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1 somedial prefrontal cortex and reward PEs in ventral striatum.
2 ly modulate the connectivity between TPJ and ventral striatum.
3 n of information, involving the left lateral ventral striatum.
4 tions of adaptive coding in the midbrain and ventral striatum.
5 ward prediction error (RPE) signaling in the ventral striatum.
6 luence on the ventromedial prefrontal cortex/ventral striatum.
7 vity with the ventromedial prefrontal cortex/ventral striatum.
8 ion of dopamine neurons sending axons to the ventral striatum.
9 ight ventromedial prefrontal cortex and left ventral striatum.
10  five tested haplotypes, particularly in the ventral striatum.
11 cores, mapping specifically to the bilateral ventral striatum.
12 ed volumes of the accumbens subregion of the ventral striatum.
13 he reward-related response properties of the ventral striatum.
14 d increases in extracellular dopamine in the ventral striatum.
15 atterns in the hippocampus and the connected ventral striatum.
16 ity in the thalamus bilaterally and the left ventral striatum.
17 heir influence on target structures, such as ventral striatum.
18 ntributing to reward-related activity of the ventral striatum.
19 havior depends on its communication with the ventral striatum.
20 ivity between the midbrain, hippocampus, and ventral striatum.
21  DAT internalization in both male and female ventral striatum.
22 d by divergent anticipatory signaling in the ventral striatum.
23 d DAT surface expression in either dorsal or ventral striatum.
24 rger reward responses in the left (vs right) ventral striatum.
25  craving measures and the dorsal, but not in ventral striatum.
26 : the ventromedial prefrontal cortex and the ventral striatum.
27 d dopamine D2/3 receptor availability in the ventral striatum.
28 st notably connectivity strength in the left ventral striatum.
29 ed in the value assignment to stimuli in the ventral striatum.
30 gulate cortex, dorsal anterior striatum, and ventral striatum.
31 ithin the brain reward system, including the ventral striatum.
32 pacity (using (18)F-DOPA, n = 21) within the ventral striatum.
33 and activity of medial prefrontal cortex and ventral striatum.
34 h attenuated HFO recorded locally and in the ventral striatum.
35 bic dopamine D2-like receptors (D2Rs) in the ventral striatum.
36 ia nigra (SN) projecting either to dorsal or ventral striatum.
37 and was phase-advanced in the OB relative to ventral striatum.
38 nhibited dopamine release in dorsal, but not ventral, striatum.
39 in the caudate (0.6), putamen (1.7 and 1.4), ventral striatum (0.7), and SN (0.5 and 0.4).
40 , putamen (1.39 +/- 1.04 vs. 4.41 +/- 0.54), ventral striatum (2.26 +/- 0.93 vs. 3.30 +/- 0.46), and
41   This was driven entirely by DeltaBP(ND) in ventral striatum (-34 +/- 14% in CHR, -20 +/- 12% in HC;
42 gen level-dependent activity was measured in ventral striatum, a dopamine target area known to repres
43                 In humans, activation of the ventral striatum, a region associated with reward proces
44 functional magnetic resonance imaging (fMRI) ventral striatum activation during reward anticipation (
45                                          Low ventral striatum activation predicted transition to subt
46 tion between greater trauma and greater left ventral striatum activation to RPE was associated with a
47 search, both preclinically and clinically-on ventral striatum activity during performance of a reward
48 hat damage to the vmPFC results in decreased ventral striatum activity during reward anticipation.
49           More generally, they indicate that ventral striatum activity may contribute to conscious pe
50                                          Low ventral striatum activity was associated with anhedonia
51          Using intracranial EEG, we recorded ventral striatum activity while 7 patients performed an
52 s have demonstrated that the vmPFC modulates ventral striatum activity.
53 differences were observed between groups for ventral striatum activity.
54 ci in the ventromedial prefrontal cortex and ventral striatum, also encodes SV during cognitive effor
55 e valuation (ventromedial prefrontal cortex, ventral striatum, amygdala) and social cognition (dorsom
56  results are consistent with findings in the ventral striatum and amygdala and show that this monosyn
57 e-exploit trade-offs, we identified that the ventral striatum and amygdala differ in how they represe
58 cision making, through interactions with the ventral striatum and amygdala.
59  and punishment prediction errors within the ventral striatum and anterior insula.
