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1 d by exposure to supraphysiological doses of vasopressin.
2 ccelerates its endocytosis in the absence of vasopressin.
3 s9) to identify PKA-independent responses to vasopressin.
4 relationship with the neuropeptide modulator vasopressin.
5 melanin-concentrating hormone, oxytocin, and vasopressin.
6 al ganglion cells expresses the neuropeptide vasopressin.
7 fy genes whose transcription is regulated by vasopressin.
8 he presence or absence of norepinephrine and vasopressin.
9 endothelin-1, gentamicin, norepinephrine and vasopressin.
10                                    Concealed vasopressin (0.03 U/min.) or norepinephrine infusions.
11     Treatment with IV 1-deamino-8-D-arginine vasopressin (0.4 ug/kg) + platelet transfusion (2 U) wit
12 ly led to a low single digit nanomolar human vasopressin 1a (hV1a) receptor antagonist 38.
13  of intranasal administration of an arginine vasopressin 1A and 1B receptor agonist against 1) no tre
14 en behavioral variation and variation in the vasopressin 1a receptor gene (Avpr1a) in bonobos.
15 ectivity versus the related receptors (human vasopressin 1a receptor, human vasopressin 1b receptor,
16 e attenuated by systemic pretreatment with a vasopressin-1a-receptor antagonist.
17 A2 area of the hippocampus, particularly the vasopressin 1b receptor (Avpr1b) expressed there, is nec
18 eptors (human vasopressin 1a receptor, human vasopressin 1b receptor, and human oxytocin receptor), a
19 ginine(8) vasopressin, dDAVP, 1) is a potent vasopressin 2 (V(2)) receptor (V(2)R) agonist approved i
20 12330A or the PKA inhibitor H89, but not the vasopressin 2 receptor (V(2)R) antagonist tolvaptan.
21 only a phosphorylated C-terminal tail of the vasopressin 2 receptor activates Src as efficiently as G
22                                 Tolvaptan, a vasopressin 2 receptor blocker, has been approved by reg
23                                     The oral vasopressin-2 receptor antagonist tolvaptan causes aquar
24 that resembles binding of the phosphorylated vasopressin-2 receptor tail to beta-arrestin-1.
25 ve focused on the neuropeptides oxytocin and vasopressin(4-6), which regulate aspects of social behav
26 s increased in PKA-null cells indicates that vasopressin activates one or more members of the AMPK/SN
27    Furthermore, the effect of IRAP to reduce vasopressin activity is a physiologically important cons
28 (adjusted odds ratio, 1.5; 95% CI, 1.3-1.8), vasopressin (adjusted odds ratio, 1.5; 95% CI, 1.1-2.1),
29 These activations weakened with oxytocin and vasopressin administration such that neural responses to
30 lls describing the response to V2R-selective vasopressin agonists and antagonists, the response to CR
31                                              Vasopressin agonists boosted placebo effects in women bu
32 Drugs (caffeine, theophylline) and hormones (vasopressin, aldosterone) known to exacerbate cysts elic
33 mes between standard dose of epinephrine and vasopressin alone or in combination with epinephrine.
34 ercentage of patients receiving epinephrine, vasopressin, amiodarone, lidocaine, atropine, bicarbonat
35  latter conferred responsiveness to arginine-vasopressin (an inhibitory PKC-dependent response).
36                  We investigated whether the vasopressin-analog, terlipressin induces systemic vasoco
37                                          The vasopressin analogue desmopressin (desamino-d-arginine(8
38                  Terlipressin is a synthetic vasopressin analogue that acts, via vascular vasopressin
39 ECV transfer and report that desmopressin, a vasopressin analogue, stimulated the uptake of fluoresce
40 ich when activated result in elevated plasma vasopressin and corticosterone.
