ライフサイエンスコーパス: vasoconstriction

1 e ability of ATP to blunt alpha1 -adrenergic vasoconstriction.

2 the ability to attenuate alpha1 -adrenergic vasoconstriction.

3 e it is accompanied by reduced ET-1-mediated vasoconstriction.

4 ty, inhibition of transient BK currents, and vasoconstriction.

5 bral blood flow from hypoxia were related to vasoconstriction.

6 cy and efficacy without changing URP induced vasoconstriction.

7 at perivascular adipose tissue (PVAT) causes vasoconstriction.

8 f Ang II, and led to complete suppression of vasoconstriction.

9 linking GPR75 activation to 20-HETE-mediated vasoconstriction.

10 A1-3 agonist VPC31143 induced dose-dependent vasoconstriction.

11 t depolarize smooth muscle cells, leading to vasoconstriction.

12 hannels to cause membrane depolarization and vasoconstriction.

13 skeletal muscle to blunt alpha1 -adrenergic vasoconstriction.

14 zation that is required for pressure-induced vasoconstriction.

15 , which comprises bradycardia and peripheral vasoconstriction.

16 atment with an antioxidant regimen increased vasoconstriction.

17 in metabolic activation and blockade of skin vasoconstriction.

18 ivity are critically important for balancing vasoconstriction.

19 terial carbon dioxide (P aC O2) and cerebral vasoconstriction.

20 e; 5 microM) prevented acute hypoxia-induced vasoconstriction.

21 m and chloride delivery, leading to afferent vasoconstriction.

22 ed spontaneous neuronal activity rather than vasoconstriction.

23 mechanism capable of attenuating sympathetic vasoconstriction.

24 el blockers, such as 4-aminopyridine, induce vasoconstriction.

25 ation, which uncouples NO/Hb interaction and vasoconstriction.

26 level of sympathetic activity there is more vasoconstriction.

27 culature with an increased propensity toward vasoconstriction.

28 cular coupling response from vasodilation to vasoconstriction.

29 kinase activation, which conjointly trigger vasoconstriction.

30 ed contractions and increased URP-associated vasoconstriction.

31 s to the ability to blunt alpha1 -adrenergic vasoconstriction.

32 promotes smooth muscle cell contraction and vasoconstriction.

33 d to a marked impairment in reflex cutaneous vasoconstriction.

34 ole of astrocytes in PA flow/pressure-evoked vasoconstriction.

35 induction of inflammation and independent of vasoconstriction.

36 tiazem or nifedipine attenuated S1P-mediated vasoconstriction.

37 protein kinase A inhibitor peptide-mediated vasoconstriction.

38 ainst these alterations, including augmented vasoconstriction.

39 le cell (VSMC) TRPC3 channels participate in vasoconstriction.

40 erial innervation and sympathetic control of vasoconstriction.

41 W2871 each evoked modest afferent arteriolar vasoconstriction.

42 educed susceptibility to temperature-induced vasoconstriction.

43 exchange factor ARHGEF12 have lost myogenic vasoconstriction.

44 bile acid transfer and TC-induced placental vasoconstriction.

45 in arterial myocytes resulting in increased vasoconstriction.

46 owed the expected LOF effects in suppressing vasoconstriction.

47 s were examined during phenylephrine-induced vasoconstriction.

48 nal effectors in the development of arterial vasoconstriction.

49 ured blood vessels, are involved in arterial vasoconstriction.

50 responsible for triggering hypoxic pulmonary vasoconstriction.

51 te 20-hydroxyeicosatetraenoic acid-dependent vasoconstriction.

52 o mesenteric vessel muscle cells, leading to vasoconstriction.

53 jacent vascular smooth muscle cells, causing vasoconstriction.

54 ligatory role of AT1 Rb for pressure-induced vasoconstriction.

55 mesenteric and hindquarters, but not renal, vasoconstrictions.

56 the key signal mediating activity-dependent vasoconstrictions.

