ライフサイエンスコーパス: ventricular myocyte

1 onfirmed in a mathematical model of a rabbit ventricular myocyte.

2 rature-specific data-driven model of the rat ventricular myocyte.

3 ormal rapid activation and relaxation of the ventricular myocyte.

4 redictive in silico model of the adult human ventricular myocyte.

5 ally detailed three-dimensional model of the ventricular myocyte.

6 sing a physiologically detailed model of the ventricular myocyte.

7 d dSTORM imaging of permeabilized Wistar rat ventricular myocytes.

8 mputer simulations and experiments of rabbit ventricular myocytes.

9 re first tested by seeding with neonatal rat ventricular myocytes.

10 geneity that exists between atrial, VCS, and ventricular myocytes.

11 current, Ito, and IK,slow when compared with ventricular myocytes.

12 lly-detailed spatial Ca(2+) cycling model of ventricular myocytes.

13 [Ca(2+)]i with fluo-3 in voltage-clamped rat ventricular myocytes.

14 ains of nonfailing and failing human and rat ventricular myocytes.

15 the cardiac action potential and INa in rat ventricular myocytes.

16 isrupted intracellular Ca(2+) homeostasis in ventricular myocytes.

17 ers and its functional correlation in living ventricular myocytes.

18 2+) entry in control or STIM1 overexpressing ventricular myocytes.

19 chondria, both collected from adult rat left ventricular myocytes.

20 ata recorded from guinea pig isolated single ventricular myocytes.

21 n of GFP-RyR2 clusters was observed in fixed ventricular myocytes.

22 etween z-lines only at the periphery of live ventricular myocytes.

23 type Ca(2+) current (ICa, T) in hypertrophic ventricular myocytes.

24 kdown were examined in cultured neonatal rat ventricular myocytes.

25 handling, and ion currents were recorded in ventricular myocytes.

26 duction and Ca(2+)-wave generation in rabbit ventricular myocytes.

27 with Cav-3 was disrupted in the hypertrophic ventricular myocytes.

28 IKs amplitude in adult guinea pig and canine ventricular myocytes.

29 ns were complemented by experiments in mouse ventricular myocytes.

30 as many spontaneous Ca(2+) release events as ventricular myocytes.

31 ration in atrial and, to a lesser degree, in ventricular myocytes.

32 e not co-localized with mitochondria in live ventricular myocytes.

33 ocytes and acutely isolated adult guinea pig ventricular myocytes.

34 d Golgi PI4P depletion in neonatal and adult ventricular myocytes.

35 r measurements of cGMP in intact adult mouse ventricular myocytes.

36 C3, C6, and C10 domains was expressed in rat ventricular myocytes.

37 for 50% and 90% repolarization in Trpm4(-/-) ventricular myocytes.

38 ion to in vitro model systems such as rodent ventricular myocytes.

39 and is localized in the nuclear membrane of ventricular myocytes.

40 expressed in enzymically isolated mammalian ventricular myocytes.

41 a2+ wave probability in field stimulated rat ventricular myocytes.

42 by IL-1beta was also seen in cultured adult ventricular myocytes.

43 NKA kinetics specific to guinea- pig and rat ventricular myocytes.

44 experiments with Ca waves using isolated rat ventricular myocytes.

45 disrupted intracellular Ca2+ homeostasis in ventricular myocytes.

46 in vivo and in cultured adult feline and rat ventricular myocytes.

47 Rs are expressed and operative in human left ventricular myocytes.

48 its function as a repolarization reserve in ventricular myocytes.

49 as repolarization abnormalities in isolated ventricular myocytes.

50 2 AC isoforms in the regulation of ICa,L in ventricular myocytes.

51 NA binding activity in cultured neonatal rat ventricular myocytes.

52 eptors (RyRs) and caveolin-3 (CAV3) in mouse ventricular myocytes.

53 s to cell-wide SCWs in PLN-/-/RyR2-R4496C+/- ventricular myocytes.

54 93) cells and I(Kr) in isolated neonatal rat ventricular myocytes.

55 ment of the L-type Ca(2+) current (ICa,L) in ventricular myocytes.

56 ct compartmentalization of these isoforms in ventricular myocytes.

57 model-predicted ChR2 behavior in guinea pig ventricular myocytes.

58 s in the elimination of Ito,f in KChIP2(-/-) ventricular myocytes.

59 cal for Akt-mediated NF-kappaB activation in ventricular myocytes.

60 .2 channels form clusters of varied sizes in ventricular myocytes.