60 uli activated the striatum, specifically the ventral striatum and caudate, striatal nodes implicated
61 ate other brain regions in learning, notably ventral striatum and cerebellum [5].
62 dACC, instantaneous prediction errors in the ventral striatum and choice signals in the ventromedial
63                                              Ventral striatum and dopaminergic midbrain neurons form
64              Measures of connectivity in the ventral striatum and dorsal striatum were compared betwe
65 ked to increased dopaminergic release in the ventral striatum and excessive stimulation of dopamine D
66 d systems (dopamine, opioid peptides) in the ventral striatum and from the between-system recruitment
67 ected behaviors is a global principle of the ventral striatum and have important implications for und
68 rence reward-related learning signals in the ventral striatum and increased temporal difference-relat
69 rs, receiving relatively few inputs from the ventral striatum and instead receiving more inputs from
70 te to reward-seeking behaviours, such as the ventral striatum and midline thalamus.
71 sm, whereas buprenorphine produced increased ventral striatum and motor cortex metabolism in females,
72 vity of tracts projecting from the insula to ventral striatum and OFC.
73  we report that ex vivo dopamine tone in the ventral striatum and orbitofrontal cortex correlate with
74 tant relative deactivations were seen in the ventral striatum and orbitofrontal cortex.
75 hermore, functional connectivity between the ventral striatum and prefrontal areas exerting top-down
76 D2/3 receptor availability in the dorsal and ventral striatum and related these changes to impulsivit
77  cortex metabolism in females, and increased ventral striatum and somatosensory cortex metabolism in
78 connectivity of the subthalamic nucleus with ventral striatum and subgenual cingulate, regions simila
79 duals exhibited reduced connectivity between ventral striatum and substantia nigra.
80 he SVPE was also clearly present in both the ventral striatum and the dorsal striatum.
81 ncreased functional connectivity between the ventral striatum and the medial prefrontal and parietal
82 mus, right amygdala, right hippocampus, left ventral striatum and the right dorsal striatum.
83 key nodes of a posited "reward circuit," the ventral striatum and the ventromedial prefrontal cortex
84  connectivity between the mid-insula and the ventral striatum and ventral pallidum.
85                                          The ventral striatum and ventromedial prefrontal cortex (vmP
86 sociated with decreased connectivity between ventral striatum and ventromedial prefrontal cortex (vmP
87 increased resting cerebral blood flow in the ventral striatum and ventromedial prefrontal cortex.
88 ticularly bilateral fibre tracts between the ventral striatum and ventromedial prefrontal cortex.
89 pared with nonsmokers (caudate, putamen, and ventral striatum) and with ex-smokers (caudate and putam
90  dorsolateral prefrontal cortex, caudate and ventral striatum, and c) contribute to feature-based sti
91 ly 10%), was most prominent in the midbrain, ventral striatum, and cerebellum.
92 areas such as the anterior cingulate cortex, ventral striatum, and insula.
93 , amygdala, hippocampus, dorsal striatum and ventral striatum, and midline cerebellar vermis and subg
94 s involving the dorsal striatum, but not the ventral striatum, and no significant correlations involv
95 y within the ventromedial prefrontal cortex, ventral striatum, and other structures implicated in dec
96 ning rate and reward prediction error in the ventral striatum, and the signal of expected value in th
97 enuated adaptive coding in both midbrain and ventral striatum, and was associated with a decrease in
98 ith eigenvector centrality positively in the ventral striatum, anterior cingulate and somatosensory c
99 rolled, crossover study of DBS targeting the ventral striatum/anterior limb of the internal capsule (
100 ala (approximately 2.3%; P = .007) and right ventral striatum (approximately 3.5%; P < .005) volumes.
101                       Dopamine levels in the ventral striatum are elevated following exposure to stre
102 onal blink task, we tested the idea that the ventral striatum, because of its ability to modulate cor
103 rated prospective relationships between left ventral striatum (beta = -1.29, p = .02) and right amygd
104 he core hubs of the brain reward system (the ventral striatum), better reward learning in the Probabi
105 when experiencing social feedback increased; ventral striatum BOLD response to positive social feedba
106 ive reinforcement-related prediction errors (ventral striatum), but also aversive processing (insular
107 ession of prediction error processing in the ventral striatum by the prefrontal cortex.
