ライフサイエンスコーパス: ventricular function

1 g pulmonary hemodynamic parameters and right ventricular function.

2 on and are sometimes associated with reduced ventricular function.

3 ular remodeling and regional and global left ventricular function.

4 dequately address the effect of PVCs on left ventricular function.

5 ered, its severity, and its effects on right ventricular function.

6 smaller infarct size and preserved long-term ventricular function.

7 ship between its protein levels and systolic ventricular function.

8 ationship with age, sex, body size, and left ventricular function.

9 sudden death, and in patients with poor left ventricular function.

10 e in interstitial fibrosis and a decrease in ventricular function.

11 erstitial fibrosis, leading to improved left ventricular function.

12 gradation pattern differed depending on left ventricular function.

13 asuring annulus diameter and valve and right ventricular function.

14 nty-two (65%) had a preserved left and right ventricular function.

15 ic shunt (TIPS) creation, regardless of left ventricular function.

16 result in symptom resolution and recovery of ventricular function.

17 rtality, imparting a negative impact on left ventricular function.

18 recent-onset heart failure and reduced left ventricular function.

19 terms of clinical events or changes in left ventricular function.

20 ng both improved systolic and diastolic left ventricular function.

21 mine their association with aortic valve and ventricular function.

22 xperimental studies metformin preserves left ventricular function.

23 perimental MI had significantly reduced left ventricular function.

24 12]), after adjustment for clinical risk and ventricular function.

25 se remodeling without adverse effect on left ventricular function.

26 predictors of recurrence and impact on left ventricular function.

27 reatment should focus on the preservation of ventricular function.

28 aching of cardiac physiology focuses on left ventricular function.

29 the relationship between adverse events and ventricular function.

30 endent of the concurrent improvement in left ventricular function.

31 old-standard technique for the assessment of ventricular function.

32 gnificantly from accepted donors with normal ventricular function.

33 hypothesis that TRPM7 is required for normal ventricular function.

34 mproved in parallel with improvement in left ventricular function.

35 ents with symptoms at rest and impaired left ventricular function.

36 ype 1, but rather an improved diastolic left ventricular function.

37 ting the influence of respiration on cardiac ventricular function.

38 Tl along with contrast cineCT to assess left ventricular function.

39 to study its impact on functional class and ventricular function.

40 creased perinatal survival and impaired left ventricular function.

41 n the subset of patients with preserved left ventricular function.

42 educes adverse remodeling and preserves left ventricular function.

43 blood pressure, systolic, or diastolic left ventricular function.

44 and mortality in patients with impaired left ventricular function.

45 onary arterial compliance, and reduced right ventricular function.

46 mplantation on the basis of symptoms or left ventricular function.

47 (AMI) has led to limited improvement in left ventricular function.

48 the gold standard in the evaluation of left ventricular functions.

49 software programs were used to compare left ventricular functions.

50 nse to post-acute myocardial infarction left ventricular functions.

51 nhances neovascularization and improves left ventricular functions.

52 trophy, fibrosis and restoration of the left ventricular functions.

53 ft survival from accepted donors with normal ventricular function (10.6 years) was similar to surviva

54 weaker in severe CHF compared to normal left ventricular function; 2) increasing HRR using rate-adapt

55 reduced myocardial death, and improved left ventricular function 24 hours after I/R injury in MMVV i

56 rveillance of cardiomyopathy (45 [10.5%]) or ventricular function (36 [8.4%]).

57 cases of acute myocardial injury with normal ventricular function (4/5, 80% with late gadolinium enha

58 ar size (94% versus 80%; P=0.001), and right ventricular function (87% versus 73%; P=0.006).

59 donors with moderately-to-severely depressed ventricular function (9.1 years; P=0.13).

60 l from accepted donors with mildly depressed ventricular function (9.7 years; P=0.24) and from accept

61 including assessment of regional and global ventricular function, absolute myocardial blood flow qua

62 All had rapid and complete recovery of ventricular function after intravenous corticosteroids.

63 r formation, thereby augmenting cardiac left ventricular function after ischemia/reperfusion.

