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1 osed as indicators, and possibly drivers, of ventilator-induced lung injury.
2  and NO may contribute to the development of ventilator-induced lung injury.
3 mmatory mediator release, suggesting reduced ventilator-induced lung injury.
4 ncrease CO2 removal and therefore may reduce ventilator-induced lung injury.
5 n, multiple system organ failure, and severe ventilator-induced lung injury.
6 alveolar instability can independently cause ventilator-induced lung injury.
7 he effects of increasing inspiratory time on ventilator-induced lung injury.
8 r vessels may be important in the genesis of ventilator-induced lung injury.
9  severe and more homogeneous distribution of ventilator-induced lung injury.
10 cessive inflammatory response, can result in ventilator-induced lung injury.
11 an IL-1beta-associated complication known as ventilator-induced lung injury.
12 te ventilatory requirements while minimizing ventilator-induced lung injury.
13 volvement of eCypA in the pathophysiology of ventilator-induced lung injury.
14 neity of ventilation, potentially mitigating ventilator-induced lung injury.
15 ributes to the mortality of patients through ventilator-induced lung injury.
16 ary biotrauma, which couldtherefore decrease ventilator-induced lung injury.
17 he physiologic prerequisites for controlling ventilator-induced lung injury.
18 ation strategies aimed at further minimizing ventilator-induced lung injury.
19  and minimize/abolish the harmful effects of ventilator-induced lung injury.
20 me and driving pressure, key determinants of ventilator-induced lung injury.
21 y observed in patients with ARDS by creating ventilator-induced lung injury.
22 ated whether fat feeding protected mice from ventilator-induced lung injury.
23  stress and strain are major determinants of ventilator-induced lung injury.
24 rve as potential biomarkers and mediators of ventilator-induced lung injury.
25 associated with disease-specific features of ventilator-induced lung injury.
26 nsequences of mechanical ventilation such as ventilator-induced lung injury.
27 tory process accompanying early experimental ventilator-induced lung injury.
28 ate acute lung injury by causing a secondary ventilator-induced lung injury.
29 ked OxPAPC protection from interleukin-6 and ventilator-induced lung injury.
30 d alveolar pressure that could contribute to ventilator-induced lung injury.
31 nimal and endothelial cell culture models of ventilator-induced lung injury.
32 reviously verified two-hit model of in vitro ventilator-induced lung injury.
33 ch plays a major role in the pathogenesis of ventilator-induced lung injury.
34 r function, and inflammation associated with ventilator-induced lung injury.
35 in lung mechanics and in the pathogenesis of ventilator-induced lung injury.
36 sion blocked OxPAPC-mediated protection from ventilator-induced lung injury.
37 odels of bleomycin-induced lung fibrosis and ventilator-induced lung injury.
38  repair by confocal microscopy in a model of ventilator-induced lung injury.
39 LT(1) receptor antagonist, largely prevented ventilator-induced lung injury.
40 s and accumulated in lungs from animals with ventilator-induced lung injury.
41 aluronan synthase 3, and was associated with ventilator-induced lung injury.
42  upregulated and contribute to lung edema in ventilator-induced lung injury.
43  expression and activity in a mouse model of ventilator-induced lung injury.
44 plays a role in the inflammatory response of ventilator-induced lung injury.
45 plasma biomarkers as a surrogate outcome for ventilator-induced lung injury.
46 lung injury in a large tidal volume model of ventilator-induced lung injury.
47  the oleic acid injury (0.67 of baseline) or ventilator-induced lung injury (0.79 of baseline) models
48 r the magnitude of blood flow contributes to ventilator-induced lung injury, 14 sets of isolated rabb
49 ompared before and after the introduction of ventilator-induced lung injury alert.
