ライフサイエンスコーパス: venous return

1 infradiaphragmatic total anomalous pulmonary venous return).

2 er 3 months confirmed unobstructed pulmonary venous return.

3 stroke volume in response to an increase in venous return.

4 l (or no) consequences if they sickle in the venous return.

5 eves of the pulmonary vein (PV) and systemic venous return.

6 mining total venous stiffness and regulating venous return.

7 he regression line to quantify resistance to venous return.

8 e and, to a lesser extent, the resistance to venous return.

9 L/min/m (p = .04), indicating a decrease in venous return.

10 rial vasodilation, endothelial function, and venous return.

11 instability by decreasing vascular tone and venous return.

12 evated venous pressures to sustain effective venous return.

13 emorrhage due to the secondary reductions in venous return.

14 al lung capacity with its negative impact on venous return.

15 ncreased blood viscosity combine to diminish venous return.

16 candidate gene for total anomalous pulmonary venous return.

17 blood contributed 40+/-16% to total systemic venous return.

18 ptal defect, and totally anomalous pulmonary venous return.

19 ting an increase in the driving pressure for venous return.

20 ceptors responding to respiratory changes in venous return.

21 severe preoperative obstruction to pulmonary venous return.

22 lformation in the lung excluded from hepatic venous return.

23 a cava (13%) and partial anomalous pulmonary venous return (13%).

24 leg raising creates a reversible increase in venous return allowing for the prediction of fluid respo

25 ects, and familial total anomalous pulmonary venous return, an autosomal dominant trait with reduced

26 hreshold device (ITD) is designed to enhance venous return and cardiac output during cardiopulmonary

27 therapeutic interventions can tend to return venous return and cardiac output to appropriate values.

28 o-part review, we describe the physiology of venous return and its interaction with the right heart f

29 nd Slit3 was found in or around the systemic venous return and pericardium during development.

30 in Fontan circulation by decreasing systemic venous return and stroke volume.

31 Resistance for venous return and systemic vascular resistance increased

32 ing pressure, then calculated resistance for venous return and systemic vascular resistance.

33 Convective heat transfer through jugular venous return and the circle of Willis was simulated.

34 chest wall recoil from leaning may decrease venous return and thereby decrease blood flow.

35 or respiration is the main driving force of venous return and which one affects the periodic flow ch

36 us arteriosus, 3 partial anomalous pulmonary venous return, and 1 aortopulmonary window) who failed c

37 filling pressure), change in resistance for venous return, and baseline heart function.

38 us pooling in the abdominal cavity, impaired venous return, and decreased central blood volumes.

39 ic resonance imaging (4-dimensional flow) of venous return, and metronome-paced tachypnea to induce d

40 ulmonary arterial blood flow, total systemic venous return, and relative blood flow to each lung.

41 mean systemic pressure and the resistance to venous return, but this has not yet been investigated in

42 nificant within-breath modulation of femoral venous return by breathing, net blood flow in the steady

43 Thus, decreased venous return by itself is not sufficient to elicit USV.

44 ins play an important role in the control of venous return, cardiac output and cardiovascular homeost

45 umatic calf compression increased lower limb venous return, causing acute but transient decreases in

46 muscle pump is not obligatory for sustaining venous return, central venous pressure,stroke volume and

47 For estimating the venous return curve, we constructed the regression line

48 ay pressure affects the cardiac function and venous return curves, which govern the static and dynami

49 The resistance to venous return decreased, i.e., 1/slope decreased.

50 As a result, venous return decreased.

51 This artificial increase of venous return did not affect the rate of USV.

52 ing and extracorporeal bypass without portal venous return did not improve MAP or cardiac output.

53 logical compensation to severe reductions of venous return, duplication of this finding utilizing sho

54 the right ventricle actively contributes to venous return during exercise, along with the muscle pum

55 is provides a sound rationale for optimizing venous return during significant allergic reactions to f

56 iration), with a slight reduction in femoral venous return during the ensuing expiratory phase of the

57 esses beyond passive stiffness and increased venous return explain the development of pulmonary venou

58 be expected to result in marked increases in venous return from the arms to the superior vena cava.

59 and cardiac filling pressures, which impede venous return from the brain, leading to increased intra

60 Chronic venous disease, a disorder involving venous return from the legs, is a growing epidemic in th

61 duction is the predominant factor modulating venous return from the locomotor limb both at rest and d

62 effects of different breathing mechanics on venous return from the locomotor limbs both at rest and

63 insufficiency is characterized by inadequate venous return from the lower extremities, which may aris

64 d by partial or complete anomalous pulmonary venous return from the right or left lung into the infer

65 art disease locus, total anomalous pulmonary venous return, had been previously localized.