64 cardiac fibrosis, as well as decreased left ventricular function, after myocardial infarction.

65 aphic assessment of myocardial mass and left ventricular function also did not suggest myocardial atr

66 (R3K5A) mice leads to an improvement in left ventricular function and a partial rescue of left ventri

67 state of lipotoxicity that compromises left ventricular function and aggravate heart failure.

68 ) apical pacing may lead to deterioration in ventricular function and an increased risk of heart fail

69 myocardial infarction model can improve left ventricular function and attenuate cardiac remodeling.

70 ion were accompanied by improvements in left ventricular function and attenuation of left ventricle r

71 ardiopoietic stem cell therapy improves left ventricular function and blunts pathological remodeling.

72 ith recent-onset heart failure, reduced left ventricular function and clinically suspected myocarditi

73 gns of abnormal diastolic and systolic right ventricular function and compression of the atrioventric

74 The findings of reduced ventricular function and conduction system defects in My

75 VB3-infected mice developed an impaired left ventricular function and displayed an increased left ven

76 enital AS frequently have impaired diastolic ventricular function and exercise capacity that may be r

77 (CHD) and investigate its relationship with ventricular function and exercise capacity.

78 ited myocardial infarct size, preserved left ventricular function and had no influence on haemodynami

79 , providing a rationale for focused study of ventricular function and heart failure after targeting t

80 rdial infarction complicated by reduced left ventricular function and HF.

81 more adverse cardiac remodeling, poorer left ventricular function and higher mortality by increasing

82 rs vs. 52.3 years), had worse right and left ventricular function and higher pulmonary artery pressur

83 horacic epidural anesthesia may affect right ventricular function and interfere with the coupling bet

84 ired congenital heart disease with preserved ventricular function and isthmus-dependent re-entry, VT

85 patients scheduled for lung resection, right ventricular function and its response to increased after

86 Regional left ventricular function and late-gadolinium enhancement wer

87 Parameters of left and right ventricular function and LGE burden were measured in 205

88 lot underwent cardiac magnetic resonance for ventricular function and metabolic exercise testing.

89 ing 2 CMR studies with a 2-year interval for ventricular function and MF assessment.

90 Left ventricular function and New York Heart Association clas

91 c complications; and (5) stable, normal left ventricular function and New York Heart Association func

92 vanced methods for quantifying dyssynchrony, ventricular function and perfusion, and hybrid imaging a

93 Despite comparable right ventricular function and pulmonary regurgitant fraction,

94 lure (HF) therapy documented to improve left ventricular function and reduce mortality.

95 In addition, it partially restores ventricular function and reduces mortality.

96 We assessed glucose metabolism, ventricular function and remodeling, and cardiac gene ex

97 reduce its hemodynamic repercussions on left ventricular function and remodeling, and improve clinica

98 nd interfere with the coupling between right ventricular function and right ventricular afterload.

99 entricular longitudinal strain (LVLS), right ventricular function and right ventricular systolic pres

100 ed by echocardiographic measurements of left ventricular function and structure.

101 me, whereas other risk factors, such as left ventricular function and surgery on the thoracic aorta,

102 It also improves left ventricular function and survival in mice (n=20-22 per g

103 infarction is associated with improved left ventricular function and survival.

104 particular focus on those with improved left ventricular function and those who have not experienced

105 cts of thoracic epidural anesthesia on right ventricular function and ventricular-pulmonary coupling.

106 Secondary end points were left ventricular function and volumes by magnetic resonance i

107 eloping standard methods for assessing right ventricular function and, hopefully, its coupling to pul

108 nal, liver, and myocardial fat content, left ventricular function, and (31)P magnetic resonance spect

109 larization priority, diabetes mellitus, left ventricular function, and 3-vessel disease.