61 nglia but no Na(V)1.8 expression in isolated ventricular myocytes.

62 equence) in primary cultures of neonatal rat ventricular myocytes.

63 o be absent in tubulated atrial myocytes and ventricular myocytes.

64 2+) cycling and action potential dynamics in ventricular myocytes.

65 el interactions and elevate Ca(2+) influx in ventricular myocytes.

66 el clusters decorate T-tubule sarcolemmas of ventricular myocytes.

67 erformed using Fluo-3 in voltage clamped rat ventricular myocytes.

68 for the onset of bursting activity in mouse ventricular myocytes.

69 coupling (ECC) is strikingly different from ventricular myocytes.

70 ibrotic signaling in isolated murine primary ventricular myocytes.

71 cative of the increasing organization of the ventricular myocytes.

72 of action potential morphology in guinea pig ventricular myocytes.

73 Quantify the 5 cardiac ARs in individual ventricular myocytes.

74 =110+/-3 mumol/L) and Na(+) current in mouse ventricular myocytes.

75 s the control of diastolic [Ca(2+) ]i in rat ventricular myocytes.

76 How well can iPSC-CM model adult ventricular myocytes?

77 ated with the sarcoplasmic reticulum (SR) in ventricular myocytes; a median separation of 20 nm in 2D

78 A computational model of the human ventricular myocyte action potential suggests that the m

79 O'Hara-Rudy computational model of the human ventricular myocyte action potential, the Cav3 mutation-

80 Primary adult rat ventricular myocytes, adeno-associated virus (AAV)-media

81 ents in voltage clamped and field stimulated ventricular myocytes, along with mathematical modelling,

82 ogically detailed computational model of the ventricular myocyte and mathematical analysis.

83 y, resulting in increased Ca(2+) influx into ventricular myocytes and a positive inotropic response.

84 y, resulting in increased Ca(2+) influx into ventricular myocytes and a positive inotropic response.

85 were subsequently expressed in neonatal rat ventricular myocytes and acutely isolated adult guinea p

86 rrhythmogenic-triggered activities in female ventricular myocytes and altered myocyte Ca2+ handling,

87 4 proteins are functionally present in mouse ventricular myocytes and are activated on Ca2+ -induced

88 passing potassium ions outward to repolarize ventricular myocytes and end each beat.

89 We used permeabilized rat ventricular myocytes and fluorescently labeled DPc10, FK

90 luorescently tagged KCNQ1 and KCNE1 in adult ventricular myocytes and follow their biogenesis and tra

91 d RyR cluster sizes at Ca spark sites in rat ventricular myocytes and further tested functional impac

92 the mechanism of MANF release from cultured ventricular myocytes and HeLa cells, both of which secre

93 v3 coimmunoprecipitated with Kir2.1 in human ventricular myocytes and in heterologous expression syst

94 is-phosphorylated form in isolated adult rat ventricular myocytes and in mouse and rat ventricular my

95 voltage dependence of IH in Na/K pumps from ventricular myocytes and in ouabain-resistant pumps expr

96 luation, as well as decreasing the number of ventricular myocytes and inducing compensatory myocyte h

97 tial), and contractile force measurements in ventricular myocytes and intact cardiac muscles.

98 d refractoriness of calcium release in mouse ventricular myocytes and investigated how activation of

99 for the majority of PLA2 activity in rabbit ventricular myocytes and is responsible for arachidonic

100 lost its ability to support adhesion of rat ventricular myocytes and led to a significant reduction

101 Using adult rat ventricular myocytes and mouse-derived cardiac HL-1 card

102 We studied isolated R33Q and wild-type ventricular myocytes and observed properties not previou

103 ies of mitochondrial metabolism in the right ventricular myocytes and pulmonary vascular cells sugges

104 ed in Gq or by Gq expression in neonatal rat ventricular myocytes and reversed by CaMKIIdelta deletio

105 We employed a co-culture of neonatal ventricular myocytes and sympathetic stellate neurons fr

106 also caused loss of T tubules in rat cardiac ventricular myocytes and the open canalicular system of

107 nofluorescence in freshly isolated adult rat ventricular myocytes and those in short-term primary cul

108 gical and Ca(2+)(i) dynamic abnormalities of ventricular myocytes and trabeculae from patients with H

109 DHHC5 coimmunoprecipitates with PLM in ventricular myocytes and transiently transfected cells.

110 Ventricular myocytes and tubulated atrial myocytes addit

111 gnalling augmented CaMKII activity in rabbit ventricular myocytes and, importantly, CaMKII activation

112 ovel Myo18A transcript is expressed in mouse ventricular myocytes (and human heart).

