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1 diac index was reduced with maintained mixed venous oxygen saturation.
2  should not be used as surrogate for central venous oxygen saturation.
3 arterial lactate concentrations, and central venous oxygen saturation.
4 ul for predicting low cardiac index or mixed venous oxygen saturation.
5 ion with cardiac index, lactate, and central venous oxygen saturation.
6 ral venous oxygen saturation for a low mixed venous oxygen saturation.
7 ght atrial pressure cardiac index, and mixed venous oxygen saturations.
8 ht atrial pressure, cardiac index, and mixed venous oxygen saturations.
9 atients, 31 catheters) and 65 pairs of mixed venous oxygen saturation (20 patients, 28 catheters) wer
10 - 0.9 to 15.7 +/- 2.4; p < .0001); and mixed venous oxygen saturation (26 +/- 5 to 36 +/- 5, p < .05)
11 or mixed venous oxygen saturation or central venous oxygen saturation; -3.7% (-4.4% to -3.0%) (p < 0.
12 olume (54 +/- 27 to 65 +/- 38 ml), and mixed-venous oxygen saturation (69 +/- 8 to 73 +/- 10%), all p
13  a significantly higher mean (+/-SD) central venous oxygen saturation (70.4+/-10.7 percent vs. 65.3+/
14               Results for changes of femoral venous oxygen saturation and central venous oxygen satur
15         Correlation and agreement of femoral venous oxygen saturation and central venous oxygen satur
16 ed, including the difference between femoral venous oxygen saturation and central venous oxygen satur
17 rrelation between obtained values of femoral venous oxygen saturation and central venous oxygen satur
18   There is lack of agreement between femoral venous oxygen saturation and central venous oxygen satur
19           We concurrently determined femoral venous oxygen saturation and central venous oxygen satur
20 ermore, we determined simultaneously femoral venous oxygen saturation and central venous oxygen satur
21 ill patients, the difference between femoral venous oxygen saturation and central venous oxygen satur
22 tly increased systemic hypotension and mixed venous oxygen saturation and decreased pulmonary artery
23 ious studies have reported increased retinal venous oxygen saturation and decreased retinal blood flo
24 ose of our study was to determine if central venous oxygen saturation and femoral venous oxygen satur
25               Six-minute walk results, mixed venous oxygen saturation and initial treatment randomiza
26           We compared the ability of central venous oxygen saturation and markers of anaerobic metabo
27 e determined correlation of baseline central venous oxygen saturation and mixed venous oxygen saturat
28  DCS-derived blood flow index (BFI), and (b) venous oxygen saturation and NIRS-derived tissue saturat
29 d with ECCO(2)R (0.4 L/min), increased mixed venous oxygen saturation and oxygenation, and improved h
30 e central venous oxygen saturation and mixed venous oxygen saturation and predictive value of a low c
31 r resistance indices, and arterial and mixed venous oxygen saturation and reduced pulmonary hypertens
32 vere sepsis, the correlation between central venous oxygen saturation and tissue oxygen saturation at
33 roblems with oxygenation, ventilation, mixed venous oxygen saturation, and cardiac output.
34  P = .02), improved hemodynamics and central venous oxygen saturation, and less organ dysfunction.
35 on, oxygen delivery, mixed venous PO2, mixed venous oxygen saturation, and PmO2 responded with simila
36  change of arterial oxygen saturation, mixed venous oxygen saturation, and PO2.