57 mild exercise did not attenuate PE-mediated vasoconstriction (-32 +/- 5 and -46 +/- 3%).

58 artery vascular resistance, fetal peripheral vasoconstriction, a reduction in oxygen delivery to the

59 unctional response, termed hypoxic pulmonary vasoconstriction, activates a multitude of pathways with

60 induced an ECM-like syndrome, causing brain vasoconstriction, adherence of activated leukocytes in t

61 e TEBV exhibited flow-mediated vasodilation, vasoconstriction after exposure to 1 muM phenylephrine a

62 scopy in vivo, we demonstrated a short-onset vasoconstriction after intrathecal injection of either P

63 ASMase-mediated microcirculatory vasoconstriction after SDRT conferred an ischemic stress

64 This dysfunctional state involves widespread vasoconstriction and a general disruption of neurovascul

65 We conclude that hypoxic pulmonary vasoconstriction and a profound catecholamine surge occu

66 al mechanisms by which uric acid could cause vasoconstriction and a progressive ateriolopathy were es

67 al inputs contribute to the daily control of vasoconstriction and blood pressure and suggest that clo

68 nates alpha1-adrenergic receptor (alpha1-AR) vasoconstriction and blood pressure homeostasis.

69 with respiratory failure may cause cerebral vasoconstriction and compromise brain tissue perfusion.O

70 uced potentiation of L-type Ca(2+) channels, vasoconstriction and decreased blood flow are prevented

71 ressurized cerebral arteries rapidly induced vasoconstriction and depolarized cVSMCs.

72 in a development of significantly increased vasoconstriction and endothelial dysfunction.

73 may have opposing roles in the regulation of vasoconstriction and endothelium-dependent vasorelaxatio

74 itric oxide (NO) in the vasculature, causing vasoconstriction and eventually cardiovascular complicat

75 and changes in blood markers associated with vasoconstriction and fibrinolysis, suggesting that OO su

76 s into the bursa of wild-type mice prevented vasoconstriction and follicle rupture.

77 -body cooling would elicit greater cutaneous vasoconstriction and greater increases in skin sympathet

78 in myocardial injury as a result of coronary vasoconstriction and heightened oxidative stress.

79 a form of fetal stress that stimulates renal vasoconstriction and ischaemia as a consequence of the p

80 larial is limited by its potent induction of vasoconstriction and its rapid degradation within minute

81 n both animal models, cocaine induced severe vasoconstriction and marked reductions in cerebral blood

82 We tested the hypothesis that muscle vasoconstriction and muscle sympathetic nerve activity (

83 ebrospinal fluid (CSF) is suggested to cause vasoconstriction and neuronal toxicity, and correlates w

84 oop that are critical for alpha1-AR-mediated vasoconstriction and PANX1 channel function.

85 bolism by hypoxia to acute hypoxic pulmonary vasoconstriction and progression of pulmonary hypertensi

86 However, it enhanced ET-1 vasoconstriction and prolonged the increase in blood pre

87 Terlipressin caused systemic vasoconstriction and pulmonary vasodilation in a porcine

88 ressin-analog, terlipressin induces systemic vasoconstriction and pulmonary vasodilation in a porcine

89 esized that serelaxin could ameliorate renal vasoconstriction and renal dysfunction in patients with

90 ding follicle rupture in wild-type mice, but vasoconstriction and rupture were absent in Amhr2(cre/+)

91 ntral thermoregulatory control, and prevents vasoconstriction and shivering in blocked areas.

92 n, synchronously reducing the thresholds for vasoconstriction and shivering.

93 y circulation to a low oxygen environment is vasoconstriction and structural remodelling of pulmonary

94 hese pathological responses promote regional vasoconstriction and subsequent blood vessel remodeling.

95 iated signaling plays a key role in myogenic vasoconstriction and that myogenic tone is required to m

96 -dependent contributions to reflex cutaneous vasoconstriction and vascular adrenergic sensitivity wer

97 isolated mouse arteries for effects on both vasoconstriction and vasodilation.