113 (expression of JUP(2157del2) in neonatal rat ventricular myocytes) and a robust murine model of ACM (

114 prevents the expansion of BMP signaling into ventricular myocytes, and ectopic activation of BMP mimi

115 hypertrophy in H9c2 cardiac cells, adult rat ventricular myocytes, and human induced pluripotent stem

116 IP3R function was determined in human left ventricular myocytes, and this analysis was integrated w

117 latter effect is more potent in atrial than ventricular myocytes, and this could be explained by our

118 p to IKs amplitudes, in chronically stressed ventricular myocytes, and use COS-7 cell expression to p

119 d strands (n=152) in which host neonatal rat ventricular myocytes (AP duration=153.2+/-2.3 ms, conduc

120 ) -activated K(+) (SK) channels expressed in ventricular myocytes are dormant in health, yet become f

121 Ventricular myocytes are excitable cells whose voltage t

122 The transverse (t-) tubules of mammalian ventricular myocytes are invaginations of the surface me

123 The dominant ventricular myocyte ARs present in all cells are the bet

124 tractility in the whole rat heart, adult rat ventricular myocytes (ARVMs), and myofibrils from both s

125 mplitude and SR Ca(2+) load in permeabilized ventricular myocytes as an endogenous cell biology model

126 ed with the opposite results in neonatal rat ventricular myocytes as p65 and p50 were decreased, and

127 rrent beyond steady state in voltage-clamped ventricular myocytes as reported by others.

128 ed sodium and potassium current densities in ventricular myocytes, as well as conduction slowing in t

129 166-selected cells are able to pace neonatal ventricular myocytes at a rate faster than their own.

130 d intracellular Ca(2+) imaging from isolated ventricular myocytes at baseline and after adrenergic st

131 We conducted experiments in rabbit ventricular myocytes at body temperature and found that

132 estigated subpopulations of PLM in adult rat ventricular myocytes based on phosphorylation status.

133 istically, knockdown of ATF6 in neonatal rat ventricular myocytes blocked phenylephrine- and IGF1 (in

134 was significantly reduced by H(2)O(2) in WT ventricular myocytes, but not in MMVV, showing ox-CaMKII

135 o) during SR Ca(2+) release in intact rabbit ventricular myocytes by confocal microscopy.

136 cal microscopy and confirmed the findings in ventricular myocytes by directly observing fluorescently

137 tion of Ca(2+) spark refractoriness in mouse ventricular myocytes by examining how beta-adrenergic st

138 lation of Ca2+ spark refractoriness in mouse ventricular myocytes by examining how beta-adrenergic st

139 erate hypokalaemia, increases Ca2+ levels in ventricular myocytes by reducing the pumping rate of the

140 d significant sarcomere shortening on murine ventricular myocytes compared to control.

141 We developed a model for mouse ventricular myocyte contraction which is based on our pr

142 f variable sarcomere length into a model for ventricular myocyte contraction.

143 l pathway that constrains BMP signaling from ventricular myocytes, coordinates sarcomere assembly, an

144 In permeabilized ventricular myocytes, CPVT-CaMs at a physiological intra

145 PLCepsilon small interfering RNA (siRNA) in ventricular myocytes decreases endothelin-1 (ET-1)-depen

146 f GS-458967 on endogenous INaL in guinea pig ventricular myocytes demonstrate a robust concentration-

147 of this protocol in in vitro guinea-pig left-ventricular myocytes demonstrated that both voltage- and

148 hearts to VT/VF, patch clamped isolated SHR ventricular myocytes developed EADs and triggered activi

149 s and triggered activities in RyR2-R4496C+/- ventricular myocytes during sarcoplasmic reticulum Ca(2)

150 In ventricular myocytes, effects of SEA-0400 were tested on

151 The dyadic organization of ventricular myocytes ensures synchronized activation of

152 ics for CaM-RyR2 in normal and heart failure ventricular myocytes, estimate the percentage of Z-line-

153 triggered activity to the same extent as NR ventricular myocytes, except with larger EAD amplitude.

154 STAA ventricular myocytes exhibited arrhythmic contractions i

155 lated rabbit and rat hearts or patch-clamped ventricular myocytes exposed to hypokalemia (1.0-3.5 mmo

156 graftment of fibrin patches onto beating rat ventricular myocytes exposed to hypoxia showed an effect

157 isms leading to triggering of arrhythmias in ventricular myocytes exposed to low Ko.