108 pal output to target structures, such as the ventral striatum, by which the hippocampus may gain prio
109 AMP signaling in medium spiny neurons of the ventral striatum can effectively modulate stress-induced
110 emonstrated reductions in D2/D3R BPND in the ventral striatum compared with controls.
111 otonin receptor 2C (HTR2C) expression in the ventral striatum compared with controls.
112 d and effort expectations were integrated in ventral striatum, consistent with a computation of an ov
113  and (2) relative caudate head expansion and ventral striatum contraction in females.
114  Here, we recorded single-unit activity from ventral striatum core in rats with sham or ipsilateral n
115 yses revealed a positive correlation between ventral striatum D2R availability and subjective value-r
116 hoice in dopamine terminals in dorsal versus ventral striatum: DA terminals in ventral striatum respo
117 nly encoding-related activation in the right ventral striatum differed between schizophrenia and psyc
118      These changes are associated with lower ventral striatum dopamine activity and blunted cocaine s
119  found that associated changes include lower ventral striatum dopamine activity and lower cocaine ope
120 sed activation in reward salience circuitry (ventral striatum, dorsal caudate, anterior cingulate cor
121 sal caudate, orbitofrontal cortex, thalamus, ventral striatum, dorsal putamen, and anterior cingulate
122 sal caudate, orbitofrontal cortex, thalamus, ventral striatum, dorsal putamen, and anterior cingulate
123 uctures including the amygdala, hippocampus, ventral striatum, dorsal striatum, and thalamus subserve
124 lation of NM-MRI signal to activation of the ventral striatum during anticipation of monetary reward.
125 sistent avoidance, as did DBS applied to the ventral striatum during Ext-RP.
126 prediction errors; blunted activation of the ventral striatum during reward anticipation; blunted aut
127 ubjects showed lower neural responses in the ventral striatum during reward outcomes and higher neura
128 howed greater task-induced activation in the ventral striatum during risky decision making.
129 gene x environment interactions were seen in ventral striatum during smoking abstinence when subjects
130                                Intriguingly, ventral striatum encodes prediction error responses but
131                                    Sustained ventral striatum engagement in the laboratory positively
132 tinctness of stimulus representations in the ventral striatum even in the absence of reward.
133 f AKT1 genotype on dopamine increases in the ventral striatum (F(2,53) = 5.3, p = 0.009), with increa
134 inergic prediction-error signals rely on the ventral striatum for the former but not the latter.
135                                     Aberrant ventral striatum functional connectivity specifically pr
136                Here, we examined whether the ventral striatum, given its ability to modulate cortical
137                               In turn, lower ventral striatum gray matter volumes were associated wit
138  presence of CRF receptors in the dorsal and ventral striatum has been acknowledged, the cellular ide
139                                       DBS of ventral striatum has been previously shown to inhibit lO
140 gdala, ventral medial prefrontal cortex, and ventral striatum, has substantial connectivity with the
141 ume in a subcortical region encompassing the ventral striatum, hypothalamus and anterior thalamus.
142  hyperresponsiveness of brain regions (e.g., ventral striatum) implicated in drug effects and reward
143 ptoms showed the strongest reductions in the ventral striatum in all analyses.
144  vivo oxygen amperometry measurements in the ventral striatum in awake, behaving rats reveal reward-r
145 d distinct dopamine transients in dorsal and ventral striatum in freely moving mice performing a simp
146 nnectivity between the ventrolateral PFC and ventral striatum in healthy controls and to functional c
147 d response of the ventral tegmental area and ventral striatum in medication-free remitted recurrent d
148 in the dorsal putamen, and 17% higher in the ventral striatum in pathological gamblers compared with
149  significant hypoactivation in the vmPFC and ventral striatum in response to stress images and to alc
150 re D2/D3R activation, were also found in the ventral striatum in the CNBP sample compared with contro
151 s, which signal valence, in the midbrain and ventral striatum in the healthy controls (or patients) i
152 tivity between the right auditory cortex and ventral striatum (including the NAcc).
153 f reinforcement learning (RL) signals in the ventral striatum, including a strong and novel correlati
154 ., face-selective areas) and hippocampus and ventral striatum increased as a function of RPE value (d
155 uced activation in the orbitofrontal cortex, ventral striatum, inferior temporal gyrus, and occipital
156 or-specific conditional knockout of Cdk5, or ventral striatum infusion of a small interfering peptide
157  dopamine-excitable cells in dorsal, but not ventral, striatum inhibited sugar's ability to drive the
158 ns relevant for reward processing, including ventral striatum, insula, and thalamus.