41                            The neuropeptides vasopressin and corticotropin-releasing factor facilitat
42                        Patients who received vasopressin and its analogs had a reduced need for renal
43         In patients with distributive shock, vasopressin and its analogs use is associated with a red
44                             In rodents, both vasopressin and oxytocin magnocellular neurones are osmo
45                                Magnocellular vasopressin and oxytocin neurones in the rat hypothalamu
46 r isotocin, homologues of mammalian arginine vasopressin and oxytocin that are broadly implicated in
47 NCs, leading to a decrease in the release of vasopressin and oxytocin.
48 to 0.06 U/min) and hydrocortisone (n = 101), vasopressin and placebo (n = 104), norepinephrine and hy
49 are differences in plasma cytokine levels in Vasopressin and Septic Shock Trial for lactate less than
50 pinephrine group 28- and 90-day mortality in Vasopressin and Septic Shock Trial in lactate subgroups.
51  rates were 10-12 % higher than the original Vasopressin and Septic Shock Trial mortality.
52                 In genotyped patients in the Vasopressin and Septic Shock Trial trial, we found that
53                  In 632 patients enrolled in Vasopressin and Septic Shock Trial, Proprotein Convertas
54 essin versus norepinephrine and mortality in Vasopressin and Septic Shock Trial.
55  included genotyped patients enrolled in the Vasopressin and Septic Shock Trial.
56 l was applied to patients with AKI in VASST (Vasopressin and Septic Shock Trial; n = 271), and differ
57 mutant and wild-type mice had similar plasma vasopressin and solute excretion levels.
58 sistent with increased levels of circulating vasopressin and up-regulation of vasopressin V2 receptor
59 eptidergic neurons in the SCN: AVP (arginine vasopressin) and VIP (vasoactive intestinal polypeptide)
60 l copeptin production, a surrogate marker of vasopressin, and dependent on the experience of birth-re
61                     Many SCN neurons express vasopressin, and it has been assumed that the role of va
62 a potential clinically important benefit for vasopressin, and larger trials may be warranted to asses
63 unctions, abnormalities in central oxytocin, vasopressin, and serotonin neurotransmission, and neuroi
64 in 1776 patients enrolled in the Efficacy of Vasopressin Antagonism in Heart Failure Outcome Study Wi
65 oc analysis was performed of the Efficacy of Vasopressin Antagonism in Heart Failure: Outcome Study w
66 a early in patients selected for an enhanced vasopressin antagonism response.
67                                   Currently, vasopressin antagonists should not be offered routinely
68        By contrast, aquaretic drugs (such as vasopressin antagonists) predominantly cause water excre
69                          Upon stimulation by vasopressin, AQP2 is phosphorylated at serine 256 (S256)
70  the endogenous IRAP substrates oxytocin and vasopressin are known to facilitate learning and memory.
71 ributing to those differences and identified vasopressin as a regulator of nestbuilding behaviour.
72        In cancer patients with septic shock, vasopressin as first-line vasopressor therapy was not su
73 esearch due to the therapeutic potentials of vasopressin as well as the possibility to systematically
74 vation of both oxytocin receptors (OXTR) and vasopressin (AVP) 1a receptors (AVPR1a).
75 we report that serotonin (5-HT) and arginine-vasopressin (AVP) act in opposite ways in the hypothalam
76 olocalization with the neuropeptide arginine vasopressin (AVP) and clock proteins (PER2 and BMAL1), s
77 nse to the neurohypophyseal hormone arginine vasopressin (AVP) and in the expression of oxidative str
78                                     Arginine vasopressin (AVP) and its type-2 receptor (V2R) play an
79                            The neuropeptides vasopressin (AVP) and oxytocin (OT) have been implicated
80 s was dependent on the neuropeptide arginine vasopressin (AVP) because it was prevented by pharmacolo
81                                     Arginine-vasopressin (AVP) binding to vasopressin V2 receptors pr
82 ts, which contain a mutation in the arginine vasopressin (AVP) gene, exhibit lower behavioral arousal
83                                     Arginine vasopressin (AVP) has a key role in osmoregulation by fa
84   Prior studies show that oxytocin (Oxt) and vasopressin (Avp) have opposing actions on the skeleton
85 ts from a deficiency of the hormone arginine vasopressin (AVP) in the pituitary gland or the hypothal
86 ration of the "social" neuropeptide arginine vasopressin (AVP) is significantly lower in pediatric AS
87 accompanied by elevated circulating arginine vasopressin (AVP) levels in SHR-A3 compared with SHR-B2.