98 n VVS, as in hemorrhage, impaired adrenergic vasoconstriction and venoconstriction result in hypotens

99 amage, epicardial and microvascular coronary vasoconstriction and/or spasm, and increased cardiac wor

100 ticles that cause intrahepatic inflammation, vasoconstriction, and extrahepatic splanchnic vasodilati

101 and sEng result in endothelial dysfunction, vasoconstriction, and immune dysregulation, which can ne

102 inhibition promoted increased proliferation, vasoconstriction, and inflammation.

103 ing skeletal muscle to attenuate sympathetic vasoconstriction, and is critical to ensure proper blood

104 d hyperthermia by moderately inhibiting skin vasoconstriction, and labetalol was ineffective.

105 o TKI treatment by secreting EDN1, promoting vasoconstriction, and limiting blood and drug delivery.

106 in humans are sweating, arteriovenous shunt vasoconstriction, and shivering.

107 n on acute pulmonary hypertension induced by vasoconstriction, and to demonstrate denervation of the

108 t proliferation of blood vessels in areas of vasoconstriction (angiogenesis), CBF remained reduced ev

109 tricted diffusion, contrast enhancement, and vasoconstriction are all compatible with a diagnosis.

110 stress, including cutaneous vasodilation and vasoconstriction, are also affected by reproductive horm

111 Importantly, we identified peripheral vasoconstriction as a critical mechanism underlying the

112 We hypothesized that vasoconstriction at the apex is essential for rupture.

113 These results demonstrate that vasoconstriction at the apex of the follicle is essentia

114 Vasoconstriction blockade with the endothelin-1 inhibito

115 y, mood regulation, platelet aggregation and vasoconstriction, but its involvement in cell-adhesion r

116 y adults is not due to augmented sympathetic vasoconstriction, but rather due to impairments in local

117 led receptors have been involved in myogenic vasoconstriction, but the downstream signalling mechanis

118 Endothelin-induced vasoconstriction by hepatic stellate cells, and not plat

119 ce P) released from sensory-motor nerves and vasoconstriction caused by noradrenaline, ATP and neurop

120 Extreme renal vasoconstriction characterizes hepatorenal syndrome, a f

121 y modulation of SNA preferentially increases vasoconstriction compared to a frequency-matched tonic p

122 Our results indicate that sympathetic vasoconstriction competes with endothelium-dependent dil

123 The heart proteome included vasoconstriction, complement activation, and lipoprotein

124 00 m both hypovolaemia and hypoxic pulmonary vasoconstriction contribute to the decrease in LV fillin

125 ther augmented SNS-mediated alpha-adrenergic vasoconstriction contributes to the age-associated impai

126 te intensity exercise attenuated PE-mediated vasoconstriction (DeltaFVC: -13 +/- 1 and -19 +/- 5%, re

127 on focus on the relationship between SNA and vasoconstriction during a pressor stimulus, which increa

128 We investigated whether excessive digital vasoconstriction during acute mental stress predicts adv

129 ains the ability to blunt alpha1 -adrenergic vasoconstriction during combined KIR channel and Na(+) /

130 KCa channel remodeling, which contributes to vasoconstriction during diabetes mellitus.

131 Protocol 2) did not enhance alpha1 -mediated vasoconstriction during exercise (Protocol 1: -27 +/- 3%

132 ents with HF and SDB have more severe muscle vasoconstriction during hypoxia and hypercapnia than HF

133 one reduced feelings of warmth and increased vasoconstriction during social inclusion, especially for

134 Enhanced vasoconstrictions during hyperglycemia were prevented by

135 optosis (TWEAK), promoted pericyte-dependent vasoconstriction followed by pericyte detachment from ca

136 old-induced vascular response, consisting of vasoconstriction followed by vasodilatation, is critical

137 ing skeletal muscle to attenuate sympathetic vasoconstriction (functional sympatholysis) is critical

