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1 c status of the heart (inhomogeneity of left ventricular repolarization).
2 e effects on atrioventricular conduction and ventricular repolarization.
3 rate, which likely contributes to prolonged ventricular repolarization.
4 hythmia associated with abnormally prolonged ventricular repolarization.
5 al-specific lymphocyte infiltration, impairs ventricular repolarization.
6 tion scar, atrial fibrillation, and abnormal ventricular repolarization.
7 tion system, as well as heterogeneity of the ventricular repolarization.
8 defining gender-related differences in mouse ventricular repolarization.
9 ng the densities of K(+) currents underlying ventricular repolarization.
10 te with long QT syndrome (LQTS), a defect in ventricular repolarization.
11 nd electrocardiographic evidence for delayed ventricular repolarization.
12 al flutter or fibrillation without affecting ventricular repolarization.
13 d re-entrant excitation arising from delayed ventricular repolarization.
14 nown whether alpha-linolenic acid influences ventricular repolarization.
15 been proposed as a more valid way to assess ventricular repolarization.
16 d, or both) of the ionic currents underlying ventricular repolarization.
17 T interval is prolonged due to dysfunctional ventricular repolarization.
18 informative in pathophysiological studies of ventricular repolarization.
19 dal factors contribute to the differences in ventricular repolarization.
20 eflect different underlying abnormalities of ventricular repolarization.
21 icate that DCM leads to temporal lability in ventricular repolarization.
22 T-wave changes imply an abnormal sequence of ventricular repolarization.
23 ly impact the 24 h rhythms in heart rate and ventricular repolarization.
24 or activity of any other channel involved in ventricular repolarization.
25 R-871 with an antagomir in females shortened ventricular repolarization.
26 I(Kr) ), which plays a major role in cardiac ventricular repolarization.
27 ucible ventricular tachycardia, and abnormal ventricular repolarization.
28 n gradient (ABRG) determines the sequence of ventricular repolarization.
29 iour, have on 24 h rhythms in heart rate and ventricular repolarization.
31 ng-QT syndrome is characterized by prolonged ventricular repolarization and a variable clinical cours
32 yed-rectifier K(+) current are important for ventricular repolarization and are continuously shuttled
33 iPSC-CMD801N cells were used to validate ventricular repolarization and arrhythmic susceptibility
34 ro-arrhythmic effects of solvent aerosols on ventricular repolarization and heart rate variability de
35 and tissue excitability, while it prolonged ventricular repolarization and refractoriness by 7.5% to
36 responsible for the abnormal prolongation of ventricular repolarization and revealed associations bet
38 function in sedated mice in vivo, prolonged ventricular repolarization, and provoked ventricular arr
39 direct effects of inflammatory cytokines on ventricular repolarization are increasingly recognized.
40 However, the 24 h rhythms in heart rate and ventricular repolarization are primarily driven by auton
41 nd 3 for Tpe response to recovery modulating ventricular repolarization, as well as cardiac conductio
42 he effects of restitution in single cells on ventricular repolarization at the level of the intact he
43 s characterized by heterogeneously prolonged ventricular repolarization, besides altered contraction
44 mportant role in determining the sequence of ventricular repolarization, but the effects of sex and a
45 model reveals complex dynamic regulation of ventricular repolarization by temperature, extracellular
47 to identify the ionic and cellular basis for ventricular repolarization components on the body surfac
49 use a spectrum of diminished IKr and delayed ventricular repolarization, consistent with the prolonge
50 ed the hypothesis that heterogeneity of left ventricular repolarization creates an arrhythmogenic sub
51 3 deletion also caused aldosterone-dependent ventricular repolarization delay (19.6% mean QTc prolong
53 restitution (TMR), an ECG marker quantifying ventricular repolarization dynamics, is strongly associa
55 low delayed rectifier K+ current, in cardiac ventricular repolarization has been a subject of debate.
56 rrhythmia characterized in part by prolonged ventricular repolarization, has been linked to 5 loci, 4
57 significantly modulate spatial gradients of ventricular repolarization (ie, modulated dispersion), w
58 rhythmias by modulating spatial gradients of ventricular repolarization (ie, modulated dispersion).
59 during atrial and ventricular activation and ventricular repolarization in (i) normal heart (ii) hear
62 t a causal effect of serum calcium levels on ventricular repolarization, in a middle-aged population
64 QT prolongation, consistent with a defect in ventricular repolarization, in Kv2.1N216FLAG-expressing
66 t increased spatio-temporal heterogeneity of ventricular repolarization is a substrate for cardiovasc
67 is the electrocardiographic manifestation of ventricular repolarization, is variable under physiologi
68 se ventricular arrhythmias and augment early ventricular repolarization (J amplitude), while menthol
69 for suppressing hypertrophy (Hdac9) and for ventricular repolarization (Kcnd2) and conduction (Hcn4)
70 ed whether HIV serostatus is associated with ventricular repolarization lability by using the QT vari
72 yndrome (LQTS) is a disease in which delayed ventricular repolarization leads to cardiac arrhythmias
73 to determine spatial and dynamic changes in ventricular repolarization may help to understand arrhyt
75 ndividuals developing marked prolongation of ventricular repolarization on exposure to an offending d
76 upts the electrophysiological homogeneity of ventricular repolarization, predisposing to arrhythmia.
78 , atrioventricular conduction (PR interval), ventricular repolarization (QT interval), and ventricula
79 ogram (ECG) recordings abnormal durations of ventricular repolarization (QT interval), atrial depolar
80 Thus, 4 months sustained training lengthened ventricular repolarization (QTc: 237.1+/-3.4 ms vs. 213.
82 Long QT syndrome (LQTS) is a disorder of ventricular repolarization that predisposes affected ind
83 Ks and cause long-QT syndrome, a disorder of ventricular repolarization that predisposes affected ind
84 tigated the impact of reduced sialylation on ventricular repolarization through gene deletion of the
85 ain (beta-MHC) gene mutations exhibit labile ventricular repolarization using beat-to-beat QT variabi
88 ular and ventricular depolarization and slow ventricular repolarization, which may promote cardiac ar
89 ome is characterized by abnormally prolonged ventricular repolarization, which predisposes patients t
90 A transmural voltage gradient during initial ventricular repolarization, which results from the prese
91 ting in vivo, and a role for MiRP1 in murine ventricular repolarization with parallels to that propos
92 for the normal function of cardiac I(Kr) in ventricular repolarization, with loss of hERG1b contribu
93 heart rate, atrioventricular conduction, and ventricular repolarization, with potential implications