88 of the SCN, including VIP, GRP, and arginine vasopressin (AVP) neurons, with each ipRGC innervating s
89 asmatic nucleus output neuropeptide arginine-vasopressin (AVP) on the activity of preoptic area kissp
90                    The neuropeptide arginine vasopressin (AVP) plays significant roles in maintaining
91 a self-assembling lipidized peptide arginine vasopressin (AVP) receptor agonist, that had not been de
92                           Synthetic arginine vasopressin (AVP) receptor agonists able to induce syste
93                                     Arginine vasopressin (AVP) regulates fluid balance and blood pres
94 ogy of thirst, taste for water, and arginine-vasopressin (AVP) release: ( a) Thirst and AVP release a
95                                     Arginine vasopressin (AVP) stimulates the release of enteroendocr
96 iciency of the antidiuretic hormone arginine vasopressin (AVP) underlies diabetes insipidus, which is
97 al three distinct binding sites for arginine vasopressin (AVP) within its V2 -receptor (V2 R).
98                   Here we show that arginine vasopressin (AVP), a neuropeptide that mediates complex
99 ncy and preschool, assayed oxytocin (OT) and vasopressin (AVP), and measured coparenting and child be
100  of copeptin, a surrogate marker of arginine vasopressin (AVP), to be associated with increased risk
101 ause cAMP is a central modulator of arginine vasopressin (AVP)-induced water transport in the renal c
102 e with a reduction of parvocellular arginine vasopressin (AVP)-positive neurons in the preoptic area,
103  PDE4 caused a greater increase in basal and vasopressin (AVP)-stimulated cAMP levels and Cl(-) secre
104  first study that demonstrates that arginine vasopressin boosts placebo effects and that the effect o
105 id not detect major deficiencies in arginine vasopressin [Ca(2+)](i) signaling in split-opened collec
106                                              Vasopressin cannot correct paracellular water loss in Il
107                                     Arginine vasopressin colocalized with EGFP more often in females
108 g adults with septic shock, the early use of vasopressin compared with norepinephrine did not improve
109                                    In VASST, vasopressin compared with norepinephrine was associated
110 gnetic resonance imaging, and serum arginine vasopressin concentration) were compatible with a diagno
111 he aim of this study was to evaluate whether vasopressin could be superior to norepinephrine to impro
112       In response to 1-desamino-8-D-arginine vasopressin (DDAVP), peak VWF:Ag levels exceeded 100 IU/
113 nalogue desmopressin (desamino-d-arginine(8) vasopressin, dDAVP, 1) is a potent vasopressin 2 (V(2))
114 controls AQP2 apical membrane abundance in a vasopressin-dependent manner, allowing for urine volume
115 oosts placebo effects and that the effect of vasopressin depends upon a significant sex by treatment
116 e to sepsis-induced immunoparalysis, whereas vasopressin does not have untoward immunologic effects.
117 ropin-releasing hormone-dominant to arginine vasopressin-dominant, and cortisol levels remain raised
118                                              Vasopressin exerted no immunomodulatory effects across t
119 were randomized to receive norepinephrine or vasopressin followed by hydrocortisone or placebo.
120 ong evidence suggests that certain hormones (vasopressin), foods (fructose), and metabolic products (
121 untered by intensivists who regularly employ vasopressin for the treatment of vasoplegic shock.
122 ctivity-dependent somatodendritic release of vasopressin from paraventricular nucleus neurones.
123 iased modulation is mediated by oxytocin and vasopressin G-protein-coupled receptors.