159           The nucleus accumbens (NAc) in the ventral striatum integrates many neurochemical inputs in
160 the causal mechanisms by which the vmPFC and ventral striatum interact during the anticipation of rew
161                           This suggests that ventral striatum iron accumulation is linked to demyelin
162                                          The ventral striatum is a neural system critical for evaluat
163 o human evidence that DA transmission in the ventral striatum is affected by morphine.
164                                          The ventral striatum is believed to encode the subjective va
165     SIGNIFICANCE STATEMENT: More than 90% of ventral striatum is composed of two cell types, those ex
166                                          The ventral striatum is critical for evaluating reward infor
167 st, neural activity in a brain region in the ventral striatum is rapidly modified in a matter of minu
168 amine-HFO which can contribute to HFO in the ventral striatum, known to project diffusely to many oth
169 ctivity between bilateral insula regions and ventral striatum, left insula and middle orbitofrontal c
170      These findings suggest that lower right ventral striatum-left caudal anterior cingulate FC to lo
171 ealed that OBP had significantly lower right ventral striatum-left caudal anterior cingulate FC to lo
172               These results suggest that the ventral striatum may be part of a subcortical network th
173 metry to measure dopamine binding across the ventral striatum (medial accumbens shell, accumbens core
174 ), and aberrant, increased activation of the ventral striatum, midbrain, and other limbic regions for
175 ning from rewards; blunted activation of the ventral striatum, midbrain, and other limbic regions for
176 re, we identify a basolateral amygdala (BLA)-ventral striatum (NAc) pathway that is activated by exti
177                               Increased left ventral striatum node strength predicted increased risk
178                                          The ventral striatum (nucleus accumbens) and its role in moo
179 y knocking down Gpr88 gene expression in the ventral striatum (nucleus accumbens) in a neurodevelopme
180 VTA-innervated limbic regions, including the ventral striatum (nucleus accumbens).
181    However, no study examined Slc6a15 in the ventral striatum [nucleus accumbens (NAc)] in depression
182 lly increased functional connectivity of the ventral striatum/nucleus accumbens and ventromedial pref
183  Kmt2a, but not the ortholog Kmt2b, in adult ventral striatum/nucleus accumbens neurons markedly incr
184                                       In the ventral striatum of CHL1-deficient mice, levels of phosp
185 pathway" medium spiny neurons (iMSNs) in the ventral striatum of D2R knockout mice, this mutant resto
186 ex (IL), insula cortex, dorsal striatum, and ventral striatum of female and male mice with green fluo
187 ntly decreased in the left but not the right ventral striatum of high-impulse (HI) rats compared with
188   We use this mode to activate dorsal versus ventral striatum of individual mice and reveal different
189  electrophysiological activity of the OB and ventral striatum of male Wistar rats was examined using
190 onitor dopaminergic neurotransmission in the ventral striatum of NF1 mice during motivated behavior.
191 recorded neural activity in the amygdala and ventral striatum of rhesus macaques as they solved a tas
192 behavior via the amygdala projections to the ventral striatum or the ventral tegmental area.
193                                              Ventral striatum- or D1 dopamine receptor-specific condi
194 ween the medial prefrontal cortex (MPFC) and ventral striatum over time were associated with decrease
195 eral release differed between the dorsal and ventral striatum owing to differential regulation by D2-
196 amen (F(2,90) = 6.6, p = 0.002), but not the ventral striatum (p = 0.3).
197 ther morphological changes in the dorsal and ventral striatum/pallidum relate to or predict therapeut
198 cits structural plasticity in the dorsal and ventral striatum/pallidum.
199 refrontal cortex, orbital prefrontal cortex, ventral striatum, parietal lobe, dorsal putamen, dorsal
200                Dopamine signaling within the ventral striatum plays an important role in reward learn
201 ivity between the following areas: thalamus, ventral striatum, posterior cingulate cortex, and tempor
202               It has been suggested that the ventral striatum provides these predictions.
203  = 0.02), putamen (r = -0.62, p = 0.01), and ventral striatum (r = -0.66, p < 0.01).
204 ession was related to increased amygdala and ventral striatum reactivity under alcohol, providing evi
205 al orbitofrontal cortex (lOFC) or DBS of the ventral striatum reduced persistent avoidance.