138 le to attenuate sympathetic alpha-adrenergic vasoconstriction ('functional sympatholysis') is impaire

139 ageing, as was the decline in noradrenergic vasoconstriction; genetic deletion of IL-10 mimicked the

140 nists able to induce systemic and mesenteric vasoconstriction have shown their usefulness in reducing

141 zolamide prevent or reduce hypoxic pulmonary vasoconstriction (HPV) in dogs and humans in vivo, by a

142 h as acetazolamide inhibit hypoxic pulmonary vasoconstriction (HPV) in humans and other mammals, but

143 Hypoxic pulmonary vasoconstriction (HPV) optimizes pulmonary ventilation-p

144 -threatening hypoxemia via hypoxic pulmonary vasoconstriction (HPV) which matches perfusion to ventil

145 is proposed to result from hypoxic pulmonary vasoconstriction (HPV).

146 s been proposed to mediate hypoxic pulmonary vasoconstriction (HPV).

147 racting skeletal muscle to blunt sympathetic vasoconstriction (i.e. 'functional sympatholysis'), whic

148 Carotid vasoconstriction identifies PAD patients with a 4-fold i

149 ular effects, such as arterial and pulmonary vasoconstriction, impaired left ventricular (LV) relaxat

150 in healthy subjects is associated with acute vasoconstriction in a conductance artery and found sugge

151 cell TNF augments resistance artery myogenic vasoconstriction in a diabetes model that induces a smal

152 n astrocytes with intracellular BAPTA causes vasoconstriction in adjacent arterioles.

153 d subsequent impairments in reflex cutaneous vasoconstriction in ageing.

154 dulatory effects on hUII- and URP-associated vasoconstriction in an ex vivo rat aortic ring bioassay.

155 t, spironolactone inhibited alpha-adrenergic vasoconstriction in arterioles from mice and hypertensiv

156 et and inversion of NVC from vasodilation to vasoconstriction in brain slices obtained from subarachn

157 t necessary to properly modulate sympathetic vasoconstriction in contracting muscle, and that age-ass

158 Sympathetic vasoconstriction in contracting skeletal muscle is blunt

159 vasodilatation can blunt alpha1 -adrenergic vasoconstriction in contracting skeletal muscle of human

160 othelium-dependent regulation of sympathetic vasoconstriction in contracting skeletal muscle, and spe

161 4; 55 +/- 2 years) and (ii) augmented reflex vasoconstriction in HTN would be mediated by an increase

162 for sympathetic reflex control of cutaneous vasoconstriction in HTN.

163 ing skeletal muscle to attenuate sympathetic vasoconstriction in humans.

164 While Sphk1 is important in mediating vasoconstriction in hypertension, Sphk1(-/-) mice were c

165 ociated with decreased aortic and mesenteric vasoconstriction in hypertensive Sphk1(-/-) mice.

166 y be explained by impaired hypoxic pulmonary vasoconstriction in infected lung regions, no studies ha

167 bution of hypovolaemia and hypoxic pulmonary vasoconstriction in limiting left ventricular (LV) funct

168 XA2 antagonists can inhibit the PVAT-induced vasoconstriction in male and female PCAs, respectively.

169 ese results demonstrate that RGS2 attenuates vasoconstriction in MAs and that RGS2 LOF mutations cann

170 ced from vascular endothelial cells, induces vasoconstriction in physiological conditions.

171 TC induced vasoconstriction in placental and rat vasculature, which

172 ve activity but modulates blood pressure and vasoconstriction in POTS women during orthostatic stress

173 al activity but modulates blood pressure and vasoconstriction in POTS women during tilting.

174 0.001-10 muM) evoked concentration-dependent vasoconstriction in preglomerular microvessels, predomin

175 of sympathetic innervation and to defective vasoconstriction in resistance arteries.

