ライフサイエンスコーパス: ventricle

1 terature received the moniker "The Forgotten Ventricle".

2 The Impella unloads the left ventricle.

3 tal heart tissue with an underdeveloped left ventricle.

4 KE was quantified in the left and right ventricle.

5 he anterior SHF results in hypoplastic right ventricle.

6 the left ventricle (LV) becomes the systemic ventricle.

7 saligned, resulting in a double outlet right ventricle.

8 lumes of elevated velocity (EVV) in the left ventricle.

9 ocytin injections at the floor of the fourth ventricle.

10 revealed thrombus within the left atrium and ventricle.

11 y arteries to guide their expansion down the ventricle.

12 ac lymphatic vessels fail to expand onto the ventricle.

13 s adjacent to the anterior wall of the third ventricle.

14 e processes from a distant part of the third ventricle.

15 -reservoirs to the frontal horn of a lateral ventricle.

16 (ER) stress, and apoptosis in diabetic left ventricle.

17 ing a standard, 17-segment model of the left ventricle.

18 pendyma of the adjacent surface of the third ventricle.

19 ems from the anterolateral part of the third ventricle.

20 oding the 3-dimensional geometry of the left ventricle.

21 ed cardiomyocyte nuclear density in the left ventricle.

22 LV fibroblasts nor cardiomyocytes of either ventricle.

23 art Association 17-segment model of the left ventricle.

24 dicating the systolic force generated by the ventricle.

25 he bottom and the lateral walls of the third ventricle.

26 ss well understood in patients with a single ventricle.

27 chromatin interaction data from LA and left ventricle.

28 C-PBR28 SUVR calculated using the WB without ventricles.

29 e atrioventricular node and the fascicles or ventricles.

30 ted markers, in the developing mouse cardiac ventricles.

31 mpairment, thin corpus callosum and enlarged ventricles.

32 1 positive microglia surrounding the lateral ventricles.

33 cast that showed the shape and extent of the ventricles.

34 rves do not converge on the lateral or third ventricles.

35 affects synchronized contraction of the two ventricles.

36 ed studies of the skull, brain, and cerebral ventricles.

37 the surface of the inflow tract to reach the ventricles.

38 f the cerebrospinal fluid (CSF)-filled brain ventricles.

39 cilia beat in multiciliated cells from brain ventricles.

40 ers and ~5000 enhancers active in human left ventricles.

41 y promoter and enhancer regions used in left ventricles.

42 cipital cortex, and whole brain (WB) without ventricles.

43 white matter (26 mL; P < .001), mean lateral ventricles (2.2 mL; P < .001), and mean summated brain a

44 - 1.06 vs 15.88 +/- 0.81; p < 0.05) and left ventricles (20.14 +/- 1.40 vs 14.17 +/- 1.53; p < 0.05).

45 queductal, 32.7% periependymal along lateral ventricles, 3.4% large hemispheric, 6.0% diencephalic, 4

46 Right (73%), left (19%), or both ventricles (8%) underwent sampling.

47 In the rodent ventricle, a significant portion of genes, including som

48 A specialized neurogenic niche along the ventricles accumulates millions of progenitor cells in t

49 d the implantation of eMSCs into the lateral ventricle activated relevant pathways associated with th

50 Rather, the right ventricle actively contributes to venous return during e

51 s physiological mechanism protects the right ventricle against acute changes in preload, and its impa

52 The healthy right ventricle also demonstrates marked augmentation in lusit

53 Infants with single ventricle anatomy and ductal-dependent pulmonary blood f

54 In patients with single-ventricle anatomy and ductal-dependent pulmonary blood f

55 Patients with single ventricle anatomy and ductal-dependent pulmonary blood f

56 ultimately contraindicated because of right ventricle anatomy.

57 y pressure catheters were placed in the left ventricle and aortic root.

58 derlie hypokalemia-induced arrhythmia in the ventricle and atrium but also vary between atrial myocyt

59 xpressed in the cardiac outflow tract, right ventricle and atrium, pharyngeal mesoderm, peripheral ne

60 tion cohort, cardiac MRI mPAP model 1 (right ventricle and black blood) was defined as follows: -179

61 ted with higher cell death rates in the left ventricle and deteriorated cardiac function.

