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1 d by exposure to supraphysiological doses of vasopressin.
2 ccelerates its endocytosis in the absence of vasopressin.
3 s9) to identify PKA-independent responses to vasopressin.
4 relationship with the neuropeptide modulator vasopressin.
5 melanin-concentrating hormone, oxytocin, and vasopressin.
6 al ganglion cells expresses the neuropeptide vasopressin.
7 fy genes whose transcription is regulated by vasopressin.
8 he presence or absence of norepinephrine and vasopressin.
9 endothelin-1, gentamicin, norepinephrine and vasopressin.
11 Treatment with IV 1-deamino-8-D-arginine vasopressin (0.4 ug/kg) + platelet transfusion (2 U) wit
13 of intranasal administration of an arginine vasopressin 1A and 1B receptor agonist against 1) no tre
15 ectivity versus the related receptors (human vasopressin 1a receptor, human vasopressin 1b receptor,
17 A2 area of the hippocampus, particularly the vasopressin 1b receptor (Avpr1b) expressed there, is nec
18 eptors (human vasopressin 1a receptor, human vasopressin 1b receptor, and human oxytocin receptor), a
19 ginine(8) vasopressin, dDAVP, 1) is a potent vasopressin 2 (V(2)) receptor (V(2)R) agonist approved i
20 12330A or the PKA inhibitor H89, but not the vasopressin 2 receptor (V(2)R) antagonist tolvaptan.
21 only a phosphorylated C-terminal tail of the vasopressin 2 receptor activates Src as efficiently as G
25 ve focused on the neuropeptides oxytocin and vasopressin(4-6), which regulate aspects of social behav
26 s increased in PKA-null cells indicates that vasopressin activates one or more members of the AMPK/SN
27 Furthermore, the effect of IRAP to reduce vasopressin activity is a physiologically important cons
28 (adjusted odds ratio, 1.5; 95% CI, 1.3-1.8), vasopressin (adjusted odds ratio, 1.5; 95% CI, 1.1-2.1),
29 These activations weakened with oxytocin and vasopressin administration such that neural responses to
30 lls describing the response to V2R-selective vasopressin agonists and antagonists, the response to CR
32 Drugs (caffeine, theophylline) and hormones (vasopressin, aldosterone) known to exacerbate cysts elic
33 mes between standard dose of epinephrine and vasopressin alone or in combination with epinephrine.
34 ercentage of patients receiving epinephrine, vasopressin, amiodarone, lidocaine, atropine, bicarbonat
39 ECV transfer and report that desmopressin, a vasopressin analogue, stimulated the uptake of fluoresce
46 r isotocin, homologues of mammalian arginine vasopressin and oxytocin that are broadly implicated in
48 to 0.06 U/min) and hydrocortisone (n = 101), vasopressin and placebo (n = 104), norepinephrine and hy
49 are differences in plasma cytokine levels in Vasopressin and Septic Shock Trial for lactate less than
50 pinephrine group 28- and 90-day mortality in Vasopressin and Septic Shock Trial in lactate subgroups.
56 l was applied to patients with AKI in VASST (Vasopressin and Septic Shock Trial; n = 271), and differ
58 sistent with increased levels of circulating vasopressin and up-regulation of vasopressin V2 receptor
59 eptidergic neurons in the SCN: AVP (arginine vasopressin) and VIP (vasoactive intestinal polypeptide)
60 l copeptin production, a surrogate marker of vasopressin, and dependent on the experience of birth-re
62 a potential clinically important benefit for vasopressin, and larger trials may be warranted to asses
63 unctions, abnormalities in central oxytocin, vasopressin, and serotonin neurotransmission, and neuroi
64 in 1776 patients enrolled in the Efficacy of Vasopressin Antagonism in Heart Failure Outcome Study Wi
65 oc analysis was performed of the Efficacy of Vasopressin Antagonism in Heart Failure: Outcome Study w
70 the endogenous IRAP substrates oxytocin and vasopressin are known to facilitate learning and memory.
71 ributing to those differences and identified vasopressin as a regulator of nestbuilding behaviour.
