vascular lumen by targeting stably expressed, noninterna

26 The formation of vascular lumens by endothelial cells is a critical step

27 Endothelial cells that line the vascular lumen can express cell-surface proteins that ar

28 ion defects, regression of valves, and focal vascular lumen collapse, which triggered generalized lym

29 . Furthermore, mutants lacked blood flow and vascular lumen despite continuous peristaltic heartbeats

30 supposed to exert therapeutic action in the vascular lumen (e.g., antithrombotic proteins), to the s

31 ons in humans; the lesions display disrupted vascular lumens, enlarged capillary cavities, loss of pr

32 In-vivo, newly recruited cells on the vascular lumen express MC markers and at later times the

33 ferent peptides, screening noninvasively the vascular lumen for binding sites.

34 Here we review the recent literature on vascular lumen formation and compare it to lumen formati

35 during embryogenesis, including insufficient vascular lumen formation as well as defective arteriogen

36 that heterogeneous mechanisms contribute to vascular lumen formation in vivo.

37 herin in regulating endothelial polarity and vascular lumen formation is mediated through its interac

38 work has shown that FMNL3 is also needed for vascular lumen formation, a critical element of the form

39 the in vitro development of the vasculature (vascular lumen formation, size, distribution).

40 VE-cadherin impairs apicobasal polarity and vascular lumen formation.

41 cadherin to regulate apicobasal polarity and vascular lumen formation.

42 llular fusion of endothelial vacuoles drives vascular lumen formation.

43 Anchoring pharmacologic agents to the vascular lumen has the potential to modulate critical pr

44 s (WPBs) that release their content into the vascular lumen in response to specific agonists that rai

45 ation-induced P-selectin localization to the vascular lumen increased in time-dependent manner, until

46 oduction in the endothelial cells lining the vascular lumen is an important component of many vascula

47 on-induced localization of P-selectin to the vascular lumen is specific to the microvasculature of ma

48 lycosaminoglycan (GAG)-rich layer lining the vascular lumen, is associated with the onset of kidney i

49 loid beta (Abeta) peptide deposits along the vascular lumen, leading to degeneration and dysfunction

50 To determine whether radiation-induced vascular lumen localization of P-selectin was tumor type

51 radiation-induced P-selectin staining in the vascular lumen of neoplasms is associated with aggregati

52 were no adverse effects on the pigs and the vascular lumens of their vessels were well maintained in

53 ergo an early swelling with narrowing of the vascular lumen, resulting in prolonged renal hypoperfusi

54 hMVECs formed larger vascular lumen size than DPSC-ECs while the latter tende

55 f capillary lumens divided by length) and of vascular lumen/stroma ratio in the outer choroid were pe

56 ells (ECs) on the mouse lung, these ECs form vascular lumen structures and hypoxia upregulates PDGFB

57 step in the migration of leukocytes from the vascular lumen to the extravascular tissue, but fundamen

58 border of the choroid stroma (SCT) than the vascular lumen (VCT) or sclera (TCT).

59 and its primary branches with regard to the vascular lumen, vessel wall anatomy, and vessel wall ede

60 the opportunity to assess the progression of vascular lumen volume in vivo after balloon angioplasty.

61 P-selectin localization to the vascular lumen was present in all tumors and all species

62 Upon perfusing the vascular lumen with whole blood, the microengineered cap

63 liomas showed P-selectin localization to the vascular lumen within 1 h, whereas blood vessels in norm

64 odies in the endothelium, which moved to the vascular lumen within 30 min after irradiation.

65 was accompanied by PMN degranulation within vascular lumen without PMN transmigration, likely becaus