ライフサイエンスコーパス: vasodilation

1 ones that promote natriuresis, diuresis, and vasodilation.

2 likely due to accelerated histamine-induced vasodilation.

3 sease severity, endothelial dysfunction, and vasodilation.

4 olarizing factor, or prostaglandins to cause vasodilation.

5 gnaling, integrity and endothelium-dependent vasodilation.

6 synthesis, and impairs endothelium-dependent vasodilation.

7 due to an inability to increase NO dependent vasodilation.

8 he classic physiological response of hypoxic vasodilation.

9 le guanylate cyclase (sGC) and cGMP-mediated vasodilation.

10 P from the red cell which has been linked to vasodilation.

11 n important physiologic regulator of hypoxic vasodilation.

12 at is independent of phasic, neuronal-evoked vasodilation.

13 sion, platelet aggregation, and dysregulated vasodilation.

14 echanisms underlying GLP-1R agonist-mediated vasodilation.

15 t vascular tone following activity-dependent vasodilation.

16 g the desensitization of GPCRs important for vasodilation.

17 emia/reperfusion injury, cytoprotection, and vasodilation.

18 ylation of KV1 channels in cVSMCs to promote vasodilation.

19 xpression and diminished BK channel-mediated vasodilation.

20 tibody significantly attenuated NaHS-induced vasodilation.

21 istance were acquired at baseline and during vasodilation.

22 r quality of life despite exerting pulmonary vasodilation.

23 NO activates myocyte BK channels and induces vasodilation.

24 s released, which induces local and systemic vasodilation.

25 ) sensitivity in arterial myocytes to induce vasodilation.

26 xploit for in vitro and in vivo blood vessel vasodilation.

27 increased vasoconstriction as with decreased vasodilation.

28 ronal activation, enhancing the accompanying vasodilation.

29 thineers) using regadenoson (5ml, 0.4mg) for vasodilation.

30 steroid-sensing site in BK beta1, rendering vasodilation.

31 the ability of stored RBCs to effect hypoxic vasodilation.

32 te cyclase (sGC), resulting in cGMP-mediated vasodilation.

33 ia ratio, and maintained endothelium-derived vasodilation.

34 ng mechanisms, and how obesity disturbs this vasodilation.

35 ide, leading to smooth muscle relaxation and vasodilation.

36 annels to reduce myogenic tone, facilitating vasodilation.

37 s consistent with decreased angiogenesis and vasodilation.

38 ) sensitivity of IK and SK channels to cause vasodilation.

39 trite is a physiological effector of hypoxic vasodilation.

40 with age, which was caused by reduced local vasodilation.

41 stimulation of IK and SK channels to promote vasodilation.

42 cle surrounding cerebral arterioles, driving vasodilation.

43 this bioactivation process is essential for vasodilation.

44 to the adjacent smooth muscle cells causing vasodilation.

45 rates stimulate EC PKD2 channels, producing vasodilation.

46 ilies are clinically important modulators of vasodilation.

47 nisms produce arterial hyperpolarization and vasodilation.

48 to nitric oxide production and flow-induced vasodilation.

49 or CO(2)-induced nitric oxide production and vasodilation.

50 ity, suggesting RhoBTB1 selectively controls vasodilation.

51 ies for effects on both vasoconstriction and vasodilation.

52 1 umol/L) both displayed augmented conducted vasodilation.

53 to loss of I(Na) activation in myocytes and vasodilation.

54 I and a loss of acetylcholine (ACh)-mediated vasodilation.

55 asoconstriction, and extrahepatic splanchnic vasodilation.

56 se BP by inducing natriuresis, diuresis, and vasodilation.

57 nd R44H mutant RGS2 showed normal control of vasodilation.

58 itical for NO-stimulated cGMP production and vasodilation.

59 as the dominant mechanism underlying hypoxic vasodilation.

60 duced the following: (1) a massive cutaneous vasodilation; (2) drastic drops in deep brain temperatur

61 assessed the contribution of MPs to arterial vasodilation, a mechanism that contributes to portal hyp

62 o-photon imaging revealed prompt peritubular vasodilation after fluvoxamine treatment, which was bloc

63 lated pressure change ratio) consistent with vasodilation also decreased after milrinone.

64 nnel and eNOS activation, hyperpolarization, vasodilation and a reduction in systemic blood pressure.