110 rt Association functional class 3, poor left ventricular function, and age (all p < 0.001) along with

111 , and the remaining patients had normal left ventricular function, and all but 1 have New York Heart

112 , sex, comorbidities, insurance status, left ventricular function, and aortic stenosis severity betwe

113 with rapid changes in pulsatile blood flow, ventricular function, and downstream impedance in both i

114 ilar improvements in clinical symptoms, left ventricular function, and left ventricular reverse remod

115 diovascular parameters (carotid plaque, left ventricular function, and myocardial late gadolinium enh

116 becomes limiting remodeling, preserving left ventricular function, and preventing heart failure.

117 sibility by high-resolution ultrasound, left ventricular function, and quality of life were measured

118 proved stability of the circulation and left ventricular function, and reduced plasma levels of cytok

119 sment reveals normal coronary arteries, left ventricular function, and resting ECG.

120 olution, thromboembolic risk, preserved left ventricular function, and severely decreased levels of a

121 right ventricular systolic pressures, right ventricular function, and survival.

122 ardiographic parameters to evaluate systolic ventricular function are affected by loading conditions.

123 Potential donor hearts with depressed ventricular function are often declined.

124 infected patients showed alterations in left ventricular function as demonstrated by a lower ejection

125 imals recovered 85.4% of their baseline left ventricular function as measured by 2-dimensional transt

126 secondary end points included regional left ventricular function assessed by magnetic resonance imag

127 s 10.9+/-2.3 cm/s; P=0.01) and regional left ventricular function, assessed by peak systolic velocity

128 C) developed cardiac hypertrophy and reduced ventricular function associated with increased Orai1 exp

129 n at discharge and 1 month with normal right ventricular function at 1 year.

130 Symptoms and left ventricular function at 2 years did not differ significa

131 iPSC-EV-treated mice exhibited improved left ventricular function at 35 d after myocardial infarction

132 which translated into impaired systolic left ventricular function at 6 to 12 months post TAVR (p < 0.

133 treperfusion and significantly improved left ventricular function at 7 days after myocardial ischemia

134 rchitectural regeneration and restoration of ventricular function at day 60 (P < 0.05, n = 6-14).

135 ew LE feature a significant decrease in left ventricular function at discharge.

136 ry disease, normal regional, and global left ventricular function at rest and during stress (exercise

137 We evaluated left ventricular function at rest, maximal aerobic capacity (

138 The present study aimed to evaluate left ventricular function, baroreflex sensitivity, autonomic

139 -) cell-treated rats exhibited improved left ventricular function, blunted left ventricular hypertrop

140 rdial infarction with severely impaired left ventricular function but no inducible ventricular tachyc

141 r 12 weeks did not alter body weight or left ventricular function but reduced maximal oxygen uptake,

142 ) has been used extensively to quantify left ventricular function, but its value for RV assessment ha

143 as an increase in frequency of impaired left ventricular function, but there was no change in Logisti

144 ned physical activity level, as well as left ventricular function by cardiac magnetic resonance imagi

145 DM2 is already detectable in preserved left ventricular function by cardiovascular magnetic resonanc

146 on could synchronize contraction and restore ventricular function by electrically connecting isolated

147 toms and objective measures of the valve and ventricular function by transthoracic echocardiography.

148 ive measurement of aortic valve gradient and ventricular function by transthoracic echocardiography.

149 ted a significantly greater increase in left ventricular function compared with all the other groups

150 fibroblasts, reduced scar size, and improved ventricular function compared with wild-type mice.

151 nscriptional profile in adulthood, impairing ventricular function, conduction, and repolarization.

152 Measure of right-ventricular function could improve the risk stratificati

153 matic severe aortic stenosis and normal left ventricular function, current practice guidelines empiri

154 Although systolic right and left ventricular function declined, peak exercise capacity re

155 A compliance, LA reservoir strain, and right ventricular function decreased with increasing AF burden

156 ntation may be associated with improved left ventricular function, decreased N-terminal pro B-type na

157 Left-ventricular function did not change in any group.

158 functional class 3 or 30-d death), and left ventricular function did not differ between two groups (

159 o-B-type natriuretic peptide, left and right ventricular function) differed between both groups.