62 function has lagged behind that of the left ventricle and historically, the RV has even been referre

63 been viewed as less important than the left ventricle and in contemporary literature received the mo

64 e between occipital horn of the left lateral ventricle and internal surface of the cranium and third

65 onnection between distant parts of the third ventricle and OVLT.

66 natomical analysis suggested greater lateral ventricle and putamen volume in duplication carriers.

67 contact the cerebrospinal fluid in the third ventricle and send processes into the hypothalamic paren

68 Anatomic human ventricle and simplified tissue models were used to inve

69 ght atrium (synchrony), or for 2 weeks right ventricle and then 2 weeks normal sinus (resynchronizati

70 SHR capillary density is increased in both ventricles and at all ages, including before the onset o

71 f neurons located in close vicinity of brain ventricles and central canal.

72 inal fluid (CSF) is produced in the cerebral ventricles and circulates within the subarachnoid space

73 progressive enlargement of lateral and third ventricles and deceleration of ciliary beating on ependy

74 ent animals, and new PVS connections between ventricles and different parts of the brain parenchyma w

75 ulates a transverse section of midbrain with ventricles and gray and white matter compartments.

76 ry light chain (RLC) simultaneously in heart ventricles and in slow-twitch skeletal muscle.

77 tionship between K(+) channel mRNA levels in ventricles and peripheral blood mononuclear cells and th

78 and brain tissue (P = 0.004) to the lateral ventricles and significantly lower (11)C-PiB signal clea

79 the subventricular zone (SVZ) of the lateral ventricles and striatum of mice with genetic deletion (D

80 he brain, constantly circulating through the ventricles and subarachnoid space.

81 tical thickness and 34 surface area, lateral ventricles and total intracranial volume measures separa

82 ere evaluated, as well as the systemic, left ventricle, and adipose tissue inflammatory profile.

83 hypoplasia of the corpus callosum, enlarged ventricles, and decreased thickness of the somatosensory

84 cortical layers, enlargement of the cerebral ventricles, and microcephaly.

85 tility or an improved relaxation of the left ventricle as assessed by wave intensity analysis.

86 fied the choroid plexus of the mouse lateral ventricle as the major source of miR-204 that is release

87 revealed suggesting a possible role for the ventricles as a source or sink for solutes in the brain.

88 ppeared smaller than controls and had larger ventricles as well as thinner germinal wall.

89 the pulmonary vascular system and the right ventricle, as well as their coupling, as important conce

90 e characterized by abnormalities in the left ventricle, associated valves, and ascending aorta.

91 glucose transporter 4 expression in the left ventricle at 8 d after TAC, indicating altered glucose m

92 brain and hippocampal volume, and sulcal and ventricle atrophy using nested multivariate regression a

93 und for SUVR calculated using the WB without ventricles (average cluster, 21.6% +/- 0.1%).

94 By 24 hpf, embryos expressed ttpa in brain ventricle borders, which showed abnormal closure in E- e

95 ed fractions from syndecan-4(-/-) mouse left ventricles but increased nuclear MLP when syndecan-4 was

96 induce locomotion when injected in the brain ventricles but to inhibit fictive locomotion when bath-a

97 ft ventricular cavity, myocardium, and right ventricle by processing an incoming time series of perfu

98 educed to low levels in diastole so that the ventricle can relax and refill with blood.

99 al cardiac lesions when a circulation with 2 ventricles cannot be achieved.

100 xosome treatment significantly improved left ventricle cardiac function, inhibited cell apoptosis, re

101 function in myocardial repair, improved left ventricle cardiac function, reduced MI scar size, and en

102 us, and amygdala volumes and greater lateral ventricle, caudate, and accumbens volumes (Cohen's d val

103 interrogate these functions in adult lateral ventricle ChP in whole-mount explants and in awake mice.

104 nd impaired functional reserve of the single-ventricle circulation.

105 red pressure within the brain parenchyma and ventricle compartments.

106 on (P = 0.08) and surrogate measures of left ventricle compliance did not reach significance.