176 process, and in the physiological process of vasoconstriction in response to hypoxia, remains unclear

177 PE caused robust vasoconstriction in resting skeletal muscle during contr

178 Stimulation of alpha-adrenoceptors elicits vasoconstriction in resting skeletal muscle that is blun

179 e ability of ATP to blunt alpha1 -adrenergic vasoconstriction in resting skeletal muscle would be ind

180 X-1 ligand) increased angiotensin II-induced vasoconstriction in STBEV-incubated arteries from both m

181 egulating vascular tone are shifted to favor vasoconstriction in the absence of GRK2 expression and t

182 contractility with an exaggerated peripheral vasoconstriction in the CHF state.

183 ing exercise attenuated alpha(1) -adrenergic vasoconstriction in the contracting muscle of older adul

184 m caused vasodilatation but was converted to vasoconstriction in the presence of 1 mum BIBN4096bs, an

185 Previously, we showed in rat that CO(2)/H(+)-vasoconstriction in the retrotrapezoid nucleus (RTN) sup

186 Our findings indicate that LPA causes vasoconstriction in VSMCs, mediated by LPA1-, Gi-, and C

187 Impaired postsynaptic alpha1-adrenergic vasoconstriction in young adults with VVS can be correct

188 icantly higher pressor responses and greater vasoconstrictions in the offspring.

189 othelium-dependent dilatation and adrenergic vasoconstriction increased at 10 days in concert with my

190 s effects of angiotensin (Ang) II, including vasoconstriction, inflammation, water and salt retention

191 Therefore, we named the peptide vasoconstriction-inhibiting factor (VIF).

192 we found that PANX1-mediated ATP release and vasoconstriction involves constitutive phosphorylation o

193 Myogenic vasoconstriction is an autoregulatory function of small

194 ested the hypothesis that alpha1 -adrenergic vasoconstriction is augmented during exercise following

195 in-1 (ET-1) in levels sufficient to initiate vasoconstriction is considered to be a hallmark feature

196 Hypoxic pulmonary vasoconstriction is correlated with pulmonary vascular r

197 We tested the hypothesis that SNS-mediated vasoconstriction is greater in older than young adults a

198 Reflex cutaneous vasoconstriction is impaired in older adults; however, t

199 TGF-mediated vasoconstriction is limited by the simultaneous release

200 The initial cold-induced vasoconstriction is mediated via TRPA1-dependent superox

201 Vasoconstriction is not simply a response to reduced thi

202 from perivascular sympathetic nerves, causes vasoconstriction largely via P2X1 receptors.

203 a feed-forward loop of organ damage, due to vasoconstriction, leukocyte adherence, and activation of

204 Oxygen-induced vasoconstriction may serve as a protective mechanism to

205 ay variations in response to agonist-induced vasoconstriction, myosin phosphorylation, and ROCK2 acti

206 nded periods of waking, and terminated, with vasoconstriction, near the nadir of daily ambient temper

207 ockers on HPV and also on normoxic pulmonary vasoconstriction (NPV) stimulated by prostaglandin F2alp

208 on, dilatation and inhibition of sympathetic vasoconstriction observed in Young MAs were lost in Old

209 Hypoxic pulmonary vasoconstriction occurred following WLST, and was associ

210 r angiotensin 2) into the bursa restored the vasoconstriction of apical vessels and ovulation.

211 Vasoconstriction of apical vessels occurred within hours

212 PM4 currents in cerebral artery myocytes and vasoconstriction of cerebral arteries.

213 ological inhibition of S1P synthesis reduced vasoconstriction of mesenteric arteries.

214 nk3-mutated rats presented predisposition to vasoconstriction of pulmonary arteries and a severe loss

215 50 4 (Cyp4)-derived eicosanoid that enhances vasoconstriction of renal vessels and induces hypertensi

216 P<0.0001), in keeping with exercise-induced vasoconstriction of stenosed epicardial segments and dil

217 TGF-induced vasoconstriction of the afferent arteriole results from

218 beta-methylene ATP (300 nm) evoked sustained vasoconstrictions of 18.7 +/- 3.2% (P < 0.05).

