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1  ventricular dilatation and 20% having right ventricular dilatation.
2 with PD98059 delayed the development of left ventricular dilatation.
3 ion, fibrosis, and decompensated right-sided ventricular dilatation.
4  of the ventricular myocardium and resultant ventricular dilatation.
5 processes which occur with acute and chronic ventricular dilatation.
6 hypertrophy and associated with the onset of ventricular dilatation.
7 y as measured by dimensional indexes of left ventricular dilatation.
8 ion analysis to determine predictors of left ventricular dilatation.
9 ventricular end-diastolic pressure, and left ventricular dilatation.
10  parenchymal infarction, and posthemorrhagic ventricular dilatation.
11 leads to a significant elevation of VEGF and ventricular dilatation.
12 ar arrhythmia, even after adjusting for left ventricular dilatation.
13 djusted odds ratio 2.2; P<0.0001), and right ventricular dilatation (adjusted odds ratio 2.2; P<0.000
14 T and leads to impaired cardiac function and ventricular dilatation after cardiac injury.
15 pectively), with 26% of patients having left ventricular dilatation and 20% having right ventricular
16 ermediate-risk pulmonary embolism with right ventricular dilatation and additional clinical risk fact
17  less and is accompanied by progressive left ventricular dilatation and adverse cardiac remodeling.
18 mitral regurgitation and a high incidence of ventricular dilatation and atrial fibrillation.
19 ecause the GSK-3beta knockouts had less left ventricular dilatation and better-preserved left ventric
20 ced hypertrophy that rapidly transitioned to ventricular dilatation and contractile dysfunction.
21 (H222P/H222P) mice after they developed left ventricular dilatation and decreased ejection fraction.
22 38alpha inhibitor ARRY-371797 prevented left ventricular dilatation and deterioration of fractional s
23 ation, inhibit apoptosis, and attenuate left ventricular dilatation and disease progression.
24 ment of older patients with less severe left ventricular dilatation and dysfunction during the years.
25 e mice that survived the first 10 days, left ventricular dilatation and dysfunction remained worse in
26 post-MI, KO mice had significantly more left ventricular dilatation and dysfunction than WT mice.
27 seline evaluation, patients had marked right ventricular dilatation and dysfunction, abnormal septal
28 sis, proliferation of nonmyocyte cells, left ventricular dilatation and dysfunction, and slightly imp
29 auses significant morbidity, producing right ventricular dilatation and dysfunction, exercise intoler
30  initial clinical status and the severity of ventricular dilatation and dysfunction.
31  arrhythmia, and right and occasionally left ventricular dilatation and dysfunction.
32 larly prevalent in families with early-onset ventricular dilatation and dysfunction.
33 eased in the late stages of hypertrophy when ventricular dilatation and failure are present.
34                              Before onset of ventricular dilatation and heart failure (determined by
35 D4(+) T cells and prevented progressive left ventricular dilatation and hypertrophy, whereas adoptive
36 primary myocardial disorder characterized by ventricular dilatation and impaired systolic function.
37 after acute myocardial infarction can reduce ventricular dilatation and improve patient survival.
38 N-ASO treatment prevents progression of left ventricular dilatation and improves left ventricular con
39  treatment (n=17) significantly reduced left ventricular dilatation and increased contractility on ec
40                    It has been proposed that ventricular dilatation and neurohormonal activation duri
41 he microvasculature leading to massive right ventricular dilatation and premature death.
42 cardial infarction (AMI) is characterized by ventricular dilatation and regional akinesis.
43 ardiomyopathy (IDC) is characterized by left ventricular dilatation and systolic dysfunction after kn
44 predominantly in coronary heart disease with ventricular dilatation and systolic dysfunction.
45 major cause of heart failure (HF) defined by ventricular dilatation and systolic dysfunction.
46 8)Ga]FAPI-46 uptake was associated with left ventricular dilatation and systolic dysfunction.
47 ngs in Dhcr7(T93M/Delta3-5) mice include CNS ventricular dilatation and two to three syndactyly.
48 perior preservation of LV geometry with less ventricular dilatation and wall thinning.
49 cal (lower NYHA functional class, absence of ventricular dilatation, and absence of implantable cardi
50 oinciding with severe hypertrophy, preceding ventricular dilatation, and glucose transporter protein
51 ith predominantly endocardial fibrosis, left ventricular dilatation, and systolic dysfunction.
