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1 lar function include more than production of vasoactive substances.
2 othelial cells targeted by mast cell-derived vasoactive substances.
3 rum and tissue ACE activity as well as other vasoactive substances.
4 alyzed by the cytochrome P450 system and are vasoactive substances.
5 activating mast cells, which in turn release vasoactive substances.
6 nd alter the metabolism of a number of other vasoactive substances.
7 ssible by the ability of RBCs to release the vasoactive substance adenosine triphosphate (ATP) in res
8 astrocytic activation, stimulates release of vasoactive substances and dilation of cerebral vasculatu
9 estration allowing for passage of diffusible vasoactive substances and interface of endothelial cell
12 he general measures, ventilation strategies, vasoactive substances, and surgical as well as mechanica
14 xocytosis of Weibel-Palade bodies, releasing vasoactive substances capable of causing vascular thromb
15 xocytosis of Weibel-Palade bodies, releasing vasoactive substances capable of causing vascular thromb
16 tions examined the endothelial production of vasoactive substances during heterologous serum exposure
17 of inflammatory mediators such as cytokines, vasoactive substances, free radicals, and chemokines, wh
18 odilatation is a consequence of release of a vasoactive substance from the endothelium owing to mecha
19 from nerve terminals and histamine and other vasoactive substances from granulocytes may be responsib
22 8 days after 6-OHDA) on the distribution of vasoactive substances in ECs of the rat thoracic aorta.
25 is a dynamic tissue, secreting and modifying vasoactive substances, influencing the behavior of other
28 and then assaying the conditioned media for vasoactive substances on isolated aortic rings and small
31 (K(ATP)) channel is targeted by a variety of vasoactive substances, playing an important role in vasc
32 latation also occurs during hypoxia, and the vasoactive substance(s) responsible for skeletal muscle
33 B breakdown after intravitreous injection of vasoactive substances shows that agents can be grouped b
34 egulated by the antagonistic balance between vasoactive substances such as NO and angiotensin II (Ang
35 lude that while astrocytes can still release vasoactive substances, vascular amyloid deposits render