50         The role of sigh breaths in reducing ventilator-induced lung injury among trauma patients at
51      Seven piglets submitted to experimental ventilator-induced lung injury and five healthy controls
52  has led to advances in the understanding of ventilator-induced lung injury and in optimizing the sup
53 led to advances both in our understanding of ventilator-induced lung injury and in optimizing the sup
54 highlights strategies directed at minimizing ventilator-induced lung injury and other new adjunctive
55  including the trade-offs between minimizing ventilator-induced lung injury and the risks from strate
56 observed in vivo in the mouse 2-hit model of ventilator-induced lung injury and were linked to MT sta
57                         However, concomitant ventilator-induced lung injury and/or other causes of ac
58            We report the evolution of severe ventilator-induced lung injury associated with lethal sy
59 ils into the lung is an important feature of ventilator-induced lung injury associated with pneumonia
60 EP levels may improve oxygenation and reduce ventilator-induced lung injury but may also cause circul
61          Volutrauma and atelectrauma promote ventilator-induced lung injury, but their relative contr
62                             This may lead to ventilator-induced lung injury by altering or depleting
63  Mechanical ventilator strategies that limit ventilator-induced lung injury by avoiding hyperventilat
64 P permits unstable alveoli and may result in ventilator-induced lung injury despite improved oxygenat
65 rts, helping to advance understanding of how ventilator induced lung injury develops.
66 risk for respiratory distress, asynchronies, ventilator-induced lung injury, diaphragmatic injury, an
67  ventilation heterogeneity may contribute to ventilator-induced lung injury during high-frequency osc
68                            The potential for ventilator-induced lung injury during high-frequency osc
69 gas exchange and afford lung protection from ventilator-induced lung injury during high-pressure mech
70 ion, the respiratory rate per se may promote ventilator-induced lung injury, dynamic hyperinflation,
71 re unique to the gastric injury, whereas the ventilator-induced lung injury group displayed a unique
72                                              Ventilator-induced lung injury has substantive impact on
73                        In a classic model of ventilator-induced lung injury, high peak pressure (and
74 stigated the impact of two distinct forms of ventilator-induced lung injury, i.e., volutrauma and ate
75 imize or even abolish the harmful effects of ventilator-induced lung injury if used as an alternative
76 e intensity of lung perfusion contributes to ventilator- induced lung injury in this model.
77 mption of a high-fat diet protects mice from ventilator-induced lung injury in a manner independent o
78                    Rationale: If the risk of ventilator-induced lung injury in acute respiratory dist
79 exchange, and reduced histologic evidence of ventilator-induced lung injury in an animal model.
80 y be a promising strategy for alleviation of ventilator-induced lung injury in critically ill patient
81       Pressures and volumes needed to induce ventilator-induced lung injury in healthy lungs are far
82 nd strain heterogeneity as local triggers of ventilator-induced lung injury in large-animal lungs sim
83                     Hydrogen sulfide reduces ventilator-induced lung injury in mice.
84 strated a markedly dependent distribution of ventilator-induced lung injury in oleic acid-injured sup
85 rway pressure ventilation strategy mitigates ventilator-induced lung injury in patients with severe a
86     Fat-fed mice showed clear attenuation of ventilator-induced lung injury in terms of respiratory m
87 eatly from our previous sheep model of acute ventilator-induced lung injury in which sheep were venti
88 ad assessed interventions likely to decrease ventilator-induced lung injury, including low tidal volu
89                                              Ventilator-induced lung injury increases proinflammatory
90 riggered mechanism in the protection against ventilator-induced lung injury involves cyclooxygenase 2
91                                              Ventilator-induced lung injury is a crucial determinant
92                                              Ventilator-induced lung injury is a life-threatening com
93      The common denominator in most forms of ventilator-induced lung injury is an intense inflammator
94 eir relative contribution to inflammation in ventilator-induced lung injury is not well established.
95 It has been postulated that the mechanism of ventilator-induced lung injury is the result of heteroge
96 igned to 4 hours of ventilation of the left (ventilator-induced lung injury) lung with tidal volume o
97                                              Ventilator-induced lung injury may arise from heterogene
98                                              Ventilator-induced lung injury may be caused by overdist
99           We speculate that the mechanism of ventilator-induced lung injury may involve altered alveo
100                                              Ventilator-induced lung injury may occur in acute respir
101 of hydrogen sulfide were analyzed in a mouse ventilator-induced lung injury model in vivo as well as
102  RM caused a lasting increase of PaO2 in the ventilator-induced lung injury model, but in oleic acid
103 e by 5 mins post-RM in oleic acid injury and ventilator-induced lung injury models.
104 rs among RM methods in oleic acid injury and ventilator-induced lung injury models.
105 cute lung injury: oleic acid injury (n = 4); ventilator-induced lung injury (n = 4); and pneumonia (n
106 injury using lavage and harmful ventilation (ventilator-induced lung injury; n = 7).