158 RATIONALE: It is unknown whether every ventricular myocyte expresses all 5 of the cardiac adren

159 In adult rat ventricular myocytes expressing wild-type SERCA, H(2)O(2

160 Adult rat ventricular myocytes expressing wild-type SERCA2b or a r

161 We show here that, in rat ventricular myocytes, fluorescent antibodies map the NBC

162 report prospective markers of atrial versus ventricular myocyte formation from hPSCs and their use i

163 recycling of the beta1-AR was determined in ventricular myocytes from AKAP5(-/-) mice.

164 Ventricular myocytes from cardiac-specific adult Scn1b n

165 Ventricular myocytes from cardiac-specific adult Scn1b n

166 um (Ca(2+)) and transverse-tubule imaging of ventricular myocytes from MCM-Speg(fl/fl) mice post HF r

167 Ventricular myocytes from mice heterozygous for the muta

168 INa and INa-L were measured in left ventricular myocytes from nonfailing, DHF, and CRT dogs.

169 HDAC5 phosphorylation and nuclear export in ventricular myocytes from the adult rat heart.

170 METHODS AND We studied ventricular myocytes from wild-type mice, mice with alph

171 ics of Ca(2+)-activated force development in ventricular myocytes from wild-type, but not those from

172 larly, Kv current density was 25% greater in ventricular myocytes from young adult males (P < 0.05).

173 -CM model to predicted response of the adult ventricular myocyte given the same genetic mutation.

174 ion potentials in Wnt3a-treated neonatal rat ventricular myocytes had a lower upstroke amplitude (91

175 al myoblast differentiation and neonatal rat ventricular myocyte hypertrophy are inhibited by mAKAPbe

176 PC-1 was sufficient to trigger neonatal rat ventricular myocyte hypertrophy.

177 ncreased MEF2D-dependent gene expression and ventricular myocyte hypertrophy.

178 howed that GDF11 did not reduce neonatal rat ventricular myocytes hypertrophy, but instead induced hy

179 in suppressed L-type Ca(++) currents (rabbit ventricular myocytes, IC50=66.5+/-4 mumol/L) and IK1 (HE

180 Here, using murine ventricular myocytes, immunoblotting, proximity ligation

181 acilitation of acid/base transporters in the ventricular myocyte, implying a specific role for the in

182 dence under physiological AP clamp in rabbit ventricular myocytes in chronic pressure/volume overload

183 .5-expressing HEK 293 cells and neonatal rat ventricular myocytes in low osmolarity (LO) medium and t

184 on suppressed Bnip3 levels and cell death of ventricular myocytes in response to hypoxia.

185 ents were recorded from isolated canine left ventricular myocytes in the presence of isoproterenol.

186 g the growth in width versus length of adult ventricular myocytes in vitro and in vivo.

187 expression of STIM1 in cultured adult feline ventricular myocytes increased diastolic spark rate and

188 Single-tilt tomograms of the dyads in rat ventricular myocytes indicated that type 2 ryanodine rec

189 ell death were recapitulated in neonatal rat ventricular myocytes infected with constitutively active

190 romote ventricular arrhythmias by disrupting ventricular myocyte intercalated disk (ID) nanodomains r

191 Contraction of ventricular myocytes is controlled by the terminal part

192 Intracellular calcium dynamics of ventricular myocytes isolated from KI hearts are altered

193 ) prolonged AP duration and blocked I(Kr) in ventricular myocytes isolated from neonatal rats.

194 D We recorded Ca(2+) transients from cardiac ventricular myocytes isolated from rabbit hearts.

195 atically impaired in both the ventricles and ventricular myocytes isolated from RGS6(-/-) mice, and t

196 Confocal imaging of live ventricular myocytes isolated from the GFP-RyR2 mouse he

197 In this study, we used ventricular myocytes isolated from young (5-9 months) an

198 In adult mouse ventricular myocytes, it dose dependently increased sarc

199 Kcne5 deletion increased mean ventricular myocyte K(V) current density in the apex and

200 In adult ventricular myocytes, KCNE1 maintains a stable presence

201 de that the absence of dystrophin in the mdx ventricular myocyte leads to impaired functional communi

202 hesis that targeted block of INaL would make ventricular myocytes less susceptible to small electrica

203 ac electrogenic Cl(-)/HCO3(-) transporter in ventricular myocytes, linking the critical roles of Slc2

204 Isolated patch-clamped rabbit ventricular myocytes loaded with Fluo-4 to image intrace

205 EGFP-tagged Myo18Agamma expressed in ventricular myocytes localized to the level of A-bands i

206 he first evidence that, in cultured neonatal ventricular myocytes, LPI triggers distinct signaling pa

207 d crest areas of the sarcolemma, whereas, in ventricular myocytes, LTCCs primarily cluster in T-tubul

208 (CA) enzymes, expressed at various sites in ventricular myocytes, may affect mitochondrial CO2 clear

209 e phosphorylation and glycolysis in Neonatal ventricular myocytes measured by seahorse assay.