160 ardiac output (Qc, foreign-gas rebreathing), ventricular function (echocardiography and cardiac MRI)

161 Left ventricular function (echocardiography), clinical status

162 Left ventricular function, edema, vascular permeability, and

163 rate aortic regurgitation and preserved left ventricular function (ejection fraction >/=55%).

164 essure (mean >15 mm Hg; p = 0.04), decreased ventricular function (ejection fraction <55%; p = 0.03),

165 Patients with new LE had a decreased left ventricular function (ejection fraction: pre, 55.5 +/- 1

166 cs, risk factors, coronary anatomy, and left ventricular function, end-systolic volume index and B-ty

167 e acute cor pulmonale determination and left ventricular function estimation.

168 nsplantation may be associated with improved ventricular function, exercise tolerance, and long-term

169 stablishes coronary blood flow, and improves ventricular function following an AMI.

170 es generation, myocardial necrosis, and left ventricular function following ischemia/reperfusion inju

171 nd.LepR-KO was associated with improved left ventricular function (fractional shortening, 28.4% versu

172 Improvements in left ventricular function, functional status, and quality of

173 Ventricular function, geometry, and regional strain were

174 Patients with preserved ventricular function had a higher rate of transplant-fre

175 ratio: 1.655; p < 0.001) and impaired right ventricular function (hazard ratio: 2.360; p = 0.001).

176 nces between these mice with regards to left ventricular function, heart ultrastructure or electrocar

177 vision of volumes, diastolic function, right ventricular function, hemodynamics, and valvular regurgi

178 sition, health-related quality of life, left ventricular function, hemodynamics, endothelial function

179 nts with heart failure (HF) who recover left ventricular function (HF-Recovered) have a distinct clin

180 n was significantly associated with impaired ventricular function, highlighting the importance of thi

181 Left ventricular function, histochemical injury, and differen

182 However, activity reduced left ventricular function (ie, ejection and shortening fracti

183 ischemia and reperfusion, postischemic left ventricular function improved commensurately with an inc

184 sonance imaging was performed to assess left ventricular function in 20 women with signs and symptoms

185 creases apoptosis, and greatly enhances left ventricular function in a model of ischemic cardiomyopat

186 mokine during a 4-week period that preserves ventricular function in a rat model of myocardial infarc

187 dothelial progenitor cells and improves left ventricular function in a rat model of myocardial infarc

188 y managed aortic stenosis and preserved left ventricular function in an era where surgical aortic val

189 ss its effect on pulmonary insufficiency and ventricular function in an ovine model of chronic postop

190 -1 receptor antagonist, on coronary and left ventricular function in coronary artery disease (CAD) pa

191 at cardiomyocytes and significantly improves ventricular function in mice with heart failure.

192 Tissue Doppler showed better diastolic left ventricular function in mutation carriers (e', P=0.001;

193 n and CVB3 copy number, and an improved left ventricular function in NOD2(-/-) CVB3 mice compared wit

194 apnoea and arrhythmia incidence and improve ventricular function in pacing-induced CHF rabbits.

195 ivation of DN T cells, as well as with worse ventricular function in patients with Chagas disease.

196 to further improvement in management of left ventricular function in patients with diabetes.

197 (Evaluating Xience and Left Ventricular Function in Percutaneous Coronary Interventi

198 The EXPLORE (Evaluating Xience and Left Ventricular Function in Percutaneous Coronary Interventi

199 The full scorpion venom increased left ventricular function in sedated mice in vivo, prolonged

200 cardiomyocytes, we found severely depressed ventricular function in the Gata4-ablated mice (mutant)

201 fice area, 38+/-18 mm(2)) and preserved left ventricular function in whom initial management was expe

202 loss of myocardial tissue and depressed left ventricular function in WT mice.