107 y develop lung injury that is similar to the ventilator-induced lung injury observed in mechanically
108 nd poorly aerated regions was also higher in ventilator-induced lung injury piglets compared with con
109 oro-2-deoxy-D-glucose uptakes were higher in ventilator-induced lung injury piglets compared with con
110 effects of placental dysfunction, hyperoxia, ventilator-induced lung injury, poor nutrition, abnormal
111  limit or reverse a potentially accelerating ventilator-induced lung injury process.
112 e in children have been targeted at reducing ventilator-induced lung injury, providing treatment adju
113                                              Ventilator-induced lung injury, pulmonary hypoplasia, an
114                                              Ventilator-induced lung injury remains a key contributor
115  (ALI) associated with sepsis and iatrogenic ventilator-induced lung injury resulting from mechanical
116                         Moreover, studies of ventilator-induced lung injury revealed induction of the
117                                              Ventilator-induced lung injury risk was compared before
118 e as a direct and easily measured marker for ventilator-induced lung injury risk.
119 n, is associated with known risk factors for ventilator-induced lung injury such as ventilation with
120 use circulatory depression and contribute to ventilator-induced lung injury through alveolar overdist
121  higher [F]fluorodeoxyglucose uptake rate in ventilator-induced lung injury versus control lung (0.01
122 y intervention, which can lead to unintended ventilator induced lung injuries (VILI).
123 c blast (125 kPa) but prior to initiation of ventilator-induced lung injury (VILI) (4 h).
124 ts, but the systemic effects associated with ventilator-induced lung injury (VILI) are unexplored.
125  p44/42 MAPK activation in a murine model of ventilator-induced lung injury (VILI) correlated with in
126                                To understand ventilator-induced lung injury (VILI) during positive pr
127                 Furthermore, how to minimize ventilator-induced lung injury (VILI) for any given lung
128 We examined the role of TH metabolism during ventilator-induced lung injury (VILI) in mice.
129 hether hypercapnic acidosis protects against ventilator-induced lung injury (VILI) in vivo, we subjec
130                                              Ventilator-induced lung injury (VILI) is a major cause o
131                                              Ventilator-induced lung injury (VILI) is a major complic
132                                              Ventilator-induced lung injury (VILI) is a major complic
133                                              Ventilator-induced lung injury (VILI) is characterized b
134                                              Ventilator-induced lung injury (VILI) is currently ascri
135                                 The onset of ventilator-induced lung injury (VILI) is linked to a num
136                           However, MV causes ventilator-induced lung injury (VILI), a condition chara
137 ophil- and platelet-dependent mouse model of ventilator-induced lung injury (VILI), NETs were found i
138                    However, this can lead to ventilator-induced lung injury (VILI), which contributes
139 ey factors shown experimentally to influence ventilator-induced lung injury (VILI).
140             Pretreatment with KGF attenuated ventilator-induced lung injury (VILI).
141  power is a crucial concept in understanding ventilator-induced lung injury (VILI).
142 entilation (MV) is life-saving but may evoke ventilator-induced lung injury (VILI).
143 ignal modification during the development of ventilator-induced lung injury (VILI).
144                   However, MV can also cause ventilator-induced lung injury (VILI).
145 monitoring of compliance are used to prevent ventilator-induced lung injury (VILI).
146 njure the lung, producing an entity known as ventilator-induced lung injury (VILI).
147 -inflammatory protection in rodent models of ventilator-induced lung injury (VILI).
148 eased pulmonary edema and inflammation after ventilator-induced lung injury (VILI).
149                                        This "ventilator-induced lung injury vortex" of the shrinking
150                                              Ventilator-induced lung injury was assessed by gravimetr
151                   An in vivo rabbit model of ventilator-induced lung injury was used in which a recru
152  of ventilator care and decrease the risk of ventilator-induced lung injury, we designed and tested a
153 ts alveolar barrier disruption in a model of ventilator-induced lung injury, we examined alveolar bar
154 ONALE: In the original 1974 in vivo study of ventilator-induced lung injury, Webb and Tierney reporte
155 nfirm that unstable alveoli are subjected to ventilator-induced lung injury whereas stable alveoli ar
156 hanically ventilated patients is the risk of ventilator-induced lung injury, which is partially preve
157                                Prevention of ventilator-induced lung injury while accomplishing the e
158 nfirm that unstable alveoli are subjected to ventilator-induced lung injury while stable alveoli are
159 rotective strategy, which aims at minimizing ventilator-induced lung injury (with low Vt/high positiv

 
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