124 other GPCR agonists, including ADP, arginine vasopressin, glucagon-like peptide 1, and forskolin, and
125 ctor, dynorphin, norepinephrine, hypocretin, vasopressin, glucocorticoids, and neuroimmune factors) i
126 (interquartile range [IQR], 1 to -24) in the vasopressin group and 13 days (IQR, 1 to -25) in the nor
127 e was observed in 71 patients (56.8%) in the vasopressin group and 66 patients (52.8%) in the norepin
128 ailure was 94 of 165 patients (57.0%) in the vasopressin group and 93 of 157 patients (59.2%) in the
129 less use of renal replacement therapy in the vasopressin group than in the norepinephrine group (25.4
130 s (10.7%) had a serious adverse event in the vasopressin group vs 17 of 204 patients (8.3%) in the no
131                                              Vasopressin has achieved common usage for the treatment
132                             The neuropeptide vasopressin has an important role in this entrainment.
133 as implicated the neuropeptides oxytocin and vasopressin in the modulation of human neural activity u
134 in, and it has been assumed that the role of vasopressin in the SCN reflects the activity of these ce
135                      We have discovered that vasopressin-independent facets of this homeostatic mecha
136 genic hormones, including catecholamines and vasopressin, induce frequency-modulated cytosolic Ca(2+)
137                                              Vasopressin-induced aquaporin-2 phosphorylation within t
138 xiety and cortisol levels showed the largest vasopressin-induced modulation of placebo effects, sugge
139  ICU patients with septic shock who received vasopressin infusion added to at least one concomitant v
140  diabetes insipidus after discontinuation of vasopressin infusion among patients treated with vasopre
141  diabetes insipidus after discontinuation of vasopressin infusion among patients with septic shock as
142  diabetes insipidus after discontinuation of vasopressin infusion appears not to be a rare phenomenon
143 pressin infusion among patients treated with vasopressin infusion for shock.
144 of patients receiving norepinephrine but not vasopressin infusion for treatment of shock, criteria fo
145  diabetes insipidus after discontinuation of vasopressin infusion had undergone cardiothoracic interv
146  diabetes insipidus after discontinuation of vasopressin infusion have been published; the majority o
147  diabetes insipidus after discontinuation of vasopressin infusion remains to be elucidated but may in
148  diabetes insipidus after discontinuation of vasopressin infusion was 1.53% among all patients.
149 yscus and chemogenetics in Mus, we show that vasopressin inhibits nest building but not other parenta
150 ollecting duct; is modulated in abundance by vasopressin; interacts with aquaporin-2 (AQP2), Hsp70, a
151                            Here we show that vasopressin is also expressed in many retinal cells that
152 d to differences in nest-building behaviour, vasopressin is differentially expressed in the hypothala
153  pig neonate demonstrated that the effect of vasopressin is not dependent on the level of maturation
154                                              Vasopressin levels begin to fall within minutes of water
155 thetic nervous system activity, and elevated vasopressin levels.
156 developed polyuria despite elevated arginine vasopressin levels.
157 own insect neuropeptides except for arginine-vasopressin-like peptide (AVLP), CNMamide, neuropeptide-
158                                              Vasopressin lowered mortality versus norepinephrine if l
159                 Previous trials suggest that vasopressin may improve outcomes in patients with vasodi
160 twork describing PKA signaling that explains vasopressin-mediated regulation of membrane trafficking
161                                              Vasopressin-mediated regulation of renal water excretion
162 r genes, consistent with the conclusion that vasopressin-mediated transcriptional regulation is highl
163 e how intranasally administered oxytocin and vasopressin modulated neural activity when receiving neg
164   We localized a subpopulation of excitatory vasopressin neurons in the anterior hypothalamus that ma
165 ensitive neurons in the lamina terminals and vasopressin neurons in the paraventricular nucleus of th
166 nts of genetically identified neuroendocrine vasopressin neurons show they can anticipate osmotic cha
167 terminalis also resulted in Fos induction in vasopressin neurons within the PVN and supraoptic nucleu
168  activity (Normal: p < 0.001; HF: p < 0.05), vasopressin (Normal: p < 0.001; HF: p < 0.01), and cycli
169                               The effects of vasopressin on expectancy-induced analgesia were signifi
170 sing; however, the influence of oxytocin and vasopressin on neural activity elicited during negative
171 ur results show effects of both oxytocin and vasopressin on the brain network involved in negative so
172 repinephrine and the alternative vasopressor vasopressin on the immune response and host defense.Meth
173 e liver with physiological concentrations of vasopressin only will evoke localized cytosolic calcium
174             Patients were assigned to either vasopressin or norepinephrine as first-line vasopressor
175 ess than or equal to 2 mmol/L, randomized to vasopressin or norepinephrine.