37  and MSNA, reduced forearm velocity, lowered venous oxygen saturation, and raised venous lactate and
38 cant decrease in Pa(O2 ), arterial and mixed venous oxygen saturation, and ratio of Pa(O(2)) to fract
39 ics, as well as global cardiac output, mixed venous oxygen saturation, and systemic and cerebral oxyg
40                     Perfusion, Intravascular Venous Oxygen saturation, and T2* (PIVOT), a recently de
41 ior probability that cardiac index and mixed venous oxygen saturation are normal and physical examina
42  under the curve 0.66; 0.46-0.86) or central venous oxygen saturation (area under the curve 0.56; 0.3
43  arterial pressure, cardiac index, and mixed venous oxygen saturation, as well as significantly highe
44                              CBF and jugular venous oxygen saturation both increased significantly at
45 to 15% was not predicted by baseline central venous oxygen saturation but by high baseline lactate an
46 central venous oxygen saturation and femoral venous oxygen saturation can be used interchangeably dur
47 ery index, oxygen consumption index, central venous oxygen saturation, central venous-to-arterial car
48                          We recorded central venous oxygen saturation continuously for 0 to 6 hrs of
49 e, and low 24-hr fluid output; and low mixed venous oxygen saturation correlated with knee mottling a
50                Delta - DeltaPCO2 and central venous oxygen saturation could predict extubation failur
51          The bias and precision of the mixed venous oxygen saturation data were -0.57% and 3.76%, res
52                                      Central venous oxygen saturation decreased in the failure group
53 in DeltaPCO2 (Delta - DeltaPCO2) and central venous oxygen saturation (DeltaScvO2) during spontaneous
54 etabolites would be increased during jugular venous oxygen saturation desaturations (<50%) and determ
55 femoral venous oxygen saturation and central venous oxygen saturation.Despite significant correlation
56 ike markers of anaerobic metabolism, central venous oxygen saturation did not allow the prediction of
57  We found that Delta - DeltaPCO2 and central venous oxygen saturation, during spontaneous breathing t
58 ration and predictive value of a low central venous oxygen saturation for a low mixed venous oxygen s
59 lism should be considered instead of central venous oxygen saturation for starting hemodynamic resusc
60 n saturation was better predictor of central venous oxygen saturation &gt;70% than thenar tissue oxygen
61 fied patients with severe sepsis and central venous oxygen saturation &gt;70%.
62 venous oxygen saturation <60%, but a central venous oxygen saturation &gt;or=70% may be useful to exclud
63 HR, BP, MSNA, forearm flow velocity, forearm venous oxygen saturation, H(+), and lactate.
64 femoral venous oxygen saturation and central venous oxygen saturation in 30 surgical patients and in
65 femoral venous oxygen saturation and central venous oxygen saturation in a group of 100 stable cardia
66 of global cerebral oxygenation using jugular venous oxygen saturation in all 126 patients.
67 femoral venous oxygen saturation and central venous oxygen saturation in both stable and unstable med
68 measures continuous cardiac output and mixed venous oxygen saturation in the clinical setting.
69 femoral venous oxygen saturation and central venous oxygen saturation including its range of variatio
70  to 90 mmHg +/- 32 after thrombectomy, mixed venous oxygen saturation increased from 48% +/- 19% to 6
71        The systolic blood pressure and mixed venous oxygen saturation increased from 75 (IQR:15) mm H
72 res, pulmonary vascular pressures, and mixed venous oxygen saturation is a unique feature; c) additio
73                                      Jugular venous oxygen saturation is monitored after traumatic br
74 " is based on continuing resuscitation until venous oxygen saturation is normalized.
75 ous oxygen saturation <70% predicted a mixed venous oxygen saturation &lt;60% with a sensitivity 84%,spe
76 ation <70% does not accurately predict mixed venous oxygen saturation &lt;60%, but a central venous oxyg
77 3% and 86%) for cardiac index <2.5 and mixed venous oxygen saturation &lt;60%, respectively.
78 ation >or=70% may be useful to exclude mixed venous oxygen saturation &lt;60%.
79                                      Central venous oxygen saturation &lt;70% does not accurately predic
80                                      Central venous oxygen saturation &lt;70% predicted a mixed venous o
81 culated the area under the curve for central venous oxygen saturation &lt;70%.
82 ntricular systolic dysfunction had a central venous oxygen saturation&lt;70%.
83 changes in blood volume and measured central venous oxygen saturation (mCVO2) pre-, mid-, and post-he
84  to -0.49 L/min/m; p = 0.00001), and central venous oxygen saturation (mean difference, -5.07; 95% CI
85 system was calibrated every 24 hrs for mixed venous oxygen saturation monitoring.
86 tolic blood pressure of 50-70 mm Hg, a mixed venous oxygen saturation of 25% to 40%, and a UO <10% of
87 ransfusions or inotropes to attain a central venous oxygen saturation of at least 70% did not decreas
88 sue oxygen saturation, brain tissue PO2, and venous oxygen saturation of the superior sagittal sinus
89 ffect of mannitol on brain tissue PO2 and on venous oxygen saturation of the superior sagittal sinus
90  index; 2.9% (2.2-3.5%) (p < 0.01) for mixed venous oxygen saturation or central venous oxygen satura
91 , mean arterial pressure, and either central venous oxygen saturation or lactate clearance.
