ライフサイエンスコーパス: ventricular contraction

1 cular tachycardia, and symptomatic premature ventricular contractions).

2 el approach previously applied by neglecting ventricular contraction.

3 tentials and pace-maps matching VT/premature ventricular contraction.

4 icient cardiac function requires synchronous ventricular contraction.

5 ardiac morphogenesis following uncoordinated ventricular contraction.

6 ium mutants can maintain circulation through ventricular contraction.

7 servocommand that was still synchronous with ventricular contraction.

8 bundle branch block, and frequent premature ventricular contractions.

9 duration at 80% repolarization or premature ventricular contractions.

10 n=10) and 1 subject diagnosed with premature ventricular contractions.

11 blocks, as well as bradycardia and premature ventricular contractions.

12 s, polyphasic R-waves and frequent premature ventricular contractions.

13 (2 athletes [1.2%]), and frequent premature ventricular contractions (1 athlete [0.6%]).

14 (57.5%) patients had ablation for premature ventricular contractions, 10 (25%) patients for sustaine

15 (10 with frequent [>1000/24 hours] premature ventricular contractions, 14 with ventricular tachycardi

16 er of hourly episodes of nocturnal premature ventricular contractions (66+/-117 versus 18+/-20, P=0.0

17 consecutive patients undergoing VT/premature ventricular contraction ablation, 7 patients underwent r

18 d for left ventricle outflow tract premature ventricular contraction ablation, an aortic valve closur

19 ibrillation accounted for >50% and premature ventricular contractions accounted for <10% of CRT loss

20 diac MRI provides a mechanism to assess left ventricular contraction and diagnose inducible myocardia

21 lation plays a critical role in accelerating ventricular contraction and speeding relaxation to match

22 educed ventricular tachycardia and premature ventricular contractions and associated re-entrant condu

23 soproterenol infusion also induced premature ventricular contractions and atrioventricular heart bloc

24 including increase in frequency of premature ventricular contractions and shortening of wavelength.

25 bility, including the frequency of premature ventricular contractions and sustained ventricular arrhy

26 Premature ventricular contractions and ventricular tachycardia wer

27 d level of MLC phosphorylation, reduced left ventricular contraction, and induction of hypertrophy; h

28 ycardia/atrial fibrillation; 16.6% premature ventricular contractions; and 8.6% captured as episodes

29 Premature ventricular contractions are a common clinical presentat

30 ycardias, atrial fibrillation, and premature ventricular contractions are known to trigger a reversib

31 Ventricular contraction asynchrony with short-term RV ap

32 phic with relatively short-coupled premature ventricular contractions at onset (300-360 ms); no QT pr

33 stolic peaks and nadirs of CF are because of ventricular contractions at the large majority of pulmon

34 on elicited arrhythmias, including premature ventricular contractions, atrioventricular heart block,

35 1-2.8)], natural logarithm of 24-h premature ventricular contraction burden [HR 1.3 (95% CI: 1.1-1.4)

36 artile range, 10.67-89.79) months, premature ventricular contraction burden decreased from a median o

37 s/min, atrial fibrillation, and/or premature ventricular contractions burden >=10%.

38 Fast cine MRI can be used to assess left ventricular contraction, but its utility for detection o

39 In 3-OST-7 morphants, ventricular contraction can be rescued by overexpression

40 ycardias, atrial fibrillation, and premature ventricular contractions can induce a reversible form of

41 patients presenting with frequent premature ventricular contractions, conduction system disease, and

42 Atrial and ventricular contractions could be discerned from E9.5, a

43 eft ventricular ejection fraction, premature ventricular contraction count/24 h, amount of negative T

44 ac function, and direct measurements of left ventricular contraction demonstrated that PLCepsilon(-/-

45 ng in heart failure with dyssynchronous left ventricular contraction (DHF) and its restoration by car

46 xtures decreased LVDP, baseline rate of left ventricular contraction (dP/dtmaximum), and baseline rat

47 ific to dyssynchronous versus resynchronized ventricular contraction during hemodynamic decompensatio

48 c valve stenosis, catheter-induced premature ventricular contractions during cardiac catheterization

49 at identifying risk-related patterns of left ventricular contraction dynamics via novel volume transi

50 owed major kinetic changes in left and right ventricular contraction (ejection) and relaxation (filli

51 age 41.5+/-16 years) referred for premature ventricular contractions evaluation or suspected ARVC.