219 luence of hypovolaemia and hypoxic pulmonary vasoconstriction on the decrease in left ventricular (LV

220 h higher anxiety also experienced more rapid vasoconstriction (P = .007).

221 ents with SCD showing significantly stronger vasoconstriction (P = .019).

222 ROS in contributing to the fetal peripheral vasoconstriction, part of the fetal defence to hypoxia.

223 ations and connects a significant purinergic vasoconstriction pathway with a previously identified, y

224 maintained the ability to blunt PE-mediated vasoconstriction (PE-mediated DeltaFVC: KIR blockade alo

225 In the absence of myogenic vasoconstriction, perfusion of peripheral organs was inc

226 e to reduced thickness of the follicle wall; vasoconstriction persisted in wild-type mice when thinni

227 physiologic responses such as cell adhesion, vasoconstriction, platelet aggregation, angiogenesis, in

228 Both hypovolaemia and hypoxic pulmonary vasoconstriction play a role in the reduction of LV fill

229 Cocaine-induced vasoconstriction reduces blood flow, which can jeopardiz

230 pe should not be limited to reduced arterial vasoconstriction, reflected in TPR, but should also enco

231 Sympathetic vasoconstriction regulates peripheral circulation and co

232 ATP attenuated PE-mediated vasoconstriction relative to adenosine (ADO) and sodium

233 Vasoconstriction required metabotropic glutamate recepto

234 ed vessel density, and displayed a decreased vasoconstriction response compared to Day 0 networks.

235 mice and showed no weakness in the tail skin vasoconstriction response or thermogenic response to col

236 Arterioles displayed a vasoconstriction response to KCl and endothelin for each

237 tinib treatment attenuated hypoxic pulmonary vasoconstriction responses and increased susceptibility

238 Augmented vasoconstriction responses and progressive decreases in

239 In addition, animals with defective myogenic vasoconstriction showed aggravated hypotension in respon

240 ng mild exercise significantly attenuated PE vasoconstriction similar to levels observed during moder

241 onary arterial hypertension (PAH), promoting vasoconstriction, smooth muscle proliferation, and infla

242 Angiotensin II also reduced vasoconstriction stimulated by Psora-4 or 4-aminopyridin

243 TS: Intravascular ATP attenuates sympathetic vasoconstriction (sympatholysis) similar to what is obse

244 r ATP attenuate sympathetic alpha-adrenergic vasoconstriction (sympatholysis).

245 Reversible cerebral vasoconstriction syndrome (RCVS) is characterized by rec

246 phatics in patients with reversible cerebral vasoconstriction syndrome (RCVS) with (n = 92) or withou

247 Simultaneous [Ca(2+)]i fluorescence and vasoconstriction testing showed reduced Ca(2+), leading

248 They had faster vasoconstriction than the controls (P = .033), especiall

249 nSMase had synergistic effects on pulmonary vasoconstriction that involved TRPC6, phospholipase C, a

250 of AZ10419369 exerted an extracranial tissue vasoconstriction that was comparable to the less blood-b

251 genous nSMase caused TRPC6 translocation and vasoconstriction that were blocked by CFTR inhibition.

252 ivery of blood to active brain regions cause vasoconstriction that would limit cerebral blood flow.

253 itric oxide (NO) attenuates alpha-adrenergic vasoconstriction, thus optimizing perfusion.