107 circulation in neonatal patients with single ventricle congenital heart defects, but this complex pro

108 innate abnormalities associated with single-ventricle congenital heart disease exposes these patient

109 on; isolated lung, upper torso, direct right ventricle contrast injection, and whole body with partia

110 ablation and 5 with structurally normal left ventricles (controls) undergoing premature ventricular c

111 ograft (right and left atria, right and left ventricles, coronary arteries) compared to the native re

112 conduction delay in RVOT, but not RV or left ventricle, correlated to the degree of J-ST point elevat

113 f encapsulated MSCs (eMSCs) into the lateral ventricle counteracted depressive-like behavior and enha

114 shown that CSF flow within and across brain ventricles depends on cilia motility of the ependymal ce

115 e (SV) at 12 weeks of age and decreased left ventricle diastolic volume with subsequent reduced SV co

116 Vessel density in the left ventricle did not change during and after compaction.

117 al valve disease and may develop as the left ventricle dilates or remodels or as a result of leaflet

118 ibit TGF-beta signaling associated with left ventricle dilation and systolic dysfunction.

119 e deterioration of cardiac function and left ventricle dilation.

120 sence of periependymal lesions along lateral ventricles discriminated neuromyelitis optica patients i

121 SCPC was performed for palliation of single ventricle disease who underwent chest MRI between July 2

122 ior horn width of the right and left lateral ventricle, distance between occipital horn of the left l

123 As the left-ventricle does not have a uniform shape and functioning

124 , and maximum rate of pressure change in the ventricle (dP/dt(max) ), as well as energy utilization a

125 Pressure-volume analysis of the right ventricle, during invasive cardiopulmonary exercise test

126 ment may be a relevant contribution to right ventricle dysfunction in pulmonary hypertension.

127 ion, followed by 4 weeks pacing at the right ventricle (dyssynchrony), right atrium (synchrony), or f

128 Left ventricle ejection fraction <55% was strongly associated

129 there was a significant improvement in left ventricle ejection fraction from 30+/-11% to 42+/-15%.

130 y disease, a reduction in the estimated left ventricle epicardial volume correlated with a loss of pu

131 in the critical tasks of segmenting the left ventricle, estimating ejection fraction and assessing ca

132 t the implantation of eMSCs into the lateral ventricle exerted antidepressant effects likely acting v

133 ients had significant enlargement of lateral ventricles (F(1,59) = 48.89; p < 0.001) and reduction of

134 infused in vivo into the CSF-filled lateral ventricle followed by ex vivo high-resolution MR imaging

135 Morbidity in patients with single-ventricle Fontan circulation is common and includes arrh

136 In the absence of a subpulmonary ventricle, Fontan circulation is characterized by chroni

137 e soma and processes of NPCs dividing at the ventricle for over 50 mum along the radial axis.

138 active, or anatomical properties of the left ventricle for reproducing measured patient phenotypes.

139 ction of right ventricular diameter as right ventricle free wall thickness was increased and an incre

140 ctile regulatory gene cmlc2 was also seen in ventricles from 2.75% HF-FW-exposed mahi.

141 ing cells to ependymal regions bordering the ventricles from the forebrain to the hindbrain, and obse

142 istomicrograph of transverse sections of the ventricles from the Mb group did not show abnormalities.

143 cardiac fibrosis response and preserves left ventricle function as compared to control-exosome admini

144 t disease are more likely to experience left ventricle function recovery with successful AF ablation.

145 07), of which 51% (28/55) had preserved left ventricle function.

146 centric myocardial hypertrophy, altered left ventricle geometry, perturbed systolic and diastolic fun

147 atterns of a healthy and ischemic human left ventricle geometry.

148 turbances, thin corpus callosum, and widened ventricles); global delay with significant alteration of

149 siological properties in failing human heart ventricles have not been examined for a circadian expres

150 fects [ASD], aortic arch defects, and single-ventricle heart) and subgroups of specific heart defects

151 counts, pulmonary vessel density, and right ventricle hypertrophy (RVH).Measurements and Main Result

152 e stress allowed myocardial energetics, left ventricle hypertrophy, and diastolic dysfunction to reco

153 the potentially lethal expansion of cerebral ventricles if not treated.

154 reduced inflammatory parameters in the left ventricle (IFN-gamma, IL-6, and IL-1beta), as well as in

155 of these cytotoxic amyloids into the lateral ventricle impairs learning and memory in mice.

156 wed a hypodense area in the apex of the left ventricle in a 57-year-old man with a history of anterio

157 n (EF), were measured for the left and right ventricle in both end-expiration and end-inspiration.

158 r native T1 and ECV measurements of the left ventricle in health adult study participants.

