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1 mporal vein, suggesting mixed arteriolar and venous occlusion.
2 are at high risk for thrombotic arterial and venous occlusions.
3  in improved specificity in the detection of venous occlusions (0.99 vs 0.96, P = .03), in reader con
4 lar stroke volume and their changes during a venous occlusion and release maneuver to a calibrated ao
5 alfunction (30%), and other (device upgrade, venous occlusion, and advisory leads; 20%).
6  occlusions (pulmonary atresia, arterial and venous occlusion, and iatrogenic graft occlusion), trave
7 urements on melanocytic nevus, vitiligo, and venous occlusion conditions were performed in volunteers
8                                  Presumably, venous occlusion could occur owing to thrombus formation
9 venting excessive tissue swelling leading to venous occlusion during catheter ablation procedures.
10 plored the potential for tissue swelling and venous occlusion during radiofrequency (RF) catheter abl
11 e treated due to symptomatic portomesenteric venous occlusion of thrombotic origin.
12  proliferative diabetic retinopathy, retinal venous occlusion or wet age-related macular degeneration
13 lium-dependent vasodilation, by both forearm venous occlusion plethysmography (93 +/- 67% vs. 145 +/-
14 asma fibrinolytic factors were assessed with venous occlusion plethysmography and blood sampling duri
15                                      Forearm venous occlusion plethysmography and intra-arterial infu
16 sured resting forearm blood flow (FBF) using venous occlusion plethysmography before and after intra-
17  and 45% of maximal voluntary contraction by venous occlusion plethysmography before and after region
18 ndependent vasodilatation were assessed with venous occlusion plethysmography before and during intra
19 t period, forearm blood flow was measured by venous occlusion plethysmography during an intrabrachial
20           Forearm blood flow was measured by venous occlusion plethysmography during intrabrachial in
21 ceptor antagonist BQ-123 were assessed using venous occlusion plethysmography in 10 patients with syn
22         Forearm blood flow was measured with venous occlusion plethysmography in 12 cigarette smokers
23           Forearm blood flow was measured by venous occlusion plethysmography in 16 volunteers during
24           Forearm blood flow was measured by venous occlusion plethysmography in response to serial i
25 erial vasodilator responses were assessed by venous occlusion plethysmography in the brachial circula
26         Forearm blood flow was measured with venous occlusion plethysmography in the resting forearm.
27                                       Supine venous occlusion plethysmography showed no differences b
28 ine gingival capillary density (GCD); and 3) venous occlusion plethysmography to assess endothelium-d
29 al (water, Na(+), and K(+)) analyses; and 2) venous occlusion plethysmography to assess peripheral mi
30 l function was assessed by bilateral forearm venous occlusion plethysmography using acetylcholine and
31                                              Venous occlusion plethysmography was used to assess fore
32                                              Venous occlusion plethysmography was used to assess resi
33                    To address this question, venous occlusion plethysmography was used to measure for
34 conduit vessel response), and in 6 subjects, venous occlusion plethysmography was used to measure for
35                                              Venous occlusion plethysmography was used to measure pea
36                   Subjects (n=157) underwent venous occlusion plethysmography with acetylcholine, bra
37 al function was assessed by standard forearm venous occlusion plethysmography with acetylcholine, nit
38  was measured simultaneously in both arms by venous occlusion plethysmography with mercury-in-Silasti
39 trials by measuring forearm blood flow (FBF; venous occlusion plethysmography) after 5 minutes of art
40 ocol 1, we measured forearm blood flow (FBF; venous occlusion plethysmography) and calculated the vas
41              We measured forearm blood flow (venous occlusion plethysmography) and calculated vascula
42 ilution), forearm vascular conductance (FVC, venous occlusion plethysmography) and cutaneous vascular
43 ulated from forearm blood flow (measured via venous occlusion plethysmography) and intra-arterial blo
44 acebo, allopurinol improved peak blood flow (venous occlusion plethysmography) in arms (+24%, P=0.027
45  healthy young men, forearm blood flow (FBF; venous occlusion plethysmography) responses to brachial
46                          Forearm blood flow (venous occlusion plethysmography) was measured during br
47 e (BP), heart rate (HR), forearm blood flow (venous occlusion plethysmography), FVR, and MSNA (obtain
48         Forearm blood flow was determined by venous occlusion plethysmography, and dose-response curv
49                   Blood flow was measured by venous occlusion plethysmography, and the percentage of
50           Forearm blood flow was measured by venous occlusion plethysmography, before, and 8 min afte
51 erial pressure were measured using bilateral venous occlusion plethysmography, bioimpedance cardiogra
52 ar pressure, and cardiac output were made by venous occlusion plethysmography, Doppler flow wire and
53 Endothelial function was assessed by forearm venous occlusion plethysmography, flow-mediated dilation
54 orearm and calf blood flow were evaluated by venous occlusion plethysmography, MSNA by microneurograp
55 and sodium nitroprusside were assessed using venous occlusion plethysmography.
56 al function was assessed by standard forearm venous occlusion plethysmography.
57 ity, carotid-radial pulse wave velocity, and venous occlusion plethysmography.
58 elation was computed for every subject using venous occlusion plethysmography.
59 earm blood flow was measured using bilateral venous occlusion plethysmography.
60 ceptor antagonist (0.3 and 1 nmol/min) using venous occlusion plethysmography.
61  of patients with coronary artery disease by venous occlusion plethysmography.
62 FBF was measured in 10 cirrhotic patients by venous occlusion plethysmography.
63         Forearm blood flow was determined by venous occlusion plethysmography.
64 red in the resting non-dominant forearm with venous occlusion plethysmography.
65 dothelial nitric oxide production by forearm venous occlusion plethysmography.
66 hile forearm blood flow (FBF was measured by venous occlusion plethysmography.
67 rm blood flow (FBF) measured by strain-gauge venous occlusion plethysmography.
68        In the remaining six patients, portal venous occlusion precluded access to the extrahepatic po
69 the dependent human foot using a single-step venous occlusion protocol.
70                RFA and EVLT offer comparable venous occlusion rates at 3 months after treatment of pr
71        Forearm blood flow was measured using venous-occlusion, strain-gauge plethysmography.
72 ortic access, recanalization of arterial and venous occlusions, transseptal access, and many other te
73 cclusive peak reactive hyperemia) and during venous occlusion (venous congestion), as assessed with s
74  (MVC) immediately followed by either a: (i) venous occlusion (VO); (ii); arterial occlusion (AO); or