73 esearch due to the therapeutic potentials of vasopressin as well as the possibility to systematically
75 we report that serotonin (5-HT) and arginine-vasopressin (AVP) act in opposite ways in the hypothalam
76 olocalization with the neuropeptide arginine vasopressin (AVP) and clock proteins (PER2 and BMAL1), s
77 nse to the neurohypophyseal hormone arginine vasopressin (AVP) and in the expression of oxidative str
80 s was dependent on the neuropeptide arginine vasopressin (AVP) because it was prevented by pharmacolo
82 ts, which contain a mutation in the arginine vasopressin (AVP) gene, exhibit lower behavioral arousal
84 Prior studies show that oxytocin (Oxt) and vasopressin (Avp) have opposing actions on the skeleton
85 ts from a deficiency of the hormone arginine vasopressin (AVP) in the pituitary gland or the hypothal
86 ration of the "social" neuropeptide arginine vasopressin (AVP) is significantly lower in pediatric AS
87 accompanied by elevated circulating arginine vasopressin (AVP) levels in SHR-A3 compared with SHR-B2.
88 of the SCN, including VIP, GRP, and arginine vasopressin (AVP) neurons, with each ipRGC innervating s
89 asmatic nucleus output neuropeptide arginine-vasopressin (AVP) on the activity of preoptic area kissp
91 a self-assembling lipidized peptide arginine vasopressin (AVP) receptor agonist, that had not been de
94 ogy of thirst, taste for water, and arginine-vasopressin (AVP) release: ( a) Thirst and AVP release a
96 iciency of the antidiuretic hormone arginine vasopressin (AVP) underlies diabetes insipidus, which is
99 ncy and preschool, assayed oxytocin (OT) and vasopressin (AVP), and measured coparenting and child be
100 of copeptin, a surrogate marker of arginine vasopressin (AVP), to be associated with increased risk
101 ause cAMP is a central modulator of arginine vasopressin (AVP)-induced water transport in the renal c
102 e with a reduction of parvocellular arginine vasopressin (AVP)-positive neurons in the preoptic area,
103 PDE4 caused a greater increase in basal and vasopressin (AVP)-stimulated cAMP levels and Cl(-) secre
104 first study that demonstrates that arginine vasopressin boosts placebo effects and that the effect o
105 id not detect major deficiencies in arginine vasopressin [Ca(2+)](i) signaling in split-opened collec
108 g adults with septic shock, the early use of vasopressin compared with norepinephrine did not improve
110 gnetic resonance imaging, and serum arginine vasopressin concentration) were compatible with a diagno
111 he aim of this study was to evaluate whether vasopressin could be superior to norepinephrine to impro
113 nalogue desmopressin (desamino-d-arginine(8) vasopressin, dDAVP, 1) is a potent vasopressin 2 (V(2))
114 controls AQP2 apical membrane abundance in a vasopressin-dependent manner, allowing for urine volume
115 oosts placebo effects and that the effect of vasopressin depends upon a significant sex by treatment
116 e to sepsis-induced immunoparalysis, whereas vasopressin does not have untoward immunologic effects.
117 ropin-releasing hormone-dominant to arginine vasopressin-dominant, and cortisol levels remain raised
120 ong evidence suggests that certain hormones (vasopressin), foods (fructose), and metabolic products (
124 other GPCR agonists, including ADP, arginine vasopressin, glucagon-like peptide 1, and forskolin, and
125 ctor, dynorphin, norepinephrine, hypocretin, vasopressin, glucocorticoids, and neuroimmune factors) i
126 (interquartile range [IQR], 1 to -24) in the vasopressin group and 13 days (IQR, 1 to -25) in the nor
127 e was observed in 71 patients (56.8%) in the vasopressin group and 66 patients (52.8%) in the norepin
128 ailure was 94 of 165 patients (57.0%) in the vasopressin group and 93 of 157 patients (59.2%) in the
129 less use of renal replacement therapy in the vasopressin group than in the norepinephrine group (25.4
130 s (10.7%) had a serious adverse event in the vasopressin group vs 17 of 204 patients (8.3%) in the no
133 as implicated the neuropeptides oxytocin and vasopressin in the modulation of human neural activity u
134 in, and it has been assumed that the role of vasopressin in the SCN reflects the activity of these ce
136 genic hormones, including catecholamines and vasopressin, induce frequency-modulated cytosolic Ca(2+)
138 xiety and cortisol levels showed the largest vasopressin-induced modulation of placebo effects, sugge
139 ICU patients with septic shock who received vasopressin infusion added to at least one concomitant v
140 diabetes insipidus after discontinuation of vasopressin infusion among patients treated with vasopre
141 diabetes insipidus after discontinuation of vasopressin infusion among patients with septic shock as