206                                 In vmPFC and ventral striatum, representations of prediction error al
207 sal versus ventral striatum: DA terminals in ventral striatum responded more strongly to reward consu
208    Both types of social information modulate ventral striatum response.
209 is that the vmPFC is necessary for enhancing ventral striatum responses to the anticipation of reward
210 responsivity to angry facial expressions and ventral striatum responsivity to monetary rewards.
211 with inflammation, and the same was true for ventral striatum responsivity to reward.
212 ocial interaction, whereas deletion from the ventral striatum resulted in repetitive grooming.
213 ntributed to an excessive stimulation of the ventral striatum resulting in impulsive compulsive behav
214                       Dopamine levels in the ventral striatum rise before initiating a reliably reinf
215 s in the olfactory tubercle subregion of the ventral striatum robustly encode the onset and progressi
216 erent, and efferent similarities between the ventral striatum's nucleus accumbens and olfactory tuber
217  outcome interaction for both right and left ventral striatum seeds.
218 tivity in ventromedial prefrontal cortex and ventral striatum showed a marked reduction in (1) neural
219                         Both hippocampus and ventral striatum showed increased synchronization betwee
220 Among 11 reward-network nodes, only the left ventral striatum significantly predicted depression.
221 egions of interest for the caudate, putamen, ventral striatum, SN, and cerebellum were drawn on coreg
222 d correlation among the thalamus, dorsal and ventral striatum, somatomotor, temporal and prefrontal c
223 trol subjects showed increased activation of ventral striatum specifically for cues predicting erotic
224               The olfactory tubercle (OT), a ventral striatum structure that receives monosynaptic in
225 prediction errors correlate with activity in ventral striatum/subgenual anterior cingulate cortex, wh
226 egions of interest for the caudate, putamen, ventral striatum, substantia nigra (SN), and cerebellum
227  dopamine-excitable cells in dorsal, but not ventral, striatum substituted for sugar in its ability t
228 nd are in accordance with the principle that ventral striatum substructures may cooperate to guide mo
229  in the dorsal anterior cingulate cortex and ventral striatum, such that the normal (vs. slow) genoty
230  decrease in opioid-evoked DA release in the ventral striatum, suggesting that the occurrence of chro
231 ome encoding from the anterior insula to the ventral striatum, suggesting that value contextualizatio
232 ntrolled trial of DBS at the ventral capsule/ventral striatum target for TRD.
233 ncludes nodes in the amygdala, hypothalamus, ventral striatum, thalamus, and specific brainstem nucle
234 ility on all striatal sub-regions except for ventral striatum than did age- and sex-matched normal co
235 the dorsal anterior cingulate cortex and the ventral striatum that negatively correlated with increas
236 d functionally segregated regions within the ventral striatum that separately encoded effort activati
237  the dopaminergic midbrain, hippocampus, and ventral striatum (the SN/VTA-Hippocampal loop) when succ
238                       In both the dorsal and ventral striatum, the ratio increased from adolescence.
239 e anterior limb of the internal capsule, the ventral striatum, the subthalamic nucleus, and a midbrai
240 rt a role for OFC in shaping activity in the ventral striatum to represent the biological significanc
241 tion of functional connectivity of bilateral ventral striatum to right anterior ventromedial subthala
242 nd decreased effective connectivity from the ventral striatum to the thalamus independently of seroto
243 sured choline at rest in both the dorsal and ventral striatum using magnetic resonance spectroscopy.
244  in the anterior limb of the ventral capsule/ventral striatum (VC/VS).
245  in the efficacy of ventral internal capsule/ventral striatum (VCVS) DBS in both major depression (MD
246 ile the origin of the association with right ventral striatum volumes was unclear.
247 rticular as manifest by relatively increased ventral striatum (VS) activity and relatively decreased
248  determine the relative contributions of the ventral striatum (VS) and amygdala to appetitive RL, we
249 eptors (KORs) are highly enriched within the ventral striatum (VS) and are thought to modulate striat
250 elatively low reward-related activity of the ventral striatum (VS) and high threat-related reactivity
251 ain stimulation (DBS) of the ventral capsule/ventral striatum (VS) as a possible treatment for drug a
252 alcohol cue-elicited activation of the right ventral striatum (VS) between baseline and week 2 and re
253 ted by increasing evidence of reward-related ventral striatum (VS) dysfunction in depression, we inve
254 ociated with prediction error signals in the ventral striatum (VS) in both contexts.