159 tial and longitudinal strain within the left ventricle in healthy Chinese subjects.

160 microglia, that are located near the lateral ventricle in the prenatal neocortex.

161 crest contribution to cardiomyocytes of the ventricles in Gallus gallus, supported by Wnt1-Cre linea

162 Infusion of neurotensin into the fourth ventricle induced NREM sleep-like cortical activity, whe

163 Using a zebrafish hindbrain ventricle infection model, we demonstrate that the T7SS

164 travenous injection), and brain (via lateral ventricle infusion).

165 rdiovascular magnetic resonance at the right ventricle insertion site.

166 r cell, which strongly correlated to that in ventricles, inversely associated to CRP and IL (interleu

167 farction border zone and in the hypertrophic ventricle, involving regulatory sequences proximal to Np

168 at irregular deposition of Sspo within brain ventricles is associated with idiopathic-like scoliosis

169 Cerebrospinal fluid (CSF) flow in the brain ventricles is critical for brain development.

170 brillation(AF) while amyloid deposits in the ventricles is increasingly being diagnosed in patients w

171 phalus (CH), characterized by enlarged brain ventricles, is considered a disease of excessive cerebro

172 ved in fibrosis and adhesion, whereas in the ventricles, it controlled inflammation and endocytosis.

173 on the body surface, coronary sinus and left ventricle leads, requires a delivered charge of 0.04+/-0

174 em cell-derived SAN-like pacemaker cells and ventricle-like cells and identified thousands of putativ

175 ned for the investigated organs (brain, left ventricle, liver, and muscle) due to different animal ha

176 men including, at least partly, cardiac left ventricle, liver, spleen, and kidney (n = 2) or three 10

177 mal, parotid, and submandibular glands; left ventricle; liver; spleen; kidneys; bowel; urinary bladde

178 ocardium patterning and, consequently, heart ventricle lumen formation.

179 Samples of left and right ventricle (LV and RV) free wall collected from 32 Wistar

180 monary circulation after birth when the left ventricle (LV) becomes the systemic ventricle.

181 usion alone, mechanically unloading the left ventricle (LV) before reperfusion reduces infarct size a

182 When remodeling and enlargement of the left ventricle (LV) cause annular dilatation and tethering of

183 cient myocardial perfusion and improved left ventricle (LV) diastolic function in HFpEF.

184 palmitate can, at least in part, offset left ventricle (LV) dysfunction in hearts from diabetic mice,

185 c MRI data included segmentation of the left ventricle (LV) in cardiac MRI perfusion and cardiac MRI

186 Remodeling of the left ventricle (LV) in response to pressure overload leads to

187 An understanding of left ventricle (LV) mechanics is fundamental for designing be

188 rtery diameter and right ventricle (RV)/left ventricle (LV) ratio.

189 Sex differences in the left ventricle (LV) size could provide an explanation for the

190 (18)F-flurpiridaz PET MPI according to left ventricle (LV) size.

191 7) was significantly higher in the male left ventricle (LV) than in the female LV.

192 entricle (RV) does not respond like the left ventricle (LV) to guideline-directed medical therapy of

193 hypertrophy were present throughout the left ventricle (LV).

194 he transcriptional profile of the human left ventricle (LV, n=4) and right ventricle (RV, n=4) after

195 e pulmonary disease (COPD) have smaller left ventricles (LVs) due to reduced preload.

196 ation did not increase cAMP in USP20-KO left ventricles (LVs), whereas NKH477-induced adenylyl cyclas

197 s bind differently to syndecan-4 in the left ventricle lysate from aortic-banded heart failure (ABHF)

198 on of the immune cell population in the left ventricle manifested by lowered abundance of proinflamma

199 measure of inflammation and with higher left ventricle mass and volumes.

200 Lower MFR was associated with higher left ventricle mass index and higher left ventricle volumes b

201 (mean % change +/- SE = 13.3 +/- 1.9), third ventricle (mean % change +/- SE = 10.4 +/- 1.1), and the

202 torial ventricular structures, i.e., lateral ventricle (mean % change +/- SE = 13.3 +/- 1.9), third v

203 +/- SE = 7.7 +/- 1.6; P = 0.0009), the third ventricle (mean % change +/- SE = 4.7 +/- 1.3; P = 0.006

204 residual increases in volume for the lateral ventricle (mean % change +/- SE = 7.7 +/- 1.6; P = 0.000

205 .001) and ratio of scar volume to total left ventricle myocardial volume (%LGE) (r = 0.91, P < .001).