52 wn of 3-OST-7 in zebrafish uncouples cardiac ventricular contraction from normal calcium cycling and

53 patients with frequent symptomatic premature ventricular contractions (>5000/24 h) and no known ische

54 patients presenting with frequent premature ventricular contractions have underlying myocardial infl

55 ar apical (RVA) pacing creates abnormal left ventricular contraction, hypertrophy, and reduced pump f

56 kinje system (HPS) is required for efficient ventricular contraction in an apex-to-base direction.

57 Resynchronization of ventricular contraction in patients with heart failure i

58 red with 0 of 30 (0%) (p = 0.048), premature ventricular contractions in 17 of 30 (57%) compared with

59 ed in 3 of 18 (17%), superior axis premature ventricular contractions in 21 of 25 (84%), and new term

60 The return of ventricular contractions in four of these 18 patients a

61 2-stimulation promoted Ca-mediated premature ventricular contractions in heart failure.

62 de, or hypovolemia, and signal the return of ventricular contractions in patients with initially abse

63 In contrast, VT or premature ventricular contractions in the setting of a structurall

64 gnificance of neural remodeling in premature ventricular contraction-induced cardiomyopathy (PVC-CM)

65 ith systolic dysfunction, known as premature ventricular contraction-induced cardiomyopathy.

66 ents (62%), whereas 6 patients had premature ventricular contraction-induced ventricular fibrillation

67 Ventricular contraction is roughly proportional to the a

68 25+/-7 mm Hg and increased to post-premature ventricular contraction mean gradient of 32+/-10 mm Hg,

69 25+/-7 mm Hg to 36+/-11 mm Hg; pre-premature ventricular contraction mean gradient was 25+/-7 mm Hg a

70 delay of the pressure wave, produced by left ventricular contraction, measured between a proximal and

71 ricular tachycardia (n=9/102, 9%), premature ventricular contraction (n=6/102, 6%), and cardiac neuro

72 us pauses, atrioventricular block, premature ventricular contractions, non-sustained ventricular arrh

73 he high daily burden of multifocal premature ventricular contractions observed on 24-hour dynamic ECG

74 Superior axis premature ventricular contractions occurred only in gene carriers.

75 epilepsy (petit mal) episodes, and premature ventricular contractions of the heart.

76 icular ectopic activity, including premature ventricular contractions of the outflow tract alternatin

77 ma (PI3Kgamma(-/-)) showed runs of premature ventricular contractions on adrenergic stimulation that

78 36 (90%) patients (elimination of premature ventricular contraction or noninducibility of ventricula

79 t ventricles (controls) undergoing premature ventricular contraction or VT ablation.

80 on 59+/-7.3%) with drug refractory premature ventricular contractions or ventricular tachycardia unde

81 orderline if polymorphic couplets, premature ventricular contractions, or nonsustained monomorphic VT

82 vs. 1 of 10 [10%], p = 0.006), and premature ventricular contractions originating from the outflow tr

83 Premature ventricular contractions originating in the left ventric

84 ization and increased incidence of premature ventricular contractions (P=0.003), whereas acetylcholin

85 imulation reduced the incidence of premature ventricular contractions (P=0.034) and partially reverse

86 ventricular arrhythmias (-70+/-22% premature ventricular contractions; P<0.05).

87 icular pacing may promote a more coordinated ventricular contraction pattern in these patients.

88 Three diverse ventricular contraction patterns were defined by cardiov

89 lity and efficacy of focal PFA for premature ventricular contraction (PVC) ablation.

90 Variability in premature ventricular contraction (PVC) coupling interval (CI) inc

91 Premature ventricular contraction (PVC) is characterized by early

92 Large-scale data on incidental premature ventricular contraction (PVC) prevalence and morphologie

93 in inflammatory pathway induction, premature ventricular contractions (PVC) and ventricular tachycard

94 High idiopathic premature ventricular contractions (PVC) burden has been associate

95 ght to examine whether suppressing premature ventricular contractions (PVC) using radiofrequency abla

96 achycardia (NSVT) in patients with premature ventricular contractions (PVCs) and heart failure treate

97 for catheter ablation suggest that premature ventricular contractions (PVCs) are a modifiable risk fa

