ライフサイエンスコーパス: ventricular end-diastolic volume

1 dysfunction by reducing the unstressed left ventricular end-diastolic volume.

2 6+/-58 mm(3) at follow-up, P=0.006) of right ventricular end-diastolic volume.

3 ricular ejection fraction, and indexed right ventricular end-diastolic volume.

4 emoglobin mass, total blood volume, and left-ventricular end-diastolic volume.

5 including after adjustment for baseline left ventricular end-diastolic volume.

6 ed as an increase of 15% or more in the left ventricular end-diastolic volume.

7 ter mean LVMT was associated with lower left ventricular end-diastolic volume (0.01 mm/mL; P<0.01), a

8 sponse (defined as percent reduction in left ventricular end-diastolic volume 1 year after CRT-D impl

9 s had mildly dilated right ventricles (right ventricular end-diastolic volume=101+/-26 mL/m(2)) with

10 stroke volume (SV) without a change in left ventricular end diastolic volume (102+/-16% change from

11 rsus 1.4 respectively; P=0.01) as were right ventricular end-diastolic volumes (113.7 30.9 versus 90

12 Significant reductions in right ventricular end-diastolic volume (12% reduction; P < 0.0

13 rdial function on cardiac MRI including left ventricular end diastolic volume (-13 [28] vs 10 [26] mL

14 133 beats per minute +/- 19, P < .001), left ventricular end-diastolic volume (140 mL +/- 32 vs 148 m

15 There was a larger increase in right ventricular end-diastolic volume (15 mL; 95% confidence

16 0.4 +/- 2 to 7.7 +/- 4 mL; p < .05) and left ventricular end-diastolic volume (18.5 +/- 3 to 14.2 +/-

17 with placebo, a significant decrease in left ventricular end-diastolic volume (-18 mL; P=0.009) and e

18 shown by increased E-wave velocity and left ventricular end-diastolic volume, 2) exhibit a higher pl

19 ventricular remodeling (>20% change in left ventricular end-diastolic volume; 21.91 [2.75-174.29]; P

20 tolic pressure by 40% to 60% (p < .05), left ventricular end-diastolic volume 25 +/- 8%, and stroke v

21 Fluid loading increased right ventricular end-diastolic volume (+31 +/- 13 mL; p = 0.0

22 a was linearly related to reductions in left ventricular end-diastolic volume (-4.1 ml; 95% confidenc

23 ificantly (P<0.05) greater reduction in left ventricular end-diastolic volume (-49+/-16% versus -35+/

24 Left ventricular end-diastolic volume (72.5 to 77.4 mL/m2, P=

25 84.0% versus 87.0%) and improvement in left ventricular end-diastolic volume (-8.0 mL versus -12.7 m

26 Diuretic treatment decreased right ventricular end-diastolic volume (-84 +/- 11 mL; p < 0.0

27 (54 +/- 10 to 87 +/-6 mL; p < .05) and right ventricular end-diastolic volume (90 +/-11 to 128 +/- 18

28 Right ventricular end-diastolic volume (99.9 to 89.7 mL/m2, P<

29 the parasternal short axis view, to the left ventricular end-diastolic volume acquired using the bipl

30 with change in RV ejection fraction and left ventricular end-diastolic volume, although correlation c

31 On echocardiography, left ventricular end diastolic volume and deceleration time i

32 was evaluated as the change in indexed left ventricular end diastolic volume and LVEF.

33 odilation reflected by a 10% decline in left ventricular end-diastolic volume and a 30% fall in atria

34 improved cardiac function, measured by left ventricular end-diastolic volume and cardiac output.