65 convection capacity associated with systemic vasodilation and an increase in cardiac output were sepa

66 s revealed a hypoxia response and changes in vasodilation and angiogenesis genes that strongly suppor

67 fy the inflammatory response, but also cause vasodilation and angiogenesis.

68 of such association may compromise cerebral vasodilation and blood flow.

69 the red blood cell (RBC) to mediate hypoxic vasodilation and cardioprotection.

70 2Y2 or Gq/G11 deficiency lacked flow-induced vasodilation and developed hypertension that was accompa

71 SAH, signaling events that normally lead to vasodilation and enhanced delivery of blood to active br

72 in women, where estradiol appears to promote vasodilation and heat dissipation.

73 Here we show in rats that NMDA-induced vasodilation and hemodynamic responses evoked by whisker

74 s is controversial because of concerns about vasodilation and hypotension.

75 nd in a crossover design augmented conducted vasodilation and improved LV diastolic function.

76 O2/H(+) where an increase in CO2/H(+) causes vasodilation and increased blood flow.

77 bsequently, increased inflow from splanchnic vasodilation and increased cardiac output lead to a furt

78 effect in normoxia, but produced significant vasodilation and increased nitrosylation during hypoxaem

79 decrease RV afterload by promoting pulmonary vasodilation and inhibiting vascular remodeling but are

80 anylyl cyclase generates cGMP, which induces vasodilation and inhibits platelet activation.

81 (RHI), which measures endothelium-dependent vasodilation and is a surrogate marker of endothelial fu

82 opressor system that induces hypotension and vasodilation and is degraded by ACE and enhanced by the

83 brachial artery macrovascular flow-mediated vasodilation and microvascular reactive hyperemia (p < 0

84 impact of IFN-alpha on mediators that induce vasodilation and modulate inflammation, including endoth

85 e diverse physiological functions, including vasodilation and neurotransmission.

86 e-induced improvement in nutrient-stimulated vasodilation and nutrient delivery to muscle rather than

87 may be a novel mechanism underlying impaired vasodilation and oxygen delivery during hypoxemia with a

88 CB(1) cannabinoid receptors (CB(1)R) induces vasodilation and reduces blood pressure, we have tested

89 n, which acted to limit the magnitude of the vasodilation and served to reset vascular tone following

90 vascular smooth muscle is implicated in the vasodilation and vascular hyporeactivity underlying sept

91 ibution of purinergic signaling to disturbed vasodilation and vascular remodeling during atherosclero

92 We observe entrainment of vasodilation and vasoconstriction with pupil diameter an

93 onses to thermal stress, including cutaneous vasodilation and vasoconstriction, are also affected by

94 rease that results primarily from peripheral vasodilation, and a subsequent increase driven by metabo

95 rescued endothelial AKAP150-TRPV4 signaling, vasodilation, and blood pressure in obesity.

96 malizing endothelial TRPV4 channel activity, vasodilation, and blood pressure in obesity.

97 the modern era that is related to excessive vasodilation, and most frequently caused by obesity, art

98 ocytic cells activates NADPH oxidase, limits vasodilation, and promotes renal IRI.

99 r glucose uptake and utilization, autophagy, vasodilation, and proton removal, as demonstrated by qua

100 quantify plaque area, vascular permeability, vasodilation, and stiffness and post-triggering to ident

101 ignificant mediator of nitroglycerin-induced vasodilation, and tolerance to nitroglycerin is associat

102 ose produced by angiotensin-(1-7), including vasodilation, antifibrosis, antihypertensive, and centra

103 g other biological functions of NO including vasodilation, antimicrobial, anticancer, and neurotransm

104 The initial vasodilation appears caused by respiratory depression-in

105 for this stress-induced shift in mediator of vasodilation are proposed.

106 physiological ramifications of RBC-mediated vasodilation are unknown, and the apparently essential n

107 d endothelial markers, endothelium-dependent vasodilation, arteriolar glycocalyx size, and glomerular

108 solution of retinal detachment and choroidal vasodilation as well as improved visual acuity.

109 The concurrent vasodilation associated with increases in Ca(2+) event f

110 liotransmitters is widely assumed to trigger vasodilation associated with rapid increases in neuronal

111 othelial function, measured by flow-mediated vasodilation before and 6 hours after severe acute pancr

112 nsduces signals important for flow-dependent vasodilation, blood vessel remodeling, and atheroscleros

113 We found a markedly reduced conducted vasodilation both in obese ZSF1 rats and in patients wit

114 ards protocell-mediated nitric-oxide-induced vasodilation by constructing a new synthetic cell model

115 ctive in selectively enhancing the pulmonary vasodilation by imatinib and sildenafil.