176 eleasing hormone and do not express arginine vasopressin or oxytocin.
177  responses were provoked by stimulation with vasopressin or serum.
178 ized to a 5-hour infusion of norepinephrine, vasopressin, or saline and intravenously challenged with
179 luenced response to either norepinephrine or vasopressin, or to corticosteroids in septic shock.
180              We isolated the insect oxytocin/vasopressin orthologue inotocin from the black garden an
181 e social behavior have mainly focused on the vasopressin-oxytocin family.
182 ntagonism acting on loci within the arginine vasopressin-oxytocin pathway explains how genetic divers
183 ted to gain novel insights into the oxytocin/vasopressin peptide-receptor interaction, which led to t
184        Treatment with 1-deamino-8-D-arginine vasopressin + platelet transfusion was not associated wi
185  for the clearance of misfolded pro-arginine vasopressin (proAVP) in the ER.
186 nd functional analysis of thirst neurons and vasopressin-producing neurons.
187        Reciprocally, Gs enhances IP1 through vasopressin receptor (V1A-R) but not alpha1 adrenergic r
188 VPA exposure downregulated prefrontal cortex vasopressin receptor (V1aR) and methyl CpG-binding prote
189 tissues through the inhibition of the type-2 vasopressin receptor (V2R) constitutes a validated strat
190                   Here we demonstrate that a vasopressin receptor (V2R) mutant with truncated third i
191                       Expression of arginine vasopressin receptor 1a (Avpr1a) and oxytocin receptor (
192 ingle 20 mg intravenous dose of the arginine vasopressin receptor 1A (V1a) antagonist, RG7713, on exp
193  treatment of mpkCCD14 cells with the type 2 vasopressin receptor agonist dDAVP increased mRNA and pr
194                                              Vasopressin receptor antagonists, urea, and loop diureti
195 expression variations of the oxytocin and/or vasopressin receptor genes OXTR and AVPR1A, respectively
196 e value in testing medications targeting the vasopressin receptor in high stress, alcohol-dependent p
197  selected for greater potential benefit from vasopressin receptor inhibition, tolvaptan was not assoc
198 n important role for development of oxytocin/vasopressin receptor modulators that would enable clear
199           In the present study, we show that vasopressin receptor type-2 (V2R) is localized to cilia
200                                   First, the vasopressin receptor-2 agonist, dDAVP, was delivered to
201 lial cells expressing the Galphas-coupled V2 vasopressin receptor.
202                                              Vasopressin receptors appear to have a regulatory role i
203 zation of the parathyroid hormone and type 2 vasopressin receptors.
204  properties on the insect and human oxytocin/vasopressin receptors.
205                The analysis revealed only 35 vasopressin-regulated genes (of 3659) including Aqp2.
206               These data are consistent with vasopressin-regulated uptake of ECVs in vivo We conclude
207                                              Vasopressin regulates phosphorylation at four sites with
208                                              Vasopressin regulates renal water excretion by binding t
209    These anticipatory signals for thirst and vasopressin release concentrate on the same homeostatic
210                                 Light-evoked vasopressin release contributes to the responses of SCN
211                                Light-induced vasopressin release enhances the responses of SCN neuron
212 sepsis features impaired thirst and enhanced vasopressin release, the basis for these defects is unkn
213 nabled a distinctive regulation of pituitary vasopressin release.
214 which increases histone H3K27 acetylation of vasopressin-responsive genes (confirmed by ChIP-seq).
215 otic stress, posterior pituitary-projecting, vasopressin-secreting neurons (VPpp neurons) counter osm
216 tinct cellular processes to regulate thirst, vasopressin secretion and autonomic function.