254 Hg) of intravascular pressures, and reduced vasoconstriction to iberiotoxin and vasodilation to NS16

255 scle cells responsible for hypoxic pulmonary vasoconstriction to limit perfusion of poorly ventilated

256 Robust vasoconstriction to PE was observed during vasodilator i

257 Vasodilatation to ACh and vasoconstriction to phenylephrine (10(-9) to 10(-5) m) w

258 to caffeine increased pressor responses and vasoconstrictions to phenylephrine, accompanied by enhan

259 ed all these effects of capsaicin as well as vasoconstriction triggered by lysophosphatidic acid, a b

260 y and increased endothelin 1 (ET-1)-mediated vasoconstriction, two abnormalities contributing to vasc

261 levations in brain temperature and sustained vasoconstriction, two critical factors associated with M

262 ET-1 contributes to vasoconstriction, vascular and cardiac hypertrophy, infl

263 gh Tie2-CYP4F2-Tr aortas displayed increased vasoconstriction, vasorelaxation and blood pressure were

264 sient BK currents in myocytes and stimulated vasoconstriction via a PKC-dependent mechanism that requ

265 travascular ATP modulates alpha1 -adrenergic vasoconstriction via pathways independent of KIR channel

266 Vasoconstriction was also attenuated in kidneys with URC

267 Surprisingly, vasoconstriction was also diminished in vessels lacking

268 Vasoconstriction was attenuated in O.

269 Here, PE-mediated vasoconstriction was blunted at rest (blockade: -20 +/-

270 In vitro, flow/pressure-evoked vasoconstriction was blunted when the astrocytic syncyti

271 PE-mediated vasoconstriction was calculated (%DeltaFVC) in each cond

272 BAPTA-induced vasoconstriction was eliminated by a general COX blocker

273 PE-mediated vasoconstriction was not attenuated by mild or moderate

274 f local application of ZM323881, significant vasoconstriction was observed in the venules of diabetic

275 Diesel exhaust-related vasoconstriction was primarily observed in the variant a

276 In contrast, ACh-mediated vasoconstriction was restored by expression of RGS2 WT,

277 rmalised to SL values, and hypoxic pulmonary vasoconstriction was reversed by administration of silde

278 Similarly, PGF2alpha-mediated vasoconstriction was symmetrically regulated by co-treat

279 channels in buffering phenylephrine-induced vasoconstrictions was decreased, whole cell BKCa current

280 is, where respiratory modulated increases in vasoconstriction were mediated by a noradrenergic mechan

281 Forearm and calf alpha1-adrenergic vasoconstriction were unimpaired in VVS and unaffected b

282 Vasoconstrictions were also observed in response to sing

283 agonist suramin blunted flow/pressure-evoked vasoconstriction, whereas K(+) and 20-HETE signaling blo

284 vasodilations were followed by a VP-mediated vasoconstriction, which acted to limit the magnitude of

285 drugs also induced dose-dependent peripheral vasoconstriction, which appears to be a primary mechanis

286 eading depolarizations along with subsequent vasoconstriction, which in turn enlarged the perivascula

287 ABSTRACT: Myogenic vasoconstriction, which reflects the intrinsic ability o

288 This anomalous vasoconstriction, which would potentiate hypoxia, raises

289 in their environment: increases in PO2 cause vasoconstriction while decreases in PO2 result in vasodi

290 sed endothelial proliferation, survival, and vasoconstriction while decreasing angiogenic potential.

291 ntioxidants augmented diesel exhaust-related vasoconstriction with a mean change in BAd of -0.18 mm (

292 Greater peripheral vasoconstriction with mental stress, denoted by a low sP

293 erquartile range, 0.48-0.88), indicating 32% vasoconstriction with mental stress.

294 We observe entrainment of vasodilation and vasoconstriction with pupil diameter and measure 3D bloo

295 Therapeutic targeting of renal vasoconstriction with serelaxin in the rat models increa

296 urrent thunderclap headaches and evidence of vasoconstriction with subsequent resolution.

297 Regional reductions in CBF, and associated vasoconstriction, within the default mode network in hyp

298 In vivo hyperglycemia enhanced these vasoconstrictions without altering respective receptor m

299 nSMase- and hypoxia-induced vasoconstriction, yet not TRPC6 translocation, were bloc

300 ng skeletal muscle to blunt the stimulus for vasoconstriction, yet the underlying signalling of this