35 ndary end points, including structural (left ventricular end-diastolic volume and left ventricular ej

36 on compared with placebo did not change left ventricular end-diastolic volume and left ventricular ma

37 Indexed left ventricular end-diastolic volume and mass correlated wit

38 d 10 weeks after infarction showed that left ventricular end-diastolic volume and mass increased and

39 was associated with modest reduction in left ventricular end-diastolic volume and preserved global lo

40 and 24+/-5 g/m(2); P<0.001) and indexed left ventricular end-diastolic volume and right ventricular e

41 -of-life score, 6-minute walk distance, left ventricular end-diastolic volume, and left ventricular e

42 s showed a borderline (16%) increase in left ventricular end-diastolic volume (angiography), whereas

43 The limits of agreement between the left ventricular end-diastolic volume as estimated by bioimpe

44 me unloading results in a reduction in right ventricular end-diastolic volume, as expected, but left

45 erved between changes in TR volume and right ventricular end-diastolic volume at 30 days.

46 ction fraction (beta=-0.02/%; P=0.015), left ventricular end-diastolic volume (beta=0.01/mL; P<0.0001

47 there was a 48% (P < .001) reduction in left ventricular end diastolic volume, beyond that achievable

48 right ventricular end-diastolic volume (left ventricular end-diastolic volume/body surface area, 104+

49 104+/-13 and 69+/-18 mL/m(2); P<0.001; right ventricular end-diastolic volume/body surface area, 110+

50 r end-diastolic pressure by 30% to 40%, left ventricular end-diastolic volumes by 33 +/- 9%, and tran

51 Pulmonary capillary wedge pressures and left ventricular end-diastolic volumes by use of echocardiogr

52 t ventricular remodelling, with reduced left ventricular end-diastolic volume (by 8%, P < 0.001) and

53 The relation between stroke volume and left ventricular end-diastolic volume, by the Frank-Starting

54 timates of ventricular filling pressures and ventricular end-diastolic volumes/cardiac performance va

55 interval, -4.02 to -0.23; P=0.028) and left ventricular end-diastolic volume (coefficient, 7.85; 95%

56 ociated with a significant reduction in left ventricular end-diastolic volume compared with the nonca

57 end-diastolic pressure (LVEDP), but not left ventricular end-diastolic volume, consistent with increa

58 Left ventricular end-diastolic volume decreased (from 125.1 +

59 Cardiac index increased and left ventricular end-diastolic volume decreased significantly

60 ipping led to a significant increase in left ventricular end-diastolic volume, demonstrating increase

61 The mean change in left ventricular end-diastolic volume differed significantly

62 sociation with an expected decrease in right ventricular end-diastolic volume during lower-body sucti

63 Patients who increased left ventricular end-diastolic volume during lower-body sucti

64 sonance imaging demonstrated increasing left ventricular end-diastolic volumes, end-systolic volumes,

65 on >=10% and improvement in the indexed left ventricular end diastolic volume >=10% at 12 months.

66 rior hospitalization for heart failure, left ventricular end-diastolic volume >/=125 mL/m(2), and lef

67 emodeling was defined as an increase in left ventricular end-diastolic volume >/=20% at 6 months.

68 CMR-derived left ventricular end-diastolic volume >246 mL had good, altho

69 A reduced left ventricular end diastolic volume, impaired early filling

70 r pressure increase and dilatation, but left ventricular end-diastolic volume improved because of red

71 Left ventricular end-diastolic volume improved from 172 ml to

72 ts, there was a paradoxical increase in left ventricular end-diastolic volume in association with an

73 mm Hg lower-body suction, on right and left ventricular end-diastolic volumes in 21 patients with ch

74 to 43+/-14 mL/m(2), P<0.0001), whereas left ventricular end diastolic volume increased (66+/-12 to 7

75 Right ventricular end diastolic volume increased by 49.4% afte

76 After 17 cycles of trastuzumab, indexed left ventricular end diastolic volume increased in patients t

77 Left ventricular end-diastolic volume increased in the rapid

78 Maximal left atrial volume index and left ventricular end diastolic volume index, ambulatory pulse

79 ing without surgery at 5 years, 90% for left ventricular end-diastolic volume index <100 mL/m(2) vers

80 us-ICD group had greater improvement in left ventricular end-diastolic volume index (-26.2 versus -7.