203 rd volumetric MRI demonstrated improved left ventricular functioning in hydrogel treated mice followe

204 ctive study, we investigated changes of left ventricular functions including speckle tracking in pati

205 fractional shortening and other measures of ventricular function, indicating that miR combo promotes

206 Impaired left ventricular function is associated with worse prognosis

207 Furthermore, although improvement in left ventricular function is one of the goals of revasculariz

208 We demonstrate that systemic right ventricular function is preserved (and may be improved)

209 feasible in TGA patients, but its effect on ventricular function is unclear.

210 The use of donors with depressed ventricular function is uncommon in pediatric heart tran

211 hy but exhibited significantly improved left ventricular function, less cardiac fibrosis and cardiomy

212 on of NaHS improved survival, preserved left ventricular function, limited infarct size, and improved

213 ment in AF symptoms, o2peak, left atrial and ventricular function, lipid levels, and QoL.

214 vs. 14%; p = 0.0027, respectively), and left ventricular function </=35% (26% vs. 10%; p = 0.0071).

215 Left ventricular function <30% was observed in 44.1% of patie

216 mild symptoms, a low PVC burden, and normal ventricular function may be best served with simple reas

217 blation before ICD implantation because left ventricular function may improve, consequently decreasin

218 nonsurgical candidates with compromised left ventricular function, MitraClip therapy could be conside

219 transplantation significantly improved left ventricular function, myocardial metabolism, and arterio

220 arction results in divergent effects on left ventricular function, myocardial remodeling, and monocyt

221 erior descending coronary disease and normal ventricular function (n=13) were recruited prospectively

222 dial biopsies from patients with normal left ventricular function (n=2) and from donor hearts (n=3) s

223 als satisfied major repolarization (n=4) and ventricular function (n=5) criteria, but this prevalence

224 l retention rates and greater improvement in ventricular function, N-terminal pro-brain natriuretic p

225 y of data regarding characteristics of right ventricular function - namely contractile and lusitropic

226 e.g., beta-blockers) partially preserve left ventricular function, novel strategies are needed to lim

227 nsfer may thus underlie the early decline in ventricular function observed in diabetic cardiomyopathy

228 Autonomic control of ventricular function occurs through regulation of cAMP-d

229 TR2), were less likely to have improved left ventricular function (odds ratio: 0.40 per minor allele;

230 atus or that the deleterious effects on left ventricular function offset any salutary vascular effect

231 Ventricular function on early echocardiograms, defined a

232 hibitor therapy in patients with normal left ventricular function on the progression of myocardial fi

233 a reported no effect of cell therapy on left ventricular function or clinical outcome.

234 it (7.6% vs. 9.0%, P < 0.01) strains but not ventricular function or myocardial damage.

235 mbined clinical scores and measures of right-ventricular function or strain.

236 re denied surgery due to increased age, poor ventricular function, or associated comorbidities, putti

237 rategy does not affect mortality, interstage ventricular function, or interstage AVVR in children wit

238 , new PPI did have a negative effect on left ventricular function over time.

239 the most important factor in improving left ventricular function (p < 0.001) and 30-day combined end

240 myocardial salvage (P<0.001), impaired left ventricular function (P<0.001), and more pronounced repe

241 ng status (P<0.0001), severely impaired left ventricular function (P<0.01), pulmonary hypertension an

242 placebo pigs had persistently depressed left ventricular function (P<0.05).

243 n independent predictor of recovery of right ventricular function (p=0.02).

244 ular hypertrophy, but systolic and diastolic ventricular function parameters were similar in diabetic

245 th CA results in significant improvements in ventricular function, particularly in the absence of ven

246 arkedly limits remodeling and preserves left ventricular function post-MI.

247 s did not affect cardiac hypertrophy or left ventricular function post-stress.

