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1 a consequence of thrombosis within the dural venous sinuses.
2 trigeminal ganglion that innervate the dural venous sinuses.
3 porting cerebrospinal fluid (CSF) into dural venous sinuses.
6 lity indexes from 13 major arteries and four venous sinuses and total cerebral blood flow were collec
7 ected in the pia mater, in the arachnoid, in venous sinuses, and among the layers of the dura mater.
9 ature, rotation of the horns of the systemic venous sinus around the pulmonary portal, expansion of t
10 ne produced by the nearby auditory canal and venous sinus artifacts, an observation that may account
11 in the choroid plexus, pituitary gland, and venous sinuses as expected from the pharmacology of dihy
12 hlight the role of the skull bone marrow and venous sinuses as pivotal sites for peripheral and centr
13 s in mice and humans that aligned with dural venous sinuses but not with nasal CSF outflow, and we di
14 pread include hematogenous dissemination via venous sinuses, cerebrospinal fluid seeding in high-grad
15 implication that individuals with increased venous sinus compliance may be at increased risk of deve
16 les which create artery, vein, and cavernous venous sinus (CVS) segmentation masks from unlabeled CTA
17 0) nor predominance of unilateral transverse venous sinus drainage (R(2) = 0.07, p = 0.45) was relate
18 This paradigm is exemplified by the dural venous sinus IgA defense system, where the antibody repe
19 w dural lymphatic structures along the dural venous sinuses in dorsal regions and along cranial nerve
20 cerebri because placing a stent in stenosed venous sinuses is a novel treatment option in patients w
23 h protein expression in liver sinusoids, the venous sinuses of the red pulp in spleen, and the medull
24 g to the presence of a fracture near a dural venous sinus or jugular bulb or a high index of clinical
27 with the endothelial lining of the meningeal venous sinus permits direct exchange of small solutes be
29 hy of these vessels, running alongside dural venous sinuses, recapitulates the meningeal lymphatic sy
30 These cells are positioned adjacent to dural venous sinuses: regions of slow blood flow with fenestra
31 vessels in close anatomic proximity to dural venous sinuses, required for a functional meningeal lymp
32 Partial forms' lack significant shared dural venous sinuses (SDVS) and 'Total forms' with SDVS also e
37 include venous sinus stenting in cases with venous sinus stenosis, and bariatric surgery for weight
39 as cerebrospinal fluid diversion procedures, venous sinus stenting and bariatric surgery but there ar
44 s also been an emerging interest in cerebral venous sinus stenting, though its role and utility remai
47 series that looked at patients treated with venous sinus stents show encouraging results in decreasi
49 volume vs astronauts without SANS for all 3 venous sinus structures: superior sagittal sinus (13.40%
51 rare, and sometimes fatal, cases of cerebral venous sinus thrombosis (CVST) and thrombocytopenia foll
54 re subsets of the primary outcomes: cerebral venous sinus thrombosis (CVST), ischaemic stroke, myocar
59 arditis (0%; 95% CI, 0%-0.01%), and cerebral venous sinus thrombosis (no individuals) consistent with
60 CPyV encephalopathy associated with cerebral venous sinus thrombosis and disseminated primary JCPyV i
61 es must also be considered, such as cerebral venous sinus thrombosis and reversible cerebral vasocons
63 sex- and age-adjusted incidence of cerebral venous sinus thrombosis before the COVID-19 pandemic wit
64 consecutive patients diagnosed with cerebral venous sinus thrombosis between January 1987 and March 2
65 participating in the International Cerebral Venous Sinus Thrombosis Consortium from Finland, the Net
66 bosis, with the most frequent being cerebral venous sinus thrombosis in combination with pulmonary em
68 venience sample of 93 patients with cerebral venous sinus thrombosis included in the laboratory analy
69 subarachnoid hemorrhage and extensive dural venous sinus thrombosis involving the superior sagittal
70 a safe and effective treatment for cerebral venous sinus thrombosis not responding to anticoagulatio
72 t case of coinciding cerebral infarction and venous sinus thrombosis unveiling the diagnosis of celia
76 orway, Germany, and the UK reported cerebral venous sinus thrombosis with thrombocytopenia and anti-p
78 symptoms and were confirmed to have cerebral venous sinus thrombosis, 12 (30%) had clinical deteriora
79 ry occlusion and left transverse and sigmoid venous sinus thrombosis, along with left jugular vein th
81 er-associated deep vein thrombosis, cerebral venous sinus thrombosis, and for patients with atrial fi
82 , central retinal artery occlusion, cerebral venous sinus thrombosis, and left ventricular thrombus.
83 I], 1.4 to 5.2) among patients with cerebral venous sinus thrombosis, by a factor of 1.7 (95% CI, 1.3
84 , unruptured intracranial aneurysm, cerebral venous sinus thrombosis, cervical artery dissection, acu
85 increased alanine aminotransferase, cerebral venous sinus thrombosis, grade 3 increased amylase, and
86 myocardial infarction, Bell palsy, cerebral venous sinus thrombosis, Guillain-Barre syndrome, myocar
87 equency of stroke (arterial, n = 3; cerebral venous sinus thrombosis, n = 4), thrombocytopenia (media
88 of clinical manifestations included cerebral venous sinus thrombosis, splanchnic vein thrombosis, art