ライフサイエンスコーパス: ventricular end-systolic volume

1 ponse was defined as a 15% reduction in left ventricular end-systolic volume.

2 MSC group, because of a preservation of left ventricular end-systolic volume.

3 e brain natriuretic peptide levels, and left ventricular end-systolic volume.

4 The primary end point was left ventricular end-systolic volume.

5 ic volume (0.01 mm/mL; P<0.01), a lower left ventricular end-systolic volume (-0.01 mm/mL; P=0.01), a

6 SMT decreased both left ventricular end-systolic volume (10.4 +/- 2 to 7.7 +/- 4

7 and was associated with a reduction in left ventricular end-systolic volume (-24.8 +/- 3.0 ml vs. -8

8 unction as indicated by an increase in right ventricular end-systolic volume (54 +/- 10 to 87 +/-6 mL

9 in +/- 3.9, P < .001) and a decrease in left ventricular end-systolic volume (58 mL +/- 18 vs 46 mL +

10 ic volume (-49+/-16% versus -35+/-20%), left ventricular end-systolic volume (-59+/-20 versus -37+/-2

11 ignificant differences were observed in left ventricular end-systolic volumes (-6.4 mL [95% CI, -18.8

12 ctions (42% versus 69%), higher indexed left ventricular end-systolic volumes (96 versus 40 mL), and

13 stolic and end-diastolic dimensions and left ventricular end-systolic volume also decreased after 12

14 with pulmonary regurgitation, elevated right ventricular end systolic volumes and reduced right and l

15 luded all-cause mortality and change in left ventricular end-systolic volume and end-diastolic volume

16 VEF) and the noninvasive calculation of left ventricular end-systolic volume and left ventricular end

17 left ventricular end-diastolic volume, left ventricular end-systolic volume, and left ventricular ej

18 left ventricular end-diastolic volume, left ventricular end-systolic volume, and LVEF were not stati

19 e hospitalization, or <15% reduction of left ventricular end-systolic volume assessed at 12 months.

20 S-LVRR (defined as >/=15% reduction in left ventricular end-systolic volume at 1-year of follow-up)

21 dians (quartiles 1 and 3) for change in left ventricular end-systolic volume at 6 months for the Smar

22 No difference in improvement in left ventricular end-systolic volume at 6 months was observed

23 sponse was defined as >15% reduction in left ventricular end-systolic volume at 6 months.

24 osite score and > or = 15% reduction in left ventricular end-systolic volume at 6 months.

25 The effect of cooling on left ventricular end-systolic volume at a pressure of 100 mm

26 ft ventricular contractility increased (left ventricular end-systolic volume at a pressure of 100 mm

27 actility was assessed by the calculated left ventricular end-systolic volume at an end-systolic left

28 ollow-up echocardiography compared with left ventricular end-systolic volume at baseline.

29 962-C were also associated with reduced left ventricular end-systolic volume (beta [SE], -1.90 [0.65]

30 ic volume (beta=0.01/mL; P<0.0001), and left ventricular end-systolic volume (beta=0.01/mL; P<0.001)

31 left ventricular end-diastolic volume, left ventricular end-systolic volume, cardiac index, dP/dt ma

32 mproved in 69% of 426 patients, whereas left ventricular end-systolic volume decreased > or = 15% in

33 Echocardiographic assessments of left ventricular end-systolic volume, end-diastolic volume, m

34 during the first month (120% increased left ventricular end-systolic volume [ESV; P<0.01]), but shun

35 A change in left ventricular end-systolic volume from intermediate stage

36 ess of CRT was defined as a decrease in left ventricular end-systolic volume >15% at follow-up echoca

37 RSs for hypertrophic cardiomyopathy and left ventricular end-systolic volume improved AUC in CCSS (0.

38 ated with the right atrial volume than right ventricular end-systolic volume in AF-TR (P<0.001).

39 Isoprenaline decreased left ventricular end-systolic volume in wild-type hearts (10.

40 aced QRS duration, and smaller baseline left ventricular end systolic volume index also were also ass

41 d from 29 +/- 10.4 to 39 +/- 12.4%, and left ventricular end systolic volume index decreased from 109

42 hanges in the clinical composite score, left ventricular end systolic volume index, 6-minute walk tim

43 GFI <37 (hazard ratio, 2.52; P=0.004), right ventricular end-systolic volume index >85 mL/m(2) (hazar

44 lume index (-26.2 versus -7.4 mL/m(2)), left ventricular end-systolic volume index (-28.7 versus -9.1

45 Empagliflozin significantly reduced left ventricular end-systolic volume index (-4.3 [95% CI, -8.

46 ) at follow-up; p = 0.0012), as did the left ventricular end-systolic volume index (48.4 +/- 19.7 ml/

47 The primary end point was the left ventricular end-systolic volume index (LVESVI) at 12 mon

48 The left ventricular end-systolic volume index (LVESVI) decreased

49 found no significant difference in the left ventricular end-systolic volume index (LVESVI) or surviv

50 Patients with a left ventricular end-systolic volume index (LVESVI) reduction

51 Longitudinal data analysis of left ventricular end-systolic volume index (LVESVi) was perfo

52 The primary end point was the left ventricular end-systolic volume index (LVESVI), a measur

53 found no significant difference in the left ventricular end-systolic volume index (LVESVI), survival

54 ienced a 25.3-mL/m(2) mean reduction in left ventricular end-systolic volume index (P<0.0001), wherea