142 diabetes insipidus after discontinuation of vasopressin infusion appears not to be a rare phenomenon
144 of patients receiving norepinephrine but not vasopressin infusion for treatment of shock, criteria fo
145 diabetes insipidus after discontinuation of vasopressin infusion had undergone cardiothoracic interv
146 diabetes insipidus after discontinuation of vasopressin infusion have been published; the majority o
147 diabetes insipidus after discontinuation of vasopressin infusion remains to be elucidated but may in
149 yscus and chemogenetics in Mus, we show that vasopressin inhibits nest building but not other parenta
150 ollecting duct; is modulated in abundance by vasopressin; interacts with aquaporin-2 (AQP2), Hsp70, a
152 d to differences in nest-building behaviour, vasopressin is differentially expressed in the hypothala
153 pig neonate demonstrated that the effect of vasopressin is not dependent on the level of maturation
157 own insect neuropeptides except for arginine-vasopressin-like peptide (AVLP), CNMamide, neuropeptide-
160 twork describing PKA signaling that explains vasopressin-mediated regulation of membrane trafficking
162 r genes, consistent with the conclusion that vasopressin-mediated transcriptional regulation is highl
163 e how intranasally administered oxytocin and vasopressin modulated neural activity when receiving neg
164 We localized a subpopulation of excitatory vasopressin neurons in the anterior hypothalamus that ma
165 ensitive neurons in the lamina terminals and vasopressin neurons in the paraventricular nucleus of th
166 nts of genetically identified neuroendocrine vasopressin neurons show they can anticipate osmotic cha
167 terminalis also resulted in Fos induction in vasopressin neurons within the PVN and supraoptic nucleu
168 activity (Normal: p < 0.001; HF: p < 0.05), vasopressin (Normal: p < 0.001; HF: p < 0.01), and cycli
170 sing; however, the influence of oxytocin and vasopressin on neural activity elicited during negative
171 ur results show effects of both oxytocin and vasopressin on the brain network involved in negative so
172 repinephrine and the alternative vasopressor vasopressin on the immune response and host defense.Meth
173 e liver with physiological concentrations of vasopressin only will evoke localized cytosolic calcium
178 ized to a 5-hour infusion of norepinephrine, vasopressin, or saline and intravenously challenged with
179 luenced response to either norepinephrine or vasopressin, or to corticosteroids in septic shock.
182 ntagonism acting on loci within the arginine vasopressin-oxytocin pathway explains how genetic divers
183 ted to gain novel insights into the oxytocin/vasopressin peptide-receptor interaction, which led to t
188 VPA exposure downregulated prefrontal cortex vasopressin receptor (V1aR) and methyl CpG-binding prote
189 tissues through the inhibition of the type-2 vasopressin receptor (V2R) constitutes a validated strat
192 ingle 20 mg intravenous dose of the arginine vasopressin receptor 1A (V1a) antagonist, RG7713, on exp
193 treatment of mpkCCD14 cells with the type 2 vasopressin receptor agonist dDAVP increased mRNA and pr
195 expression variations of the oxytocin and/or vasopressin receptor genes OXTR and AVPR1A, respectively
196 e value in testing medications targeting the vasopressin receptor in high stress, alcohol-dependent p
197 selected for greater potential benefit from vasopressin receptor inhibition, tolvaptan was not assoc
198 n important role for development of oxytocin/vasopressin receptor modulators that would enable clear
209 These anticipatory signals for thirst and vasopressin release concentrate on the same homeostatic
212 sepsis features impaired thirst and enhanced vasopressin release, the basis for these defects is unkn
214 which increases histone H3K27 acetylation of vasopressin-responsive genes (confirmed by ChIP-seq).
215 otic stress, posterior pituitary-projecting, vasopressin-secreting neurons (VPpp neurons) counter osm
218 xtracellular osmolarity stimulate thirst and vasopressin secretion through a central osmoreceptor; ho
223 hage studies, adjunct 1-deamino-8-D-arginine vasopressin showed no benefit in limiting hematoma expan
224 have been made available online for modeling vasopressin signaling and signaling downstream from othe
227 n mouse collecting duct cells to ask whether vasopressin signaling selectively increases Aqp2 gene tr
228 ice to discover an important contribution of vasopressin signaling to the evolution of nest building.
229 -like 3 by PKC or PKA downstream of AngII or vasopressin signaling, respectively, abrogates binding.