217                       Osmotically stimulated vasopressin secretion promotes antidiuresis while oxytoc
218 xtracellular osmolarity stimulate thirst and vasopressin secretion through a central osmoreceptor; ho
219 ey forebrain neurons that control thirst and vasopressin secretion.
220 other osmolytes readily stimulate thirst and vasopressin secretion.
221                         Alternatives such as vasopressin/selepressin, angiotensin II, and phenylephri
222                                 In contrast, vasopressin, serotonin, and testosterone play only limit
223 hage studies, adjunct 1-deamino-8-D-arginine vasopressin showed no benefit in limiting hematoma expan
224 have been made available online for modeling vasopressin signaling and signaling downstream from othe
225                                              Vasopressin signaling is initiated by binding to a G-pro
226           The results show that V2R-mediated vasopressin signaling is predominantly, but not entirely
227 n mouse collecting duct cells to ask whether vasopressin signaling selectively increases Aqp2 gene tr
228 ice to discover an important contribution of vasopressin signaling to the evolution of nest building.
229 -like 3 by PKC or PKA downstream of AngII or vasopressin signaling, respectively, abrogates binding.
230 rvation of the 600-million-year-old oxytocin/vasopressin signalling system.
231                  Here, we show that arginine vasopressin specifically activates interneurons to suppr
232 ng PKA, treatment with antidiuretic hormone (vasopressin) stimulated within 5-minutes proteasomal act
233 level in wild-type mice but does not restore vasopressin-stimulated levels of urea permeability.
234 model predicts that, through PKA activation, vasopressin stimulates AQP2 exocytosis by inhibiting MAP
235  also predicts that, through PKA activation, vasopressin stimulates Aqp2 transcription through induct
236                                        Since vasopressin stimulates renal water and sodium reabsorpti
237 was similar to levels in wild-type mice, but vasopressin stimulation of urea permeability in wild-typ
238           During perfusion with subthreshold vasopressin, sympathetic stimulation converted asynchron
239 pothalamus (PVN) that appear to synapse onto vasopressin-synthesizing neurons.
240 ediated inhibition of oxytocin- and arginine-vasopressin-synthesizing paraventricular nucleus (PVN) m
241 thin the domain of social communication, the vasopressin system is implicated in social cognition and
242 o effects by targeting pharmacologically the vasopressin system, characterized by a sexually dimorphi
243 ease of cross-reactivity with its correlated vasopressin system.
244               We used optogenetics to excite vasopressin terminals, originating from the hypothalamic
245 ultured cells that indicates aldosterone and vasopressin, the two major hormones regulating sodium re
246 different clinical outcomes and responses to vasopressin therapy.
247 ated by the neurohypophyseal peptide hormone vasopressin through actions in renal collecting duct cel
248          Patients were randomly allocated to vasopressin (titrated up to 0.06 U/min) and hydrocortiso
249 ounts for the ability of the peptide hormone vasopressin to regulate water excretion via a phosphoryl
250 ugh these findings do not support the use of vasopressin to replace norepinephrine as initial treatme
251                                  We compared vasopressin-to-norepinephrine group 28- and 90-day morta
252 ctivity of the net transcriptional response, vasopressin treatment was associated with increased RNA
253 tor with the C-terminal tail of the arginine vasopressin type 2 receptor (beta(2)V(2)R).
254 mbrane-delimited G protein-coupled receptor (vasopressin type 2 receptor; V(2)R) and a cytosolic prot
255           In this study, we examined whether vasopressin type-2 receptor (V2R) activity in cystic epi
256  vasopressor in septic shock; however, early vasopressin use has been proposed as an alternative.
257 onists to determine the role of oxytocin and vasopressin V(1a) receptors.
258 tin recruitment to the angiotensin AT(1) and vasopressin V(2) receptors.
259 vasopressin analogue that acts, via vascular vasopressin V1 receptors, as a systemic vasoconstrictor.