210 ity changes of APs observed in a paced human ventricular myocyte model by decreasing and/or increasin

211 ing components into our previously developed ventricular myocyte model consisting of a three-dimensio

212 In this study, we developed a spatiotemporal ventricular myocyte model that integrates mitochondria-r

213 ts, a 3-dimensional physiologically-detailed ventricular myocyte model, and a coupled map lattice mod

214 t this new, to our knowledge, spatiotemporal ventricular myocyte model, incorporating properties of m

215 a pig myocytes, combined with a quantitative ventricular myocyte model, reveals a threshold behaviour

216 Using computer simulations of a ventricular myocyte model, we show that initiation and t

217 orporated this NCX model into an established ventricular myocyte model, which allowed us to predict r

218 345 ms was observed using the human cardiac ventricular myocyte model.

219 incorporated into a physiologically detailed ventricular myocyte model.

220 by performing a comprehensive analysis of 13 ventricular myocyte models.

221 r simulations using a realistic neonatal rat ventricular myocyte monolayer model provided crucial ins

222 ent protein (GFP) expression in neonatal rat ventricular myocyte monolayers.

223 We monitored Ca(2+) transients in ventricular myocytes near the adenovirus-injection sites

224 lectrophysiological property of neonatal rat ventricular myocyte (NRVM) cultures.

225 Treatment of confluent neonatal rat ventricular myocyte (NRVM) monolayers with astemizole or

226 system applied to monolayers of neonatal rat ventricular myocytes (NRVM).

227 lly, MANF knockdown in cultured neonatal rat ventricular myocytes (NRVMs) impaired protein folding in

228 (J(leak)) from the sarcoplasmic reticulum of ventricular myocytes occurs in spark and nonspark forms.

229 In ventricular myocytes of large mammals with low T-tubule

230 athematical models of Ca(2+) dynamics in the ventricular myocytes of the control, four-week, and seve

231 APs were recorded in rabbit ventricular myocytes paced at cycle lengths (CLs) decrea

232 al-time qPCR, echo- and electrocardiography, ventricular myocyte patch-clamp, coronary artery ligatio

233 In cultured adult feline ventricular myocytes, PKA inhibition protected myocytes

234 3-dimensional nanostructure of TT in rabbit ventricular myocytes, preserved at different stages of t

235 c models, we show that beta-catenin controls ventricular myocyte proliferation during development and

236 Our objective is to understand how adult ventricular myocytes regulate the IKs amplitudes under b

237 In isolated adult ventricular myocytes, relaxin upregulated Nav1.5 (EC(50)

238 and the apoptotic response to doxorubicin in ventricular myocytes required RGS6-dependent ROS product

239 AP recordings in rabbit ventricular myocytes revealed intermittent EADs, with sl

240 hannels (K(ATP)), hugely abundant in cardiac ventricular myocytes, sense the local myocyte metabolic

241 At the level of ventricular myocytes, Stim1 silencing or inhibition abro

242 patial localisation of [H(+)]i regulation in ventricular myocytes, suggesting that, by guarding pHi,

243 sonance energy transfer imaging of adult rat ventricular myocytes, surprisingly suggest that beta-blo

244 ription of the death gene Bnip3 resulting in ventricular myocyte survival.

245 aling and mTOR, the balance which determines ventricular myocyte survival.

