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1                                              Venous stasis activated fibrinolysis, measured by D-dime
2                We conclude that experimental venous stasis activates the fibrinolytic system to block
3  lymph node (RPLN) metastases that may cause venous stasis and increase the risk of VTE development.
4  sequential compression devices to minimizes venous stasis, and optimize intravascular volume to mini
5 erative joint disease, urinary incontinence, venous stasis, and pseudotumor cerebri.
6 ding diabetes, hypertension, hyperlipidemia, venous stasis, and sleep apnea.
7 mple of Virchow's triad: hypercoagulability, venous stasis, and vascular damage; together these facto
8 Surgically induced weight loss corrected the venous stasis disease in almost all patients as well as
9             The GBP database was queried for venous stasis disease including pretibial venous stasis
10 ificantly more frequent in the patients with venous stasis disease than for those without.
11                                       Severe venous stasis disease was associated with a significantl
12 is associated with a risk of lower extremity venous stasis disease, pretibial ulceration, cellulitis,
13 r than in patients who underwent GBP without venous stasis disease.
14 he consequential CRVO presumably advanced by venous stasis due to decrease in arterial inflow.
15 h the development of the upright posture and venous stasis in the lower extremities.
16  hypertension and general infradiaphragmatic venous stasis may compound the damage.
17 estive heart failure (n = 47 [39.2%]) and/or venous stasis (n = 16 [13.3%]).
18                     Local hypoxia, caused by venous stasis, plays a critical role in deep vein thromb
19 e increased intraabdominal pressure enhances venous stasis, reduces intraoperative portal venous bloo
20 horoidal neovascular membrane (CNVM; 10,15), venous stasis retinopathy (VSR; 10,2), choroidal infarct
21              All eyes had varying degrees of venous stasis retinopathy and intraretinal edema overlyi
22             We present an impressive case of venous stasis retinopathy in a 10-year-old boy with ocul
23                                      By now, venous stasis retinopathy in children due to Valsalva ma
24 ntral vein, wich are subsumed under the term venous stasis retinopathy, may occur as transient blurre
25 ed by Valsalva maneuver is a risk factor for venous stasis retinopathy.
26 caused by infection or intravenous devices), venous stasis (such as from chronic venous insufficiency
27 ation of P multocida via his cat licking the venous stasis ulcers on his legs.
28                                              Venous stasis ulcers resolved in all but three patients.
29          Thirty-seven patients had pretibial venous stasis ulcers, 4 had bronze edema, 23 had both, a
30 or venous stasis disease including pretibial venous stasis ulcers, bronze edema, and cellulitis.
31  complexes also occurred in wound fluid from venous stasis ulcers, but neither was detected in mastec
32 eep apnea, obesity hypoventilation syndrome, venous stasis ulcers, intestinal leak, small bowel obstr
33        Examining T-cell recruitment in human venous stasis, we show that superficial varicose veins p