255                                          The ventral striatum (VS) is a central node within a distrib
256                                          The ventral striatum (VS) is a key brain center regulating r
257 ated the performance of rhesus macaques with ventral striatum (VS) lesions on a two-arm bandit task t
258 ATEMENT Reinforcement learning models of the ventral striatum (VS) often assume that it maintains an
259 t OFC encoded values in a similar way to the ventral striatum (VS) or the anterior insula (AI) during
260 hed findings, we found an association of the ventral striatum (VS) with reward processing.
261 suggests that reward-related activity of the ventral striatum (VS), a brain region critical for motiv
262 er an index of reward system function in the ventral striatum (VS), a key reward circuitry region, mo
263  that signed RPEs (SRPEs) are encoded in the ventral striatum (VS), and crucially, that SRPE VS activ
264 sion-making, the orbitofrontal cortex (OFC), ventral striatum (VS), and dorsal striatum (DS), while m
265  in key reward processing areas, such as the ventral striatum (VS), as measured with functional magne
266                            We focused on the ventral striatum (VS), due to its association with incen
267 n function, particularly the activity of the ventral striatum (VS), has been identified as a potentia
268                                          The ventral striatum (VS), like its cortical afferents, is c
269  studies have not looked specifically at the ventral striatum (VS), which plays an important role in
270 ms and activation of presynaptic KORs in the ventral striatum (VS).
271 ic (PL) PFC, basolateral amygdala (BLA), and ventral striatum (VS).
272 ly, we observe that SRPEs are encoded in the ventral striatum (VS).
273 ite dynamics in dopamine axon signals in the ventral striatum ('VS dopamine') and the posterior tail
274  known reward-processing neurocircuitry (eg, ventral striatum, VS) has been reported among medicated
275 tral tegmental area (SN/VTA) (+20%; p=0.02), ventral striatum (VST) (+14%; p<0.01), and pallidum (+11
276 ls (LFPs) recorded from the human and rodent ventral striatum (vStr) exhibit prominent, behaviorally
277 ated limbic areas.SIGNIFICANCE STATEMENT The ventral striatum (vStr) is an area of anatomical converg
278                     In contrast, activity in ventral striatum was associated with subjects' global SP
279                                     The left ventral striatum was more active when the chosen option
280 alyses revealed that D2R availability in the ventral striatum was positively correlated with subjecti
281                              Activity in the ventral striatum was reduced in participants with subthr
282 ity between prefrontal brain regions and the ventral striatum was significantly diminished in the ris
283 duced upregulation of silent synapses in the ventral striatum; we show it can occur in the dorsal str
284 urthermore, higher levels of iron within the ventral striatum were accompanied by a negative correlat
285 ced increases in connectivity with bilateral ventral striatum were observed across a network of regio
286 ment and reduced rCBF in the hippocampus and ventral striatum were observed.
287         Responses of the NAc and surrounding ventral striatum were quantified during a monetary incen
288 The changes of rs-fc among the PAG, rACC and ventral striatum were significantly associated with head
289 ia Nigra/Ventral Tegmental Area (SN/VTA) and ventral striatum were steeper for prediction errors occu
290 s is accompanied by enhanced activity in the ventral striatum when curiosity or hunger was elicited,
291 addiction showed increased activation in the ventral striatum, whereas individuals with gambling addi
292 r, task engagement relies on function of the ventral striatum, whereas the basolateral amygdala media
293 rcement-learning strategies supported by the ventral striatum, whereas younger adults use attention p
294 were also found in the prefrontal cortex and ventral striatum which, although of smaller amplitude, w
295 ain stimulation (DBS) of the ventral capsule/ventral striatum, which facilitates patients' response t
296      We recorded in these regions and in the ventral striatum, which has not been associated previous
297 gative correlation of iron and myelin in the ventral striatum, which predicted individual memory perf
298 ve relationship of midbrain RSFC to the left ventral striatum with cognitive impulsivity, whereas a n
299  subjects there was greater perfusion in the ventral striatum with fructose relative to glucose inges
300 rate depression showed intact RPE signals in ventral striatum (z = 3.16; P = .002) that did not diffe

 
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