116 nitrate, GTN) in blood vessels, resulting in vasodilation by nitric oxide (NO) or a related species.

117 a role in S-nitrosothiol (SNO)-based hypoxic vasodilation by RBCs.

118 that astrocytes provide tonic, steady-state vasodilation by releasing prostaglandin messengers.

119 he nervous and circulatory systems including vasodilation, cardioprotection, and pain transmission.

120 We found that DAR induced hepatic sinusoidal vasodilation, caused more transplanted cells to be depos

121 tor and better understanding of its roles in vasodilation, cell permeability, platelet function, infl

122 pragmatic approach of maximal and sustained vasodilation combining individualized doses of sublingua

123 it causes a strong side effect of cutaneous vasodilation, commonly called flushing.

124 a strategy of early intensive and sustained vasodilation, compared with usual care, did not signific

125 Local increases in blood flow--'hypoxic vasodilation'--confer cellular protection in the face of

126 leads to a predominant nitric oxide-mediated vasodilation (Delta19 +/- 2%).

127 nel and transient BK current activation, and vasodilation did not involve Rab11A S177.

128 ertension and impaired endothelium-dependent vasodilation due to uncoupled NO synthase (NOS).

129 us CAD, resulting in an altered capacity for vasodilation during disease.

130 ic agonist albuterol would improve pulmonary vasodilation during exercise in patients with HFpEF, wit

131 xtensive evidence now indicates that hypoxic vasodilation entails S-nitrosothiol-based (SNO-based) va

132 ounds (922+/-38mg GAE/kg), it did not induce vasodilation, even at the highest dose tested (0.206g/L)

133 Large vasodilation events could be followed by post-stimulus c

134 Propionate, a SCFA shown to induce vasodilation ex vivo, produces an acute hypotensive resp

135 atus was assessed by measuring flow-mediated vasodilation (FMD), brachial pulse wave velocity (bPWV),

136 the change in brachial artery flow-mediated vasodilation (FMD).

137 of ADK (adenosine kinase) augments conducted vasodilation for a more efficient myocardial perfusion a

138 Recent work has proposed iFR as a vasodilation-free alternative to FFR for making mechanic

139 erutz, to blood flow autoregulation (hypoxic vasodilation governing oxygen delivery) observed by Guyt

140 (2+) signaling in endothelial cells promotes vasodilation has led to the hypothesis that endothelial

141 5% CI, 0.90-1.31; P=0.39), and flow-mediated vasodilation (HR, 0.85; 95% CI, 0.69-1.04; P=0.12) were

142 itric oxide levels and endothelium-dependent vasodilation in a murine model and ex vivo human tissues

143 used systemic vasoconstriction and pulmonary vasodilation in a porcine in vivo model of acute pulmona

144 uces systemic vasoconstriction and pulmonary vasodilation in a porcine model of acute pulmonary embol

145 peroxide and inhibited nitric oxide-mediated vasodilation in a SIRP-alpha-dependent manner.

146 (JNK) in db/db PVAT restored insulin-induced vasodilation in an adiponectin-dependent manner.

147 re-establishing a physiological mechanism of vasodilation in arterioles from subjects with CAD.

148 , elevation of external K(+) to 10 mM caused vasodilation in brain slices from control animals but ca

149 Despite the importance of vasodilation in cardiovascular homeostasis and therapy,

150 expression and BK-channel-mediated coronary vasodilation in diabetic mice.

151 on and restored BK channel-mediated coronary vasodilation in diabetic mice.

152 into liposomal vesicles results in prolonged vasodilation in distal pulmonary arterioles.

153 ng, and, hence, reduced NO-mediated coronary vasodilation in DM patients.

154 Coronary PVAT also diminished H2O2-mediated vasodilation in lean and, to a lesser extent, in obese a

155 ulatory mechanism for processes ranging from vasodilation in mammals to communal behavior in bacteria

156 formulations produced pulmonary preferential vasodilation in MCT induced PAH rats.