98 achycardia (VT) and high burden of premature ventricular contractions (PVCs) are common in arrhythmog

99 Frequent premature ventricular contractions (PVCs) are often amenable to ca

100 his study was to determine whether premature ventricular contractions (PVCs) arising from the aortic

101 A high burden of premature ventricular contractions (PVCs) at disease diagnosis has

102 Frequent premature ventricular contractions (PVCs) can induce cardiomyopath

103 initiated by short-coupled trigger premature ventricular contractions (PVCs) for which the term short

104 icular (LV) dyssynchrony caused by premature ventricular contractions (PVCs) has been proposed as a m

105 Monomorphic premature ventricular contractions (PVCs) have been shown to initi

106 e consequences of exercise-induced premature ventricular contractions (PVCs) in asymptomatic individu

107 The prognosis of exercise-induced premature ventricular contractions (PVCs) in asymptomatic individu

108 tenolol-sensitive tachycardia with premature ventricular contractions (PVCs) in conscious SH rats.

109 ventricular tachycardia (VT), >500 premature ventricular contractions (PVCs) on 24h-Holter, or a rece

110 er, certain patients with frequent premature ventricular contractions (PVCs) or VT and tachycardiomyo

111 ic ventricular tachycardia (VT) or premature ventricular contractions (PVCs) originating from the myo

112 F), dyssynchrony, tachycardia, and premature ventricular contractions (PVCs), are present in most pat

113 Premature ventricular contractions (PVCs), couplets, and nonsustai

114 of VT or cardiomyopathy related to premature ventricular contractions (PVCs).

115 tachycardia with both AV block and premature ventricular contractions (PVCs).

116 tricular tachycardia (NSVT), or 3) premature ventricular contractions (PVCs).

117 ars, 21 female) with outflow tract premature ventricular contractions (PVCs)/VT.

118 ) and frequent ventricular ectopy (premature ventricular contractions [PVCs] >10/h) was assessed from

119 Premature ventricular contraction rate increased with exercise, an

120 associated with 154 and 102 daily premature ventricular contractions, respectively (rate ratio, 1.51

121 ycardia, ventricular couplets, and premature ventricular contractions showed greater amounts of inter

122 ion and instead would estimate the degree of ventricular contraction, similar to a human expert train

123 shape deepening despite similar magnitude of ventricular contraction, suggestive of ventricular-annul

124 rous cardiac arrhythmias including premature ventricular contractions, tachycardia, and high-degree h

125 rdia by delivering His-synchronous premature ventricular contractions that either delayed the subsequ

126 tricular tachycardia, couplets, or premature ventricular contractions, the extent of intermediate LGE

127 se of beta-blockers to the reduction of left ventricular contractions, three case studies with differ

128 e (AVN) coordinates the timing of atrial and ventricular contraction to optimize cardiac performance.

129 henotype variability, ranging from premature ventricular contractions to sudden cardiac death and hea

130 ycardia, 1 long QT syndrome, and 1 premature ventricular contraction-triggered ventricular fibrillati

131 cardia, and 7 (17.5%) patients for premature ventricular contraction-triggered ventricular fibrillati

132 the development of closely coupled premature ventricular contractions via a phase 2 reentrant mechani

133 A premature ventricular contraction was induced by intentional cathe

134 Normal left-ventricular contraction was shown in seven patients exam

135 e base of the heart in synchrony with native ventricular contractions was evaluated with the use of a

136 ory of hypertension and occasional premature ventricular contractions was found on routine blood work

137 with left ventricle outflow tract premature ventricular contraction were included.

138 Premature ventricular contractions were also strongly associated w

139 Frequent premature ventricular contractions were common among patients with

140 Premature ventricular contractions were common in ARVC patients wi

141 s in the electrocardiogram caused by delayed ventricular contraction (wide QRS complex), is a common

142 used to predict whether regions of abnormal ventricular contraction will improve after revasculariza

143 there were either (1) polymorphic premature ventricular contractions with >/=1 couplet or (2) sustai

144 an acute increase in frequency of premature ventricular contractions with coffee consumption.

145 logy of ventricular tachycardia or premature ventricular contractions with left bundle branch block/i

146 inus beats competing with numerous premature ventricular contractions with right and/or left bundle b