52  ventricular diastolic dysfunction and right ventricular dilatation are the echocardiographic variabl
53  troponin T (deltaLys210) caused early-onset ventricular dilatation (average age at diagnosis, 24 yea
54 c left ventricular remodeling, greater right ventricular dilatation (base, 34+/-7 versus 31+/-6 and 3
55 minent in animals that had more initial left ventricular dilatation (both P<0.05).
56 e conclude that ALPK3 cardiomyopathy induces ventricular dilatation caused by insufficient myomesin-m
57                   Athletes with CTE had more ventricular dilatation, cavum septum pellucidum, thalami
58                         From 5 weeks onward, ventricular dilatation, decreased contractility, and dim
59 sation in most animals characterized by left ventricular dilatation, depressed systolic function, and
60 TnI1-193) in the heart were found to develop ventricular dilatation, diminished contractility, and re
61 omed to the heart and induced long-term left ventricular dilatation, dysfunction, and fibrosis in nai
62    Right ventricular injury (including right ventricular dilatation/dysfunction/limitation/failure) a
63 in dyssynchrony >200 ms, lack of severe left ventricular dilatation (end-systolic dimension index <29
64 mice developed severe heart failure and left ventricular dilatation even under baseline conditions an
65 ructural remodeling is characterized by left ventricular dilatation, fibrosis, and hypertrophy of the
66 : 1.91; 95% CI: 1.19-3.05; P = 0.007), right ventricular dilatation &gt;42 mm (OR: 3.35; 95% CI: 1.37-9.
67 monary hypertension is associated with right ventricular dilatation, high right-sided filling pressur
68 flow velocities, valvular regurgitation, and ventricular dilatation; however, its ability to visualiz
69  iASPP deficiency specifically induces right ventricular dilatation in mouse embryos at embryonic day
70 centric left ventricular remodeling and less ventricular dilatation in response to arterial hypertens
71  experienced periventricular haemorrhage and ventricular dilatation in the neonatal period showed the
72 of the extracellular matrix may help prevent ventricular dilatation in the pressure-overloaded RV.
73 mber per heart, contractile dysfunction, and ventricular dilatation in zebrafish.
74 mitral E-to-strain-rate e'-wave ratio, right ventricular dilatation (increased right ventricular end-
75                                         Left ventricular dilatation is a well-recognized precursor of
76     Hydrocephalus, characterized by cerebral ventricular dilatation, is routinely attributed to prima
77                        At both ages, lateral ventricular dilatation, necrotic foci, periventricular c
78 isease, before fixed atrial fibrillation and ventricular dilatation occur.
79 ic volumes revealed that the least amount of ventricular dilatation occurred in the MN group (P<0.05)
80  evidence of periventricular haemorrhage and ventricular dilatation on neonatal ultrasound exhibited
81 tudy establishes the role of very early left ventricular dilatation on outcome in MI and may be usefu
82 curs soon after MI, the effect of early left ventricular dilatation on outcome is not established and
83                           The effect of left ventricular dilatation on the risk of heart failure in p
84 e findings challenge the paradigm that right ventricular dilatation on ultrasound during cardiopulmon
85  3-4, i.e., intraventricular hemorrhage with ventricular dilatation or intraparenchymal involvement).
86 riant of PPCM in mice, characterized by left ventricular dilatation (postpartum second delivery: left
87    The magnitude of elevation predicted left ventricular dilatation (r=.80 when variables were treate
88 hages or intraventricular hemorrhage without ventricular dilatation, respectively); and 3) severe int
89    Neonatal periventricular haemorrhage with ventricular dilatation revealed by ultrasound may be ass
90 on secondary to increased afterload and left ventricular dilatation secondary to volume overload.
91                                   The marked ventricular dilatation seen in the untreated fistula gro
92 eficient mice showed heart failure with left ventricular dilatation, systolic dysfunction and lung co
93 onses culminate in diminished pump function, ventricular dilatation, wall thinning, and fibrosis, and
94 tion, left ventricular hypertrophy, and left ventricular dilatation were associated with the developm
95 e-treated HF mice displayed progressive left ventricular dilatation with significantly increased end-