81 [95% CI, -8.5 to -0.1] mL/m2; P = .04), left ventricular end-diastolic volume index (-5.5 [95% CI, -1

82 a brain natriuretic peptide (79 pg/mL), left ventricular end-diastolic volume index (110 mL/m2), and

83 cause the degree of SMR relative to the left ventricular end-diastolic volume index (LVEDVi) was subs

84 not correlate with six-month changes in left ventricular end-diastolic volume index (p = 0.26), LVESV

85 phy showed a small, stable reduction in left ventricular end-diastolic volume index (P<0.001), with a

86 ncomitant small stable increase in the right ventricular end-diastolic volume index (P<0.001).

87 RV stroke volume index (P<0.0001), and left ventricular end-diastolic volume index (P=0.0015).

88 3.2 +/- 2.9 years, LVEF 0.21 +/- 0.07, left ventricular end-diastolic volume index 180 +/- 64 ml/m2.

89 Patients with a right ventricular end-diastolic volume index above 150 ml/m(2)

90 Patients with a right ventricular end-diastolic volume index above 150 ml/m(2)

91 .001) increase in change from baseline right ventricular end-diastolic volume index and a 429 ml (P <

92 The relationship between left ventricular end-diastolic volume index and stroke volume

93 The left ventricular end-diastolic volume index decreased (90.1 +

94 ulmonary artery occlusion pressure, and left ventricular end-diastolic volume index failed to correla

95 Resting left ventricular end-diastolic volume index increased (P=0.00

96 Right ventricular end-diastolic volume index measured by means

97 lic volume index threshold of 227% or a left ventricular end-diastolic volume index of 58 ml/m(2) ide

98 ry outcome was change from baseline in right ventricular end-diastolic volume index versus placebo.

99 f SCA were PET sympathetic denervation, left ventricular end-diastolic volume index, creatinine, and

100 MP-2 levels were associated with larger left ventricular end-diastolic volume index, greater left ven

101 atrial area, left atrial volume index, left ventricular end-diastolic volume index, peak E wave, and

102 tion, initial central venous pressure, right ventricular end-diastolic volume index, pulmonary artery

103 B-type natriuretic peptide, and larger left ventricular end-diastolic volume index.

104 er left atrial volume index, and larger left ventricular end-diastolic volume index.

105 95% CI, 1.8-12.8]; P=0.002) and greater left ventricular end-diastolic volume indexed (hazard ratio,

106 remained predictive after adjusting for left ventricular end-diastolic volume indexed (hazard ratio,

107 olute ventilatory anaerobic threshold, right ventricular end-diastolic volume indexed, ventilatory an

108 t ventricular end-diastolic volume and right ventricular end-diastolic volume (left ventricular end-d

109 zone and its percentage of myocardium, left ventricular end-diastolic volume, left ventricular end-s

110 rdial scar volume by CMR is superior to left ventricular end-diastolic volume, left ventricular end-s

111 cant improvements in ejection fraction, left ventricular end-diastolic volume, left ventricular end-s

112 the following quantitative results: (a) left ventricular end-diastolic volume (LVDV) of 165 mL (LVDV/

113 the following quantitative results: (a) left ventricular end-diastolic volume (LVDV) of 165 mL (LVDV/

114 itative results were obtained with MRI: Left ventricular end-diastolic volume (LVDV) was 40 mL (LVDV

115 itative results were obtained with MRI: Left ventricular end-diastolic volume (LVDV) was 40 mL (LVDV

116 entricular ejection fraction (LVEF) and left ventricular end diastolic volume (LVEDV) on cardiac magn

117 Cut off values for change in left ventricular end-diastolic volume (LVEDV) and LV end-syst

118 onist tolvaptan (30 mg/day) on reducing left ventricular end-diastolic volume (LVEDV) compared with p

119 less than 40% to 50% or greater, whose left ventricular end-diastolic volume (LVEDV) had normalised,

120 Gated SPECT measurements for left ventricular end-diastolic volume (LVEDV) index, left ven

121 ry capillary wedge pressure (PCWP), SV, left ventricular end-diastolic volume (LVEDV), and left ventr

122 ing a multivariable MR framework (e.g., left ventricular end-diastolic volume (LVEDV), Beta = 0.33, 9

123 Left ventricular end-diastolic volume (LVEDV), left ventricul

124 with CRT, defined as percent change in left ventricular end-diastolic volume (LVEDV), was analyzed i

125 eft ventricular end-systolic volume and left ventricular end-diastolic volume (LVEDV).