55 0.01) and was associated with increased left ventricular end-systolic volume index (r=0.62, P<0.01),

56 nt MRI predictors of death (P < 0.01): right ventricular end-systolic volume index adjusted for age a

57 In a multivariable regression model, left ventricular end-systolic volume index and left atrial vo

58 Right ventricular end-systolic volume index and left ventricul

59 p = 0.004), but not when combined with right ventricular end-systolic volume index and strain-rate e'

60 Percentage-predicted right ventricular end-systolic volume index can identify a hig

61 patients requires further scrutiny; 4) left ventricular end-systolic volume index has emerged as an

62 Percentage-predicted right ventricular end-systolic volume index independently pred

63 e remodeling, defined as an increase in left ventricular end-systolic volume index of >15% at 24 mont

64 Left ventricular end-systolic volume index remained unchanged

65 d Main Results: A percentage-predicted right ventricular end-systolic volume index threshold of 227%

66 vorably affected by VNS (p < 0.05), but left ventricular end-systolic volume index was not different

67 as also a strong predictor of change in left ventricular end-systolic volume index with monotonic inc

68 ight ventricular dilatation (increased right ventricular end-systolic volume index), high Acute Physi

69 t age, sex, ST or T changes on ECG, and left ventricular end-systolic volume index, LGE maintained a

70 MVR group compared with the CABG group: left ventricular end-systolic volume index, MR volume, and pl

71 ox regression analyses, the MRI-derived left ventricular end-systolic volume index, RV, and OMR categ

72 erapy, or a 15% or more increase in the left ventricular end-systolic volume index.

73 eatures (decreasing order of strength: right ventricular end-systolic volume indexed, right ventricul

74 Corrected right ventricular end-systolic volume is a strong prognostic m

75 ufficient for classification (left and right ventricular end-systolic volumes, left atrial volume, my

76 icacy end point was the 6-month indexed left ventricular end-systolic volume (LVESV) adjusted for bas

77 r (CRT-D), defined as reduction in both left ventricular end-systolic volume (LVESV) and left atrial

78 Left ventricular end-systolic volume (LVESV) and LVSV were ob

79 lar end-diastolic volume (LVEDV) index, left ventricular end-systolic volume (LVESV) index, and LVEF

80 The primary endpoint was change in left ventricular end-systolic volume (LVESV) on cardiac magne

81 entricular ejection fraction (LVEF) and left ventricular end-systolic volume (LVESV) relative to base

82 as measured as reductions in atrial and left ventricular end-systolic volumes (LVESV) at 1 year.

83 % and <25% reductions, respectively, in left ventricular end-systolic volume [LVESV] at 1 year compar

84 idated polygenic risk score for indexed left ventricular end-systolic volume (LVESVi-PRS), previously

85 mL/m(2); normal range, 64-100 mL/m(2)), left ventricular end-systolic volume (LVSV) of 80 mL (LVSV/BS

86 mL/m(2); normal range, 64-100 mL/m(2)), left ventricular end-systolic volume (LVSV) of 80 mL (LVSV/BS

87 r body surface area [BSA], 25 mL/m(2)); left ventricular end-systolic volume (LVSV), 21 mL (LVSV/BSA,

88 r body surface area [BSA], 25 mL/m(2)); left ventricular end-systolic volume (LVSV), 21 mL (LVSV/BSA,

89 ar arterial elastance (p = 0.003), and right ventricular end-systolic volume (p = 0.020) while right

90 d for age, sex, and body surface area, right ventricular end-systolic volume (P=0.004) strongly predi

91 patients with crypts had lower indexed left ventricular end-systolic volumes (P=0.042) and higher in

92 ng (left ventricular ejection fraction, left ventricular end-systolic volume), plus clinical outcomes

93 Left ventricular end-systolic volume progressively increased

94 ; P<0.001) and related to the change in left ventricular end-systolic volume (r=-0.53; P<0.001).

95 estimate, 21.34 mL; bias, -4.93 mL) and left ventricular end-systolic volume (r=0.96; standard error

96 alue of systolic blood pressure-indexed left ventricular end-systolic volume ratio, or cardiac contra

97 Echocardiographic left ventricular end-systolic volume reduction >=15% after 6

98 egments at peak stress, and an abnormal left ventricular end-systolic volume response to stress were

99 ranging from 35% to 91%; for predicting left ventricular end-systolic volume response, sensitivity ra

100 L/m(2); normal range, 63-111 mL/m(2)), right ventricular end-systolic volume (RVSV) of 81 mL (RVSV/BS

101 L/m(2); normal range, 63-111 mL/m(2)), right ventricular end-systolic volume (RVSV) of 81 mL (RVSV/BS

102 V) was 262 mL (RVDV/BSA, 164 mL/m(2)); right ventricular end-systolic volume (RVSV), 198 mL (RVSV/BSA

103 etralogy of Fallot in women had larger right ventricular end-systolic volumes (standard deviation sco

104 A polygenic score of MRI-derived left ventricular end systolic volume strongly associates with

105 tricular ejection fraction, from which right ventricular end-systolic volume was derived, was measure

106 left ventricular end-diastolic volume, while ventricular end-systolic volume was reduced by 24 +/- 6%

107 owed that incremental 10% reductions in left ventricular end-systolic volume were associated with cor

108 hic variables, especially the change in left ventricular end-systolic volume with exercise and the ex