206 ly (n=44), septal defects (n=39), and single ventricle (n=36).

207 cribing myocyte orientations) of healthy rat ventricles-obtained using diffusion tensor imaging at 10

208 The anterior chamber and lateral ventricle of anaesthetized Brown-Norway rats were cannul

209 serelaxin's antifibrotic actions in the left ventricle of mice with cardiomyopathy, indicating that c

210 ression was significantly suppressed in left ventricle of mice with transverse aortic constriction-in

211 CaM-M37Q, into the anterior wall of the left ventricle of RyR2 wild type or mutant mouse hearts.

212 Fibroblasts were isolated from the left ventricle of the explanted hearts of transplant recipien

213 xenon dissolved in the blood inside the left ventricle of the heart, it is possible to directly measu

214 The right ventricle of the mammal heart is highly sensitive to the

215 RT2104) is injected into the mPFC or lateral ventricle of wild-type mice, it reverses chronic unpredi

216 implanted epicardially on the right and left ventricles of a porcine model and were inductively power

217 itude of (11)C-PiB PET signal in the lateral ventricles of an independent group of Alzheimer and mild

218 of samples was acquired from the nonfailing ventricles of brain-dead donors.

219 s show a circadian expression pattern in the ventricles of failing human hearts, which may underlie a

220 s the distribution of CM size throughout the ventricles of intact mouse heart, in which cells in the

221 entricular zones along the lateral forebrain ventricles of mice.

222 e alpha(1C) I-II loop, which is increased in ventricles of patients with end-stage heart failure, inc

223 cells divide in the ventricular zone at the ventricles of the embryonic brain, self-renew and genera

224 form pool of progenitor cells that lines the ventricles of the forebrain.

225 l brain, neurons begin to aggregate near the ventricles of treated animals.

226 Z NPCs when directly injected in the lateral ventricles of uninjured mice.

227 adaptive atlas algorithm to segment the left ventricle on CAC-CT, extracting 107 radiomics features f

228 ral blood oxygenation in fetuses with single ventricle or aortic obstruction.

229 ricular arrhythmias originate from the right ventricle outflow tract (RVOT).

230 y and late remodelling of the left and right ventricle over the course of monocrotaline-induced PAH t

231 11)C-PiB signal clearance out of the lateral ventricles (P = 0.002) in Alzheimer subjects than in hea

232 ation in stellate ganglia (p = 0.02) but not ventricles (p = 0.2) of PVC-CM and recovered animals ver

233 ay severe OFT and RV hypoplasia and a single ventricle phenotype due to decreased proliferation of Se

234 performed for surgical palliation of single ventricle physiology.

235 and quality of life for patients with single ventricle physiology.

236 ior horn width of the right and left lateral ventricle, posterior horn width of the right and left la

237 tance 1.5[2.2, 0.9] WU, and transmural right ventricle pressure 10[15, 6] mmHg during exhalation.

238 P(PL) demonstrated unchanged transmural left ventricle pressure and systemic blood pressure after LRM

239 ds and Results We used a mouse model of left ventricle pressure overload coupled to in vitro studies

240 Cardiac ventricles printed with human cardiomyocytes showed sync

241 econd model, cardiac MRI mPAP model 2 (right ventricle pulmonary artery), which excludes the black bl

242 tay using the Pediatric Heart Network Single Ventricle Reconstruction trial dataset.

243 alysis of the Pediatric Heart Network Single Ventricle Reconstruction trial.

244 Lateral ventricle regions of interest were generated manually fr

245 ofiles are associated with aorta dilatation, ventricle remodeling, aneurysms, and development of athe

246 f the cerebrospinal fluid (CSF)-filled brain ventricles resulting from failed CSF homeostasis.

247 ol, 14.6 ng) ouabain into each lateral brain ventricle results in increased locomotor activity, stere

248 enitor pool leads to an underdeveloped right ventricle (RV) and outflow tract (OFT).

249 e and function in children with single right ventricle (RV) anomalies may be influenced by shunt type

250 ial for normal outflow tract (OFT) and right ventricle (RV) development.