157 gates whether PVAT regulates insulin-induced vasodilation in muscle, the underlying mechanisms, and h

158 emonstrate that GrC activity causes a robust vasodilation in nearby capillaries via the NMDA receptor

159 ODS AND H2O2 induced endothelium-independent vasodilation in non-CAD adipose arterioles, which was re

160 H2O2 induced endothelium-independent vasodilation in non-CAD adipose arterioles, which was re

161 uated endothelial TRPV4 channel activity and vasodilation in obese animals.

162 DK mitigates adenosine-facilitated conducted vasodilation in obese ZSF1 rats and in patients with HFp

163 We assessed conducted vasodilation in obese ZSF1 rats that develop LV diastoli

164 -mediated dilation and endothelium-dependent vasodilation in response to acetylcholine were improved

165 ction by measuring the endothelial-dependent vasodilation in response to acetylcholine.

166 eters of athletes would be larger, have less vasodilation in response to cuff occlusion, but more con

167 lost the ability to induce NO formation and vasodilation in response to flow and consequently develo

168 ithin the T cell compartment in mice ablated vasodilation in response to infection, impaired the migr

169 eled by measuring parenchymal arteriole (PA) vasodilation in response to neuronal stimulation in amyg

170 giotensin II-induced hypertension, decreased vasodilation in response to NO correlates with GC1 thiol

171 sting arteriole diameter but fails to affect vasodilation in response to vibrissae stimulation.

172 icited more pulmonary specific and sustained vasodilation in SUGEN-5416/hypoxia-induced PAH rats than

173 as signalling in the respiratory system and vasodilation in the cardiovascular system.

174 ra tissue in Franz diffusion cells suggested vasodilation in the conjunctival vasculature in the pres

175 igh sodium diets, and ADRB2-mediated forearm vasodilation in the high sodium condition.

176 indicate that obestatin causes NO-dependent vasodilation in the human circulation.

177 cellular calcium was not required for normal vasodilation in the IP3R2-KO animals.

178 lower endothelium-dependent and -independent vasodilation in the macrocirculation.

179 signed to right heart catheterization in the Vasodilation in the Management of Acute Congestive Heart

180 on and with decreased acetylcholine-mediated vasodilation in the renal microvasculature.

181 The endothelin-1-induced vasodilation in these PCOS subject, was dependent on the

182 s significantly to nitrite-dependent hypoxic vasodilation in vivo and ex vivo.

183 omote increases in NO production and enhance vasodilation in vivo.

184 ls leading to impaired endothelium-dependent vasodilation, increased vascular tone, and hypertension.

185 ly partially restores eNOS-mediated coronary vasodilation, indicating that other critical factors tri

186 xide release, improved endothelial-dependent vasodilation, inhibited vascular inflammation, and suppr

187 Impaired endothelium-dependent vasodilation is a hallmark of obesity-induced hypertensi

188 We sought to confirm that early hypoxic vasodilation is a nitric oxide (NO)-mediated phenomenon

189 Hypoxic vasodilation is a physiological response to low oxygen t

190 In conclusion, acute hypoxic vasodilation is an adaptive NO-mediated response conferr

191 In the postischemic heart, coronary vasodilation is impaired due to loss of endothelial nitr

192 Our findings indicate that vasodilation is largely mediated by endothelial cells th

193 We hypothesized that nitroglycerin-induced vasodilation is mediated by disulfide activation of PKG1

194 Vasodilation is observed to be a major determinant of ba

195 Notably, hypoxic vasodilation is recapitulated by native S-nitrosothiol (

196 CGRP-mediated vasodilation is, however, a critical rescue mechanism in

197 anoid PGE(2), which induces angiogenesis and vasodilation, is diminished in diabetic skin, suggesting

198 hysiological role in processes as diverse as vasodilation, maintenance of vascular tone, neurotransmi

199 ith control, increased endothelium-dependent vasodilation (mean difference 4.1%-units of measurement;

200 lves increasing intestinal perfusion through vasodilation mediated by nitric oxide and hydrogen sulfi

201 Reactive vasodilation merits further investigation as a novel ris

202 Interventions that enhance pulmonary vasodilation might be beneficial in this cohort but coul

203 e diverse physiological processes, including vasodilation, neurotransmission, and myocardial function

204 s in diverse physiological processes such as vasodilation, neurotransmission, and the innate immune r

205 Larval pdx1 (-/-) mutants prominently show vasodilation of blood vessels through increased vascular

206 of the hemodynamic (mostly mediated through vasodilation of capacitance and conductance arteries) an

207 After stress, vasodilation of PAs to neuronal stimulation was greatly

208 The effect of ROCK inhibition on the vasodilation of porcine conjunctival vasculature was ass

209 thelial function was explored by testing the vasodilation of the liver circulation to increasing conc

210 ative modifications in endothelium-dependent vasodilation of the principal nutrient artery (PNA) of t

211 Functionally, the intrinsic vasodilation of the vessel wall decreased at 12 weeks co

212 ne (P < 0.001) but did not induce additional vasodilation (P > 0.05) during obestatin.