92 oid tissue oxygen saturation but not central venous oxygen saturation or thenar tissue oxygen saturat
93 ue hypoxia (reflected by lactate and central venous oxygen saturation), organ dysfunction, and mortal
94 s physiologic monitoring (arterial and mixed venous oxygen saturation, oxygen consumption, etc.) may
95                                        Mixed venous oxygen saturation percentage and pulmonary capill
96  cardiac output (r(s) =.52, p<.05) and mixed venous oxygen saturation (r(s) =.61, p<.05).
97 oxygen transport (r=-.68, P=.005), and mixed venous oxygen saturation (r=-.79, P<.0001).
98 x, and PVRI, whereas cardiac index and mixed venous oxygen saturation remained unchanged.
99                                 When central venous oxygen saturation remains low, despite achieving
100 femoral venous oxygen saturation and central venous oxygen saturation (rs = 0.55; p < .001), the limi
101 (P(SL)CO2), lactate concentration, and mixed venous oxygen saturation (S(MV)O2) in hemodynamically un
102 dissociation curves, we plotted arterial and venous oxygen saturation (SaO2 and SvO2 ) against oxygen
103 determine the diagnostic accuracy of central venous oxygen saturation (Scv o2 ) in estimating mixed v
104 To understand the relationship among central venous oxygen saturation (Scv(O(2))), lactate, and base
105 ial CO(2) difference ( - PCO(2)) and central venous oxygen saturation ( ScvO(2)) induced by volume ex
106                                      Central venous oxygen saturation (ScvO2) in the superior vena ca
107 e (or lactate >4 mM), and continuous central venous oxygen saturation (Scvo2) monitoring for quantita
108 itiate central venous pressure (CVP)/central venous oxygen saturation (Scvo2) monitoring within 2 hrs
109                                Thus, femoral venous oxygen saturation should not be used as surrogate
110 sure, urine output, central venous (or mixed venous) oxygen saturation should be targeted.
111 an arbitrary choice of Sat(Thresh), systemic venous oxygen saturation (SsvO(2)) may be a useful param
112  simultaneous reflectance spectrophotometry (venous oxygen saturation StO2 and relative tissue hemogl
113 gen saturation (Scv o2 ) in estimating mixed venous oxygen saturation (Sv o2 ) and cardiac index in c
114 w), continuous cardiac output (Q), and mixed venous oxygen saturation (Sv(O(2))) were measured by flo
115                                   Perfusion, venous oxygen saturation SvO2, and T2* were each quantif
116 termining weaning outcome, we recorded mixed venous oxygen saturation (SvO2) continuously in eight ve
117                                        Mixed venous oxygen saturation (SVO2) was also compared with P
118 nts were transfused on bypass solely for low venous oxygen saturation (SvO2), ie, <55%, without regar
119 -1.5 mmol/L, respectively, and minimum mixed venous oxygen saturation, systemic oxygen delivery, and
120 ectrocardiogram, electroencephalogram, mixed venous oxygen saturation, temperature (core and blood),
121 0 and 2016, we calculated the ratio of mixed venous oxygen saturation-to-pulmonary capillary wedge pr
122           Cardiac index decreased, but mixed venous oxygen saturation was maintained.
123  patients at H0 and H6, whereas mean central venous oxygen saturation was preserved but significantly
124                                A low jugular venous oxygen saturation was treated in both groups, min
125 nuous monitoring of cardiac output and mixed venous oxygen saturation was used with either an 8.5-Fr
126 tissue hypoxia (as quantified by low central venous oxygen saturation) was not associated with major
127 femoral venous oxygen saturation and central venous oxygen saturation were assessed, including the di
128 femoral venous oxygen saturation and central venous oxygen saturation were similar.
129 onary artery pressure, heart rate, and mixed venous oxygen saturation, were obtained at baseline, aft
130 l venous pressure, fluid output, and central venous oxygen saturation with parameters from a pulmonar

 
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