248 Left ventricular function predicts cardiovascular mortality b

249 Furthermore, simple NI estimates of ventricular function proved useful in this setting.

250 Furthermore, left ventricular function recovered within 72 hours of cardio

251 dical and reperfusion therapy, assessment of ventricular function, referral to cardiac rehabilitation

252 Ventricular function remodeling was similar in Cav3.1(-/

253 myopathy characterized by impaired diastolic ventricular function resulting in a poor clinical progno

254 Impaired ventricular function (right or left ventricular ejection

255 Optimal thresholds for ventricular function (RV EF <30%: hazard ratio, 3.90; 95

256 were associated with measures of worse right ventricular function (RV s', r=-0.39, P<0.0001) and pred

257 Right ventricular function (RVF) is an important determinant o

258 AFCA provides long-term benefits on left ventricular function, significantly reducing the number

259 accurately and noninvasively determine right ventricular function, such as cardiac magnetic resonance

260 (CAD), atrial fibrillation, or reduced left ventricular function, suggesting shared genetic aetiolog

261 cluded cine steady-state free precession for ventricular function, T2* for myocardial iron quantifica

262 cantly compromise left ventricular and right ventricular function through different mechanisms that a

263 Conclusively, Cfz decreased left ventricular function through increased PP2A activity and

264 linking fibrosis, inflammation, and impaired ventricular function to DPP4 inhibition in preclinical s

265 294-treated mice significantly improved left ventricular functions together with decreased infarct si

266 spid regurgitation velocity; and worse right ventricular function (tricuspid annular plane systolic e

267 2 weeks at multivariable analysis: (1) right ventricular function (tricuspid annular plane systolic e

268 activation in CHF patients with reduced left-ventricular function using SPECT to precisely quantify s

269 ne on the vasculature and its effects on the ventricular function using wave intensity analysis.

270 ntriculography is a method of measuring left ventricular function usually performed at the discretion

271 r cardiac repair in vivo with regard to left ventricular function, vascularization, and amelioration

272 re laboratory-masked analyses for quantified ventricular function, volumes, infarct size, microvascul

273 Ventricular functions, volumes, and dimensions were meas

274 ollow-up of 36 months, normalization of left ventricular function was achieved in >90% of patients wi

275 Left ventricular function was assessed by conventional and de

276 Cardiac ventricular function was evaluated with transthoracic ec

277 The decline in left ventricular function was further supported by significan

278 Left ventricular function was impaired (ejection fraction <50

279 quently, infarct size was increased and left ventricular function was impaired 28 days after myocardi

280 ewer hearts were functional during EVHP, and ventricular function was increasingly impaired.

281 Ventricular function was mildly depressed in 245 (7%) an

282 e (71%); left atrium was 43+/-6 mm; and left ventricular function was normal (59+/-9%).

283 On propensity-matched analysis, donor ventricular function was not associated with graft survi

284 Ventricular function was preserved in all 5.

285 Left ventricular function was restored in the 25 of 35 of tho

286 Myocardial perfusion and left ventricular function were also investigated using a newl

287 Cardiac fibrosis and ventricular function were assessed by histology and echo

288 After the MI, the autonomic and ventricular function were evaluated, as well as the syst

289 , and echocardiographic measurements of left ventricular function were greater in the AMD3100-treated

290 Myocardial blush grade and left ventricular function were significantly lower in patient

291 Left ventricular functions were consistently depressed in mic

292 ondary outcomes, including symptoms and left ventricular function, were also evaluated.

293 c MRI assessed myocardial viability and left ventricular function, whereas bioluminescence imaging as

294 nds on the actual stenosis severity and left ventricular function, which is of prognostic importance.

295 A8 on protein level, showed an improved left ventricular function, which was associated with a reduce

296 ficacy end points were functional status and ventricular function while temporarily weaned from LVAD

297 nt arrhythmia can result in rapid decline in ventricular function with development of heart failure,

298 mice fed a high-fat diet exhibited depressed ventricular function with reduced fractional shortening

299 hemodynamics showing normalization of right ventricular function with right atrial pressure <8 mm Hg

300 ansplant recipients of grafts with depressed ventricular function would have comparable survival with