213 erine administration resulted in significant vasodilation (p < 0.05).

214 n gene-related peptide artificially activate vasodilation pathways in rat brain and induce contrast c

215 s in cardiovascular physiology, specifically vasodilation, platelet aggregation, and leukocyte rollin

216 regulate physiological functions, including vasodilation, platelet aggregation, and neurotransmissio

217 trol diverse physiological processes such as vasodilation, platelet aggregation, and synaptic plastic

218 rdiopulmonary bypass, levosimendan induces a vasodilation, preferentially of preglomerular resistance

219 ular processes, including neurotransmission, vasodilation, proliferation, and apoptosis in various ce

220 ease of the drug, produce pulmonary specific vasodilation, reduce the systemic exposure of the drug,

221 rlying red blood cell (RBC)-mediated hypoxic vasodilation remain controversial, with separate roles f

222 ated, mechanisms that initiate CO(2)-induced vasodilation remain unclear.

223 ivate beta1-containing BK channels and evoke vasodilation remain unknown.

224 Vasodilation required neuronal NMDARs and NOS stimulatio

225 Rac1 in vSMCs causes defective nitric oxide vasodilation responses and hypertension.

226 sfusion and effects on endothelial-dependent vasodilation responses to acetylcholine have not been fu

227 In vivo and ex vivo vasodilation responses, the reduction of nitrite to NO.,

228 these observations and its ability to induce vasodilation, resveratrol induced oxidative activation o

229 Despite increasing NO-dependent vasodilation, SIL unexpectedly elevated mean arterial bl

230 ide contributed to hypoxia-mediated coronary vasodilation similarly in IGF-1-treated and Control fetu

231 Hypoxic vasodilation studies in myoglobin and endothelial and in

232 s do not contribute to acetylcholine-induced vasodilation, suggesting stimulus-specific function.

233 to hypercapnia, which triggers blood vessel vasodilation, suggests its dependence on vascular effect

234 taCys93Ala mutation are deficient in hypoxic vasodilation that governs blood flow autoregulation, the

235 al for therapeutic augmentation of conducted vasodilation, thereby improving tissue perfusion and LV

236 may be related to increased CBF rather than vasodilation; these results contradict most previous stu

237 from C57BL/6 mice uncovered insulin-induced vasodilation; this vasodilation was abrogated with PVAT

238 pregnancy, contributing to profound maternal vasodilation through endothelial and nitric oxide (NO)-d

239 esis involves impaired endothelium-dependent vasodilation through reactive oxygen species (ROS) forma

240 Although astrocytes can induce vasodilations through the release of prostaglandins, the

241 a strategy of early intensive and sustained vasodilation throughout the hospitalization (n = 386) or

242 Hyperemic enhancement of rCBF and vasodilation throughout the vascular network was observe

243 portal perfusion pressure and a decrease in vasodilation to acetylcholine (sinusoidal endothelial dy

244 pendent signalling mechanism is required for vasodilation to ADO.

245 l brain blood flow by providing steady-state vasodilation to arterioles via resting astrocyte Ca(2+)

246 ling mechanism linking endothelium-dependent vasodilation to bone remodeling.

247 omega)-nitro-l-arginine methyl ester reduced vasodilation to flow in adipose as well as atrial vessel

248 ventions to correct deficits in RBC-mediated vasodilation to improve oxygen delivery-steps toward eff

249 EET) substrates, so they accumulate inducing vasodilation to lower blood pressure (BP).

250 reduced vasoconstriction to iberiotoxin and vasodilation to NS1619, BK channel inhibitors and activa

251 However, the endothelium-independent vasodilation to sodium nitroprusside was unaffected.

252 astrocyte endfeet and inversion of NVC from vasodilation to vasoconstriction in brain slices obtaine

253 of the neurovascular coupling response from vasodilation to vasoconstriction.