126 mL, P < 0.001; [95% CI 113 to 455]) and left ventricular end-diastolic volume (LVEDV; +10 mL, P < 0.0

127 -3.46 mL/m(2) [-5.8 to -1.2], P=0.003, right ventricular end-diastolic volume/m(2): -4.2 mL/m(2) [-6.

128 -6.8% to 2.1%]; P = .29) or changes in left ventricular end-diastolic volume (mean difference, 13.4

129 ratio, 1.16; P=0.002), exercise indexed left ventricular end-diastolic volume (odds ratio, 1.04; P=0.

130 ) was the only predictor of increase in left ventricular end-diastolic volume (p < 0.001).

131 VOT) gradient (P = 0.01), and increased left ventricular end-diastolic volume (P = 0.02) and stroke v

132 end-diastolic volume, as expected, but left ventricular end-diastolic volume paradoxically increases

133 ular end-diastolic pressure (LVEDP) and left ventricular end-diastolic volume (preload) in CHF rats,

134 n fraction (r = -0.606), higher indexed left ventricular end-diastolic volume (r = 0.572), and higher

135 thod (n=53, r=0.85, P<0.0001), and with left ventricular end-diastolic volume (r=0.81, P<0.0001).

136 -beam computed tomography for measuring left ventricular end-diastolic volume (r=0.96; standard error

137 splayed an additional decline in the RV/left ventricular end-diastolic volume ratio (P=0.05) and tren

138 ricular ejection fraction, and indexed right ventricular end-diastolic volume resulted in significant

139 ; and ejection fraction of 52% and (b) right ventricular end-diastolic volume (RVDV) of 163 mL (RVDV/

140 , and ejection fraction of 52% and (b) right ventricular end-diastolic volume (RVDV) of 163 mL (RVDV/

141 Right ventricular end-diastolic volume (RVDV) was 262 mL (RVDV

142 Right ventricular end-diastolic volume (RVEDV) was reduced in

143 ange or change of 15 to 30 mL in 3DTTE right ventricular end-diastolic volume; sample sizes were 2x t

144 ow obstruction are inversely related to left ventricular end-diastolic volume, stroke volume, and car

145 earts increased relaxation velocity and left ventricular end diastolic volume to produce higher left

146 aval occlusion (VCO) was used to reduce left ventricular end-diastolic volume to 70 +/- 5% of baselin

147 s minor, and a decrease in the ratio of left ventricular end-diastolic volume to body weight reflecte

148 ume (angiography), whereas the ratio of left ventricular end-diastolic volume to body weight was redu

149 The mean left ventricular end-diastolic volume was 108 +/- 47 mL, as e

150 regurgitant fraction was 34+/-17%, and right ventricular end-diastolic volume was 114+/-39 cc/m(2).

151 genic right ventricular dysplasia, but right ventricular end-diastolic volume was decreased in RyR2(R

152 However, the change in right ventricular end-diastolic volume was similar in the two

153 By CMR, right ventricular end-diastolic volumes were 118 +/- 31 cc/m(2

154 Functional and atrialized right ventricular end-diastolic volumes were 84 15 and 21 13 m

155 At 6 months follow-up, left atrial and left ventricular end-diastolic volumes were significantly mor

156 eat stress did not significantly change left ventricular end-diastolic volume, while ventricular end-

157 most recent echocardiogram, the median left ventricular end-diastolic volume z score was +1.7 (range

158 with published normal values, left and right ventricular end-diastolic volume z scores were mildly en

159 nsion z-score (OR=2.2, P=0.02) or lower left ventricular end-diastolic volume z-score (OR=1.9, P=0.03