66 e data examining the effects of breathing on venous return have been derived from anaesthetized or re

67 mmon arterial trunk, and anomalous pulmonary venous return have not previously been described.

68 ve intrathoracic pressure to enhance cardiac venous return, improve stroke volume, and reduce heart r

69 noses included PVS associated with anomalous venous return in 51%, PVS associated with other congenit

70 ay act as adaptive mechanisms in maintaining venous return in Fontan circulation.

71 e important influences on infradiaphragmatic venous return in Fontan patients.

72 for the modulation of venous capacitance and venous return in health and disease.

73 e calf muscle pump is a major determinate of venous return in the legs but has not been studied as a

74 ay also be an adaptive mechanism to maintain venous return in these patients while standing.

75 temic vascular resistance and resistance for venous return increased and stroke volume variation decr

76 fraction (HFpEF) or due to reduced pulmonary venous return indicated by small LV size.

77 To test whether venous return is a necessary mechanism for USV productio

78 s with Fontan circulation, it is unclear how venous return is augmented to increase stroke volume and

79 d because IVC contribution to total systemic venous return is smaller than that of the SVC.

80 However, the amount of venous return may vary in various clinical settings pote

81 We evaluated the gradient for venous-return (mean systemic filling pressure-central ve

82 deling additionally indicated that increased venous return observed in the KO mice helps maintain a h

83 Combined with devices that enhance venous return out of the brain and into the thorax, prec

84 al correction of partial anomalous pulmonary venous return (PAPVR), involving transection and reanast

85 beat provides new insights into the abnormal venous return patterns that may be responsible for adver

86 of arterial pressure; and (3) reductions of venous return, probably induced by intense LBNP, disrupt

87 real bypass, including both caval and portal venous return, produced significant increases in MAP and

88 efect, repair of partial anomalous pulmonary venous return, reconstruction of the pulmonary venous co

89 d cardiac index, mean systemic pressure, and venous return resistance (the latter two estimated throu

90 nous pressure) gradient by 28% (23-86%), and venous return resistance by 37% (17-77%).

91 adient by 144% (83-215)%, while it increased venous return resistance by 71% (60-154%).

92 eload-responsive patients if the increase in venous return resistance was lower than the increase in

93 eload responsiveness, and of the increase in venous return resistance.

94 e demonstrate how these shock states perturb venous return/right heart interactions.

95 Upright posture reduces venous return, stroke volume, and cardiac output (CO) wh

96 th standard (STD) CPR, ITPR-CPR will enhance venous return, systemic arterial pressure, and vital org

97 Total anomalous pulmonary venous return (TAPVR) is a congenital heart defect inher

98 ts of a cardiovascular response to decreased venous return, the abdominal compression reaction.

99 suggest that a Valsalva manoeuvre, blocking venous return through the superior vena cava, may allow

100 oracic pressure during inspiration increases venous return to the chest.

101 Its most common trigger is a reduction in venous return to the heart due to excessive venous pooli

102 nd heart rate declines, infant pups maintain venous return to the heart with a mechanical maneuver th

103 uction of speech, straining, facilitation of venous return to the heart, and reaction to vestibular s

104 oning the mother on her left side to improve venous return to the heart, maintaining a minimum matern

105 pressure is proportional to venodilation and venous return to the heart, we hypothesized that altered

106 sity, which is an important factor affecting venous return to the heart.

107 producibility, and examine associations with venous return to the right heart in individuals with chr

108 To test the hypothesis, venous return was decreased in infant rats while USV and

109 tely -6 cmH(2)O; T(I)/T(TOT) = 0.5), femoral venous return was markedly impeded (net retrograde flow

110 T)) = 0.5), a slight facilitation of femoral venous return was observed during inspiration (65% of al

111 The bradykinin and BK1-5 levels in forearm venous return were quantified by liquid chromatography-m

112 emoved from rats and splanchnic and systemic venous returns were then reestablished using a conduit o

113 tetralogy of Fallot, and anomalous pulmonary venous return, were also associated with smaller HC.

114 of both effects and the resulting effect on venous return when decreasing the dose of norepinephrine

115 significant detrimental effect on lower body venous return, which is more marked in APC than in TCPC