246 s and detailed mathematical models of rabbit ventricular myocytes that replicate the observed behavio

247 sed to develop models of isolated guinea pig ventricular myocytes that simulate the electrophysiologi

248 inculin (Vcl) plays a key structural role in ventricular myocytes that, when disrupted, can lead to c

249 In ventricular myocytes, the physiological function of stro

250 In isoproterenol-stimulated ventricular myocytes, the RyR2-A4860G mutation decreased

251 epending on initial ion circumstances within ventricular myocytes, these multi-stable AP states might

252 logy approach to test the hypothesis that in ventricular myocytes, these two apparently conflicting m

253 ed by the channel opener pinacidil in rabbit ventricular myocytes, through reducing the occurrence an

254 We developed an ionic model of the mouse ventricular myocyte to interrogate this potentially arrh

255 highly detailed mathematical model of mouse ventricular myocytes to disclose the key mechanisms unde

256 We used cultured neonatal rat ventricular myocytes to examine how chronic beta-AR stim

257 Exposing neonatal rat ventricular myocytes to hypo-osmotic medium resulted in

258 We subjected neonatal rat ventricular myocytes to mechanical stretch by exposing t

259 nged from minor alterations in anterior left ventricular myocytes to nearly complete loss of the t-sy

260 ies using phenylephrine-treated neonatal rat ventricular myocytes, to explore the putative antihypert

261 TV1 RNA sequencing dataset from neonatal rat ventricular myocytes transduced with Etv1 showed recipro

262 h factor 1 (IGF-1) treatment in neonatal rat ventricular myocytes, translocates to mitochondria and i

263 sting of monolayers of cultured neonatal rat ventricular myocytes treated with anthopleurin-A (AP-A).

264 Neonatal ventricular myocytes treated with miR-294 showed elevate

265 in hERG-HEK cells as well as in neonatal rat ventricular myocytes treated with the muscarinic agonist

266 of intracellular Ca(2+) cycling dynamics in ventricular myocytes under periodic pacing have been dev

267 vestigated SERCA dimerization in rabbit left ventricular myocytes using a photoactivatable cross-link

268 ans were studied in single rabbit atrial and ventricular myocytes using combined [Ca(2+)]i and electr

269 Ca(2+) dynamics in intact and permeabilized ventricular myocytes using confocal microscopy.

270 Ks was recorded from freshly isolated rabbit ventricular myocytes using whole-cell patch clamp.

271 um signals during wave propagation in rabbit ventricular myocytes, using high resolution fluorescence

272 s oocytes, and of native NKA isozymes in rat ventricular myocytes, using normal mammalian physiologic

273 bicin in the ventricles of mice and isolated ventricular myocytes via a posttranscriptional mechanism

274 voltage-clamp recordings from cGOF or icGOF ventricular myocytes (VM) show increased basal L-type Ca

275 ) -activated K(+) (SK) channels expressed in ventricular myocytes (VMs) are dormant in health, yet be

276 more susceptible to Ca(2+) dysfunction than ventricular myocytes (VMs).

277 u is a phase of high resistance, which makes ventricular myocytes vulnerable to small electrical pert

278 all CA activity in the cytoplasm of isolated ventricular myocytes was found to be modest (2.7-fold ab

279 +) and nitric oxide (NO) imaging in isolated ventricular myocytes was performed together with protein

280 ing an in-silico subcellular model of rabbit ventricular myocyte, we show that the high dimensional n

281 In cultured neonatal rat ventricular myocytes, we compared the responses to IGF1

282 ) ([Ca(2+)]SR) decline during rest in rabbit ventricular myocytes, we found that approximately 76% of

283 Using rabbit ventricular myocytes, we show that this eigenvalue can b

284 Isolated patch-clamped rabbit ventricular myocytes were exposed to either oxidative st

285 Furthermore, ventricular myocytes were found to be an important sourc

286 Isolated adult rat ventricular myocytes were infected with wild-type (WT)-K

287 ll shortening for epicardial and endocardial ventricular myocytes were investigated.

288 Ventricular myocytes were isolated from wildtype (C57Bl/

289 Primary adult rat ventricular myocytes were studied for morphology and int

290 Neonatal mouse ventricular myocytes were treated with a pathophysiologi

291 Neonatal cardiac ventricular myocytes were treated with atorvastatin and

292 istribution along the T-tubule sarcolemma of ventricular myocytes where nanometer proximity between c

293 otein localized to the intercalated discs in ventricular myocytes, where K(V)2.1 was also detected in

294 (2+)) alternans is a dynamical phenomenon in ventricular myocytes, which is linked to the genesis of

295 dings of action potentials on isolated mouse ventricular myocytes, which may contribute to the develo

296 rfusion or enzymatically digested to isolate ventricular myocytes, which were subsequently fixed at 0

297 aracterized by subcellular remodeling of the ventricular myocyte with a reduction in the scaffolding

298 cium concentration was measured in adult rat ventricular myocytes with a genetically targeted fluores

299 Treatment of neonatal rat ventricular myocytes with either recombinant Wnt3a prote

300 Infection of 2D cultures of neonatal rat ventricular myocytes with WT and mutant channels increas