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1 aling for the development and maintenance of venous valves.
2  and in the atrioventricular, semilunar, and venous valves.
3 lantation, and confirmed Prox1 expression in venous valves.
4 itical for the development or maintenance of venous valves.
5          Intriguingly, developing and mature venous valves also expressed a repertoire of proteins, i
6 ecame preferentially localized to the dorsal venous valve and atrioventricular canal regions.
7 alization of individual arterioles, venules, venous valves and millimetre-scale arteries and veins to
8 ptum, display dilated atria with hypoplastic venous valves, and exhibit blood backflow from the heart
9 ion, fusion with the inferior margins of the venous valves, and formation of the mouth of the coronar
10 ions are not well understood, but congenital venous valve aplasia or agenesis may play a role in some
11 reover, we show that in Cx37-deficient mice, venous valves are entirely absent.
12                                         Deep venous valves are frequent sites of deep venous thrombos
13                           The right and left venous valves are prominent structures in the fetal hear
14                                 However, the venous valves can persist leading to a range of anatomic
15 therefore identified molecular regulators of venous valve development and maintenance and highlighted
16 eads to severe hypoplasia of the SAN and the venous valves, dis-regulation of the SAN genetic network
17 evated capillary pressures due to failure of venous valves, elevated capillary permeability from loca
18                  Unexpectedly, we found that venous valve endothelial cells closely resemble lymphati
19 thesis that FOXC2 mutations may be linked to venous valve failure and reflux.
20 s have been strongly associated with primary venous valve failure in both the superficial and deep ve
21 describe various presentations of persistent venous valves, focusing on the right venous valve in thi
22  regulating the formation and maintenance of venous valves have not been studied.
23 sistent venous valves, focusing on the right venous valve in this illustrated multimodality imaging a
24 e (reflux), and FOXC2 is highly expressed on venous valves in mouse embryos, we tested the hypothesis
25       This review focuses on the role of the venous valves in the genesis of thrombus formation in ve
26                            A cage comprising venous valve-inspired directional micro-structure is fab
27 at we believe to be the first description of venous valve morphogenesis and identify signaling pathwa
28                                              Venous valves play a crucial role in blood circulation,
29 oclivity for venous thrombosis to develop in venous valve pockets and suggest an alternative strategy
30 hat aberrant flow patterns that may occur in venous valve pockets of individuals with common stasis-r
31 ntrolling the development and maintenance of venous valves remain poorly understood.
32                             Using global and venous valve-selective knockout mice, we further demonst
33               By preventing retrograde flow, venous valves spare capillaries and venules from being s
34 maintenance of lymphovenous valves (LVV) and venous valves (VV) is unknown.
35                                              Venous valves (VVs) prevent venous hypertension and ulce
36 d that the endothelial cells surrounding the venous valve, where DVTs originate, express high levels