232 ng PKA, treatment with antidiuretic hormone (vasopressin) stimulated within 5-minutes proteasomal act
233 level in wild-type mice but does not restore vasopressin-stimulated levels of urea permeability.
234 model predicts that, through PKA activation, vasopressin stimulates AQP2 exocytosis by inhibiting MAP
235 also predicts that, through PKA activation, vasopressin stimulates Aqp2 transcription through induct
237 was similar to levels in wild-type mice, but vasopressin stimulation of urea permeability in wild-typ
240 ediated inhibition of oxytocin- and arginine-vasopressin-synthesizing paraventricular nucleus (PVN) m
241 thin the domain of social communication, the vasopressin system is implicated in social cognition and
242 o effects by targeting pharmacologically the vasopressin system, characterized by a sexually dimorphi
245 ultured cells that indicates aldosterone and vasopressin, the two major hormones regulating sodium re
247 ated by the neurohypophyseal peptide hormone vasopressin through actions in renal collecting duct cel
249 ounts for the ability of the peptide hormone vasopressin to regulate water excretion via a phosphoryl
250 ugh these findings do not support the use of vasopressin to replace norepinephrine as initial treatme
252 ctivity of the net transcriptional response, vasopressin treatment was associated with increased RNA
254 mbrane-delimited G protein-coupled receptor (vasopressin type 2 receptor; V(2)R) and a cytosolic prot
256 vasopressor in septic shock; however, early vasopressin use has been proposed as an alternative.
259 vasopressin analogue that acts, via vascular vasopressin V1 receptors, as a systemic vasoconstrictor.
261 olished by pretreatment of the RVLM with the vasopressin V1a receptor antagonist, SR 49059 (-1 +/- 1
262 t vascular tone through interaction with the vasopressin V1a receptor but that [Pyr(1) ]apelin-13-ind
263 activated the insect inotocin and the human vasopressin V1b receptors, but inhibited the human V1aR.
266 hanistic implications on the emerging use of vasopressin V2 receptor antagonists such as tolvaptan as
267 vated betaarr1 upon its interaction with the vasopressin V2 receptor C-terminal phosphopeptide (V2Rpp
270 tream from a G-protein-coupled receptor, the vasopressin V2 subtype receptor, and its role of the reg
271 atinine clearance, >/=60 ml per minute), the vasopressin V2-receptor antagonist tolvaptan slowed the
272 definition (lactate > 2 mmol/L) differ from vasopressin versus norepinephrine and mortality in Vasop
274 gnificantly (p = 0.028) lower mortality with vasopressin versus norepinephrine in lactate less than o
275 variants were further assessed in the VASST (Vasopressin versus Norepinephrine Infusion in Patients w
276 St Paul's Intensive Care Unit 2 (n=203), and Vasopressin Versus Norepinephrine Infusion in Patients W
277 d a continuous infusion of norepinephrine or vasopressin via microosmotic pumps and were challenged w
278 y and control the synthesis and secretion of vasopressin (VP) and oxytocin (OT) by the neurohypophysi
279 y of activity-dependent dendritic release of vasopressin (VP) from hypothalamic neurones in brain sli
280 escribe a critical role for the neuropeptide vasopressin (VP) in social buffering of synaptic metapla
281 (AQP2) is crucial for water homeostasis, and vasopressin (VP) induces AQP2 membrane trafficking by in
282 units (alpha, beta and gamma) are located in vasopressin (VP) magnocellular neurons in the hypothalam
287 than in the norepinephrine group (25.4% for vasopressin vs 35.3% for norepinephrine; difference, -9.
288 ized clinical trial in septic shock (VANISH [Vasopressin vs. Norepinephrine as Initial Therapy in Sep
291 een pSTAT5 and vesicular GABA transporter or vasopressin were observed, whereas approximately 20% and
292 gned to mimic the N terminus of oxytocin and vasopressin, were assessed and compared based on their a
293 ased on norepinephrine equivalents excluding vasopressin, were significantly lower at 24 hours in the
294 physiological concentrations of glucagon and vasopressin, where glucagon also facilitated the recruit
297 lly, we observed robust induction of NHA2 by vasopressin, which is physiologically consistent with in
298 tem inhibitor therapy versus those receiving vasopressin who were not on chronic renin-angiotensin-al
299 pressure in septic shock patients receiving vasopressin who were on chronic renin-angiotensin-aldost
300 response in septic shock patients receiving vasopressin who were on chronic renin-angiotensin-aldost