260                     Selepressin, a selective vasopressin V1a receptor agonist, is a noncatecholaminer
261 olished by pretreatment of the RVLM with the vasopressin V1a receptor antagonist, SR 49059 (-1 +/- 1
262 t vascular tone through interaction with the vasopressin V1a receptor but that [Pyr(1) ]apelin-13-ind
263  activated the insect inotocin and the human vasopressin V1b receptors, but inhibited the human V1aR.
264 iven fluid secretion (PDE3), and response to vasopressin V2 receptor activation (both).
265  mice before and after administration of the vasopressin V2 receptor antagonist tolvaptan.
266 hanistic implications on the emerging use of vasopressin V2 receptor antagonists such as tolvaptan as
267 vated betaarr1 upon its interaction with the vasopressin V2 receptor C-terminal phosphopeptide (V2Rpp
268 circulating vasopressin and up-regulation of vasopressin V2 receptors in ADPKD.
269        Arginine-vasopressin (AVP) binding to vasopressin V2 receptors promotes redistribution of the
270 tream from a G-protein-coupled receptor, the vasopressin V2 subtype receptor, and its role of the reg
271 atinine clearance, >/=60 ml per minute), the vasopressin V2-receptor antagonist tolvaptan slowed the
272  definition (lactate > 2 mmol/L) differ from vasopressin versus norepinephrine and mortality in Vasop
273            Our first hypothesis was that the vasopressin versus norepinephrine comparison and 28-day
274 gnificantly (p = 0.028) lower mortality with vasopressin versus norepinephrine in lactate less than o
275 variants were further assessed in the VASST (Vasopressin versus Norepinephrine Infusion in Patients w
276 St Paul's Intensive Care Unit 2 (n=203), and Vasopressin Versus Norepinephrine Infusion in Patients W
277 d a continuous infusion of norepinephrine or vasopressin via microosmotic pumps and were challenged w
278 y and control the synthesis and secretion of vasopressin (VP) and oxytocin (OT) by the neurohypophysi
279 y of activity-dependent dendritic release of vasopressin (VP) from hypothalamic neurones in brain sli
280 escribe a critical role for the neuropeptide vasopressin (VP) in social buffering of synaptic metapla
281 (AQP2) is crucial for water homeostasis, and vasopressin (VP) induces AQP2 membrane trafficking by in
282 units (alpha, beta and gamma) are located in vasopressin (VP) magnocellular neurons in the hypothalam
283                                      Because vasopressin (VP) neurons play a pivotal role in coordina
284 s insipidus (NDI) is caused by impairment of vasopressin (VP) receptor type 2 signaling.
285                     Here, we show that local vasopressin (VP) signaling in PVN buffers the short-term
286                 ABSTRACT: Oxytocin (OT)- and vasopressin (VP)-secreting magnocellular neurons of the
287  than in the norepinephrine group (25.4% for vasopressin vs 35.3% for norepinephrine; difference, -9.
288 ized clinical trial in septic shock (VANISH [Vasopressin vs. Norepinephrine as Initial Therapy in Sep
289                   Somatodendritic release of vasopressin was rarely observed during continuous firing
290 ich has been linked to nonosmotic release of vasopressin) was assessed.
291 een pSTAT5 and vesicular GABA transporter or vasopressin were observed, whereas approximately 20% and
292 gned to mimic the N terminus of oxytocin and vasopressin, were assessed and compared based on their a
293 ased on norepinephrine equivalents excluding vasopressin, were significantly lower at 24 hours in the
294 physiological concentrations of glucagon and vasopressin, where glucagon also facilitated the recruit
295 c perturbations by altering their release of vasopressin, which controls renal water excretion.
296                                              Vasopressin, which is a nonapeptide with substantial str
297 lly, we observed robust induction of NHA2 by vasopressin, which is physiologically consistent with in
298 tem inhibitor therapy versus those receiving vasopressin who were not on chronic renin-angiotensin-al
299  pressure in septic shock patients receiving vasopressin who were on chronic renin-angiotensin-aldost
300  response in septic shock patients receiving vasopressin who were on chronic renin-angiotensin-aldost

 
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