254 The endothelium-dependent NO-mediated vasodilations to bradykinin and stepwise increases in lu

255 (2+)-activated K(+)- and KV (KV1.5)-mediated vasodilation toward a large-conductance Ca(2+)-activated

256 cium-evoked release of PgE2 is decreased and vasodilation triggered by increased astrocyte [Ca(2+)]ii

257 ations of short dosing intervals, peripheral vasodilation, unwanted side effects, and restricted use

258 the therapeutic potential of selective renal vasodilation using serelaxin as a new treatment for rena

259 The TEBV exhibited flow-mediated vasodilation, vasoconstriction after exposure to 1 muM p

260 ng blood-brain barrier dysfunction, impaired vasodilation, vessel stiffening, dysfunctional blood flo

261 via A2A receptors located on ECs to produce vasodilation via activation of KATP channels located on

262 rite may contribute to physiological hypoxic vasodilation via reactions with vascular myoglobin to fo

263 Effect of a strategy of comprehensive vasodilation vs usual care on mortality and heart failur

264 se events with early intensive and sustained vasodilation vs usual care were hypokalemia (23% vs 25%)

265 Early intensive and sustained vasodilation was a comprehensive pragmatic approach of m

266 uncovered insulin-induced vasodilation; this vasodilation was abrogated with PVAT from db/db mice.

267 Impaired conducted vasodilation was accompanied by increased vascular ADK e

268 Acetylcholine-induced vasodilation was also impaired in ECSHIP2(Delta/+) mice,

269 nt of endothelial TRPV4 channel activity and vasodilation was also observed in the arteries from obes

270 Cinaciguat-induced vasodilation was associated with a time- and concentrati

271 Insulin-mediated vasodilation was blunted in ECSHIP2(Delta/+) mice, as wa

272 ogenous estrogen (i.e., intact and old OVX), vasodilation was correlated with BV/TV (R(2) = 0.630; P<

273 In SRF(SMKO), vasodilation was decreased in aorta and carotid arteries

274 In young rats, vasodilation was diminished by OVX and restored with est

275 ponse to endothelium-dependent microvascular vasodilation was greater after the apples [WA: 853 perfu

276 Tonic vasodilation was insensitive to TTX, as well as a variet

277 Additionally, conducted vasodilation was measured in arterioles isolated from th

278 ith this hypothesis, intact stimulus-induced vasodilation was observed in inositol 1,4,5-triphosphate

279 This vasodilation was predominantly mediated by enhanced NO a

280 ; P<0.0001), whereas endothelium-independent vasodilation was similar in the 2 groups.

281 Finally, hypoxic microvascular vasodilation was studied in vivo with a murine dorsal sk

282 Endothelium-dependent and independent vasodilation was tested by selective infusion of acetylc

283 In old animals, vasodilation was unaffected by OVX but enhanced with est

284 ous and exogenous nitric oxide (NO)-mediated vasodilation were both impaired by repeated hypoglycemia

285 ulated vascular cGMP signals associated with vasodilation were detected in vivo in an acutely untouch

286 of cell-signaling pathways and induction of vasodilation were examined in vitro and in isolated arte

287 Activity-dependent vasodilations were followed by a VP-mediated vasoconstri

288 red direct NO donor and endothelial-mediated vasodilation which were accompanied on structural and mo

289 an important mediator of VSMC relaxation and vasodilation, which acts by increasing cyclic GMP (cGMP)

290 VAT from obese mice inhibits insulin-induced vasodilation, which can be restored by inhibition of JNK

291 enoic acid (EET) induces mesenteric arterial vasodilation, which contributes to the onset of portal h

292 We sought to determine if conducted vasodilation, which coordinates microvascular resistance

293 the ability to induce endothelium dependent vasodilation, which is dependent on guanylyl cyclase but

294 receptors WIN55212, a CB(1)R agonist, caused vasodilation, which was absent in CB(1)R knock-out mice.

295 Hypoxaemia caused a rapid and sustained vasodilation, which was only partially reversed by non-s

296 ere made at baseline and under pharmacologic vasodilation with adenosine.

297 baseline hemodynamics and acute responses to vasodilation with intravenous sodium nitroprusside in pa

298 y of vascular smooth muscle (VSM) allows for vasodilation without lowering of cytosolic Ca(2+).

299 Considering that CO2/H(+)-induced vasodilation would accelerate removal of CO2/H(+) and po

300 Handgrip and forearm vasodilation yielded no haplotype differences, and no co