251 The failing right ventricle (RV) does not respond like the left ventricle

252 rioration, specifically a reduction in right ventricle (RV) ejection fraction and stroke volume (SV)

253 orated into a patient-specific silicon right ventricle (RV) emulating severe FTR, on which Kay repair

254 Until recently, the right ventricle (RV) has often been viewed as less important t

255 recognition of the crucial role of the right ventricle (RV) in determining functional status and prog

256 The right ventricle (RV) is involved in systemic circulation in th

257 as those with tetralogy of Fallot, the right ventricle (RV) is subject to pressure overload stress, l

258 ionale: Remodeling and fibrosis of the right ventricle (RV) may cause RV dysfunction and poor surviva

259 o confirm HB capture/exclude that only right ventricle (RV) myocardial septal pacing is present.

260 unction and structure were assessed in right ventricle (RV) myocardium collected from patients with R

261 The right ventricle (RV) of the mammal heart is highly sensitive t

262 Right ventricle (RV) systolic performance at 24 hours was subs

263 odelling of the pulmonary arteries and right ventricle (RV), which leads to functional decline of car

264 as main pulmonary artery diameter and right ventricle (RV)/left ventricle (LV) ratio.

265 the human left ventricle (LV, n=4) and right ventricle (RV, n=4) after 0, 4, and 8 hours of cold stor

266 Patients with systemic morphological right ventricles (RVs), including congenitally corrected trans

267 fusion of antibodies into the mouse cerebral ventricle(s) or intrathecal space.

268 ook transcriptome analysis of human DMD left ventricle samples and found that DMD hiPSC-derived cardi

269 Entrainment from the right ventricle showed ventricular fusion in 4 out of 5 cases.

270 caques had persistent enlargement of lateral ventricles, smaller volumes and altered functional conne

271 HD were categorized into those with a single ventricle (SV) or two ventricles (TVs) and those with ao

272 In patients with left ventricle systolic dysfunction, 37% (10/27) showed an im

273 oblasts and cardiomyocytes between atria and ventricles that are distinct from those of skeletal musc

274 te administration of liraglutide into fourth ventricle, the area with easy access to the AP and NTS,

275 and cell contractility specific to the right ventricle; these changes could explain the lower cardiac

276 d we also find that LBBB will cause the left ventricle to contract later than the right ventricle, wh

277 oxygenated blood is delivered from the left ventricle to end organs with each cardiac cycle (200 mil

278 to those with a single ventricle (SV) or two ventricles (TVs) and those with aortic obstruction (AO)

279 we compared well and poorly healing cardiac ventricles using a transgenic fish model that exhibits h

280 e close relationship between the RV and left ventricle (ventricular interdependence) and its coupling

281 y matter and amygdala volume, larger lateral ventricle volume, and lower structural connectivity in p

282 er left ventricle mass index and higher left ventricle volumes but not with ejection fraction or dias

283 = 0.036; CVR, 1.30; p < 0.001), and lateral ventricles (VR, 1.56; p = 0.004).

284 aller hearts compared to males, with thinner ventricle walls and more mononucleated cardiomyocytes.

285 f pre-determined neural stem cells along the ventricle walls.

286 The right ventricle was dilated without evidence of right ventricu

287 magnitude of (11)C-PiB signal in the lateral ventricles was calculated as area under the curve from 3

288 90 min after injection using the WB without ventricles were calculated.

289 More specifically, cardiac ventricles were smaller in SMA hearts, whilst liver and

290 is greater than 25% of the perimeter of the ventricle, when the width is approximately half that of

291 t ventricle to contract later than the right ventricle, which in turn affects synchronized contractio

292 d-contacting DA nuclei surrounding the 3(rd) ventricle, which lack distal projections outside of the

293 lly and functionally different from the left ventricle, which precludes direct extrapolation of our k

294 lity of the ependymal cells lining the brain ventricles, which play a crucial role to maintain patenc

295 nd internal surface of the cranium and third ventricle width depending on the severity of leukoaraios

296 deos, our model accurately segments the left ventricle with a Dice similarity coefficient of 0.92, pr

297 and normal size of the left atrium and left ventricle with a normal ejection fraction.

298 We examined the right ventricle with cardiac magnetic resonance imaging in the

299 animals developed dilated and hypertrophied ventricles with preserved systolic functions due to comp

300 and nodoventricular AP appear to connect the ventricles with the CS musculature in the region of the